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1 xpression by creating a binding site for the transcription factor Oct-1.
2 nes contain binding sites for the ubiquitous transcription factor Oct-1.
3 egulator of the broadly expressed POU domain transcription factor Oct-1.
4 in did not inhibit the expression of nuclear transcription factor Oct-1.
5 amer sequence, which recruits the POU domain transcription factor Oct-1.
6 GRE in vitro along with GR is the POU domain transcription factor Oct-1.
7 ng site for the broadly expressed POU domain transcription factor Oct-1.
8 and was recruited as a coactivator with the transcription factor Oct-1.
10 ith two cellular factors-the POU homeodomain transcription factor Oct-1 and the cell proliferation fa
11 f BRCA1, we demonstrated that BRCA1 binds to transcription factor OCT-1 and up-regulates the transcri
12 ctivator that functions with octamer binding transcription factors (Oct-1 and Oct-2) to mediate effic
14 o the nucleolus and shares homology with the transcription factors Oct-1 and -2, Pit-1, and POU-M1.
15 ts have presented the novel observation that transcription factors Oct-1 and NF-YA participate in the
16 Physical associations of BRCA1 protein with transcription factors Oct-1 and NF-YA, which directly bi
19 histocompatibility genes and octamer-binding transcription factors Oct-1 and Oct-2, respectively.
21 ein partially recognized by antibody against transcription factor Oct-1 binds to the LREAA element co
22 his report, we demonstrate that the cellular transcription factor Oct-1 cooperates with the EBV immed
23 However, the role for the octamer-binding transcription factor Oct-1 in the DNA damage-activated r
24 ave presented the novel observation that the transcription factor Oct-1 is induced after cells are ex
26 ve BRAF upregulates HMGCL through an octamer transcription factor Oct-1, leading to increased intrace
27 fore, these results have implicated that the transcription factor Oct-1 might participate in cellular
28 he host cell factor HCF-1 and the POU domain transcription factor Oct-1, on TAATGARAT-containing sequ
29 y the interaction of a POU domain containing transcription factor Oct-1 or Oct-2, with the B-cell-spe
30 activation, on the DNA binding of the basal transcription factor Oct-1, or on hydrogen peroxide-indu
33 f the ubiquitously expressed POU-homeodomain transcription factor, Oct-1, strongly represses transcri
34 FPIV) with a binding site for the ubiquitous transcription factor Oct-1 that appears crucial for horm
35 phism (SNP) that causes the helix-turn-helix transcription factor OCT-1 to bind to a novel region of
36 rs and enhancers is regulated by two related transcription factors, Oct-1 (ubiquitous) and Oct-2 (B l
37 xpression of certain genes by activating the transcription factor Oct-1, while p53 has been reported
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