ライフサイエンスコーパス: trans fatty acid

1 (containing only 16:0, 18:0, and 16:1,Delta3-trans fatty acids).

2 and n-6 cis polyunsaturated fatty acids and trans fatty acids.

3 ed States are attributable to consumption of trans fatty acids.

4 changes occurred when PO was substituted for trans fatty acids.

5 might be used to replace fats and oils with trans fatty acids.

6 ulation of products to reduce the content of trans fatty acids.

7 rtant new tool for assessing usual intake of trans-fatty acids.

8 , saturated fat (1.43; 1.25-1.64; P < .001), trans-fatty acids (1.03; 1.01-1.05; P < .001), and chole

9 The associations were limited to the trans fatty acids 16:1t (0.12 units; 95% CI: 0.02, 0.22

10 MUFA; 23 vs 21 g/100g fatty acids) and total trans fatty acid (6.5 vs 4.5 g/100g fatty acids) concent

11 ecrease in the intake of saturated (52%) and trans fatty acids (92%), energy (14%) and sodium (47%),

12 nimal fats and gradually reducing intakes of trans fatty acids, a one-third reduction in cholesterol-

13 ited associative data have addressed whether trans fatty acids adversely affect fetal and infant neur

14 ew studies addressed the question of whether trans fatty acids adversely affect human fetal growth.

15 e there are no known nutritional benefits of trans fatty acids and clear adverse metabolic consequenc

16 found between polyunsaturated fatty acids or trans fatty acids and glycemic control in this populatio

17 dothelial cells as a model, the influence of trans fatty acids and magnesium on cell membrane composi

18 the consumption of products that are low in trans fatty acids and saturated fat has beneficial effec

19 in the delta nomenclature usually applied to trans fatty acids and used herein) arouses great scienti

20 nd WT mice a high-fat diet supplemented with trans-fatty acids and fructose (TFF).

21 correlations between total dietary intake of trans-fatty acids and total trans-fatty acid levels in a

22 mendations are to keep saturated fatty acid, trans fatty acid, and cholesterol intakes as low as poss

23 ived stress; alcohol use; intakes of energy, trans fatty acids, and saturated fatty acids; and use of

24 ence of dietary fatty acids, alternatives to trans fatty acids, and the use of alternatives in food m

25 , specific macronutrients, such as fructose, trans-fatty acids, and saturated fat, may contribute to

26 diets as well as limit intakes of fructose, trans-fatty acids, and saturated fat.

27 studies whether total or common subtypes of trans fatty acids are associated with fetal growth.

28 ions of alternative oils and fats to replace trans fatty acids are available or in development.

29 trans Fatty acids are formed during the process of parti

30 be suggestive, then the question of whether trans fatty acids are indeed harmful to human population

31 units) for each 1% increment in energy from trans fatty acids as a replacement for carbohydrates.

32 ietary fat components, such as saturated and trans fatty acids, as well as cholesterol.

33 ffects suggest that not only should the free trans fatty acids be studied but also monitoring the pre

34 Several vanaspati samples exhibited trans fatty acid beyond the permitted limit while trace

35 These include saturated fatty acids and trans fatty acids, both of which raise serum cholesterol

36 taining margarines in the United States (17% trans fatty acids by dry wt).

37 ic data associated with the possibility that trans fatty acids compromise fetal and infant early deve

38 FFQ by comparing the dietary estimates with trans-fatty acid concentrations in adipose tissue.

39 We assessed trans fatty acid consumption by using a validated semiqu

40 Total trans fatty acid consumption during the second trimester

41 observed no associations of first-trimester trans fatty acid consumption with fetal growth.

42 rs further investigated the relation between trans-fatty acid consumption and colorectal neoplasia by

43 trans-Fatty acid consumption is known to have detrimenta

44 trans-Fatty acid consumption, energy adjusted by the res

45 association between colorectal adenomas and trans-fatty acid consumption, the authors utilized data

46 ditional support to recommendations to limit trans-fatty acid consumption.

