ライフサイエンスコーパス: transferase

1 systems operating in vivo (xanthophyll acyl transferase).

2 CD2, CD3, CD4, and terminal deoxynucleotidyl transferase.

3 amorphous aggregation of human glutathione S-transferase.

4 re proficient than terminal deoxynucleotidyl transferase.

5 nsferase and an increase in serine palmitoyl transferase.

6 tion of methyl halides using a methyl halide transferase.

7 examine the substrate specificity of mouse R-transferase.

8 onol glycosides and some associated glycosyl transferases.

9 ivity against a panel of pure histone methyl transferases.

10 thus relies on exonucleases and nucleotidyl transferases.

11 ' headgroup positions by phosphoethanolamine transferases.

12 udies demonstrated that serine hydroxymethyl transferase 1 (SHMT1) was necessary for ovarian cancer t

13 Because the carnitine palmitoyl transferase 1a (CPT1a) is a protein that catalyzes the r

14 n of the phase II-metabolizing enzyme sulfur transferase 1A1 does.

15 e we demonstrate that the polypeptide GalNAc-transferase 2 (GalNAc-T2) specifically O-glycosylates be

16 Nicotinamide mononucleotide adenylyl transferase 2 (NMNAT2) is a key neuronal maintenance fac

17 Nicotinamide mononucleotide adenylyl transferase 2 (NMNAT2) is neuroprotective in numerous pr

18 ns in the gene coding for protein O-mannosyl-transferase 2 (POMT2) are known to cause severe congenit

19 s (lactoperoxidase, microsomal glutathione S-transferase 2 and 3, glutathione S-transferase peroxidas

20 ated fat liquid diet to female glutathione-S-transferase 4-4 (Gsta4(-/-))/peroxisome proliferator act

21 nsferases, including human adenosine ribosyl transferase 5 (ART5) and Cholera toxin subunit A (CTA),

22 us 27 +/- 10 IU/L (P = 0.81), gamma-glutamyl transferase 54 +/- 138 versus 49 +/- 35 IU/L (P = 0.72),

23 The patient's gamma-glutamyl transferase (77 U/L [1.28 mukat/L]; normal level, <55 U/

24 s mediated by the Ate1 arginyltransferase (R-transferase), a component of the Arg/N-end rule pathway.

25 the p-coumaroyl-CoA:agmatine N4-p-coumaroyl transferase ACT for the biosynthesis and of the MATE tra

26 in a UDP-GlcNAc-dependent fashion within the transferase active site of O-GlcNAc-transferase (OGT).

27 followed by its transmission to the glycosyl transferase active site.

28 The Kdo transferase activities of these proteins were determined

29 ooth muscle cells (VSMC), has histone methyl transferase activities, and acts as a transcriptional su

30 eins associated with binding, oxidoreductase/transferase activities, cytoskeletal and lipid/carbohydr

31 on promoters in vitro Inhibition of O-GlcNAc-transferase activity and O-GlcNAcylation prevents pol II

32 Evidence for AlmG glycyl to lipid substrate transferase activity is demonstrated in vivo by heterolo

33 These data indicate that O-GlcNAc-transferase activity is essential for RNA pol II promote

34 We further find that Poltheta terminal transferase activity is most efficient on DNA containing

35 olymerases and is known for its deoxycytidyl transferase activity that incorporates dCMP into DNA and

36 e phenotype with normal serum gamma-glutamyl transferase activity without intestinal disease.

37 ulopathy, low-to-normal serum gamma-glutamyl transferase activity, elevated serum alpha-fetoprotein a

38 occur independently of its poly(ADP-ribosyl) transferase activity.

39 spects of UGT functions diverging from their transferase activity.

40 e phenotype with normal serum gamma-glutamyl transferase activity.

41 ng between three different modes of terminal transferase activity: non-templated extension, templated

42 syl-l-methionine pocket of catechol O-methyl transferase allowed the identification of structurally r

43 Glutathione S-transferase alpha 4 (GSTA4) is a phase II detoxifying en

44 ly member 6 (HDAC6) and alpha-tubulin acetyl transferase (alpha-TAT1).

