ライフサイエンスコーパス: transcranial Doppler

1 measured in the middle cerebral artery using transcranial Doppler.

2 d significant right-to-left shunt defined by transcranial Doppler.

3 stroke risk in patients deemed high risk by transcranial Doppler.

4 measured in the middle cerebral artery using transcranial Doppler.

5 these fields to elucidate the exact role for transcranial Doppler.

6 d and cerebral blood flow was estimated with transcranial Doppler.

7 Preoperative embolization was recorded by transcranial Doppler.

8 disease, particularly the prognostic use of transcranial Doppler, (2) oral contraceptives as a strok

9 With transcranial Doppler and magnetic resonance imaging (MRI

10 .0-23.1]), but without previous stroke, with transcranial doppler and overnight pulse oximetry.

11 n the middle cerebral artery was measured by transcranial Doppler at 12, 24, 36, 48, 60, 72, 84, 96,

12 ive of nine with delayed stroke had positive transcranial Dopplers (at least one microembolus detecte

13 ty (FVsv), and methods derived from arterial transcranial Doppler (aTCD) on the middle cerebral arter

14 ients with acute ischemic stroke, continuous transcranial Doppler augments t-PA-induced arterial reca

15 Transcranial Doppler based static autoregulation measure

16 lated vertebral artery injuries had positive transcranial Dopplers before stroke, and positive transc

17 ers (at least one microembolus detected with transcranial Dopplers) before stroke, compared with 46 o

18 Transcranial Doppler can be a useful tool to detect both

19 Noninvasive ICP estimation using transcranial Doppler can have a role as a screening tool

20 n injury were the pressure reactivity index, transcranial Doppler-derived mean velocity index based o

21 lateralization was measured with functional transcranial Doppler during language production.

22 cardiopulmonary exercise tests with contrast transcranial Doppler, esophageal, and gastric balloon ca

23 Study evaluations included: 1) transcranial Doppler evaluations to determine middle cer

24 han 24-hour time frame provides a window for transcranial Doppler examinations and therapeutic interv

25 Serial transcranial Doppler examinations before, during, and 3

26 penicillin up to the age of 5 years, annual transcranial Doppler examinations from the ages of 2 to

27 le cerebral artery blood flow measured using transcranial Doppler flowmetry.

28 study, which has substantial advantages over transcranial Doppler for the assessment of CBF.

29 Transcranial Doppler has an established role in diagnosi

30 surement of cerebral blood velocity (CBV) by transcranial Doppler has been used to identify patients

31 However, transcranial Doppler has poor specificity, and transfusi

32 Although there are several settings in which transcranial Doppler has well established usefulness, th

33 e cerebral artery blood flow velocity (MCAv; transcranial Doppler), heart rate (ECG), blood pressure

34 Procedural HITS were detected by transcranial Doppler in all patients.

35 With the advent of transcranial Doppler, measurement of cerebral blood flow

36 oplethysmographic mean arterial pressure and transcranial Doppler middle cerebral artery blood flow v

37 Early recanalization (ER) was identified by transcranial Doppler monitoring during the first 2 hours

38 Continuous transcranial Doppler monitoring of middle cerebral arter

39 the microembolic signals (MESs) detected by transcranial Doppler on the use of different ablation te

40 I, 1.01-1.05) and with persistently positive transcranial Dopplers over multiple days (risk ratio, 16

41 without additional vessel injuries, positive transcranial Dopplers predicted stroke after adjusting f

42 he control group, whereas cardiac output and transcranial Doppler readings were similar.

43 Transcranial Doppler recordings were made from the ipsil

44 We studied cerebral hemodynamics with transcranial Doppler recordings, and we closely examined

45 dren with sickle cell anemia, routine use of transcranial Doppler screening, coupled with regular blo

46 velocity was measured in 6 patients through transcranial Doppler sonography of the middle cerebral a

47 aterality assessment using coarse functional transcranial Doppler sonography should be interpreted wi

48 Previous research using functional transcranial Doppler sonography showed that blood flow v

49 erebral angiography, perfusion scintigraphy, transcranial Doppler sonography, CT angiography and MR a

50 than clinical or haematological features, or transcranial doppler sonography.

