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1 ernal-carotid or middle-cerebral arteries on transcranial doppler ultrasonography.
2 and vasomotor reactivity were measured with transcranial Doppler ultrasonography.
3 signals (ES) may be detected with the use of transcranial Doppler ultrasonography.
4 kle cell anemia who have abnormal results on transcranial Doppler ultrasonography.
5 rtery mean blood flow velocity (MCAVm) using transcranial Doppler ultrasonography, and expressed resp
6 li burden, assessed noninvasively by bedside transcranial Doppler ultrasonography, correlates with ri
7 ocardiogram with second harmonic imaging and transcranial Doppler ultrasonography during a standardiz
8 Jugular venous bulb oximetry, transcranial Doppler ultrasonography, electroencephalogr
9 investigated this proposal using functional transcranial Doppler ultrasonography (fTCD), which asses
10 markers, urine osmolality, neurodevelopment, transcranial Doppler ultrasonography, growth, and mutage
12 6 months over the following 18 months using transcranial Doppler ultrasonography in 144 patients wit
13 ic polysomnography for sleep apnea underwent transcranial Doppler ultrasonography of the middle cereb
14 t was complete recanalization as assessed by transcranial Doppler ultrasonography or dramatic clinica
15 amentarium in Parkinson's disease, including transcranial Doppler ultrasonography, radiolabeled trace
17 imary stroke prevention has occurred through transcranial Doppler ultrasonography screening, but util
18 tion Trial has confirmed that utilization of transcranial Doppler ultrasonography (TCD), which examin
20 andomly assigned to receive continuous 2-MHz transcranial Doppler ultrasonography (the target group)
21 iddle cerebral arteries were insonated using transcranial Doppler ultrasonography to calculate mean m
26 ess contraindicated, and 82% underwent daily transcranial Doppler ultrasonography with embolic monito
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