45 ructose-1,6-bisphosphatase and glutathione-S-transferase-alpha Additionally, we quantified urinary ex

46 ructose-1,6-bisphosphatase and glutathione-S-transferase-alpha release exclusively within 7 days afte

47 the liver (increased placental glutathione S-transferase and cytokeratin 8-18 activity; starting at 1

48 um levels of the liver enzyme gamma-glutamyl transferase and fecal virus shedding were significantly

49 In contrast to aspartate-amino-transferase and M65, M30 levels increased significantly

50 can affect the rate of Nt-arginylation by R-transferase and thereby the rate of degradation of a sub

51 Analysis of TSG-6 mutants (with impaired transferase and/or hyaluronan-binding functions) reveale

52 se domain with structural homology to GalNAc transferases and a predicted capsule synthesis domain wi

53 the mechanism of action of many other prenyl transferases and may also be of use in engineering new m

54 s of years, driven by CG-specific DNA methyl transferases and spontaneous methyl-cytosine deamination

55 ivities of two enzymes, MCAT (malonylCoA:ACP transferase) and PDH (pyruvate dehydrogenase).

56 ses, carboxyl/cholinesterases, glutathione-S-transferases, and ATP-binding cassette transporters.

57 result in inhibition of mTOR-mediated acetyl-transferase ARD1 S228 phosphorylation, leading to ARD1-d

58 ical") residues that are Nt-arginylated by R-transferase are N-terminal Asp, Glu, and (oxidized) Cys.

59 ed GCN5 bromodomain-containing lysine acetyl transferases are members of subfamily I of the bromodoma

60 s, and the corresponding inverting alpha-Kdo transferases are well characterized.

61 cosyltransferase 4-amino-4-deoxy-L-arabinose transferase (ArnT).

62 ve TCA cycle, in which acetate:succinate CoA-transferase (ASCT) replaces the enzymatic step typically

63 chrome P450 monooxygenases and glutathione S-transferases associated with detoxification are found.

64 lyketide synthases that lack integrated acyl transferase (AT) domains.

65 dergoes efficient arginylation by an arginyl transferase (ATE1).

66 and GGTA1/ CMAH /b1,4 N-acetylgalactosaminyl transferase (B4GalNT2) KO pigs were screened for human a

67 ession of mitochondrial branched chain amino transferase (BCAT) which produces KIC in skeletal muscle

68 SE TO ABA1 (ERA1), that encodes the farnesyl transferase beta-subunit.

69 a tight docking of the conserved nucleotidyl transferase bi-domain module with a RET1-specific C2H2 z

70 Loss of O-GlcNAc transferase blocked T cell progenitor renewal, malignant

71 antially through blocking the gamma-glutamyl transferase catalysis of the first breakdown step of mod

72 We present terminal deoxynucleotidyl transferase-catalyzed enzymatic polymerization (TcEP) fo

73 sion of a third toxin, known as C. difficile transferase (CDT), is increasingly common.

74 ADP ribosylating toxin Clostridium difficile transferase (CDT).

75 nd of the A-site tRNA away from the peptidyl transferase center (PTC) is functionally significant.

76 ynthesis inhibitors that target the peptidyl transferase center (PTC) on the large subunit of the rib

77 isruption of the A-site side of the peptidyl-transferase center (PTC).

78 yzing peptide bond formation at the peptidyl transferase center (PTC).

79 protein synthesis by targeting the peptidyl transferase center of the bacterial ribosome.

80 ng the neighborhood surrounding the peptidyl transferase center, and stable association of ribosomal

81 ed near the decoding center and the peptidyl transferase center, respectively.

82 y between the nascent chain and the peptidyl transferase center.

83 ction of the distance away from the peptidyl transferase center.

84 dation of decoding factors into the peptidyl transferase center.

85 ks the catalytic GGQ motif into the peptidyl-transferase center.

86 se, glutathione peroxidase and glutathione S transferase compared with the control, and decreased the

87 e are potent inhibitors of catechol-O-methyl transferase (COMT) for the treatment of Parkinson's dise

88 copy number elevations of catechol-O-methyl-transferase (COMT) or Tbx1, two genes encoded in the two

89 hat enzymatic catalysis by catechol-O-methyl-transferase (COMT) predominates over DAT re-uptake.