51 analization at 24 hours on CTA regardless of transcranial Doppler status was labeled as CTR.

52 history of stroke had to have undergone two transcranial Doppler studies that showed that the time-a

53 icroembolic signals to the brain detected by transcranial Doppler study and can predict the developme

54 ents (55 children) for indications including transcranial Doppler (TCD) abnormalities, AIS, or previo

55 Transcranial Doppler (TCD) and the National Institutes o

56 on of asymptomatic embolic signals by use of transcranial doppler (TCD) could predict stroke risk in

57 In 1992, transcranial Doppler (TCD) evidence of elevated intracra

58 ing/magnetic resonance angiography (MRA) and transcranial Doppler (TCD) exams were performed at entry

59 sickle cell anemia (SCA), but its effects on transcranial Doppler (TCD) flow velocities remain undefi

60 r children with sickle cell anaemia and high transcranial doppler (TCD) flow velocities, regular bloo

61 , computed tomographic angiography (CTA) and transcranial Doppler (TCD) have been advocated as altern

62 In France, transcranial Doppler (TCD) is not recognized as a legal

63 Early transcranial Doppler (TCD) screening of the Creteil sick

64 With transcranial Doppler (TCD) screening, we can identify ch

65 ic attack has not been compared with that of transcranial Doppler (TCD) using a comprehensive meta-an

66 sickle cell anemia (SCA), predicted by high transcranial Doppler (TCD) velocities, is prevented by t

67 n with sickle cell anemia (SCA) and abnormal transcranial Doppler (TCD) velocities.

68 oke prevention in children with SCA and high transcranial Doppler (TCD) velocities; after at least a

69 uantitative analyses of angiograms and daily transcranial Doppler (TCD) were performed.

70 ng, white matter hyperintensities (WMHs) and transcranial doppler (TCD) were used as control conventi

71 , transesophageal echocardiography (TEE) and transcranial Doppler (TCD), with saline contrast.

72 Forty-four underwent transcranial Doppler (TCD).

73 le-cell anemia at high risk as determined by transcranial Doppler (TCD).

74 crease in cerebral microemboli detectable by transcranial doppler (TCD).

75 to 16 years selected for high stroke risk by transcranial Doppler (TCD).

76 ty (TAMV) of 200 cm/s or more as measured by transcranial Doppler (TCD).

77 ransesophageal echocardiography and contrast transcranial doppler to detect patent foramen ovale.

78 contrast transthoracic echocardiography and transcranial Doppler to identify PFO.

79 investigated this proposal using functional transcranial Doppler ultrasonography (fTCD), which asses

80 tion Trial has confirmed that utilization of transcranial Doppler ultrasonography (TCD), which examin

81 andomly assigned to receive continuous 2-MHz transcranial Doppler ultrasonography (the target group)

82 ocardiogram with second harmonic imaging and transcranial Doppler ultrasonography during a standardiz

83 Since its introduction in 1982, transcranial Doppler ultrasonography has become an impor

84 6 months over the following 18 months using transcranial Doppler ultrasonography in 144 patients wit

85 ic polysomnography for sleep apnea underwent transcranial Doppler ultrasonography of the middle cereb

86 t was complete recanalization as assessed by transcranial Doppler ultrasonography or dramatic clinica

87 Primary stroke prevention through transcranial Doppler ultrasonography screening may ultim

88 imary stroke prevention has occurred through transcranial Doppler ultrasonography screening, but util

89 Transcranial Doppler ultrasonography that is aimed at re

90 iddle cerebral arteries were insonated using transcranial Doppler ultrasonography to calculate mean m

91 This experiment used functional transcranial Doppler ultrasonography to demonstrate that

92 We used transcranial Doppler ultrasonography to identify childre

93 Transcranial Doppler ultrasonography was used to determi

94 Transcranial Doppler ultrasonography was used to measure

95 ess contraindicated, and 82% underwent daily transcranial Doppler ultrasonography with embolic monito

96 rtery mean blood flow velocity (MCAVm) using transcranial Doppler ultrasonography, and expressed resp

97 li burden, assessed noninvasively by bedside transcranial Doppler ultrasonography, correlates with ri

98 Jugular venous bulb oximetry, transcranial Doppler ultrasonography, electroencephalogr

99 markers, urine osmolality, neurodevelopment, transcranial Doppler ultrasonography, growth, and mutage

100 amentarium in Parkinson's disease, including transcranial Doppler ultrasonography, radiolabeled trace

101 ernal-carotid or middle-cerebral arteries on transcranial doppler ultrasonography.

