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1 f IAP transcripts, Gag proteins, and reverse transcription products.
2 ntry, before the generation of viral reverse transcription products.
3 ability of MLV particles to generate reverse transcription products.
4 prising approximately 80% of the total viral transcription products.
5 mate size expected for a full-length reverse transcription product and with plus-strand strong-stop D
7 decreased amounts of early and late reverse transcription products and integrated virus relative to
8 xosomes reduce accumulation of HIV-1 reverse transcription products and steady-state levels of HIV-1
9 ing strategy to characterize nascent reverse transcription products and their precise 3'-termini in H
10 , results in reduced accumulation of reverse transcription products, and is dominant in heterokaryons
11 tilize an unspliced version of their primary transcription product as an RNA template for synthesis o
13 is to investigate the length distribution of transcription products at various times following initia
15 and MDDCs revealed similar levels of reverse transcription products, but increased nuclear import in
16 es by preventing the accumulation of reverse transcription products, but the underlying mechanism rem
17 evealed that the generation of early reverse transcription products coincides with the timing of unco
18 integration, indicating that a viral reverse transcription product comprises the HIV-1 pathogen-assoc
19 ll mutants tested produced levels of reverse transcription products either similar to or only somewha
21 the normal generation of HIV-1 late reverse transcription products, even though HIV-1 infection and
22 llowing a blood meal, and those accumulating transcription products exclusively or preferentially in
24 how a decreased tendency to produce aberrant transcription products from DNA templates having protrud
26 e was determined by real-time PCR on reverse transcription products from RNA isolated from human corn
29 e conserved exonic IES determine alternative transcription products in the developing macronucleus; s
31 aggregates promote synthesis of long reverse transcription products in vitro by concentrating nucleic
33 te the synthesis of RNA polymerase (Pol) III transcription products, including tRNAs and 5S rRNAs, wa
34 tive real-time PCR analysis of early reverse transcription products indicated that HIV-1 reverse tran
36 iant was impaired in generating late reverse transcription products, indicating that replication was
37 , and mediate the integration of the reverse-transcription product into the genome of the host cell.
39 multiple ways and that inhibition of reverse transcription products is not necessary for TRIM5-mediat
40 tantially reduced, neither the amount of HBV transcription products nor the core polypeptide was dete
42 s oxyR homolog and provide evidence that the transcription product of this gene binds to the B. abort
45 antitative real-time PCR analysis of reverse transcription products revealed that the mutant RTs were
47 e, and cell death through an undefined CIITA transcription product that may serve as a new antiviral
48 iency virus type 1 (HIV-1), allowing reverse transcription products to accumulate even though viral i
49 ication, including synthesis of late reverse transcription products, two-long terminal repeat circles
50 ve methods that monitor formation of reverse transcription products, two-LTR circles and integrated p
51 microring resonators to detect cDNA reverse transcription products via a subsequent enzymatic signal
54 indicate that faithful, full-length reverse transcription products were underrepresented in the abse
55 ction and quantitative PCR for HIV-1 reverse transcription products, whereas treatment of the target
56 tive real-time PCR analysis of early reverse transcription products, with initiation at the HIV-1 PBS
57 thesis.IMPORTANCE The fates of HIV-1 reverse transcription products within infected cells are not wel
58 r viral DNA as well as intracellular reverse transcription products, without affecting HBV RNAs or cc
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