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1 timulation technology called high-definition transcranial alternating current stimulation (HD-tACS).
2 t in human auditory cortex with non-invasive transcranial alternating current stimulation (TACS) [28-
3 determined by increased sigma activity after transcranial alternating current stimulation (tACS) appl
5 transcranial magnetic stimulation (rTMS) and transcranial alternating current stimulation (tACS) have
6 cortical excitability.SIGNIFICANCE STATEMENT Transcranial alternating current stimulation (tACS) is a
7 ntrainment of gamma or beta oscillations via transcranial alternating current stimulation (tACS) over
8 th short bursts of high frequency (>/=80 Hz) transcranial alternating current stimulation (tACS) over
9 we measured rs-fMRI in humans while applying transcranial alternating current stimulation (tACS) to e
14 and beta-band rhythms were manipulated with transcranial alternating current stimulation (tACS) whil
16 rmed the same task while receiving occipital transcranial alternating current stimulation (tACS), to
17 a oscillatory activity in the human M1 using transcranial alternating current stimulation (tACS).
18 rticospinal tES known so far, which is 20 Hz transcranial alternating current stimulation (tACS).
19 hermore, tremor was selectively entrained by transcranial alternating current stimulation applied ove
20 transcranial direct current stimulation, and transcranial alternating current stimulation are used in
21 Second, we found that brief application of transcranial alternating current stimulation at 10 Hz re
30 hat brief application of 2 mA (peak-to-peak) transcranial currents alternating at 10 Hz significantly
35 vestigated the potential of slow oscillatory transcranial direct current stimulation (so-tDCS), appli
38 Working memory (WM) training paired with transcranial direct current stimulation (tDCS) can impro
40 Non-invasive stimulation of the brain using transcranial direct current stimulation (tDCS) during mo
41 interface-assisted motor imagery (MI-BCI) or transcranial direct current stimulation (tDCS) has been
47 ed to the aging brain.SIGNIFICANCE STATEMENT Transcranial direct current stimulation (tDCS) modulates
49 In the present experiment, we tested whether transcranial direct current stimulation (tDCS) of the dl
50 interest in alternative treatments, such as transcranial direct current stimulation (tDCS) of the do
53 stion by applying double-blind bihemispheric transcranial direct current stimulation (tDCS) over both
56 that honesty can be increased in humans with transcranial direct current stimulation (tDCS) over the
61 the non-invasive brain stimulation technique transcranial direct current stimulation (tDCS) targeting
62 ntic information by applying high-definition transcranial direct current stimulation (tDCS) to an fMR
63 the possibility of suppressing the DLPFC by transcranial direct current stimulation (tDCS) to facili
68 ts performing similar value decisions during transcranial direct current stimulation (tDCS), a non-in
70 nce supports application of one type of tCS, transcranial direct current stimulation (tDCS), for majo
71 invasive neuromodulatory techniques, such as transcranial direct current stimulation (tDCS), have sho
72 invasive neuromodulatory techniques, such as transcranial direct current stimulation (tDCS), have sho
77 we show that modulation of this region with transcranial direct current stimulation alters both acti
78 participants received 30 min of real or sham transcranial direct current stimulation applied to the l
79 ine working memory task performance, but the transcranial direct current stimulation group demonstrat
80 ced activation in the left cerebellum in the transcranial direct current stimulation group, with no c
84 ween noise and motor cost, we used bilateral transcranial direct current stimulation of the motor cor
86 nd randomized sham controlled pilot study of transcranial direct current stimulation on a working mem
87 earchers have used brain stimulation such as transcranial direct current stimulation on human subject
88 euronal excitability with anodal or cathodal transcranial direct current stimulation over right front
92 re, we induced neuronal excitation by anodal transcranial direct current stimulation versus sham, exa
94 including transcranial magnetic stimulation, transcranial direct current stimulation, and deep brain
95 s such as transcranial magnetic stimulation, transcranial direct current stimulation, and transcrania
96 suspect that emerging technology, including transcranial direct current stimulation, will follow a s
102 ty (FVsv), and methods derived from arterial transcranial Doppler (aTCD) on the middle cerebral arter
103 r children with sickle cell anaemia and high transcranial doppler (TCD) flow velocities, regular bloo
106 ic attack has not been compared with that of transcranial Doppler (TCD) using a comprehensive meta-an
107 sickle cell anemia (SCA), predicted by high transcranial Doppler (TCD) velocities, is prevented by t
109 oke prevention in children with SCA and high transcranial Doppler (TCD) velocities; after at least a