47 ximated the average trans monoene content of trans fatty acid-containing margarines in the United Sta

48 hrocyte n-3 fatty acids of marine origin and trans fatty acid content are suitable biomarkers for lon

49 lity, polar fraction content and unsaturated trans fatty acid content in any samples.

50 le cardiovascular risk factors, higher total trans fatty acid content in erythrocytes was associated

51 Total trans fatty acid content in erythrocytes was significant

52 rom the lowest to highest quartiles of total trans fatty acid content in erythrocytes were 1.0 (refer

53 A survey to determine the trans fatty acid content of a range of processed foods w

54 ion be minimized and that information on the trans fatty acid content of foods be available to consum

55 ed a relation between preterm births and the trans fatty acid content of maternal plasma.

56 One study reported a correlation between the trans fatty acid content of plasma and birth weight of p

57 by the food industry to lower the artificial trans fatty acid content of processed products.

58 reported an inverse association between the trans fatty acid content of tissue lipids and measures o

59 en hindered by the lack of a database on the trans-fatty acid content of various foods.

60 We assessed the hypothesis that higher trans fatty acid contents in erythrocytes were associate

61 For polyunsaturated and trans fatty acids, correlations between intakes and biom

62 A high intake of trans fatty acids decreases HDL cholesterol and is assoc

63 ull-fat dairy products and naturally derived trans fatty acids did not cause significant changes in c

64 ntinue to confirm previous observations that trans fatty acids elevate low density lipoprotein choles

65 trans Fatty acids elicit effects that are intermediate t

66 A higher maternal intake of trans fatty acids, especially 16:1t and 18:2tc, during t

67 The mean consumption of total trans-fatty acids estimated from the FFQ was 2.24 g per

68 from the possible association found between trans fatty acid exposure and lower n-3 and n-6 long-cha

69 This technique allows the separation of trans fatty acids (FAs) and polyunsaturated FAs (PUFAs)

70 trans Fatty acids (FAs) have been identified as negative

71 ietary and the NO(2)-dependent mechanisms of trans fatty acid formation and will be useful in definin

72 Whereas the negative effect of consuming trans fatty acids found in partially hydrogenated vegeta

73 se trans fatty acids than did an estimate of trans fatty acids from animal sources.

74 CVD) risk is well established, the effect of trans fatty acids from ruminant sources (rTFAs) on CVD r

75 e placed on a diet containing high levels of trans fatty acids, fructose, and cholesterol (HTF-C diet

76 ivation in livers of mice fed a diet rich in trans-fatty acids, fructose, and cholesterol.

77 onclusions: 1) There is little evidence that trans fatty acids have an adverse effect on carcinogenes

78 Unsaturated trans fatty acids have been linked to a higher incidence

79 Trans fatty acids have been shown to increase the produc

80 The effects of trans fatty acids have rarely been studied, but there ar

81 t (bovine), which serve as common origins of trans fatty acids in a typical Western diet that include

82 owed that the amounts of polyunsaturated and trans fatty acids in adipose tissue reflect dietary inta

83 n the fatty acid composition of retail milk, trans fatty acids in particular, and how these change th

84 the need to reduce intakes of saturated and trans fatty acids in the diet.

85 The reduction of trans fatty acids in the food supply is a complex issue

86 The ever-increasing amount of trans fatty acids in the human diet has been linked to a

87 The mean concentration of total trans-fatty acids in buttock adipose tissue was 4.7% of

88 Higher total trans-fatty acids in red blood cell membranes was associ

89 ary fat and specific fatty acids, especially trans fatty acids, in altering concentrations of markers

90 Saturated fatty acids C12:0 and C14:0, trans-fatty acids including conjugated linoleic acid (CL

91 h risk of type 2 diabetes in women, but that trans fatty acids increase and polyunsaturated fatty aci

92 udies has provided unequivocal evidence that trans fatty acids increase plasma concentrations of low-

93 In these same studies, trans fatty acids increased the plasma ratio of total to

94 Animal studies provide little evidence that trans fatty acids influence growth, reproduction, or gro

95 tance of n-3 and n-6 fatty acids; shown that trans fatty acids inhibit delta6 desaturation of linolei

96 k chips rich in PUFA and low in saturated or trans fatty acids instead of high-saturated fatty acid a

97 ve not established a causal relation between trans fatty acid intake and early development.

