ライフサイエンスコーパス: transcription factor E2F-1

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1 itory complex, and partial activation of the transcription factor E2F-1.

2 f the cell cycle by its association with the transcription factor E2F [1-3].

3 The transcription factor E2F-1, a downstream regulator of th

4 In contrast, overexpression of transcription factor E2F-1, a known target for the activ

5 Transcription factor E2F-1 and its interaction with pRb

6 e regulators: the Rb family of proteins, the transcription factor E2F-1, and the p21 family of protei

7 een the retinoblastoma protein (pRb) and the transcription factor E2F-1 are thought to be important f

8 ion analyses revealed that ASA inhibited the transcription factor E2F-1 binding activity to the survi

9 iously been shown that overexpression of the transcription factor E2F-1 can induce apoptosis in quies

10 The transcription factor E2F-1 has been postulated to play a

11 y reported that forced overexpression of the transcription factor E2F-1 in human HT-1080 fibrosarcoma

12 binding of the retinoblastoma protein to the transcription factor E2F-1 in vivo, and induced the acti

13 The transcription factor E2F-1 induces both cell-cycle progr

14 The transcription factor E2F-1 induces cell cycle progressio

15 The transcription factor E2F-1 interacts stably with cyclin

16 nce of hypophosphorylated pRb and binding to transcription factor E2F-1 is consistent with ZD1694-ind

17 The transcription factor E2F-1 is implicated in the activati

18 Recent reports show that the transcription factor, E2F-1, may play a role in mediatin

19 Transcription factor E2F-1 mediates apoptosis and suppre

20 The transcription factor E2F-1 plays a central role in the c

21 The transcription factor E2F-1 plays a key role in regulatin

22 The transcription factor E2F-1 promotes S-phase entry and de

23 The transcription factor E2F-1 promotes vascular smooth musc

24 Overexpression of the transcription factor E2F-1 regulates growth of human cor

25 The cellular transcription factor E2F-1, which is a downstream effect

26 nant-negative mutants of the Rb gene, or the transcription factor E2F-1, which is a downstream target

27 prevented induction of the expression of the transcription factor E2F-1, which positively regulates t

28 30 induction also decreased cyclin A and the transcription factor E2F-1 while increasing cyclin E at

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