ライフサイエンスコーパス: transepidermal water loss

1 primary cell population responsible for high transepidermal water loss.

2 oglobulin levels, skin symptom scores, or on transepidermal water loss.

3 id envelope and die shortly after birth from transepidermal water loss.

4 icle-treated animals), assessed as increased transepidermal water loss.

5 tures improve barrier repair, as assessed by transepidermal water loss.

6 Our study showed that transepidermal water loss, a parameter of skin barrier i

7 recovery of barrier function as measured by transepidermal water loss after tape stripping.

8 the delayed barrier recovery as measured by transepidermal water loss after tape-stripping.

9 unction, as measured by corneometry, pH, and transepidermal water loss also normalized with treatment

10 Transepidermal water loss and a(*) (skin redness) decrea

11 rier development, evidenced by a decrease in transepidermal water loss and an enhanced outside-in bar

12 We assessed here, first, baseline transepidermal water loss and barrier recovery kinetics

13 These deposits increased transepidermal water loss and caused irritation, particu

14 This was associated with increased transepidermal water loss and development of eczematous

15 of the epidermis associated with a decreased transepidermal water loss and increased proinflammatory

16 Bacterial diversity correlated with transepidermal water loss and pH level but not with corn

17 to barrier disruption, indicated by elevated transepidermal water loss and reduced lipid synthesis en

18 was to investigate the relationship between transepidermal water loss and skin permeability to triti

19 certained using confocal Raman spectroscopy; transepidermal water loss and skin surface pH were measu

20 more prematurely have elevated rates of both transepidermal water loss and transcutaneous heat loss,

21 on of the barrier function of skin increases transepidermal water loss and up-regulates inflammatory

22 rations, skin barrier dysfunction (increased transepidermal water loss), and AD in infancy.

23 l epidermis, corneocyte fragility, increased transepidermal water loss, and local inflammation in the

24 measurements of percentage of skin redness, transepidermal water loss, and participant-assessed pain

25 permeability indicated by higher pH, greater transepidermal water loss, and reduced lipid synthesis e

26 Changes in total sign scores (TSSs), transepidermal water loss, and tissue biomarkers (determ

27 ates erythema (IASI-E) and scaling (IASI-S); transepidermal water loss; and pruritus.

28 d to interpret the significance of measuring transepidermal water loss by evaporimetry.

29 These data indicate that transepidermal water loss cannot be unconditionally ascr

30 ximately 0.5 U); (ii) enhanced SC integrity (transepidermal water loss change with sequential tape st

31 ing IL-13Ralpha2 had significantly increased transepidermal water loss, cutaneous inflammation, perip

32 mal permeability barrier defects with severe transepidermal water loss, decreased intercellular lipid

33 We then assessed skin pH and transepidermal water loss every 12 hours on the burn wou

34 lents in SCID/NOD mice demonstrated enhanced transepidermal water loss following s.c. administration

35 fetal rats displayed no measurable barrier (transepidermal water loss > 10 mg per cm2 per h), a meas

36 Previous measurements of transepidermal water loss have suggested that, regardles

37 m, with SerpinB2(-/-) mice showing increased transepidermal water loss, increased overt loss of strat

38 hat cutaneous barrier formation, measured as transepidermal water loss, is delayed in male fetal rats

39 active plaque phenotypes displayed elevated transepidermal water loss levels, increased numbers of e

40 se curve, with maximal sweating (measured as transepidermal water loss) (mean 70 g m(-2) hour(-1)) af

41 quency impedance spectroscopy, to complement transepidermal water loss measurements.

42 sebopsoriasis displayed a lesser increase in transepidermal water loss, normal numbers of lamellar bo

43 asol, respectively, with parallel changes in transepidermal water loss (P < .05).

44 present in these dogs, as well as increased transepidermal water loss, particularly in sites charact

45 tic administration of all activators tested (transepidermal water loss range 4.0-8.5 mg per cm2 per h

46 There was no correlation between transepidermal water loss rate and 3H2O permeability fol

47 Similarly, there was no correlation between transepidermal water loss rates and the 3H2O permeabilit

48 No correlation was found between basal transepidermal water loss rates and the permeability of

49 While basal transepidermal water loss rates are near normal, barrier

50 Transepidermal water loss rates declined during explant

51 Whereas baseline transepidermal water loss rates were elevated by approxi

52 exercise, on several dermatologic measures: transepidermal water loss, recovery of skin barrier func

53 e markers also significantly correlated with transepidermal water loss, suggesting a link between the

54 ] and urocanic acid [UCA]) using UPLC-MS/MS, transepidermal water loss (TEWL) and epidermal pH.

55 ipid barrier as measured by skin resistance, transepidermal water loss (TEWL) and Fourier transform i

56 The effects were quantified using transepidermal water loss (TEWL) and were correlated wit

57 ering techniques of laser Doppler imaging, a transepidermal water loss (TEWL) device and a skin therm

58 tional Endpoints (BASELINE) birth cohort had transepidermal water loss (TEWL) measured in the early n

59 Transepidermal water loss (TEWL) measures were collected

60 rthermore, to determine whether increases in transepidermal water loss (TEWL) predate the development

61 of the skin and measurements of the rate of transepidermal water loss (TEWL) were recorded sequentia

62 RCM, transepidermal water loss (TEWL), and fluorescence excit

63 We measured water flux across the SC, transepidermal water loss (TEWL), in six women, in vivo.

64 rneum was assessed by measuring capacitance, transepidermal water loss (TEWL), rates of absorption-de

65 sure in wild-type and Serpinb3a-null mice on transepidermal water loss (TEWL), sensitization, and inf

66 e associated with atopic dermatitis (AD) and transepidermal water loss (TEWL).

67 n barrier function was assessed by measuring transepidermal water loss (TEWL).

68 versely correlated (r=-0.654, P=0.0004) with transepidermal water loss (TEWL).

69 ion examining the permeability barrier using transepidermal water loss (TEWL).

70 by reduced transglutaminase (TGM) activity, transepidermal water loss, up-regulation of the proinfla

71 orneocytes and its contribution to increased transepidermal water loss was confirmed by tape strippin

72 Transepidermal water loss was measured on unaffected for

73 Transepidermal water loss was normal in all conditions.

74 In an acute AD model, skin transepidermal water loss was significantly attenuated i

75 nce analysis to measure hydration status and transepidermal water loss, we show that the epidermal ba

76 ith severe dehydration, suggesting excessive transepidermal water loss, which was confirmed by in vit

77 y normal, and exhibited an increase in basal transepidermal water loss without alteration in basal mi