ライフサイエンスコーパス: transcription by

1 -copies of the MSR1 element can repress gene transcription by 50 to 115-fold; (3) the higher-expressi

2 is positively autoregulated at the level of transcription by a mechanism that requires cAMP receptor

3 studies demonstrate that MUC1-C drives BMI1 transcription by a MYC-dependent mechanism in breast and

4 mouse O4(+) cells inhibited TH-induced mMog transcription by a yet unknown mechanism.

5 MLL-ENL and MLL-AF10 constitutively activate transcription by aberrantly inducing both AEP-dependent

6 After blocking transcription by actinomycin D, R38X appeared to acceler

7 manganese superoxide dismutase (MnSOD) gene transcription by activating MKK4 via redox control of Cy

8 ltaneously, fumarate increases ferritin gene transcription by activating the NRF2 (nuclear factor [er

9 Enhanced AR gene transcription by ADT provides a prerequisite condition t

10 1 domain and suppresses ribosomal RNA (rRNA) transcription by affecting the protein level of UBF.

11 o acid determinant for maximal activation of transcription by AgrA and provides novel insights into s

12 tion, DNA packing and repair, as well as for transcription by allowing changes in DNA topology.

13 e transcript, working in cis, regulated HAS2 transcription by altering the chromatin structure around

14 in vitro, suggesting that IdeR induces bfrB transcription by antagonizing the repressor activity of

15 d subnuclear compartments, and regulation of transcription by antisense transcription.

16 tion of the 25-kb Lockd locus reduced Cdkn1b transcription by approximately 70% in an erythroid cell

17 elayed by the apparent lack of resumption of transcription by arrested polymerases.

18 nal regulators RbpA and CarD act to modulate transcription by associating to the initiation complex a

19 se (RNAP) accomplishes multiple tasks during transcription by assuming different structural forms.

20 talk logic imparting differential control of transcription by AT1R, integrins, and calcium channels.

21 te that epigenetic upregulation of Npr1 gene transcription by ATRA and NaBu leads to attenuation of r

22 histone phosphorylation, in turn, regulates transcription by attenuating the effect of local arginin

23 tein produced in the very late phase of gene transcription by Autographa californica multiple nucleop

24 interplay between these two elements during transcription by bacterial RNAP.

25 Tat, a small protein that facilitates viral transcription by binding an RNA structure (trans-activat

26 Bacterial 6S RNAs globally regulate transcription by binding RNA polymerase (RNAP) holoenzym

27 cle regulatory factors drive tissue-specific transcription by binding selectively near muscle-specifi

28 Under normal conditions, AmpR represses ampC transcription by binding the PG precursor UDP-N-acetylmu

29 igations revealed that IRF8 suppressed STAT1 transcription by binding the STAT1 promoter.

30 BBR inhibits gene transcription by binding the TATA boxes in the transcrip

31 In contrast, menin promotes gene transcription by binding the transcription activator MLL

32 racts with its target mRNAs initially during transcription by binding through its N-terminal prion-li

33 Most activated response regulators regulate transcription by binding tightly to promoter DNA via a p

34 RNA has been shown to potently repress mRNA transcription by binding tightly to RNA polymerase II (P

35 tailed in this review, estrogen can regulate transcription by binding to 2 nuclear receptors, ERalpha

36 series antimicrobials allosterically inhibit transcription by binding to a conserved alpha helix (bet

37 First, KLF11 activates transcription by binding to a distinct Sp-KLF site withi

38 Intranuclear uPA modulates gene transcription by binding to a subset of transcription fa

39 We show here that Bmh inhibits transcription by binding to Adr1 at promoters that conta

40 ranscription factor complex to activate CD73 transcription by binding to an intronic enhancer.

41 assical biotype V. cholerae repressed vieSAB transcription by binding to its promoter.

42 RA activates transcription by binding to nuclear RA receptors (RARs)

43 DksA alters transcription by binding to RNA polymerase and allosteri

44 tors (TFs) regulate complex programs of gene transcription by binding to short DNA sequence motifs.

45 we demonstrate that FOXO3a regulates its own transcription by binding to the conserved response eleme

46 The first is that ILF3 activates uPA transcription by binding to the CTGTT sequence in the nu

47 SypG is predicted to activate syp transcription by binding to the syp enhancer (SE), a con

48 lity, in erythroid cells, Sp1 activates PIGM transcription by binding upstream of (but not to) the -2

49 We found that p38gamma increases EGFR transcription by c-Jun-mediated promoter binding and sti

50 Transcription by "cap-snatching" requires that host-cell

51 ylases (HDACs) are believed to regulate gene transcription by catalyzing deacetylation reactions.