111 ng, white matter hyperintensities (WMHs) and transcranial doppler (TCD) were used as control conventi
112 g optic nerve sheath diameter (ONSD), venous transcranial Doppler (vTCD) of straight sinus systolic f
114 han 24-hour time frame provides a window for transcranial Doppler examinations and therapeutic interv
116 dren with sickle cell anemia, routine use of transcranial Doppler screening, coupled with regular blo
117 ess contraindicated, and 82% underwent daily transcranial Doppler ultrasonography with embolic monito
118 li burden, assessed noninvasively by bedside transcranial Doppler ultrasonography, correlates with ri
125 splant indications included stroke (n = 12), transcranial Doppler velocity >200 cm/s (n = 2), >/=3 va
127 n injury were the pressure reactivity index, transcranial Doppler-derived mean velocity index based o
129 ive of nine with delayed stroke had positive transcranial Dopplers (at least one microembolus detecte
130 lated vertebral artery injuries had positive transcranial Dopplers before stroke, and positive transc
131 I, 1.01-1.05) and with persistently positive transcranial Dopplers over multiple days (risk ratio, 16
132 without additional vessel injuries, positive transcranial Dopplers predicted stroke after adjusting f
133 cranial Dopplers before stroke, and positive transcranial Dopplers were not associated with delayed s
134 ers (at least one microembolus detected with transcranial Dopplers) before stroke, compared with 46 o
135 l properties of electric fields arising from transcranial electric stimulation (TES) in a nonhuman pr
137 the following working hypothesis: in humans, transcranial electric stimulation (tES) with a time cour
140 Widespread enthusiasm for low-intensity transcranial electrical current stimulation (tCS) is ref
141 In contrast to optogenetic interventions, transcranial electrical stimulation (TES) does not requi
147 above described stimulation, which we named transcranial individual neurodynamics stimulation (tIDS)
148 ENT This study demonstrated that, in humans, transcranial individual neurodynamics stimulation (tIDS)
149 tments currently under investigation include transcranial magnetic or electrical brain stimulation, a
153 in stimulation (STN-DBS) with motor cortical transcranial magnetic stimulation (M1-TMS) at specific t
155 rally patterned waveforms such as repetitive transcranial magnetic stimulation (rTMS) and transcrania
156 nciple trials suggest efficacy of repetitive transcranial magnetic stimulation (rTMS) for the treatme
157 Although several strategies of repetitive transcranial magnetic stimulation (rTMS) have been inves
158 inical and cognitive responses to repetitive transcranial magnetic stimulation (rTMS) in bipolar II d
159 n-invasive brain stimulation like repetitive transcranial magnetic stimulation (rTMS) is an increasin
161 ate the effects of high-frequency repetitive transcranial magnetic stimulation (rTMS) of the right do
162 rtex for treating depression with repetitive transcranial magnetic stimulation (rTMS) remains unknown
163 etic resonance imaging (fMRI) and repetitive transcranial magnetic stimulation (rTMS) to examine the
164 essed this question by combining theta burst transcranial magnetic stimulation (TBS) with fMRI to tes
165 estingly, disrupting cerebellar activity via transcranial magnetic stimulation (TMS) abolished the ad
166 ral prefrontal cortex (DLPFC) using combined transcranial magnetic stimulation (TMS) and electroencep
169 exposure group (N=17) underwent single-pulse transcranial magnetic stimulation (TMS) concurrent with
170 tested whether high-frequency, non-invasive transcranial magnetic stimulation (TMS) delivered twice
172 ous, causal test by combining the FCM with a transcranial magnetic stimulation (TMS) intervention tha
176 rformed the sequential task while undergoing transcranial magnetic stimulation (TMS) of the RLPFC ver
177 s studies have shown asymmetrical effects of transcranial magnetic stimulation (TMS) on task performa
178 Along this scheme, we tested the effect of transcranial magnetic stimulation (TMS) over the hand ar
179 ere we explored this possibility by means of transcranial magnetic stimulation (TMS) over the hand ar
185 In the current study, we used MRI-guided transcranial magnetic stimulation (TMS) to assess whethe
186 s subjects underwent MRI-guided single-pulse transcranial magnetic stimulation (TMS) to co-localise p
188 ed the virtual lesion methodology offered by transcranial magnetic stimulation (TMS) to explore the i
189 e and female participants using single-pulse transcranial magnetic stimulation (TMS) to interfere wit
191 sed peripheral nerve stimulation paired with transcranial magnetic stimulation (TMS) to primary motor
192 ale and female) brain noninvasively, we used transcranial magnetic stimulation (TMS) to probe the exc
197 ronometry of the process by combining online transcranial magnetic stimulation (TMS) with computation
199 ntal eye field (FEF) by combining repetitive transcranial magnetic stimulation (TMS) with subsequent
200 motivation, we hypothesized that inhibitory transcranial magnetic stimulation (TMS) would reduce app