90 of the DA catalytic enzyme catechol-O-methyl-transferase (COMT), but negligible expression of the dop

91 Nicotinamide adenylyl transferase condenses nicotinamide mononucleotide and (t

92 TE4 (which encodes a gamma-tocopherol methyl transferase converting gamma-tocopherol into alpha-tocop

93 The histone acetyl transferases CREB-binding protein (CBP) and its paralog

94 the precisely timed expression of the S-acyl-transferase DHHC10.

95 peroxidase system, HOAS/dihydrolipoyl acetyl transferase (DlaT)/alkylhydroperoxide reductase colorles

96 erized a Drosophila melanogaster glutathione transferase (DmGSTE6) which has activity towards TNT.

97 ddition, HFD increased binding of DNA methyl-transferases (DNMTs) 3a and 3b and methyl-CpG-binding do

98 ion, mutation of the DXD motif in the GalNAc transferase domain and of the HP motif in the Kdo transf

99 ferase domain and of the HP motif in the Kdo transferase domain resulted in a loss of encapsulation.

100 strate-triggered movement of phosphatase and transferase domains creating a solvent inaccessible cavi

101 Terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) and phospho-h

102 re associated with terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL)-positive soma

103 nd stereology, and terminal deoxynucleotidyl transferase dUTP nick end labeling assay was used to ass

104 and apoptosis via terminal deoxynucleotidyl transferase dUTP nick end labeling staining and caspase-

105 ctivity and TUNEL [terminal deoxynucleotidyl transferase dUTP nick end labeling])-positive cells) of

106 IRI as measured by terminal deoxynucleotidyl transferase dUTP nick-end labeling (TUNEL) staining, cir

107 th, as measured by terminal deoxynucleotidyl transferase dUTP nick-end labeling, was significantly de

108 inol adenosine synthase and other cis-prenyl transferases (e.g. cis-farnesyl, decaprenyl, undecapreny

109 quitin-ligase (DZIP3) and the histone methyl transferase (EHMT1).

110 COQ2 (p-hydroxybenzoate polyprenyl transferase) encodes the enzyme required for the second

111 MCR-1 is a member of the phosphoethanolamine transferase enzyme family, with expression in E coli res

112 because of the diversity of numerous GalNAc transferase enzymes that initiate O-linked carbohydrate

113 Small molecule inhibitors of prenyl transferases, enzymes that catalyze the post-translation

114 A 1-phosphatase (LpxE) and a lipid A 1 P-Etn transferase (EptA).

115 57630 was named Extensin Arabinose Deficient transferase, ExAD, accordingly.

116 ation, oxidative stress, serum alanine amino transferase, expression of tumor necrosis factor alpha,

117 Histone methyl transferase EZH2 (Enhancer of Zeste Homolog 2) is genera

118 The histone methyl transferase EZH2 is frequently altered in many cancers,

119 In Arabidopsis thaliana, a glycosyl transferase family 37 (GT37) fucosyltransferase 1 (AtFUT

120 a pharmacological approach for the ppGalNAc-transferase family and suggest that targeting specific p

121 nding site architecture of the glutathione S-transferase family.

122 ited by members of the membrane-bound O-acyl transferase family.

123 eptidoglycan precursor used by the aminoacyl-transferase FemXWv for synthesis of the bacterial cell w

124 -O-Me globally by inhibiting the rRNA methyl-transferase fibrillarin in human cells.

125 The protein adopts the zeta or cis-prenyl transferase fold but remarkably, unlike tuberculosinol a

126 c rhythmicity by acting as a rhythmic acetyl-transferase for metabolic enzymes.

127 of a full-length lipid A phosphoethanolamine transferase from Neisseria meningitidis, determined to 2

128 onal modification of ribosomal peptides, and transferases from various biosynthesis pathways.

129 Ac-P-P-Und) produced by the GlcNAc-phosphate transferase GacO and GlcNAc-phosphate-undecaprenol (GlcN

130 be disease-relevant targets in epimerase and transferase galactosemias, and identify UGP as promising

131 The polypeptide GalNAc-transferases (GalNAc-Ts), that initiate mucin-type O-gly

132 The histone acetyl transferase GCN5 and the histone deacetylase HDA19 are r

133 s, chromatin occupancy of the histone acetyl-transferase Gcn5 is controlled by the GG-NER complex, wh

134 hogen-inducible promoter, from glutathione S-transferase gene from potato.