98 epidemiologic data it has been claimed that trans fatty acid intake causes coronary artery disease (

99 We examined associations of maternal trans fatty acid intake during pregnancy with fetal grow

100 An estimate of trans fatty acid intake from vegetable sources correlate

101 Current estimates of trans fatty acid intake in developed countries range fro

102 s suggest that current efforts at decreasing trans fatty acid intake in foods should take into consid

103 trimester, the estimated mean (+/-SD) total trans fatty acid intake was 2.35 +/- 1.07 g/d, of which

104 The best indicators of total trans fatty acid intake were ct18:2n-6 and tc18:2n-6 (r

105 The relation of trans-fatty acid intake to life-threatening arrhythmias

106 the US Department of Agriculture to estimate trans-fatty acid intake using a self-administered food f

107 Past studies of the association of trans-fatty acid intake with coronary heart disease have

108 We investigated the association of trans-fatty acid intake, assessed through a biomarker, w

109 he time of an interview (controls) to assess trans-fatty acid intake.

110 The increased presence of synthetic trans fatty acids into western diets has been shown to h

111 Intake of trans fatty acids is associated with increased risk of c

112 indings suggest that dietary intake of total trans-fatty acids is associated with modest increase and

113 e estimated that replacing 2% of energy from trans fatty acids isoenergetically with polyunsaturated

114 a6 desaturation of linoleic acid; identified trans fatty acid isomers in fetal, infant, and maternal

115 Adverse effects of industrially produced trans fatty acids (iTFAs) on the risk of coronary artery

116 Both adipose and plasma trans fatty acid levels reflect dietary intake.

117 ietary intake of trans-fatty acids and total trans-fatty acid levels in adipose tissue were 0.67 (95%

118 R technique can be used to rapidly determine trans fatty acids <1% in oils/fat.

119 Dietary trans fatty acids may depress milk lipid synthesis under

120 suggest that consumption of high amounts of trans-fatty acid may increase the risk of colorectal neo

121 Actions to reduce trans fatty acids need to carefully consider both intend

122 For intakes of red and processed meat and trans fatty acids, no association was found in women, wh

123 I review the effects of trans fatty acids, oleic acid, n-3 polyunsaturated fatty

124 d the effects of diets with a broad range of trans fatty acids on serum lipoprotein cholesterol level

125 ids instead of high-saturated fatty acid and trans fatty acid or low-fat snacks leads to improvements

126 re is little evidence to suggest that either trans fatty acids or oleic acid has any specific effect

127 income, and adipose tissue linoleic acid and trans fatty acids (OR for the top versus lowest quintile

128 nificantly lower with a higher proportion of trans fatty acids (OR, 0.24; 95% CI, 0.07 to 0.77), as a

129 Endogenous trans fatty acids originate from diet, but recent studie

130 he fat contributed as soybean oil (<0.5 g of trans fatty acid per 100 g of fat), semiliquid margarine

131 iments all confirm the basic hypothesis that trans fatty acids produce membrane properties more simil

132 al 18:1 trans fatty acid (r = 0.45) and 16:1 trans fatty acid (r = 0.16) were the next best indicator

133 nd tc18:2n-6 (r = 0.58 for each); total 18:1 trans fatty acid (r = 0.45) and 16:1 trans fatty acid (r

134 Total trans fatty acids (r(s)=0.43) and total 18:1 trans isome

135 Whether different classes of trans fatty acids show similar associations is unclear.

136 mparison of PO-rich diets with diets rich in trans fatty acids showed significantly higher concentrat

137 and n-6 fatty acids; appropriate labeling of trans fatty acids, stearic acid, and other non-cholester

138 dence from cohort studies has suggested that trans fatty acid (TFA) consumption may be associated wit

139 In this work, a strategy to produce trans fatty acid (TFA) free (or low TFA) products from p

140 The adverse relation between dietary trans fatty acid (TFA) intake and coronary artery diseas

141 trans fatty acid (TFA) intake increases systemic inflamm

142 trans Fatty acid (TFA) intake predicts risks of coronary

143 The consumption of trans fatty acid (TFA) is linked to the elevation of LDL

144 Trans fatty acids (TFA) are strongly correlated with an

145 ay play a role in the adverse impact dietary trans fatty acids (TFA) have on biological function.

