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1                     In these cells, although transforming growth factor alpha (TGF-alpha) and epiderm
2                           ADM in response to transforming growth factor alpha was also suppressed in
3 fibroblast growth factor 2, IL-7, IL-15, and transforming growth factor alpha) but lower levels of th
4 okines were increased, whereas the levels of transforming growth factor-alpha (TGF-alpha) and platele
5 ung disease induced by the overexpression of transforming growth factor-alpha (TGF-alpha), establishe
6 deleted fibroblasts had higher expression of transforming growth factor-alpha (Tgfa) mRNA and secrete
7 E-536) is a novel fusion protein that blocks transforming growth factor beta (TGF beta) superfamily i
8 induction of SP-A expression is repressed by transforming growth factor beta (TGF-beta) and by hypoxi
9 reatment there was a significant increase in transforming growth factor beta (TGF-beta) and concomita
10                                Tumor-derived transforming growth factor beta (Tgf-beta) decreased Sat
11                    EMT is induced by soluble transforming growth factor beta (TGF-beta) family member
12      To understand the cell-specific role of transforming growth factor beta (TGF-beta) in the myeloi
13                                              Transforming growth factor beta (TGF-beta) is an establi
14                                              Transforming growth factor beta (TGF-beta) isoforms are
15                                              Transforming growth factor beta (TGF-beta) pathways are
16 l as thymic stromal lymphopoietin (TSLP) and transforming growth factor beta (TGF-beta) plasma levels
17                    We show that the type III transforming growth factor beta (TGF-beta) receptor (Tbe
18 ave previously determined that type I and II transforming growth factor beta (TGF-beta) receptors (Tb
19  in genes encoding various components of the transforming growth factor beta (TGF-beta) signaling cas
20 , which in turn activates the STAT3-mediated transforming growth factor beta (TGF-beta) signaling pat
21 plays a central role in the amplification of transforming growth factor beta (TGF-beta) signaling res
22 d pathways: NF-kappaB (p65) transactivation, transforming growth factor beta (TGF-beta) signaling, an
23  extracellular matrix, or decrease canonical transforming growth factor beta (TGF-beta) signaling.
24 xpression, a microRNA cluster that regulates transforming growth factor beta (TGF-beta) signaling.
25 been linked to NR4A1-dependent regulation of transforming growth factor beta (TGF-beta) signaling.
26  Kruppel-like factor 15 (Klf15), by both the transforming growth factor beta (TGF-beta) transcription
27 the prototypical prosclerotic growth factor, transforming growth factor beta (TGF-beta), is thought t
28 cules include collagen types I, III, and IV, transforming growth factor beta (TGF-beta), TGF-beta rec
29           Notably, expansion was mediated by transforming growth factor beta (TGF-beta)-containing ex
30  ShcA is an important mediator of ErbB2- and transforming growth factor beta (TGF-beta)-induced breas
31                                              Transforming growth factor beta (TGF-beta)-induced migra
32 er and serves as a common downstream node of transforming growth factor beta (TGFbeta) and bone morph
33 ber of key signaling pathways, including the transforming growth factor beta (TGFbeta) and epithelial
34                                              Transforming growth factor beta (TGFbeta) and fibroblast
35                               Members of the transforming growth factor beta (TGFbeta) cytokine famil
36 e that low-dosage/short-duration exposure to transforming growth factor beta (TGFbeta) induces partia
37                                              Transforming growth factor beta (TGFbeta) is important i
38                                              Transforming growth factor beta (TGFbeta) is instrumenta
39 mong tumour-associated inflammatory factors, transforming growth factor beta (TGFbeta) is regarded as
40                     Here we demonstrate that transforming growth factor beta (TGFbeta) is required fo
41 a family of signal transduction molecules in transforming growth factor beta (TGFbeta) ligand pathway
42                                          The transforming growth factor beta (TGFbeta) pathway plays
43 as TIEG1, plays essential roles in mediating transforming growth factor beta (TGFbeta) signaling and
44       Microarray studies identified impaired transforming growth factor beta (TGFbeta) signaling in c
45            In pausing-deficient embryos, the transforming growth factor beta (TGFbeta) signaling is e
46 dependent protein kinase (PDK1) and enhanced transforming growth factor beta (TGFbeta) signaling rath
47 d that these proteoglycans were dependent on transforming growth factor beta (TGFbeta) signaling.