135 Induction of hepatic glutathione transferase genes and elevated total glutathione level w

136 ns are modified by variants in Glutathione S-Transferase genes.

137 Prenatal air pollution exposures, DNA methyl transferase genotypes, and associations with newborn LIN

138 aenoic acid) was catalyzed by gamma-glutamyl transferase (GGT) in human macrophages.

139 Elevated gamma-glutamyl transferase (GGT) levels are associated with higher risk

140 iver enzyme levels, including gamma-glutamyl transferase (GGT), alanine aminotransferase (ALT), and a

141 of the UAG-activating enzyme ghrelin O-acyl transferase (GOAT), which is located in the membranes of

142 ncept, a reversible label-free glutathione-S-transferase (GST) biosensor is demonstrated.

143 ve cooperativity occurs in human glutathione transferase (GST) GSTP1-1 when it binds and neutralizes

144 ies availability of functional glutathione S-transferase (GST) metabolic activity, the key activation

145 000 ng of purified recombinant glutathione-S-transferase (GST) proteins and could particularly detect

146 Coimmunoprecipitation and glutathione S-transferase (GST) pulldown assays revealed that GBP1 int

147 cide and fungus, activities of glutathione-S-transferase (GST), general esterases (ESTs) and phenol o

148 uitin-related modifier (Sumo), glutathione S-transferase (GST), maltose-binding protein (MBP), N-util

149 n, and rhythmic translation of glutathione S-transferase (GST-3) from constitutive mRNA levels in viv

150 Glutathione S-transferases (GST) were evaluated as biomarkers of AKI.

151 Cs resemble the omega class of Glutathione S-transferases (GST), yet differ from them in their abilit

152 Glutathione S-transferases (GSTs) comprise a diverse family of phase I

153 sed expression and activity of glutathione S-transferases (GSTs).

154 uence similarities of GDAP1 to glutathione S-transferases (GSTs).

155 ferase (AAEL014279-RA) and the glutathione-S transferases GSTS1 and GSTT3.

156 monstrate a role for the yeast glutathione S-transferase Gtt1p in glutathionylation.

157 The genes encoding the histone acetyl-transferases (HATs) CREB binding protein (CREBBP) and EP

158 show that the four major isoforms of mouse R-transferase have similar Nt-arginylation specificities i

159 Two hydroxycinnamoyl-CoA transferases (HCT/HQT) have been involved in CGA product

160 ), which was associated with aspartate amino transferase, hemoglobin and ferritin levels (two studies

161 coexpression of homogentisate geranylgeranyl transferase (HGGT), stacked with carotenoid biosynthesis

162 that is catalyzed by protein geranylgeranyl transferase I (GGTase I).

163 oincides with a shift of carnitine palmitoyl transferase I from muscle to increased liver isoforms.

164 Acutely increased carnitine palmitoyl transferase I in normal rodent hearts has been shown to

165 phate synthase inhibitors or geranylgeraniol transferase I inhibitors, we evaluated combinations of p

166 wed increased expression of kynurenine amino transferase II (KATII) and kynurenine-3-monooxygenase (K

167 AD(+) also serves as a substrate for ribosyl transferases, including human adenosine ribosyl transfer

168 th the vemurafenib group were gamma-glutamyl transferase increase (36 [15%] in the cobimetinib and ve

169 ophosphatase, and mimicked by geranylgeranyl transferase inhibition.

170 a single-arm, open-label trial of a farnesyl transferase inhibitor for patients with HRAS mutations.

171 ocalization with GGTi-2418, a geranylgeranyl transferase inhibitor, sensitizes xenotransplanted tumou

172 ring this mutation are resistant to peptidyl-transferase inhibitors (e.g., anisomycin).

173 the fundamental mechanisms by which peptidyl transferase inhibitors modulate the catalytic activity o

174 O-GlcNAc transferase is responsible for O-GlcNAc addition to seri

175 en proposed that FKRP, a ribitol-5-phosphate transferase, is a participant in alpha-dystroglycan (alp

176 cally possible that the repertoire of GalNAc transferase isoforms in natural target cells for HIV and

177 polypeptide, isoatp4056 and kynurenine amino transferase (kat), a gene involved in the production of

178 ltheta as among the most proficient terminal transferases known.

