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1 logic manipulator apart from its function as trans-activator.
2 to selective induction of pro-apoptotic UPR trans-activators.
3 rotein complexes are proposed to function as trans-activators.
6 s expressing an iron-independent form of the trans-activator, Aft1, a protein that regulates the expr
7 the gene is induced by the same iron-sensing trans-activator, Aft1p, that regulates CCC2 and FET3.
8 osaccharomyces pombe is dependent on certain trans activators and a specific nucleotide sequence, whi
9 ce the role of ATF-1 as a hypoxia-responsive trans-activator and identifies a novel regulatory progra
11 n-2A2 (BTN2A2) are regulated by the class II trans-activator and regulatory factor X, two transcripti
12 g proteins such as Bright, a B-cell-specific trans-activator, and the Drosophila melanogaster dead ri
13 y repressed in glioblastomas, the absence of trans-activators appears to be key to the production of
14 evidence that VP16 and the promiscuous viral trans-activator (bICP0) are expressed during the escape
15 The human immunodeficiency virus type 1 Rev trans activator binds directly to unspliced viral mRNA i
16 ntly, the EICP0 and IE proteins are powerful trans-activators but do not function synergistically, (i
17 ree promoters were activated by MHC class II trans-activator, but not CREB-binding protein, whereas I
22 rated that major histocompatibility class II trans-activator (CIITA) is crucial in mediating interfer
25 tive in the transcription factor or class II trans-activator (CIITA) were used to reveal a requiremen
29 stimulating protein 1 (Sp1) as an important trans-activator essential for constitutive activity in c
30 ar factor 1alpha (HNF1alpha) as an obligated trans-activator for PCSK9 gene expression and demonstrat
31 says identified HNF1alpha as the predominant trans-activator for PCSK9 gene working through this sequ
36 ression of mRNAs encoding the liver-enriched trans activators HNF-1, HNF-4, HNF-3 alpha, and HNF-3 be
37 ine the role played by this lineage-inducing trans-activator in the establishment and maintenance of
38 varicella-zoster virus (VZV), a major viral trans-activator, initiates the virus life cycle and is a
39 al activation of endogenous genes by a viral trans-activator is sufficient to induce gene repositioni
40 reby the human immunodeficiency virus type-1 trans-activator might impair tumor suppressor functions
42 ts of HAD, we have used the HIV-1 neurotoxin trans activator of transcription protein (Tat) to invest
46 gamma Lys(49) acetylation were blocked using trans-activator of HIV transcription factor 1 peptides c
47 he 26-kDa protein has now been shown to be a trans-activator of late transcription and it is the prod
49 , our data suggest that Ets-1 functions as a trans-activator of the DOR promoter in the neonatal mous
51 ption factors, C/EBP alpha was the strongest trans-activator of the PEPCK-C gene promoter in the NIH3
55 mmunodeficiency virus (HIV)-specific protein trans-activator of transcription (Tat) can contribute to
56 that the neurotoxic HIV-1 regulatory protein trans-activator of transcription (Tat) continues to be e
57 al and electrophysiological effects of HIV-1 trans-activator of transcription (Tat) in dopamine subty
58 he presence of viral proteins, including the trans-activator of transcription (Tat), has been reporte
59 ed a cell-permeable peptide, which we termed trans-activator of transcription (TAT)-AKAD for TAT-conj
60 peptide modified to include a TAT sequence (Trans-Activator of Transcription gene in HIV; TAT-4BB) a
63 D) tagged with a protein transduction domain trans-activator of transcription was transduced in cultu
64 ate-early (IE) and EICP0 proteins are potent trans-activators of EHV-1 promoters; however, in transie
66 Sp1 (specific protein-1) and Sp3, acting as trans-activators of type I collagen expression in ANF an
67 a null mutant of another endosperm-specific trans-activator Opaque2 (O2) has been shown to be requir
68 disruption of genes for either MHC class II trans-activator or I-A beta) showed no signs of lesion d
69 y is the presence or absence of a particular trans-activator or repressor relevant in determining gen
71 OS gene transcription by S-nitrosylating the trans-activator PARP-1 and decreasing its binding and/or
74 -1 subgenomic promoter and the 34-nucleotide trans-activator prevent expression of a reporter gene.
75 inducing survival and cell division; and as trans-activator, prolonging IFN-gamma synthesis through
76 the iron response element (IRE), and of HIV trans-activator protein (Tat) to the HIV trans-activatio
77 o the arginine-rich motif present in the Tat trans-activator protein of human immunodeficiency virus
78 59-residue stem-loop interacts with the HIV trans-activator protein Tat and other cellular factors t
79 lar factors in addition to the virus-encoded trans-activator protein Tat and the RNA element TAR.
80 groups responsible for interaction with the trans-activator protein Tat, including conformations sim
81 e acidic activation domain of the prototypic trans-activator protein VP16, can itself bind to RPA.
82 th a single vector encoding the tetracycline trans-activator protein, controlled by a constitutive pr
88 is complete before the binding of end-stage trans-activators, supporting the notion that heritable c
89 using a binary lentivector system i.e., the trans-activator, Switch, and the inducible promoter-tran
93 Human immunodeficiency virus (HIV)-encoded trans-activator (Tat) acts through the trans-activation
94 ukemia virus type 1 (HTLV-1) transcriptional trans-activator Tax has been demonstrated to have transf
96 man T-cell lymphotropic virus, type (HTLV)-1 trans-activator, Tax, coordinates with cAMP-responsive e
97 The human T-lymphotropic retrovirus type-I trans-activator, Tax, has been shown to interact directl
98 cell lymphotropic retrovirus type I (HTLV-I) trans-activator, Tax, interacts specifically with the ba
100 he intein-mediated splicing of an artificial trans-activator that signals to a genetic circuit contai
102 GBP1 and heart-specific reverse tetracycline trans-activator transgenes, we expressed human CUGBP1 in
103 While the RNA-sequence specific HIV-1 Tat trans-activator was also shown to enhance processivity i
104 ut also revealed Sp2 to be a relatively weak trans-activator with little or no capacity for additive
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