48 2-HER3 signaling, or A83-01, an inhibitor of transforming growth factor beta (TGFbeta) signaling.
49                                              Transforming growth factor beta (TGFbeta) signalling is
50                                              Transforming growth factor beta (TGFbeta) signalling is
51  in (IL6) beta2SP(+/-) LSCs was activated by transforming growth factor beta (TGFbeta)-activated kina
52 SCs, both TANGO1 and the UPR were induced by transforming growth factor beta (TGFbeta).
53 hat significantly increased with exposure to transforming growth factor beta 1 (TGF-beta1), a potent
54  SMAD family member 2 (SMAD2), SMAD3, SMAD4, transforming growth factor beta 1 (TGFB1), TGFB2, TGFB3,
55  death-ligand 1, programmed cell death 1, or transforming growth factor beta 1 inhibitors.
56 e subclass expressed many genes regulated by transforming growth factor beta 1 that mediate immunosup
57    Plasmid transfection was used to modulate transforming growth factor beta 2 (TGF-beta2) gene expre
58   Connective tissue growth factor (CTGF) and transforming growth factor beta 3 (TGFbeta3) were then d
59 lated type 2 (IL-4 and IL-5) and regulatory (transforming growth factor beta [TGF-beta]) cytokines.
60 ding osteopontin, and LTBP4, encoding latent transforming growth factor beta [TGFbeta]-binding protei
61 yte-macrophage colony-stimulating factor and transforming growth factor beta alone, whereas CD14(+) c
62                                     However, transforming growth factor beta and bone morphogenetic p
63                   To investigate the role of transforming growth factor beta and bone morphogenetic p
64 D206 and arginase-1) and secretory products (transforming growth factor beta and interleukin-6) and d
65 in-1, p21-activated kinase, microRNA 21, and transforming growth factor beta are also being explored
66 as attenuated by thrombin-induced release of transforming growth factor beta by platelets.
67 sition on CTCs through platelet secretion of transforming growth factor beta in response to CTC activ
68 ted immune-stimulatory cytokines and reduced transforming growth factor beta in the serum.
69                               Treatment with transforming growth factor beta induced some cells to ad
70                                          The transforming growth factor beta isoforms, TGF-beta1, -be
71      Subsequent analysis suggested that anti-transforming growth factor beta neutralizing antibody, w
72 art by defective desmosomes and dysregulated transforming growth factor beta production and signaling
73  growth factor beta 1 (TGFB1), TGFB2, TGFB3, transforming growth factor beta receptor 1 (TGFBR1), and
74 bsence of CLCa was attributable to increased transforming growth factor beta receptor 2 (TGFbetaR2) s
75                                              Transforming growth factor beta receptor II interacting
76 lished a mechanism of negative regulation of transforming growth factor beta signaling mediated by th
77 iptional regulator SMAD6, which inhibits the transforming growth factor beta signaling pathway, is re
78 iptional regulator SMAD6, which inhibits the transforming growth factor beta signaling pathway, is re
79            Furthermore, we demonstrated that transforming growth factor beta stimulation resulted in
80 ced by a combination of ErbB2 activation and transforming growth factor beta stimulation, which is kn
81 (or GDF8) are closely related members of the transforming growth factor beta superfamily and are ofte
82  epiregulin (EREG), and other members of the transforming growth factor beta superfamily.
