ライフサイエンスコーパス: transepidermal

1 Transepidermal inoculation of vaccinia virus (VV), or sc

2 fetal rats displayed no measurable barrier (transepidermal water loss > 10 mg per cm2 per h), a meas

3 asol, respectively, with parallel changes in transepidermal water loss (P < .05).

4 ] and urocanic acid [UCA]) using UPLC-MS/MS, transepidermal water loss (TEWL) and epidermal pH.

5 ipid barrier as measured by skin resistance, transepidermal water loss (TEWL) and Fourier transform i

6 The effects were quantified using transepidermal water loss (TEWL) and were correlated wit

7 ering techniques of laser Doppler imaging, a transepidermal water loss (TEWL) device and a skin therm

8 tional Endpoints (BASELINE) birth cohort had transepidermal water loss (TEWL) measured in the early n

9 Transepidermal water loss (TEWL) measures were collected

10 rthermore, to determine whether increases in transepidermal water loss (TEWL) predate the development

11 of the skin and measurements of the rate of transepidermal water loss (TEWL) were recorded sequentia

12 RCM, transepidermal water loss (TEWL), and fluorescence excit

13 We measured water flux across the SC, transepidermal water loss (TEWL), in six women, in vivo.

14 rneum was assessed by measuring capacitance, transepidermal water loss (TEWL), rates of absorption-de

15 sure in wild-type and Serpinb3a-null mice on transepidermal water loss (TEWL), sensitization, and inf

16 e associated with atopic dermatitis (AD) and transepidermal water loss (TEWL).

17 n barrier function was assessed by measuring transepidermal water loss (TEWL).

18 versely correlated (r=-0.654, P=0.0004) with transepidermal water loss (TEWL).

19 ion examining the permeability barrier using transepidermal water loss (TEWL).

20 recovery of barrier function as measured by transepidermal water loss after tape stripping.

21 the delayed barrier recovery as measured by transepidermal water loss after tape-stripping.

22 unction, as measured by corneometry, pH, and transepidermal water loss also normalized with treatment

23 Transepidermal water loss and a(*) (skin redness) decrea

24 rier development, evidenced by a decrease in transepidermal water loss and an enhanced outside-in bar

25 We assessed here, first, baseline transepidermal water loss and barrier recovery kinetics

26 These deposits increased transepidermal water loss and caused irritation, particu

27 This was associated with increased transepidermal water loss and development of eczematous

28 of the epidermis associated with a decreased transepidermal water loss and increased proinflammatory

29 Bacterial diversity correlated with transepidermal water loss and pH level but not with corn

30 to barrier disruption, indicated by elevated transepidermal water loss and reduced lipid synthesis en

31 was to investigate the relationship between transepidermal water loss and skin permeability to triti

32 certained using confocal Raman spectroscopy; transepidermal water loss and skin surface pH were measu

33 more prematurely have elevated rates of both transepidermal water loss and transcutaneous heat loss,

34 on of the barrier function of skin increases transepidermal water loss and up-regulates inflammatory

35 d to interpret the significance of measuring transepidermal water loss by evaporimetry.

36 These data indicate that transepidermal water loss cannot be unconditionally ascr

37 ximately 0.5 U); (ii) enhanced SC integrity (transepidermal water loss change with sequential tape st

38 We then assessed skin pH and transepidermal water loss every 12 hours on the burn wou

39 lents in SCID/NOD mice demonstrated enhanced transepidermal water loss following s.c. administration

40 Previous measurements of transepidermal water loss have suggested that, regardles

41 active plaque phenotypes displayed elevated transepidermal water loss levels, increased numbers of e

42 quency impedance spectroscopy, to complement transepidermal water loss measurements.

43 tic administration of all activators tested (transepidermal water loss range 4.0-8.5 mg per cm2 per h

44 There was no correlation between transepidermal water loss rate and 3H2O permeability fol

45 Similarly, there was no correlation between transepidermal water loss rates and the 3H2O permeabilit

46 No correlation was found between basal transepidermal water loss rates and the permeability of

47 While basal transepidermal water loss rates are near normal, barrier

48 Transepidermal water loss rates declined during explant

49 Whereas baseline transepidermal water loss rates were elevated by approxi

50 orneocytes and its contribution to increased transepidermal water loss was confirmed by tape strippin

51 Transepidermal water loss was measured on unaffected for

52 Transepidermal water loss was normal in all conditions.

53 In an acute AD model, skin transepidermal water loss was significantly attenuated i

54 y normal, and exhibited an increase in basal transepidermal water loss without alteration in basal mi

55 se curve, with maximal sweating (measured as transepidermal water loss) (mean 70 g m(-2) hour(-1)) af

56 rations, skin barrier dysfunction (increased transepidermal water loss), and AD in infancy.

57 Our study showed that transepidermal water loss, a parameter of skin barrier i

58 l epidermis, corneocyte fragility, increased transepidermal water loss, and local inflammation in the

59 measurements of percentage of skin redness, transepidermal water loss, and participant-assessed pain

60 permeability indicated by higher pH, greater transepidermal water loss, and reduced lipid synthesis e

61 Changes in total sign scores (TSSs), transepidermal water loss, and tissue biomarkers (determ

62 ing IL-13Ralpha2 had significantly increased transepidermal water loss, cutaneous inflammation, perip

63 mal permeability barrier defects with severe transepidermal water loss, decreased intercellular lipid

64 m, with SerpinB2(-/-) mice showing increased transepidermal water loss, increased overt loss of strat

65 hat cutaneous barrier formation, measured as transepidermal water loss, is delayed in male fetal rats

66 sebopsoriasis displayed a lesser increase in transepidermal water loss, normal numbers of lamellar bo

67 present in these dogs, as well as increased transepidermal water loss, particularly in sites charact

68 exercise, on several dermatologic measures: transepidermal water loss, recovery of skin barrier func

69 e markers also significantly correlated with transepidermal water loss, suggesting a link between the

70 by reduced transglutaminase (TGM) activity, transepidermal water loss, up-regulation of the proinfla

71 nce analysis to measure hydration status and transepidermal water loss, we show that the epidermal ba

72 ith severe dehydration, suggesting excessive transepidermal water loss, which was confirmed by in vit

73 primary cell population responsible for high transepidermal water loss.

74 oglobulin levels, skin symptom scores, or on transepidermal water loss.

75 id envelope and die shortly after birth from transepidermal water loss.

76 icle-treated animals), assessed as increased transepidermal water loss.

77 tures improve barrier repair, as assessed by transepidermal water loss.

78 ates erythema (IASI-E) and scaling (IASI-S); transepidermal water loss; and pruritus.