52 e context, however, the reduction of COOLAIR transcription by cdkc;2 disrupts a COOLAIR-mediated repr

53 Ongoing transcription by cellular RNA polymerase II (Pol II) is

54 Precise transcription by cellular RNA polymerase requires the ef

55 n was directly linked to CREB-dependent gene transcription by chromatin immunoprecipitation that show

56 transcription factors play key roles in HAEC transcription by co-binding enhancers associated with EC

57 pha was shown to directly regulate Mfn1 gene transcription by coactivating the estrogen-related recep

58 ur model predicted the correct gating of CBF transcription by cold only when the cold signal originat

59 ase pair regulates the efficiency of initial transcription by controlling multiple steps including do

60 tion functions to indirectly maintain active transcription by counteracting H3K9 dimethylation and ge

61 CBP; an essential cofactor for activation of transcription by CREB) impair long-term synaptic plastic

62 cate a mechanism for selective regulation of transcription by CtBP and LSD1 involving their associati

63 nistically, Sirt6 inhibits Notch1 and Notch4 transcription by deacetylating histone H3K9.

64 revents the premature termination of reverse transcription by degrading the dNTPase SAMHD1.

65 inding factor ETS-1, and it represses miR-31 transcription by delivering the H3K4me3 demethylase JARI

66 mb histone methyltransferase Ezh2 stabilizes transcription by depositing repressive marks during deve

67 sis further shows that FACT facilitates gene transcription by destabilizing the tetranucleosomal unit

68 nsistent with a role for USP42 in regulating transcription by deubiquitylating histones.

69 We determine that PhoP activates transcription by different mechanisms, and that ancestra

70 y Klf5 as an activator of Dmp1 and Dspp gene transcriptions by different mechanisms and demonstrate t

71 ry protein CueR both represses and activates transcription by differentially modulating local DNA str

72 ining DNA binding domains generally regulate transcription by direct interaction with DNA.

73 report that nuclear CaMKII activates cardiac transcription by directly binding to chromatin and regul

74 Our results suggest that Xist silences transcription by directly interacting with SHARP, recrui

75 Mechanistically, PBX1 plays a dual role in transcription by directly repressing or activating genes

76 esence of CRY, nuclear entry of PER inhibits transcription by displacing CLOCK-BMAL1 from the promote

77 e human MMP13 and IL1B genes modulates their transcription by distinct mechanisms.

78 c transcription factors (GSTFs) control gene transcription by DNA binding and specific protein comple

79 tigated and the regulatory mechanism of TNFA transcription by DNA methylation is explored.

80 We find that repression of reaper transcription by Dpp is mediated by Schnurri.

81 gs suggest a mechanism for the repression of transcription by E2F7.

82 that Olfm2 mediates TGF-beta-induced SM gene transcription by empowering SRF binding to CArG box in S

83 ts greatest antiviral potency during reverse transcription by enhancement of G-to-C transversion muta

84 hat testosterone robustly represses hepcidin transcription by enhancing Egfr signaling in the liver a

85 histone methyltransferase to repress NEDD4L transcription by enhancing histone H3 lysine 27 trimethy

86 ce that GIV positively autoregulates its own transcription by enhancing STAT3 activation via its guan

87 suggested that A20 knockdown increased STAT1 transcription by enhancing TBK1 activation and subsequen

88 hal initiation, suggesting that Hir1 affects transcription by establishing transcriptional thresholds

89 molecular players involved in initiation of transcription by eukaryotic RNA polymerase II (Pol II) a

90 chromatin environment is thought to inhibit transcription by excluding transcription factors and RNA

91 These factors switch on and off transcription by exerting dynamic control of DNA stereoc

92 -equilibrium description informs our view of transcription by explicitly considering time- and energy

93 sults suggest that CTCF promotes HSV-1 lytic transcription by facilitating the elongation of RNA Pol

94 s usually achieved by exogenous induction of transcription by factors acting in the nucleus.

95 ve uncovered a mechanism for effective viral transcription by focal assembly of RNA polymerase II aro

96 d, glucocorticoid response element-dependent transcription by formoterol was displaced to the left by

97 wn regulation of Cln3 translation and Cln1,2 transcription by glucose is sufficient to explain the ex

98 a previously unrecognized regulation of gene transcription by GPCR-cAMP signaling through augmentatio

99 element mediated cooperative enhancement of transcription by GR and NF-kappaB that required the pres

100 RNAs modify chromatin structure and silence transcription by guiding Argonaute-containing complexes

101 h X chromosomes of hermaphrodites to repress transcription by half.