83 lowing isotropic chondrogenic induction with transforming growth factor beta to set up a dual-compart
84                                              Transforming growth factor beta treatment induced gene-b
85                                              Transforming growth factor beta type III receptor (Tbeta
86  cytokines (interleukin 6, 1beta, and 23 and transforming growth factor beta) restored CD4+ Th17 cell
87  local fibroblast activation by secretion of transforming growth factor beta, and a preneoplastic or
88 hogenetic protein, fibroblast growth factor, transforming growth factor beta, and Wnt signaling, and
89 h as proliferation-progenitor, proliferation-transforming growth factor beta, and Wnt-catenin beta1.
90 rences cannot be explained by a differential transforming growth factor beta, bone morphogenetic prot
91  that these HCV-infected hepatocytes express transforming growth factor beta, which activates stromal
92                                              Transforming growth factor beta-1 (TGF-beta1) induces FA
93                                              Transforming growth factor beta-1 (TGFbeta-1)-induced ph
94 V independently induce profibrogenic markers transforming growth factor beta-1 (TGFbeta1) (mediated b
95                                              Transforming growth factor beta-activated kinase 1 (TAK1
96 V-A6, and EV-D68 3C(pro) proteins all cleave transforming growth factor beta-activated kinase 1 (TAK1
97                                              Transforming growth factor beta-activated kinase 1 (TAK1
98 th death domain as an upstream regulator and transforming growth factor beta-activated kinase 1 as a
99                              LZTFL1 inhibits transforming growth factor beta-activated mitogen-activa
100 CSF-1)-dependent donor macrophages, induce a transforming growth factor beta-high environment locally
101  during glucose starvation of HeLa cells and transforming growth factor beta-induced epithelial-to-me
102 scle (Asm) area with decreased periostin and transforming growth factor beta-positive cells within As
103 an CFC tissue, but surprisingly, more active transforming growth factor beta.
104 e activity of nuclear factor-kappaB, but not transforming growth factor beta.
105 -1beta; 2) IL-6; 3) IL-17A; 4) IL-23; and 5) transforming growth factor- beta.
106 e found to be an important source of cardiac transforming growth factor-beta (TGF-beta) and PAI-1 reg
107  the various regulatory factors tested, only transforming growth factor-beta (TGF-beta) demonstrated
108 ave markers that denote tissue residency and transforming growth factor-beta (TGF-beta) imprinting.
109         Furthermore, reduction of nephrin by transforming growth factor-beta (TGF-beta) in podocytes
110                                          The transforming growth factor-beta (TGF-beta) network of li
111                                       In the transforming growth factor-beta (Tgf-beta) pathway, expo
112  screen in human FA fibroblasts, we identify transforming growth factor-beta (TGF-beta) pathway-media
113  of both XPC and DDB1 through activating the transforming growth factor-beta (TGF-beta) pathway.
114                                              Transforming growth factor-beta (TGF-beta) plays an impo
115 nd that human PDACs with impaired epithelial transforming growth factor-beta (TGF-beta) signaling hav
116                                  The role of transforming growth factor-beta (TGF-beta) signaling in
117                                              Transforming growth factor-beta (TGF-beta) signaling is
118                                          The transforming growth factor-beta (TGF-beta) signaling pat
119                           Hyperactivation of transforming growth factor-beta (TGF-beta) signaling pat
120 teins are central mediators in the canonical transforming growth factor-beta (TGF-beta) signaling pat
121 SIGNIFICANCE STATEMENT We show that reducing Transforming growth factor-beta (TGF-beta) signaling pro
122                           Here, we show that transforming growth factor-beta (TGF-beta) signaling via
123 umulation, extracellular matrix degradation, transforming growth factor-beta (TGF-beta) signaling, co
124 clear-shuttling transcriptional mediators of transforming growth factor-beta (TGF-beta) signaling.
125 ere we show that epidermal Hedgehog (Hh) and Transforming growth factor-beta (TGF-beta) signalling me
126 eover, RNA-sequencing analysis shows altered transforming growth factor-beta (TGF-beta) signalling.
