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1 lear factor erythroid 2-related factor 2 and transcription factor 4.
2 ogen species and regulated by the activating-transcription factor-4.
3 eracting RNA clusters (3.6-fold) and curated transcription factors (4.1-fold), including zinc-finger
4 factor of activated T cells (NFAT) family of transcription factors [4, 5], we have quantified protein
6 acid deprivation did not require activating transcription factor 4, a critical component of the conv
7 iolized cells stained with antibody to helix transcription factor 4, a factor associated with the cil
8 sitol-requiring enzyme 1alpha and activating transcription factor 4 abrogates autophagosome formation
9 nes (C/EBP homologous protein and activating transcription factor 4), accumulation of ceramide, loss
10 itol requiring kinase 1alpha, and activating transcription factor 4, all of which are featured change
11 factor 2alpha phosphorylation and activating transcription factor 4 and C/EBP homologous protein up-r
12 pha (eiF2alpha) and expression of activating transcription factor 4 and tribbles 3 were elevated in H
14 B, high mobility group AT-hook 2, spalt-like transcription factor 4, and alpha-fetoprotein expression
16 pseudokinase tribbles homolog 3, activating transcription factor 4, and transcription factor CCAAT/-
17 iched for components of the ATF4 (activating transcription factor 4) arm of the unfolded protein resp
18 log 1) was regulated by the ATF4 (activating transcription factor 4) arm of the unfolded protein resp
20 stress/UPR genes, including BiP, activating transcription factor 4 (ATF-4), ATF-6, and a spliced for
22 by demonstrating the induction of activating transcription factor-4 (ATF-4) and growth arrest DNA dam
23 stent eif2-alpha phosphorylation (activating transcription factor 4 [ATF-4], C/EBP homologous transcr
24 rd increasing downstream signals, activating transcription factor 4 (ATF4) and ATF6 indicating potent
25 ial translation of mRNAs, such as activating transcription factor 4 (ATF4) and C/EBP-homologous prote
26 ivated, as the expression of both activating transcription factor 4 (ATF4) and CHOP (DDIT3), critical
27 hesis but enhances translation of activating transcription factor 4 (ATF4) and is a crucial evolution
28 In silico analysis identified activating transcription factor 4 (ATF4) as a potential transcripti
30 ochondrial stressors, we identify activating transcription factor 4 (ATF4) as the main regulator of t
31 ranscription was conferred by two activating transcription factor 4 (ATF4) binding sites in the FGF21
33 s pathway, linked functionally to activating transcription factor 4 (ATF4) following treatment with o
34 response (UPR) and an increase in activating transcription factor 4 (ATF4) has been previously report
35 nvestigate the protective role of activating transcription factor 4 (ATF4) in controlling the hepatic
36 We evaluate a potential role of activating transcription factor 4 (Atf4) in invertebrate and mammal
37 ling pathway of ER stress-induced activating transcription factor 4 (ATF4) in the regulation of Mulle
44 HRI also activates the eIF2alphaP-activating transcription factor 4 (ATF4) ISR in primary erythroid p
46 ively enhances the translation of activating transcription factor 4 (ATF4) mRNA to induce stress resp
47 trast, obese mice lacking hepatic activating transcription factor 4 (Atf4) showed an exaggerated ISR
49 nase RNA-like ER kinase (PERK) or activating transcription factor 4 (ATF4) significantly reduced the
50 f AP20187 significantly increased activating transcription factor 4 (ATF4) translation and concomitan
51 are transcriptionally induced by activating transcription factor 4 (Atf4) via C/ebp-Atf-Response-Ele
52 of the bZip transcription factor activating transcription factor 4 (ATF4) was induced by glutathione
53 eIF2alpha phosphorylation induces activating transcription factor 4 (ATF4), a basic leucine zipper tr
54 ed cells stimulated expression of activating transcription factor 4 (ATF4), a master transcription fa
56 of triglyceride metabolism by the activating transcription factor 4 (ATF4), a member of the basic leu
57 important effector of the ISR is activating transcription factor 4 (ATF4), a transcription factor th
58 element binding protein 2 (CREB2)/activating transcription factor 4 (ATF4), a transcriptional repress
59 eases translation of the metazoan activating transcription factor 4 (ATF4), activating the integrated
60 rsenite, in a manner dependent on activating transcription factor 4 (ATF4), an important mediator of
61 ing protein-1 mRNA, expression of activating transcription factor 4 (ATF4), and cleavage of ATF6 were
62 ulator of amino acid homeostasis, activating transcription factor 4 (ATF4), is dysfunctional in HD be
63 lectively increase translation of Activating Transcription Factor 4 (ATF4), resulting in the inductio
65 cts with the transcription factor Activating Transcription Factor 4 (ATF4), which is involved in the
66 rine synthesis were stimulated by activating transcription factor 4 (ATF4), which was activated by mT
67 e histone demethylase JMJD3 is an activating transcription factor 4 (ATF4)-dependent target gene.