179 ) T-cell count, HCV genotype, gamma-glutamyl transferase level, and baseline APRI.

180 atidylglycerol:prolipoprotein diacylglyceryl transferase (Lgt) recognizes a conserved lipobox motif w

181 show the presence of numerous choline acetyl transferase-like immunoreactive en plaque motor endplate

182 Apolipoprotein N-acyl transferase (Lnt) catalyses the third step in this pathw

183 younger age, higher levels of gamma-glutamyl transferase, lower pretherapeutic hemoglobin, a higher G

184 sed to large proteins, such as glutathione S-transferase, maltose-binding protein, or thioredoxin, or

185 oxylase (MdcD-MdcE) and acyl-carrier protein transferase (MdcA) catalytic activities.

186 jury, the number of terminal deoxynucleotide transferase-mediated deoxyuridine triphosphate nick end-

187 ved caspase-3, and terminal deoxynucleotidyl transferase-mediated deoxyuridine triphosphate nick-end

188 analysis, including immunohistochemistry and Transferase-mediated deoxyuridine triphosphate-biotin ni

189 by flow cytometry, terminal deoxynucleotidyl transferase-mediated dUTP nick end labeling (TUNEL) stai

190 DNase levels, and terminal deoxynucleotidyl transferase-mediated dUTP nick end-labeling staining.

191 NF-kappaB-p65, and terminal deoxynucleotidyl transferase-mediated dUTP nick-end labeling assay was pe

192 -67, and increased terminal deoxynucleotidyl transferase-mediated dUTP nick-end labeling staining.

193 tokine expression, terminal deoxynucleotidyl transferase-mediated dUTP nick-end labeling, and Ki-67 i

194 s confirmed by the terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling assay

195 ), N-glycans by targeting the beta1,2 GlcNAc-transferase (MGAT1) and GSLs by deleting UDP-glucose cer

196 rates that are not substrates for DNA methyl transferases mimic the CG suppression of their hosts.

197 Glutathione S-transferase mu 1 (GSTM1) encodes an enzyme that catalyze

198 OL1) high-risk alleles and the glutathione-S-transferase-mu1 (GSTM1) null allele have been shown sepa

199 by introducing a dominant negative O-GlcNAc transferase mutant (F460A) restored Ogg1 enzymatic activ

200 changes, but had lower nicotinamide N-methyl transferase (NNMT) levels and were predisposed to APAP-i

201 mine (O-GlcNAc) processing enzymes, O-GlcNAc-transferase (OGT) and O-GlcNAcase (OGA), interact with t

202 The mechanisms by which the O-GlcNAc transferase (OGT) and the O-GlcNAcase (OGA), the enzymes

203 pathway (YAP) is O-GlcNAcylated by O-GlcNAc transferase (OGT) at serine 109.

204 O-Linked N-acetylglucosamine transferase (OGT) catalyzes O-GlcNAcylation of target pr

205 etylglucosamine (UDP-GlcNAc),O-linked-GlcNAc transferase (OGT) catalyzes Ser/ThrO-GlcNAcylation of ma

206 O-GlcNAc transferase (OGT) catalyzes the posttranslational modifi

207 STATEMENT We show the importance of O-GlcNAc transferase (OGT) for sensory neuron health and survival

208 Deletion of the O-GlcNAc transferase (ogt) gene responsible for the modification

209 ly, many transcriptional effects of O-GlcNAc transferase (OGT) inhibition were due to the activation

210 O-Linked beta-N-acetylglucosamine transferase (OGT) is an essential human enzyme that glyc

211 O-GlcNAc transferase (OGT) is elevated in multiple cancers and in

212 O-linked N-acetylglucosamine transferase (OGT) is found in all metazoans and plays an

213 O-linked beta-N-acetylglucosamine (O-GlcNAc) transferase (OGT) is required for glucagon-stimulated li

214 anges in the relative expression of O-GlcNAc transferase (OGT) isoforms and accumulation of OGT at th

215 umor cells with O-linked N-acetylglucosamine transferase (OGT) knockdown grew significantly slower th

216 The nutrient sensing protein O-GlcNAc transferase (OGT) mediates post-translational O-linked N

217 O-linked-beta-N-acetylglucosamine (O-GlcNAc) transferase (OGT) modifies intracellular proteins with N

218 of the O-GlcNAc-regulating enzymes O-GlcNAc transferase (OGT) or O-GlcNAcase (OGA) impairs mitochond

219 O-GlcNAc transferase (OGT) regulates a wide range of cellular pro

220 N-Acetylglucosamine (O-GlcNAc) transferase (OGT) regulates protein O-GlcNAcylation, an

221 the O-linked N-acetylglucosamine (O-GlcNAc) transferase (OGT) SECRET AGENT (SEC) in Arabidopsis.