127               We have shown a vital role for transforming growth factor-beta (TGF-beta) signals in sa
128                                              Transforming growth factor-beta (TGF-beta) signals throu
129 o known as MIC-1, is a distant member of the transforming growth factor-beta (TGF-beta) superfamily a
130 lammatory drug-activated gene-1 (NAG-1) is a transforming growth factor-beta (TGF-beta) superfamily p
131                                    ABSTRACT: Transforming growth factor-beta (TGF-beta), RhoA/Rho-kin
132                                              Transforming growth factor-beta (TGF-beta), serine prote
133 f regulatory T cells (Treg cells) induced by transforming growth factor-beta (TGF-beta), we identifie
134 t analysis of gene expression changes during transforming growth factor-beta (TGF-beta)-induced EMT i
135  that the STAT3 signaling pathway attenuates transforming growth factor-beta (TGF-beta)-induced respo
136 injury produced by airway disease triggers a transforming growth factor-beta (TGF-beta)-mediated epig
137      Papillary thyroid carcinoma overexpress transforming growth factor-beta (TGF-beta).
138  and also required a novel intersection with transforming growth factor-beta (TGF-beta)/SMAD signalin
139 ced expression of Wnt target genes (axin-2), transforming growth factor-beta (TGF-beta1) and collagen
140                     Activin, a member of the transforming growth factor-beta (TGFB) family, might be
141 d that DMF blocks the profibrotic effects of transforming growth factor-beta (TGFbeta) in SSc skin fi
142  integrin alphavbeta8-mediated activation of transforming growth factor-beta (TGFbeta).
143   Hepatic stellate cell (HSC) activation and transforming growth factor-beta 1 (TGF-beta1) expression
144 asuring fibrosis markers, proliferation, and transforming growth factor-beta 1 secretion.
145 le actin, fibronectin, collagen type 1a, and transforming growth factor-beta 1.
146 tine leads to release of fossilized factors (transforming growth factor-beta [TGF-beta] and bone morp
147 lation of IL-1 receptor-associated kinase 1, transforming growth factor-beta activated kinase-1, Ikap
148 expression, and profibrogenic cytokines (eg, transforming growth factor-beta and IL-13) and alteratio
149 vs E12.5, while abundance of elements of the transforming growth factor-beta and insulin-like growth
150             RDEB mice express high levels of transforming growth factor-beta and significantly lower
151 diac reprogramming was similarly enhanced on transforming growth factor-beta and WNT inhibition and w
152                                              Transforming growth factor-beta and WNT inhibitors joint
153 pilla inductive signaling pathways including transforming growth factor-beta and Wnt/beta-catenin.
154                                  Endoglin, a transforming growth factor-beta co-receptor, is highly e
155                                              Transforming growth factor-beta decreased RXFP1 in both
156 nished lung injury, collagen production, and transforming growth factor-beta expression and signaling
157 ial cell expansion, and collagen type IV and transforming growth factor-beta expression were signific
158 tensin-aldosterone system and members of the transforming growth factor-beta family play an important
159  the mature growth factor domains across the transforming growth factor-beta family.
160 hat further induction of Mmp-2 expression by transforming growth factor-beta I was blocked by Meg3 si
161 y both in the absence and in the presence of transforming growth factor-beta I.
162           We found that a combination of the transforming growth factor-beta inhibitor SB431542 and t
163 tion of cytotoxic T-lymphocyte antigen 4 and transforming growth factor-beta partially abrogated the
164 capacity to suppress multiple genes from the transforming growth factor-beta pathway and the producti
165 tracellular space, such as modulation of pro-transforming growth factor-beta processing, activation o
166 ted with reduced macrophage infiltration and transforming growth factor-beta production.
167 eceptor type 1C (ACVR1C), a component of the transforming growth factor-beta receptor superfamily.