102 MeCP2 represses transcription by histone deacetylase (HDAC)-dependent an

103 Major features of transcription by human RNA polymerase II (Pol II) remain

104 reveal new insights into cell type-specific transcription by identifying novel transcription units,

105 link between the negative regulation of Rag transcription by IL-7 and a novel repressive pathway inv

106 es cerevisiae ATPase Mot1 globally regulates transcription by impacting the genomic distribution and

107 demonstrated that entinostat enhanced NKG2D transcription by increasing acetylation of Histones H3 a

108 the BH conformation, whereas CBRs deregulate transcription by increasing coupling between the BH and

109 Here, we report that Snai1 inhibits beta4 transcription by increasing repressive histone modificat

110 gX regulation revealed that it can stimulate transcription by increasing the binding of RNA polymeras

111 s release of paused polymerase and activates transcription by increasing the number of transcribing p

112 Zinc treatment induces Dnmt1 transcription by increasing the occupancy of MTF-1 on th

113 AT can prevent TRAP-mediated termination of transcription by inducing dissociation of TRAP from the

114 e and also to the nascent RNA and influences transcription by inducing pausing and facilitating the p

115 est that TRIM5alpha(rh) blocks HIV-1 reverse transcription by inducing premature viral uncoating in t

116 st, and Chikungunya viruses inhibit cellular transcription by inducing rapid degradation of Rpb1, a c

117 , antisense transcription can regulate sense transcription by induction of epigenetic modifications.

118 H4K16 acetylation (H4K16Ac) activates gene transcription by influencing both chromatin structure an

119 omoter of its target gene Nfia and represses transcription by influencing epigenetic events.

120 that p21 can indirectly block HIV-1 reverse transcription by inhibiting host cofactors supporting HI

121 1,25(OH)2D3 downregulates bic transcription by inhibiting NF-kappaB activation, which

122 r results demonstrate that PKA regulates frq transcription by inhibiting RCM-1 activity through RCM-1

123 tly been shown to exert selective effects on transcription by inhibiting the function of the alpha su

124 rions, it also significantly restricts HIV-1 transcription by inhibiting the NF-kappaB pathway.IMPORT

125 on increased, whereas inhibition of INS gene transcription by INS promoter targeting siRNA decreased

126 ERbeta promotes PHD2 transcription by interacting with a unique estrogen resp

127 unliganded PRA paradoxically activates Cx43 transcription by interacting with FRA2/JUND heterodimers

128 MAF1 represses Pol III-mediated transcription by interfering with TFIIIB and Pol III.

129 rsional stress from DNA and facilitates gene transcription by introducing transient DNA double-strand

130 brain is masculinized by direct induction of transcription by ligand-activated nuclear steroid recept

131 rotein complex, plays a pivotal role in gene transcription by linking gene-specific transcription fac

132 Bacterial transcription activators regulate transcription by making essential protein-protein intera

133 biological response toward DNA damage during transcription by manipulating their gene expression.

134 We observed upregulation of IL-4 transcription by MCp in mLN and spleens of helminth-infe

135 1) has been reported to repress and activate transcription by mediating histone H3K4me1/2 and H3K9me1

136 Induction of Duox2 transcription by microbial colonization did not require

137 roth are required for activation of vps gene transcription by Mlc.

138 which are accessory proteins that antagonize transcription by modifying chromatin structure.

139 p of activators maintain heritable states of transcription by modifying nucleosomal histones or remod

140 whether intergenic lncRNAs commonly regulate transcription by modulating chromatin at genomically dis

141 that Sp100 isoforms differentially regulate transcription by modulating lysine acetylation.

142 trand reduces the yield of T7 RNA polymerase transcription by more than an order of magnitude when po

143 mensional chromosomal conformations regulate transcription by moving enhancers and regulatory element

144 However, the mechanism of gene transcription by MTA1/NuRD remains enigmatic.

145 Ebf1 and c-Myb antagonize Rag transcription by negatively regulating the binding of Fo

146 Although the regulation of BDNF transcription by neuronal activity has been widely studi

147 ults suggest that the downregulation of Ezh2 transcription by NFIB is an important component of the p

148 Thus repression of Fbw7beta transcription by Notch signalling is an essential mechan

149 This suggests transcription by novel promoters for 72% of Arabidopsis

150 ostasis is regulated largely at the level of transcription by nuclear receptors, particularly the pri

151 tion of RNA polymerase II (Pol II)-dependent transcription by nucleating pre-initiation complex (PIC)

152 assessed the role of LRPPRC in mitochondrial transcription by performing size exclusion chromatograph

153 Furthermore, we show that activation of transcription by PF1088 is dependent upon the presence o

154 nique strategy for selectively targeting MYC transcription by pharmacological means as a potential tr

155 n/Wnt activities and p38gamma stimulates Wnt transcription by phosphorylating beta-catenin at Ser605.