146 Significantly higher amounts of trans fatty acids (TFA) were found in hard margarines (u

147 Intake of trans fatty acids (TFA), which are consumed by eating fo

148 Trans-fatty acid (TFA) consumption is associated with ri

149 Trans-fatty acids (TFA) have been associated with increa

150 belling policy concerning the declaration of trans fatty acids (TFAs) content in the nutritional fact

151 ure; however, effects of naturally occurring trans fatty acids (TFAs) from ruminant animals (rTFA), s

152 ecent efforts in Canada to reduce industrial trans fatty acids (TFAs) in foods include mandated inclu

153 erol-raising oil that can be used to replace trans fatty acids (TFAs) in solid fat applications.

154 Dietary trans fatty acids (TFAs) increase the risk of heart dise

155 Whether elevated intakes of trans fatty acids (TFAs) increase the risk of stroke rem

156 The overall consumption of trans fatty acids (TFAs) increases the risk of coronary

157 The consumption of trans fatty acids (TFAs) is associated with an increased

158 Although trans fatty acids (TFAs) may increase the risk of dyslip

159 ) and IHD mortality when the sum of SFAs and trans fatty acids (TFAs) was theoretically replaced by t

160 with the following: cholesterol, oleic acid, trans fatty acids (TFAs), stearic acid (STE), TFA+STE (4

161 Circulating trans fatty acids (TFAs), which cannot be synthesized by

162 nduces chemical changes such as formation of trans fatty acids (TFAs).

163 l interaction of trans isomeric fatty acids [trans fatty acids (TFAs)] with the availability of long-

164 Trans-fatty acids (TFAs) have deleterious cardiovascular

165 and concentrations of omega-3, omega-6, and trans-fatty acids (TFAs) were expressed as proportions o

166 s correlated much more strongly with adipose trans fatty acids than did an estimate of trans fatty ac

167 Sciences recommended in a position paper on trans fatty acids that models be developed to assess the

168 0.0001) and for a 2% increase in energy from trans fatty acids the RR was 1.39 (1.15, 1.67; P = 0.000

169 , and nutrient intakes (omega-3 fatty acids, trans fatty acids, total fiber, and vitamins K(1), B(6),

170 ody mass index, diet, and long-chain n-3 and trans fatty acids, total VLCSFAs in plasma were associat

171 The number and location of cis and trans fatty acid unsaturations and the absolute stereoch

172 uestionnaires was 0.40; this correlation for trans fatty acids was also 0.40.

173 Low concentrations of trans fatty acids were observed in biscuits (0.86% of to

174 Levels of trans fatty acids were reduced considerably compared wit

175 unsaturated, and 1.16 (CI, 1.06 to 1.27) for trans fatty acids when the top and bottom thirds of base

176 lts indicate a high daily intake of SFAs and trans fatty acids, which may have an unfavourable effect

177 ated the associations of plasma phospholipid trans fatty acids with fatal ischemic heart disease (IHD

178 ), percentage of energy from SFAs, and total trans fatty acids with serum PLFAs in both relative and

179 n-6 polyunsaturated fatty acids (PUFAs) and trans-fatty acids with prostate cancer risk, and whether

180 t status and future implications of reducing trans fatty acids without increasing saturated fats in t

181 fatty acids (PUFAs) and low in saturated and trans fatty acids would improve cardiovascular health.

182 hydrogenated polyunsaturated fatty acids for trans fatty acids would likely reduce the risk of type 2