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1 in transcription, as a module of the general transcription factor TFIIH.
2 odification of XPB, a subunit of the general transcription factor TFIIH.
3 CTD is phosphorylated at Ser 5 by the basal transcription factor TFIIH.
4 L interacts with p44, a subunit of the basal transcription factor TFIIH.
5 , also functions as a subunit of the general transcription factor TFIIH.
6 a DNA helicase and a subunit of the general transcription factor TFIIH.
7 e repairosome and the RNA polymerase H basal transcription factor TFIIH.
8 nstructural protein NSs and the host general transcription factor TFIIH.
9 h include the multisubunit RNA polymerase II transcription factor TFIIH.
10 nt of the CTD kinase activity of the general transcription factor TFIIH.
11 dent kinase 7 (CDK7), a subunit of the basal transcription factor TFIIH.
12 s of recruitment and activity of the general transcription factor TFIIH.
13 E. coli, and is not a component of the basal transcription factor, TFIIH.
14 versible inhibitor of the XPB subunit of the transcription factor TFIIH and initiation of RNA polymer
15 itute the TFIIK kinase subcomplex of general transcription factor TFIIH and to mutations in Cak1, whi
17 We now report that TAF7 interacts with the transcription factors, TFIIH and P-TEFb, resulting in th
18 major CTD kinase is a subunit of the general transcription factor TFIIH, and is encoded by an essenti
19 timulates the CTD kinase activity of general transcription factor TFIIH, and subsequent CTD phosphory
20 bind to XPB, the largest subunit of general transcription factor TFIIH, and to cause degradation of
21 (HSSB)], and the multisubunit (7-10) general transcription factor TFIIH are required for the dual inc
22 K, a subcomplex of RNA polymerase II general transcription factor TFIIH, are encoded by the yeast cyc
23 minary studies, we have identified the basal transcription factor TFIIH as the potential target for u
25 extracts, which was complemented by the NER/transcription factor TFIIH, but not by purified Mms19 pr
26 ith transcription sites and with the general transcription factor TFIIH, but not with the splicing fa
27 Tat can also interact with the multisubunit transcription factor TFIIH complex and increase the phos
28 nt Srb4, but not on the Kin28 subunit of the transcription factor TFIIH, even though both proteins ar
29 The DNA helicases XPB and XPD, components of transcription factor TFIIH, have been implicated in a p5
30 (OC) requires DNA unwinding mediated by the transcription factor TFIIH helicase-related subunit XPB/
31 cates that the AR interacts with the general transcription factor TFIIH in a physiological condition.
32 arge subunit of RNA polymerase II by general transcription factor TFIIH is believed to be an importan
35 XPB, the largest subunit of the eukaryotic transcription factor TFIIH, is essential for both initia
40 tations in the XPD subunit of the DNA repair/transcription factor TFIIH result in distinct clinical e
41 ction between SCL and a subunit of the basal transcription factor TFIIH, suggesting a potential means
42 CDK7 is the kinase subunit of the general transcription factor TFIIH that phosphorylates the C-ter
43 tion by disrupting the assembly of the basal transcription factor TFIIH through sequestration of its
44 rboxy-terminal domain (CTD) of Pol II by the transcription factor TFIIH through the associated CAK ki
46 ukaryotes, XPB is an integral subunit of the transcription factor TFIIH, which plays a dual role in D
47 ntosum group D (XPD) protein is a subunit of transcription factor TFIIH with DNA helicase activity.
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