156 clin-dependent kinase, CDK7, which regulates transcription by phosphorylating the carboxy-terminal do

157 VRK2 increases NFAT1-dependent transcription by phosphorylation, and this effect is onl

158 icative of a direct regulation of qseBC gene transcription by PmrAB under physiological conditions.

159 been partly attributed to the increased rDNA transcription by Pol I in cancer.

160 sequencing (GRO-seq) to measure the rates of transcription by Pol II following gene activation.

161 Here we show that non-coding transcription by Pol V is controlled by preexisting chro

162 oactivator complexes SAGA and NuA4 stimulate transcription by post-translationally modifying chromati

163 those described previously for regulation of transcription by ppGpp.

164 P(XD)-N(TAIL) interaction results in reduced transcription by preformed transcriptases, suggesting re

165 nd DNA-DNA cross-links block replication and transcription by preventing DNA strand separation, contr

166 sults in modulation of estrogen-induced gene transcription by preventing Estrogen Receptor chromatin

167 c DNA lesions that block DNA replication and transcription by preventing strand separation.

168 scriptional regulators required to fine tune transcription by preventing the spread of active chromat

169 ome remodeler SWI/SNF, facilitated divergent transcription by promoting rapid nucleosome turnover.

170 cluster potentiated bursts in CREB-mediated transcription by promoting recruitment of the CREB coact

171 discuss the potential benefits of activating transcription by promoting RNA polymerase isomerization

172 glucose catabolism, acetyl-CoA, induces CLN3 transcription by promoting the acetylation of histones p

173 This reduction in methylation could enhance transcription by promoting the binding of transcription

174 of the regulon prevented repression of SPI1 transcription by propionate.

175 into chromosome segregation genes and alter transcription by providing a premature termination site,

176 ed the stability of the viral genome and TAg transcription by quantitative reverse transcriptase PCR

177 k to understand the control of mycobacterial transcription by RbpA and CarD.

178 entary to exon 1 of GNG12-AS1 suppresses its transcription by recruiting Argonaute 2 and inhibiting R

179 ethylated DNA motif (TCCTGCNA) and represses transcription by recruiting chromatin remodeling corepre

180 activate ADD1 and CCND1 but repress p53 gene transcription by recruiting differential chromatin modif

181 nscription factor SLUG/SNAI2, repressing its transcription by recruiting HDAC1 and licensing the remo

182 nteract with acetylated histones to regulate transcription by recruiting Positive Transcription Elong

183 HLX1 represses p16(INK4a) transcription by recruiting PRCs and HDAC1.

184 th different levels of methylation affecting transcription by recruiting various factors to distinct

185 ns showed that MUC1-C acted to elevate PD-L1 transcription by recruitment of MYC and NF-kappaB p65 to

186 r localization, DNA binding, and target gene transcription by reducing AKT-dependent FOXO1 phosphoryl

187 s aggregation, and promote AR-dependent gene transcription by regulating AR-cofactor interactions.

188 el in which modulation of Kcs1 controls INO1 transcription by regulating synthesis of inositol pyroph

189 Cdc7-Dbf4 promotes NDT80 transcription by relieving repression mediated by a comp

190 hromatin remodeling complex facilitates gene transcription by remodeling chromatin using the energy o

191 sition from elongation to termination during transcription by removing RNAPII CTD serine 5 phosphoryl

192 nto the elongation and termination phases of transcription by removing the phosphate marker on serine

193 p21 inhibits RNR2 transcription by repressing E2F1 transcription factor, i

194 sitive transcriptional regulation of icaADBC transcription by repressing icaR in S. aureus strain 832

195 ment reflect a rapid silencing of new ifngr1 transcription by repressive transcriptional regulators.

196 ntributes to temporal regulation of myogenic transcription by restricting late gene expression during

197 how different forms of DNA alkylation impair transcription by RNA Pol II in cells and with the isolat

198 Initiation of gene transcription by RNA polymerase (Pol) III requires the a

199 a tiny number are estimated to be active in transcription by RNA polymerase (Pol) III.

