ライフサイエンスコーパス: transcription factor CHOP

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1 of CCAAT/enhancer-binding protein homologous transcription factor (CHOP).

2 -box binding protein-1 (Xbp-1) mRNA, and the transcription factor CHOP.

3 xhibited elevated levels of the proapoptotic transcription factor CHOP.

4 um (ER stress), is a powerful inducer of the transcription factor CHOP.

5 uppressive activity of MDSCs is regulated by transcription factor Chop.

6 an endoplasmic reticulum (ER) stress-related transcription factor, CHOP.

7 s, BiP and PERK, as well as the proapoptotic transcription factor, CHOP.

8 , but is thought to drive cell death via the transcription factor, CHOP.

9 The CAAT/enhancer binding protein homologous transcription factor CHOP, also known as GADD153, is inv

10 optotic proteins was detected, including the transcription factor CHOP and Bim, an essential factor f

11 3 induction is substantially mediated by the transcription factors CHOP and ATF4.

12 y in the UPR literature: the function of the transcription factor CHOP as a protective or a prodeath

13 s is mediated by the ER stress regulator and transcription factor CHOP, but not the tumor suppressor

14 ed by Adv-DNFoxo, including the proapoptotic transcription factor CHOP (C/EBP [CCAAT/enhancer binding

15 The transcription factor CHOP (C/EBP homologous protein 10)

16 AT, -59 to -54) that presumptively binds the transcription factor CHOP [CAAT enhancer binding protein

17 Induction of the transcription factor CHOP (CCAAT-binding homologous prot

18 The inhibition of the major stress-inducible transcription factor CHOP (CCAAT/enhancer-binding protei

19 Deletion of the UPR mediator transcription factor CHOP completely rescues the motor d

20 induced by eIF2alpha-P is that encoding the transcription factor CHOP (DDIT3/GADD153).

21 The transcription factor CHOP (GADD153) heterodimerizes with

22 After chronic UPR activation, the transcription factor CHOP (GADD153/DDIT3) triggers cell

23 ome that features elevated expression of the transcription factor CHOP (GADD153/DDIT3).

24 ce to oxidative stress, and the proapoptotic transcription factor CHOP (GADD153/DDIT3).

25 an increased expression of the pro-apoptotic transcription factor CHOP-(gadd153), a downstream event

26 er pathways, including the expression of the transcription factor CHOP/GADD153 and its downstream tar

27 posarcomas, consists of a fusion between the transcription factor CHOP/GADD153 and the N terminus of

28 The transcription factor CHOP/GADD153 gene is induced by cel

29 ted samples revealed that the stress-induced transcription factor CHOP/Gadd153 was dramatically up-re

30 ess-dependent expression of the proapoptotic transcription factor chop/gadd153.

31 he data reveal a major negative role for the transcription factor CHOP in overall survival during sep

32 scription factor 4 [ATF-4], C/EBP homologous transcription factor [CHOP], oxireductase endoplasmic re

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