222 us to predict hundreds of putative O-GlcNAc transferase (OGT) substrates.

223 GlcNAc can be added to proteins by O-GlcNAc transferase (OGT) to regulate protein activity.

224 encode enzymes for its attachment (O-GlcNAc transferase (OGT)) and removal (O-GlcNAcase (OGA)).

225 w that O-linked N-acetylglucosamine (GlcNAc) transferase (OGT), an enzyme that catalyzes O-GlcNAcylat

226 We have previously discovered that O-GlcNAc transferase (OGT), an enzyme that modifies protein funct

227 scovered the presence of functional O-GlcNAc transferase (OGT), O-GlcNAcase (OGA), and nucleocytoplas

228 O-GlcNAc transferase (OGT), the solo enzyme for O-GlcNAcylation,

229 the enzyme O-linked beta-N-acetylglucosamine transferase (OGT), whereas the enzyme O-GlcNAcase (OGA)

230 thin the transferase active site of O-GlcNAc-transferase (OGT).

231 e and threonine O-linked N-acetylglucosamine transferase (OGT).

232 O-GlcNAc transferase (OGT/SXC) is essential for Pc repression sug

233 Glutathione S-transferase omega 1 (GSTO1) is an atypical GST isoform t

234 Glutathione transferase Omega 1 (GSTO1-1) is an atypical GST reporte

235 tive stress, such as the Spt-Ada-Gcn5-acetyl transferase or Elongator complexes, respectively.

236 e no increase in nicotinamide phosphoribosyl transferase or in the NR transport protein, CD73.

237 the polytopic membrane-bound oligosaccharyl transferases (OTases) with partner substrates.

238 levated levels of antioxidants glutathione S-transferase P (GSTP) and superoxide dismutases (SOD).

239 ated by using tumor cells that glutathione S-transferase P1 (GSTP1) sequesters NO as dinitrosyl-dithi

240 , as well as the model protein glutathione S-transferase P1, in vitro.

241 ry receptors through CamK and histone acetyl transferase p300/CBP to maintain ORN-specific fru expres

242 etabolites and upregulation of glutathione S-transferase pathway genes, including Gstp1 and Gstz1, wh

243 ys-Cys-rich domain-containing Protein S-Acyl Transferases (PATs) are multipass transmembrane proteins

244 athione S-transferase 2 and 3, glutathione S-transferase peroxidase kappa 1, and glutathione peroxida

245 Phosphoglycosyl transferases (PGTs) are integral membrane proteins with

246 Glutathione S-transferase pi 1 (GSTP1) is frequently overexpressed in

247 identified a new family of DDs in trans-acyl transferase PKSs, exemplified by a matched pair from the

248 talysis of p-coumaroyl-coenzyme A monolignol transferase (PMT).

249 ds requires their modification by the O-acyl transferase Porcupine (PORCN).

250 , such as cytochrome P-450 and glutathione S-transferases, potentially involved in the PCB metabolism

251 ns (beta-trefoil fold) of polypeptide GalNAc-transferases (ppGalNAc-Ts) on catalytic activity of glyc

252 Sfp-type phosphopantetheinyl transferases (PPTases) are essential to the production o

253 e kidney-specific podocin and gamma-glutamyl transferase promoters, but found expression primarily in

254 etic enzymes revealed that the glutathione S-transferase PsoE requires participation of the bifunctio

255 Glutathione S-transferase pull-down and coimmunoprecipitation studies

256 nce energy transfer (FRET) and glutathione S-transferase pulldown analyses identified Akt1 pleckstrin

257 2 interaction was confirmed by glutathione S-transferase pulldown, coimmunoprecipitation, and laser c

258 an enzyme named quinolinate phoshphoribosyl transferase (QPRTase).

