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5 Eleven eyes shared common features with both tractional and degenerative lamellar macular holes and w
7 ation of the nerve at the elbow results in a tractional and frictional neuritis with classical sympto
8 ution, TMJ = temporomandibular joint, mu(T) =tractional coefficient, mu(s) = static coefficient of fr
9 ith air endotamponade strongly advocates the tractional component of non-RRD and retinal schisis asso
10 patients with Eales' disease, one developed tractional detachment at macula while the other, an epir
11 s of young patients (</=2.5 years) developed tractional detachment, despite prophylactic ablation in
12 retinal detachments was observed, with most tractional detachments (7 eyes) occurring by age 2.5 yea
13 tact ellipsoid layer and was associated with tractional epiretinal membranes and/or vitreomacular tra
14 associated epiretinal proliferation, whereas tractional epiretinal membranes presenting contractive p
17 fibroblasts interact with collagen matrices, tractional force exerted by the cells can couple to matr
18 Fs) on retinal pigment epithelial (RPE) cell tractional force generation and the contributions of vit
22 ty to modulate Muller cell proliferation and tractional force generation in tissue culture models.
26 its effects on Muller cell proliferation and tractional force generation, activities relevant to prol
27 Contraction of fibroblasts and the resultant tractional force is a contributing factor to fibrotic di
32 d on its ability to mechanically resist cell tractional forces and thereby produce cell and cytoskele
33 dentified matrix identity and cell-generated tractional forces as key determinants of the dedifferent
37 collagen matrix contraction culminating from tractional forces generated by fibroblasts showed that b
40 , the principal glia of the retina, generate tractional forces in response to IGF-I and platelet-deri
41 fibrocontractive ocular tissues and generate tractional forces in response to insulin-like growth fac
42 uller cells acquire the capacity to generate tractional forces in vitro and the contraction-promoting
43 se to insulin-like growth factor I, generate tractional forces of the type that cause retinal detachm
45 ittle is known about the consequences of the tractional forces that leukocytes generate on ECs as the
47 ir stress fibers and focal adhesions so that tractional forces were concentrated in these corner regi
49 filopodia were also observed, and transient tractional forces were generated by these extending proc
50 e results showed that compressive strain and tractional forces were largest for the start of movement
52 s > 27 mm/sec and AR.epsilon(3), was > 0.09, tractional forces were significantly higher (< or = 12%
60 terior displacement, followed by rebound and tractional nerve avulsion 10 mm behind the lamina after
63 luded epiretinal membrane (n = 26), diabetic tractional retinal detachment (n = 14), full-thickness m
64 = 121), vitreous floaters (n = 69), diabetic tractional retinal detachment (n = 49), vitreous hemorrh
65 inal membrane (ERM), and an additional 8 for tractional retinal detachment (RD) and/or rhegmatogenous
66 feration (FP), vitreous hemorrhage (VH), and tractional retinal detachment (TRD) were documented.
69 ive vitreoretinopathy, results in a blinding tractional retinal detachment because of the contractile
72 lete and posterior retinal reattachment; (5) tractional retinal detachment; (6) hypotony/increased in
74 rated growth corresponded to regions of high tractional stress generated within the sheet, as predict
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