200 Transcription by RNA polymerase (RNAP) in bacteria requi

201 Transcription by RNA polymerase (RNAP) is interrupted by

202 regulation in organisms minimally depends on transcription by RNA polymerase and on the stability of

203 Transcription by RNA polymerase can induce the formation

204 we report that SWI/SNF also plays a role in transcription by RNA polymerase I (Pol I) in Saccharomyc

205 ion factor A (CITFA), which is essential for transcription by RNA polymerase I (Pol I) in the parasit

206 show that the THO complex positively affects transcription by RNA polymerase I (Pol I).

207 Transcription by RNA polymerase I (Pol-I) is the main dr

208 te that DNA binding by Rrn3 is essential for transcription by RNA polymerase I.

209 Transcription by RNA polymerase II (Pol II) in eukaryote

210 Transcription by RNA polymerase II (Pol II) in metazoans

211 Transcription by RNA polymerase II (Pol II) is a complex

212 Transcription by RNA polymerase II (Pol II) is dictated

213 Transcription by RNA polymerase II (Pol II) is required

214 kely required for nucleosome survival during transcription by RNA polymerase II (Pol II) through chro

215 t of this process is repression of host cell transcription by RNA polymerase II (Pol II), which also

216 scopy, we probed the spatial organization of transcription by RNA polymerase II (RNAP II) molecules a

217 Pre-mRNA splicing is coupled to transcription by RNA polymerase II (RNAP II).

218 Initiation and regulation of transcription by RNA polymerase II (RNAPII) in eukaryote

219 Transcription by RNA polymerase II (RNAPII) is accompani

220 Histone modifications regulate transcription by RNA polymerase II and maintain a balanc

221 p50 in both locations is dependent on active transcription by RNA polymerase II and requires the N-te

222 l repeat (LTR) retrotransposons, begins with transcription by RNA polymerase II followed by reverse t

223 C is involved in CtBP-mediated regulation of transcription by RNA polymerase II in vivo.

224 We propose that transcription by RNA polymerase II is tuned to optimize

225 mplex is known to be a master coordinator of transcription by RNA polymerase II, and this complex is

226 tRNA-gene mediated (tgm) silencing of local transcription by RNA polymerase II.

227 whose function is important for terminating transcription by RNA polymerase II.

228 f human mRNA decapping factors in control of transcription by RNA polymerase II.

229 way is essential to maintain the accuracy of transcription by RNA polymerase II.

230 quently occurs at the same time and place as transcription by RNA polymerase II.

231 DNA and identification of the start site for transcription by RNA polymerase II.

232 h tRNA and 5S rRNA genes and regulates their transcription by RNA polymerase III (pol III) through di

233 ugh much is known about the initial phase of transcription by RNA polymerase III (Pol III), the enzym

234 FIIIC are multi-subunit factors required for transcription by RNA polymerase III.

235 showed that template methylation impedes U6 transcription by RNA polymerase III.

236 uence of U6 promoter is critical for optimal transcription by RNA polymerase III.

237 Transcription by RNA polymerase is a highly dynamic proc

238 Transcription by RNA polymerase may be interrupted by pa

239 ons of late genes, which would prevent their transcription by RNA polymerase.

240 Analysis of host cell transcription by RNA-seq during latency in AGS cells ide

241 Further characterization of ELL2-dependent transcription by RNA-Seq revealed that approximately 12%

242 e evidence for an involvement of the TEFs in transcription by RNAPI (or RNAPIII).

243 ssing of mRNA and the coupled termination of transcription by RNAPII requires the CF IA complex.

244 BET proteins regulate transcription by selectively recognizing acetylated lysi

245 can reverse T-cell anergy, suppresses sirt1 transcription by sequestering FoxO3a to the cytoplasm th

246 nt mediator of the regulation of PPAR-gamma2 transcription by SHP.

247 ying the circadian regulation of chloroplast transcription by SIG5 are unidentified.