259 hesis domain with structural homology to Kdo transferases, raising the possibility that this enzyme i

260 ch ribosomal RNA is responsible for peptidyl-transferase reaction catalysis.

261 rases catalyze a metal-dependent nucleotidyl transferase reaction during extension of a DNA strand us

262 D using unprecedented carboxylase and sulfur transferase reactions to form the organic component of t

263 MUM5 was identified as a putative xylosyl transferase recently characterized as MUCI21.

264 e catalytic Lys of FemXWv but not to related transferases recognizing different aminoacyl-tRNAs.

265 In addition, C3 transferase reduced brain injury and increased endotheli

266 ved arginines from nucleotide hydrolases and transferases revealed a consensus amino acid location an

267 h is mediated by GCN5 and the histone methyl transferase SDG8.

268 l. found that mutation of the histone methyl transferase SEDT2 affects alternative splicing fates of

269 a, we pursued inhibitors of serine palmitoyl transferase (SPT).

270 inhibition of farnesyl-diphosphate farnesyl transferase (squalene synthase), but not 3-hydroxy-3-met

271 An Arabidopsis mutant lacking the glucosyl-transferase, STARCH SYNTHASE 4 (SS4) is impaired in its

272 EZH2, a histone methyl transferase subunit of a Polycomb repressor complex, is

273 PARP3 is a member of the ADP-ribosyl transferase superfamily that we show accelerates the rep

274 tial role for H3K9me3 and its histone methyl transferase (SUV39H1) in mediating hippocampal memory fu

275 tionally-cloned Tst gene (Thiosulfate Sulfur Transferase, synonym Rhodanese) responsible for the Fob3

276 e (biotin-dATP) by terminal deoxynucleotidyl transferase (TdT).

277 TMEM5 is a UDP-xylosyl transferase that elaborates the structure.

278 It is an electron transferase that is able to deliver electrons to a varie

279 metabolic/nutrient sensing protein O-GlcNAc transferase that mediates the O-linked addition of N-ace

280 the gene encoding succinyl-CoA:3-oxoacid-CoA transferase, the rate-limiting enzyme for myocardial oxi

281 nce for a significant activity of the Ate1 R-transferase toward previously invoked non-canonical N-te

282 xins A (TcdA) and B (TcdB), and C. difficile transferase toxin (CDT).

283 essing the p-COUMAROYL-Coenzyme A MONOLIGNOL TRANSFERASE transgene can therefore produce monolignol p

284 ridylated by activities of terminal uridylyl transferases (TUTases) in miRNA-induced silencing comple

285 alanine aminotransferase and gamma-glutamyl transferase, two markers of fatty liver disease.

286 ated gene UDP glucose:flavonoid 3-O-glucosyl transferase (UFGT), which was dependent of the transgene

287 Sm2 encodes a rhamnosyl transferase (UGT91L1) that uses isoorientin and UDP-rham

288 is of a construct containing human A1- and H-transferases under control of the ICAM-2 promoter was pe

289 fraction-derived resveratrol 4-dimethylallyl transferase utilizes 3,3-dimethylallyl pyrophosphate as

290 Selective RhoA inhibitor C3 transferase was administered to clarify the involvement

291 ee P450s in combination with a single acetyl transferase was identified that catalyzes the conversion

292 ified DNA, whereas terminal deoxynucleotidyl transferase was used for a single-nucleotide labeling of

293 and asymptomatic increases in gamma-glutamyl transferase were observed in some patients receiving tel

294 roteome analysis revealed that Glutathione S-transferases were induced in the shoot and suggested its

295 sulfur-utilizing proteins, including sulfur transferases, were enriched at sulfidic depths.

296 otides inserted by terminal deoxynucleotidyl transferase, which resulted in a decrease of 2 to 3 amin

297 ere, we have shown that the histone dimethyl transferase WHSC1 critically drives indolent PTEN-null t

298 and suggest that targeting specific ppGalNAc-transferases will yield new therapeutics.

299 tion, primarily mediated by the protein-acyl transferase ZDHHC13, is essential for activating MC1R si

300 is mediated by the Golgi-resident palmitoyl transferases zDHHC9/14/18 and is followed by depalmitoyl