203 d the complexity of the cortical response to transcranial magnetic stimulation (TMS)--an approach tha
211 s to perturbations, as can be assessed using transcranial magnetic stimulation and electroencephalogr
213 on can be blocked in vivo using single pulse transcranial magnetic stimulation and further highlight
214 dress these issues, we combined single-pulse transcranial magnetic stimulation and motor-evoked poten
217 ulation, and non-invasive such as repetitive transcranial magnetic stimulation and transcranial direc
218 nistered post-cortical spreading depression, transcranial magnetic stimulation blocked the propagatio
219 data, a robotic arm positioned a repetitive transcranial magnetic stimulation coil over a subject-sp
220 ar inhibition (CBI): a conditioning pulse of transcranial magnetic stimulation delivered to the cereb
221 eral nerve in close temporal contiguity with transcranial magnetic stimulation delivered to the contr
228 nical neurophysiology of the brain employing transcranial magnetic stimulation has convincingly demon
229 lp electroencephalography (EEG) responses to transcranial magnetic stimulation in 22 participants dur
230 h prior findings from functional imaging and transcranial magnetic stimulation in healthy participant
231 ng functional magnetic resonance imaging and transcranial magnetic stimulation indicated the involvem
232 ysiological biomarkers were assessed using a transcranial magnetic stimulation multiparadigm approach
233 male human participants, whether repetitive transcranial magnetic stimulation of a frontal midline n
235 n motor-evoked potentials (MEPs) elicited by transcranial magnetic stimulation over the ipsilateral m
239 prefrontal cortex target, and 50 repetitive transcranial magnetic stimulation pulses were delivered
243 aging studies of phonological processing, or transcranial magnetic stimulation sites that did not use
244 ted by redefining the borders of each of the transcranial magnetic stimulation sites to include areas
245 are in agreement with functional imaging and transcranial magnetic stimulation studies in human Parki
246 inical and functional assessments along with transcranial magnetic stimulation studies were taken on
248 applying excitatory or inhibitory repetitive transcranial magnetic stimulation to a subject-specific
250 al magnetic resonance imaging and repetitive transcranial magnetic stimulation to demonstrate the rep
256 g changes in motor-cortical excitability via transcranial magnetic stimulation up to 2 h after stimul
257 los with a videoed partner, and double-pulse transcranial magnetic stimulation was applied around the
260 ohort of 57 participants, threshold-tracking transcranial magnetic stimulation was used to assess cor
262 combining inhibitory continuous theta-burst transcranial magnetic stimulation with model-based funct
263 peripheral nerve electrical stimulation and transcranial magnetic stimulation) combined with electro
264 recorded from extensor carpi radialis using transcranial magnetic stimulation, and fractional anisot
265 studies on treatment including medications, transcranial magnetic stimulation, biofeedback, target-s
266 ured corticospinal excitability at rest with transcranial magnetic stimulation, local concentrations
268 ng brain stimulation in addiction, including transcranial magnetic stimulation, transcranial direct c
269 ectromagnetic stimulation techniques such as transcranial magnetic stimulation, transcranial direct c
270 tigate the potential mechanisms of action of transcranial magnetic stimulation, using a transcortical
272 nterfering with rTPJ activity through online transcranial magnetic stimulation, we showed that partic
273 trol site by means of continuous theta-burst transcranial magnetic stimulation, while measuring effor
274 d around sites that had been identified with transcranial magnetic stimulation-based functional local
276 lthy participants, we show how damage to our transcranial magnetic stimulation-guided regions affecte
277 lly compensate for the contribution that the transcranial magnetic stimulation-guided regions make to
279 ween those with and without damage to these 'transcranial magnetic stimulation-guided' regions remain
280 ubjects, as indicated by specific markers of transcranial magnetic stimulation-induced muscle and bra
287 ral activity in the IFC using high frequency transcranial random noise stimulation (tRNS) could enhan
288 ol conditions were as follows: (1) sham, (2) transcranial random noise stimulation (tRNS) in the same
290 tigated these conflicting biases by applying transcranial random noise stimulation (tRNS) while subje
293 upled training with parietal, motor, or sham transcranial random noise stimulation, known for modulat
296 de first time evidence that slow oscillatory transcranial stimulation amplifies the functional cross-
297 es sensory adaptation.SIGNIFICANCE STATEMENT Transcranial stimulation has been claimed to improve per
299 eveloping an animal model to help understand transcranial stimulation, this study will aid the ration
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