168 own-regulation of thrombospondin 1, a latent transforming growth factor-beta receptor, and transcript
169                miR-337-3p requires Notch and transforming growth factor-beta signaling and exerts a b
170                          We demonstrate that transforming growth factor-beta signaling confers this p
171 which was partly attributable to blockade of transforming growth factor-beta signaling in dermal fibr
172 ncreased expression of profibrotic genes and transforming growth factor-beta signaling in SFBLs.
173 was determined on mitochondrial function and transforming growth factor-beta signaling in vitro and i
174 ed genes were significantly enriched in the "transforming growth factor-beta signaling pathway".
175  agonists, likely through suppression of the transforming growth factor-beta signaling pathway.
176 mitochondrial ribosomal stress and increased transforming growth factor-beta signaling.
177 lation, suggesting that miR-542-5p increased transforming growth factor-beta signaling.
178 R-29 itself is regulated by SP1 activity and transforming growth factor-beta signaling.
179 nt role in kidney fibrogenesis by modulating transforming growth factor-beta signaling.
180 actor 11 (GDF11) and GDF8 are members of the transforming growth factor-beta superfamily sharing 89%
181          Endoglin (CD105), a receptor of the transforming growth factor-beta superfamily, has been re
182  molecular markers of fibrosis (collagen and transforming growth factor-beta) and immunostaining for
183 -2 complex had no effect on profibrotic (eg, transforming growth factor-beta) or type 17 immune respo
184 e because of myeloid deficiency of TGF-beta (transforming growth factor-beta) signaling.
185 ticularly robust when cells are treated with transforming growth factor-beta, an enhancer of EMT.
186 review, we summarize how the wingless (Wnt), transforming growth factor-beta, and bone morphogenetic
187 owed tumor necrosis factor-alpha, IFN-gamma, transforming growth factor-beta, and IL-13 as potential
188  mannose receptor-1 (CD206), interleukin-10, transforming growth factor-beta, arginase-1, matrix meta
189 that it involves collagen production through transforming growth factor-beta, at least in the case of
190 tifibrotic strategies include antagonists of transforming growth factor-beta, connective tissue growt
191 in kinases, tight junctions, focal adhesion, transforming growth factor-beta, vascular smooth muscle
192             Smad7 is a negative regulator of transforming growth factor-beta, which is increased in t
193 a/b and thereby modulating the expression of transforming growth factor-beta-activated kinase 1 (TAK1
194     GCD is caused by a point mutation in the transforming growth factor-beta-induced (TGFBI) gene, lo
195     Mechanistically, sphingosine ameliorates transforming growth factor-beta-induced collagen accumul
196 rmore, mimics of miR-185 and miR-186 blocked transforming growth factor-beta-induced collagen V overe
197 based selective miRNA analysis revealed that transforming growth factor-beta-induced enhanced express
198 ced collagen V overexpression and alleviated transforming growth factor-beta-induced epithelial-mesen
199 control fibroblasts and attenuated basal and transforming growth factor-beta-induced expression of CC
200 RNA-21 levels suppressed the IL10 effects on transforming growth factor-beta-induced fibrotic signali
201 evel, IL10 treatment significantly inhibited transforming growth factor-beta-induced transdifferentia
202 tase expression was significantly induced by transforming growth factor-beta.
203 ancer, mitogen-activated protein kinase, and transforming growth factor-beta.
204  secreted proreparative cytokines, including transforming growth factor-beta.
205 anonical Hh pathway, involving TGFbeta/SMAD (transforming growth factor-beta/Sma- and Mad-related fam
206                                  KEY POINTS: Transforming growth-factor-beta (TGF-beta) and RhoA/Rho-
207 of profibrotic gene markers, fibronectin and transforming-growth-factor-beta.