102 signals (ES) may be detected with the use of transcranial Doppler ultrasonography.

103 kle cell anemia who have abnormal results on transcranial Doppler ultrasonography.

104 and vasomotor reactivity were measured with transcranial Doppler ultrasonography.

105 Blood flow was determined by transcranial Doppler ultrasound (cerebral blood flow) an

106 s using magnetic resonance imaging (n = 26), transcranial Doppler ultrasound (n = 35) and perfusion c

107 atic microembolic signals (MES), detected by transcranial Doppler ultrasound (TCD), are markers of fu

108 ith a neurologic exam, complete blood count, transcranial Doppler ultrasound (TCD), measurement of in

109 od flow velocities (CBFV) were measured with transcranial Doppler ultrasound along with noninvasive b

110 to determine sensitivity and specificity of transcranial Doppler ultrasound and cerebral angiography

111 We reviewed transcranial Doppler ultrasound data of 199 patients; 55

112 Transcranial Doppler ultrasound data were available in 2

113 bolic signals can be detected in patients by transcranial Doppler ultrasound despite aspirin and hepa

114 The sensitivity and specificity of transcranial Doppler ultrasound for anterior circulation

115 The sensitivity of transcranial Doppler ultrasound for anterior circulation

116 All patients then underwent Transcranial Doppler ultrasound measurements of OAF para

117 Daily transcranial Doppler ultrasound monitoring could provide

118 Bilateral transcranial Doppler ultrasound monitoring of the middle

119 Transcranial Doppler ultrasound recordings from the ipsi

120 Transcranial Doppler ultrasound signs of vasospasm impro

121 The mean times for symptomatic and transcranial Doppler ultrasound signs of vasospasm prese

122 Transcranial Doppler ultrasound was as sensitive as cere

123 The reliability of transcranial Doppler ultrasound was better at detecting

124 Transcranial Doppler ultrasound was performed to identif

125 Transcranial Doppler ultrasound was used to measure bloo

126 vessels, the sensitivity and specificity of transcranial Doppler ultrasound were middle cerebral art

127 omatic circulating emboli can be detected by transcranial Doppler ultrasound, are frequent in patient

128 al artery velocity (MCAV) was measured using transcranial Doppler ultrasound, as an index of CBF, in

129 nd 2 patients showed microembolic signals in transcranial Doppler ultrasound.

130 Transcranial Doppler US can be used to identify patients

131 red, one of which was detected with abnormal transcranial Doppler US findings.

132 Of the eight patients with abnormal transcranial Doppler US results who underwent MR imaging

133 sis, results of seven of the 26 preoperative transcranial Doppler US studies were abnormal, and all o

134 sis, results of six of the nine preoperative transcranial Doppler US studies were abnormal.

135 en were examined with nonimaging and imaging transcranial Doppler US techniques on the same day, for

136 Transcranial Doppler US was performed through the tempor

137 substantially between nonimaging and imaging transcranial Doppler US.

138 splant indications included stroke (n = 12), transcranial Doppler velocity >200 cm/s (n = 2), >/=3 va

139 event stroke in those children with abnormal transcranial Doppler velocity (>/=200 cm/s).

140 intracranial pressure, mean cerebral artery transcranial Doppler velocity, PaCO2, cerebral perfusion

141 g optic nerve sheath diameter (ONSD), venous transcranial Doppler (vTCD) of straight sinus systolic f

142 e, and asymptomatic) indicated that abnormal transcranial Doppler was more common in the seizure (4/6

143 Using transcranial Doppler, we compared the frequency of MES d

144 cranial Dopplers before stroke, and positive transcranial Dopplers were not associated with delayed s

145 learning difficulty, headaches, or abnormal transcranial Doppler), who also underwent bolus-tracking

146 ur shaft fracture with RLS evaluation, daily transcranial Doppler with embolus detection studies, and

147 The number of emboli detected by transcranial Doppler within 3 hours of CEA was independe