248 and the circadian regulation of chloroplast transcription by SIG5 was predominantly dependent on blu

249 , gp79, an inhibitor of host and early phage transcription by sigma(70) holoenzyme, activated transcr

250 tingly, the selective activation of IL-1beta transcription by SIRT1 deficiency is likely mediated thr

251 ical model includes a description of genetic transcription by SREBP-2 which is subsequently translate

252 1 and likely other factors dampens activated transcription by stabilizing Tup1 binding and stimulatin

253 robe all intermediate RNA transcripts during transcription by stalling elongation complexes at cataly

254 a reduction of KIT expression ablates DNMT1 transcription by STAT3 pathway leading to in-parallel mo

255 e that glucagon plus insulin increases FGF21 transcription by stimulating ATF4 expression and that ac

256 show that (1) H3K4me3 enhances p53-dependent transcription by stimulating preinitiation complex (PIC)

257 Suppression of ncRNA transcription by strand-specific CRISPR/dCas9 results in

258 In bacteria, RNA polymerase (RNAP) initiates transcription by synthesizing short transcripts that are

259 Activation of LXR-mediated transcription by synthetic agonists, such as T0901317 an

260 f primer assembly of DNA templates, in vitro transcription by T7 RNA polymerase and kit-based purific

261 and studied the effects of this structure on transcription by T7 RNA polymerase in vitro.

262 ors and CRISPR-Cas9 methods aiming to reduce transcription by targeting DNA.

263 Lysine acetylation regulates transcription by targeting histones and nonhistone prote

264 We find that both complexes influence transcription by targeting promoters and TSS-distal enha

265 n, whereas miR-122 indirectly inhibits c-Myc transcription by targeting Tfdp2 and E2f1.

266 Notably, miR-122 also repressed c-Myc transcription by targeting transcriptional activator E2f

267 osteoblasts abolished the inhibition of Ocn transcription by TGFbeta, confirming an indirect mechani

268 We found that timing of Shh transcription by the cell cycle clock can be reset, thus

269 NA can mimic B-form DNA to directly regulate transcription by the DNA-dependent RNAP.

270 the Escherichia coli btuB riboswitch during transcription by the E. coli RNA polymerase.

271 The repression of E-cadherin transcription by the EMT inducers Snail1 and Zeb2 plays

272 scription by sigma(70) holoenzyme, activated transcription by the gp36 holoenzyme in vitro.

273 , many trans-acting factors that orchestrate transcription by the individual Pols have been described

274 ysis and found that Mel-18 increased miR-205 transcription by the inhibition of DNA methyltransferase

275 l and temporal restriction of organizer gene transcription by the integration of two major signaling

276 described the augmentation of GRE-dependent transcription by the LABA formoterol.

277 of Cavalpha1.2; and promotion of Cavalpha1.2 transcription by the prevention of Kir/Gem-mediated shut

278 promoter and MUC1-C promotes TCF7L2-mediated transcription by the recruitment of beta-catenin and p30

279 in the mgtA mRNA that permits termination of transcription by the Rho protein upstream of mgtA, where

280 y in keratinocytes with 11-fold induction of transcription by the risk allele versus 8-fold by the co

281 s, this is a result of activation of class I transcription by the T cell enhanceosome consisting of R

282 al proteins and activation of endogenous RNA transcription by the TEC inside the virus.

283 le and readily displaced in the direction of transcription by the transcriptional machinery.

284 f MCL-1 mitochondrial localization and BFL-1 transcription by the viral EBNA3A protein.

285 Activation of transcription by the yeast activator Gcn4, for example,

286 lncRNAs that are targeted, at their site of transcription, by the YTH domain of the RNA-binding prot

287 unit of the Mediator complex, which controls transcription by thyroid hormone and other nuclear hormo

288 xpression, in part through inhibition of HIV transcription by TRIM22 and decrease in T-cell activatio

289 ich suggest that HiNmlR enhances target gene transcription by twisting of operator DNA sequences in a

290 CRL4(DCAF1) promotes YAP- and TEAD-dependent transcription by ubiquitylating and, thereby, inhibiting

291 nit of the RNA polymerase where it activates transcription by unknown mechanisms, and it may also inf

292 results demonstrate that regulation of gene transcription by uPA contributes to cancer stemness and

293 HP initiates reverse transcription by using itself as a protein primer (prote

294 HP initiates reverse transcription by using itself as a protein primer (prote

295 nd RNA G4-ligand interactions during reverse transcription, by using a selective reverse transcriptas

296 ein is considered a master regulator of gene transcription by virtue of its ability to modulate the e

297 global and specific alterations of host cell transcription by viruses.

298 Fenofibrate transactivates MDR3 gene transcription by way of the binding of PPARalpha to thre

299 The slowing down of Pol II transcription by ZFP-1/DOT-1.1 is associated with an inc

300 the enhancement of TCF/beta-catenin-mediated transcription by ZMIZ2.