208 kophilin-2 (PKP2) in cardiomyocytes elevates transforming growth factor beta1 (TGF-beta1) and p38 mit
209 eal injury typically involves fibrosis, with transforming growth factor beta1 (TGF-beta1) as one of i
210 al immunosensor for the determination of the transforming growth factor beta1 (TGF-beta1) cytokine co
211                                    Increased transforming growth factor beta1 (TGF-beta1) in mammary
212                                              Transforming growth factor beta1 (TGF-beta1) is a master
213 KRAS-variant with p16 status and blood-based transforming growth factor beta1 (TGF-beta1).
214                                              Transforming growth factor beta1 (TGFbeta1) is the princ
215 that the integrin alphavbeta8 and its latent transforming growth factor beta1 (TGFbeta1) protein liga
216                               High levels of transforming growth factor beta1 (TGFbeta1) were detecte
217 ide SLC4A2 expression under stimulation with transforming growth factor beta1 (TGFbeta1), a cytokine
218 gressive changes in renal mRNA expression of transforming growth factor beta1 (TGFbeta1), endothelin-
219 th factor receptor 1, 2, and 3; osteopontin; transforming growth factor beta1 and beta2; thrombospond
220 es the release of active MMP2 in response to transforming growth factor beta1 and rescues tissue inte
221    Mechanistically, this process engages the transforming growth factor beta1 protein and Notch signa
222 hemokine ligand 5 [CCL5]) and profibrogenic (transforming growth factor beta1, collagen type 4 alpha
223 o enhanced (by 20% to 40%) the expression of transforming growth factors beta1 and beta2, vascular en
224 e increase in collagen I and III expression, transforming growth factor-beta1 (TGF- beta1) expression
225 pression of Hmga2 enhances the activation of transforming growth factor-beta1 (TGF-beta1) and Cxcl12
226  interactions with soluble mediators such as transforming growth factor-beta1 (TGF-beta1) and mechani
227                     Because the signaling of transforming growth factor-beta1 (TGF-beta1) and tumor n
228                     Both a neutralizing anti-transforming growth factor-beta1 (TGF-beta1) antibody an
229 s inhibited by increased accumulation of the transforming growth factor-beta1 (TGF-beta1) in skeletal
230                                              Transforming growth factor-beta1 (TGF-beta1) may stimula
231 ibrogenic phenotype through the induction of transforming growth factor-beta1 (TGF-beta1), collagen d
232 pression variations (versus normal aorta) of transforming growth factor-beta1 (TGF-beta1), connective
233 e loss induced by AbetaOs, via production of transforming growth factor-beta1 (TGF-beta1).
234 (Mmp2) and Mmp9, which could activate latent transforming growth factor-beta1 (TGF-beta1).
235 onor corneas with interleukin-10 (IL-10) and transforming growth factor-beta1 (TGFbeta1) altered the
236 was up-regulated by the profibrotic cytokine transforming growth factor-beta1 and in PD effluent-deri
237 d investigated the interrelationship between transforming growth factor-beta1 and secreted frizzled-r
238 ponse to myelin perturbations, we identified transforming growth factor-beta1 as a partial mediator o
239           On the molecular level, microglial transforming growth factor-beta1 expression is down-regu
240 l population and increased the expression of transforming growth factor-beta1 in the liver.
241                                  The myokine transforming growth factor-beta1 increases during inflam
242                         We observed that the transforming growth factor-beta1 inhibitor, secreted fri
243                                 We show that transforming growth factor-beta1 is responsible for the
244 ional repressors, snail and slug, induced by transforming growth factor-beta1 or extracellular matrix
245                         In activation of the transforming growth factor-beta1 precursor (pro-TGF-beta
246       Gene set enrichment analysis uncovered transforming growth factor-beta1 signaling activation in
247 2 physically interacted and colocalized with transforming growth factor-beta1 through its cysteine-ri
248                                   TGF-beta1 (transforming growth factor-beta1) importantly contribute
249        Alu RNA expression is also induced by transforming growth factor-beta1, a major driver of EMT.
250 ession levels of fibroblast growth factor-2, transforming growth factor-beta1, and platelet-derived g
251 tu by antagonizing E-cadherin, combined with transforming growth factor-beta1, epidermal growth facto
252 ditioned medium resulted in up-regulation of transforming growth factor-beta1, transforming growth fa
253 ed mesenchymal cells secreted high levels of transforming growth factor-beta1, which down-regulates m
254 ecreted frizzled-related protein 2 prevented transforming growth factor-beta1-induced atrophy in C2C1
255 tablishes a positive feedback loop enhancing transforming growth factor-beta1-mediated atrophic effec
256 rizzled-related protein 2 reduction enhances transforming growth factor-beta1-mediated effects and in
257 beta6 integrin, an activator of latent local transforming growth factor-beta1.