77 leucine zipper (bZIP) domains of activating transcription factor-4 (ATF4) and CCAAT box/enhancer-bin
78 addition, the CREB-related factor activating transcription factor-4 (ATF4) has been shown to interact
79 or 2 (eIF2alpha) phosphorylation, activating transcription factor-4 (ATF4) induction, and increased e
81 of the transcriptional activators activating transcription factor 4, C/EBPalpha, Runx1, and Runx2.
82 on, particularly up-regulation of activating transcription factor-4, CHOP (C/EBP homologous protein),
83 The ER-stress element and the activating transcription factor 4 element in the CHOP promoter were
85 control nonderepressible 2 kinase-activating transcription factor 4 (GCN2-ATF4) pathway activation an
87 t in BioTapestry, includes 22 genes encoding transcription factors, 4 genes encoding known signaling
88 ose-regulated protein-78 (GRP78), activating transcription factor 4, growth arrest and DNA damage-ind
89 alian vascular factor Kruppel-like family of transcription factor 4 has a conserved role in augmentin
91 ion of WNT-dependent inhibitory Kruppel-like transcription factor 4 in miR-199a-5p-overexpressing cel
94 e inhibitor bortezomib, levels of activating transcription factor-4 increase dramatically early in dr
95 aryotic initiation factor-2alpha, activating transcription factor 4, inositol requiring kinase 1, and
97 The transcription factor ATF4 (activating transcription factor 4) is induced by multiple PD-releva
98 nduced nuclear translocation of Kruppel-like transcription factor 4 (KLF4), a known repressor of SMC
99 nscriptionally regulated by the Kruppel-like transcription factor 4 (KLF4), we determined the spatial
100 with enhanced expression of octomer-binding transcription factor 4 (OCT-4) and several other genes c
101 s well as down-regulation of octamer-binding transcription factor 4 (Oct-4) expression and matrix met
103 ve for alkaline phosphatase, octamer-binding transcription factor 4 (Oct-4), stage-specific embryonic
105 are within binding sites of octamer-binding transcription factor 4 (OCT4) and signal transducer and
106 ed stem cell markers such as octamer binding transcription factor 4 (Oct4) and stage-specific embryon
109 nd POU5F1, the gene encoding octamer-binding transcription factor 4 (Oct4; also known as POU domain,
110 ivation of the PKR-like ER kinase/activating transcription factor 4 (PERK/ATF4) ER stress pathway, in
111 y transcription factor Oct4 (octamer-binding transcription factor 4) plays an unappreciated role in t
113 e treatment and is independent of activating transcription factor 4 signaling and protein translation
118 basic helix-loop-helix transcription factor transcription factor 4 (TCF4) in the nervous system is u
120 ain-of-function of the psychiatric risk gene transcription factor 4 (TCF4) severely disrupts the colu
121 ted as inhibitors of the beta-catenin/T cell transcription factor 4 (Tcf4) signaling pathway using a
122 schizophrenia, SNPs in or in the vicinity of transcription factor 4 (TCF4), neurogranin (NRGN) and an
123 c chondrocytes showed that HIF1alpha lowered transcription factor 4 (TCF4)-beta-catenin transcription
126 1.39 (1.24-1.55)] and at 18q21 [rs1452787 at transcription factor 4 (TCF4); P = 2.61 x 10(-8) , OR (9
129 lity of the CFTR Y-box for sequence-specific transcription factors; 4) that trichostatin A, an inhibi
132 in Stat4 (Signal Transducer and Activator of Transcription Factor 4) was the causative mutation.
133 protein kinase RNA-like ER kinase, activator transcription factor 4) were assessed in transgenic mice
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