1 f transcranial magnetic stimulation, using a transcortical approach, in preclinical migraine models.

2                A significant increase in E2 (transcortical component) size of the cutaneomuscular ref

3 an activate cortical neurons interposed in a transcortical long-latency loop leading to pre-programme

4  that amphetamines, piracetam and repetitive transcortical magnetic stimulation may be effective adju

5 S is mediated, in part, by a fast conducting transcortical pathway via the primary motor cortex.

6   This study assessed the importance of fast transcortical pathways for reactive grip responses by ex

7 co-striato-pallidothalamic loops and related transcortical pathways.

8 ng electrocorticography (ECOG) and localized transcortical recordings (bipolar SEEG).

9 lex regulation and it is common to speak of "transcortical reflex loops." Such loops appear to add fl

10 or interneurons, rather than by long-latency transcortical reflex responses.

11 n force is similar to that of 'long-latency' transcortical reflexes recorded from muscles following m

12                                              Transcortical sensory aphasia (TSA) is characterized by

13                                            A transcortical stab wound divided the field of labeled co

14  (rTMS) over the motor cortex on the size of transcortical stretch and mixed nerve reflexes.

15 ) evoked by ulnar nerve stimulation, and (3) transcortical stretch or electrically elicited reflexes.

16 sponses to more 'natural' inputs involved in transcortical stretch reflexes.

17                       The reduced density of transcortical vessels may thus be attributed to impaired