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1 egatively correlated with the presence of CB tractional abnormalities (P = .002).
2                                 At baseline, tractional abnormalities of the CB were diagnosed in 58
3                                              Tractional abnormalities of the CB were identified with
4                           To investigate the tractional alterations of the central bouquet (CB) in id
5 Eleven eyes shared common features with both tractional and degenerative lamellar macular holes and w
6 es of lamellar macular hole were identified: tractional and degenerative.
7 ation of the nerve at the elbow results in a tractional and frictional neuritis with classical sympto
8 ution, TMJ = temporomandibular joint, mu(T) =tractional coefficient, mu(s) = static coefficient of fr
9 ith air endotamponade strongly advocates the tractional component of non-RRD and retinal schisis asso
10  patients with Eales' disease, one developed tractional detachment at macula while the other, an epir
11 s of young patients (</=2.5 years) developed tractional detachment, despite prophylactic ablation in
12  retinal detachments was observed, with most tractional detachments (7 eyes) occurring by age 2.5 yea
13 tact ellipsoid layer and was associated with tractional epiretinal membranes and/or vitreomacular tra
14 associated epiretinal proliferation, whereas tractional epiretinal membranes presenting contractive p
15                                 Two eyes had tractional folds in the retina with posterior extension
16                                Generation of tractional force by Muller cells primarily involves inte
17 fibroblasts interact with collagen matrices, tractional force exerted by the cells can couple to matr
18 Fs) on retinal pigment epithelial (RPE) cell tractional force generation and the contributions of vit
19  68 samples were evaluated using Muller cell tractional force generation as a target bioassay.
20 ing amounts of growth factors that stimulate tractional force generation by Muller cells.
21                  It appears that Muller cell tractional force generation in PDR is driven by vitreous
22 ty to modulate Muller cell proliferation and tractional force generation in tissue culture models.
23 F-I and -II are potent promoters of RPE cell tractional force generation in vitro.
24         Intact IGFBP-3 modulates Muller cell tractional force generation stimulated by IGF-I and IGF-
25                                              Tractional force generation was assessed as a function o
26 its effects on Muller cell proliferation and tractional force generation, activities relevant to prol
27 Contraction of fibroblasts and the resultant tractional force is a contributing factor to fibrotic di
28                                Generation of tractional force was assessed with a tissue culture assa
29                         Quantitation of cell tractional forces and cellular prestress by using tracti
30  TMJ may involve fatigue produced by surface tractional forces and compressive stresses.
31 stress-field translation on TMJ disc-surface tractional forces and stresses.
32 d on its ability to mechanically resist cell tractional forces and thereby produce cell and cytoskele
33 dentified matrix identity and cell-generated tractional forces as key determinants of the dedifferent
34                  Cells capable of generating tractional forces associated with proliferative diabetic
35        This study tested the hypotheses that tractional forces following static loading of the TMJ di
36                                              Tractional forces generated by cells incubated on three-
37 collagen matrix contraction culminating from tractional forces generated by fibroblasts showed that b
38 te behavior and EC stiffening in response to tractional forces generated by leukocytes.
39                          RPE cells generated tractional forces in response to IGF-I and -II with IGF-
40 , the principal glia of the retina, generate tractional forces in response to IGF-I and platelet-deri
41 fibrocontractive ocular tissues and generate tractional forces in response to insulin-like growth fac
42 uller cells acquire the capacity to generate tractional forces in vitro and the contraction-promoting
43 se to insulin-like growth factor I, generate tractional forces of the type that cause retinal detachm
44                                              Tractional forces on the temporomandibular joint (TMJ) d
45 ittle is known about the consequences of the tractional forces that leukocytes generate on ECs as the
46       Results showed non-linear increases in tractional forces that were positively correlated with i
47 ir stress fibers and focal adhesions so that tractional forces were concentrated in these corner regi
48                                      Maximum tractional forces were found to occur following 60 sec o
49  filopodia were also observed, and transient tractional forces were generated by these extending proc
50 e results showed that compressive strain and tractional forces were largest for the start of movement
51                                         Peak tractional forces were linearly and positively related t
52 s > 27 mm/sec and AR.epsilon(3), was > 0.09, tractional forces were significantly higher (< or = 12%
53                The results demonstrated that tractional forces were strain-related at the start of mo
54 ge thickness significantly affected TMJ disc tractional forces.
55 ndibular joint (TMJ) disc may be promoted by tractional forces.
56 roblast-like phenotype capable of generating tractional forces.
57 act extracellular matrices and thus generate tractional forces.
58        This study tested the hypotheses that tractional forces: (1) increased with stress-field veloc
59                             Degenerative and tractional lamellar macular holes may be 2 distinct clin
60 terior displacement, followed by rebound and tractional nerve avulsion 10 mm behind the lamina after
61 eria included RRD before 2009 and exudative, tractional, or traumatic retinal detachments.
62        Six eyes developed moderate to severe tractional RD or bullous RD in the control group by day
63 luded epiretinal membrane (n = 26), diabetic tractional retinal detachment (n = 14), full-thickness m
64 = 121), vitreous floaters (n = 69), diabetic tractional retinal detachment (n = 49), vitreous hemorrh
65 inal membrane (ERM), and an additional 8 for tractional retinal detachment (RD) and/or rhegmatogenous
66 feration (FP), vitreous hemorrhage (VH), and tractional retinal detachment (TRD) were documented.
67 trategy with contractile cellular forces and tractional retinal detachment (TRD).
68 cipitate fibrotic changes, drusen formation, tractional retinal detachment and so on.
69 ive vitreoretinopathy, results in a blinding tractional retinal detachment because of the contractile
70                                Patients with tractional retinal detachment, previous retinal detachme
71  tears of the retinal pigment epithelium and tractional retinal detachment.
72 lete and posterior retinal reattachment; (5) tractional retinal detachment; (6) hypotony/increased in
73       Full-thickness eyewall sections showed tractional retinal folds, tented intravitreal vasculariz
74 rated growth corresponded to regions of high tractional stress generated within the sheet, as predict
75                              The first type, tractional, was diagnosed in 43 eyes, and was characteri

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