258                   Increased levels of active transforming growth factor-beta2 (TGF-beta2) in the aque
259 ulation of transforming growth factor-beta1, transforming growth factor-beta2, and collagens I and II
260                                              Transforming growth factor-betas regulate a wide range o
261                            Here we show that transforming growth factor receptor 1 (TGFbetaR1) has an
262 es showed aberrant tumor necrosis factor and transforming growth factor signaling in metastatic cells
263  and associated with increased production of transforming growth factor (TGF) beta and interleukin (I
264 hymal self-organisation relies on restricted transforming growth factor (TGF) beta signalling, which
265 n cells were pretreated with blockers of the transforming growth factor (TGF) superfamily, purinergic
266 l-mesenchymal transition (EMT) is induced by transforming growth factor (TGF)-beta and facilitates tu
267 e further characterized by the activation of transforming growth factor (TGF)-beta and TGF-beta targe
268                                              Transforming growth factor (TGF)-beta cytokines signal v
269 n factor 15 (GDF15), a distant member of the transforming growth factor (TGF)-beta family, is a secre
270 expression in response to two members of the transforming growth factor (TGF)-beta family, TGF-beta1
271                                 The roles of transforming growth factor (TGF)-beta in extracellular m
272      In addition, culture in the presence of transforming growth factor (TGF)-beta inhibitors allows
273 promote SCC tumor initiation in concert with transforming growth factor (TGF)-beta present in the tum
274                                              Transforming growth factor (TGF)-beta signaling disorder
275              Herein, we analyzed the role of transforming growth factor (TGF)-beta signaling for CNV
276  the activity intrinsic to EMD that provokes transforming growth factor (TGF)-beta signaling in oral
277                                          The transforming growth factor (TGF)-beta signaling pathway
278      We provide evidence that Lfng regulates transforming growth factor (TGF)-beta signaling through
279                 Genes whose products mediate transforming growth factor (TGF)-beta signaling were als
280 red as having tumour-suppressive properties, transforming growth factor (TGF)-beta signalling is alte
281 hological formation, neural development, and transforming growth factor (TGF)-beta signalling of the
282                                              Transforming growth factor (TGF)-beta supports multiple
283 C or TDFSM controls, was highly dependent on transforming growth factor (TGF)-beta, but not promoted
284                            We tested whether transforming growth factor (TGF)-beta, which can functio
285                             Antioxidant- and transforming growth factor (TGF)-beta-related genes were
286 -mesenchymal transition (EMT) in response to transforming growth factor (TGF)-beta.
287 kine that induces myofibroblast formation is transforming growth factor (TGF)-beta.
288        In this review, we concentrate on the transforming growth factor (TGF)-beta/bone morphogenetic
289                           We now showed that transforming growth factor (TGF)-beta/Smad signaling par
290                   Recent findings suggesting transforming growth factor (TGF)-beta1 activation by mec
291 uction of hepatocyte growth factor (HGF) and transforming growth factor (TGF)-beta1 caused by injury.
292                                              Transforming growth factor (TGF)-beta1 contributes to au
293  first documented protein known to stimulate transforming growth factor (TGF)-beta1 to -beta3 transla
294            ADAM10 expression is increased by transforming growth factor (TGF)-beta1, and ADAM10-media
295 on of pro-fibrotic growth factors, including transforming growth factor (TGF)-beta1, in human granulo
296  disruption of cell-cell contact can promote transforming growth factor (TGF)-beta1-induced EMT and t
297 trate the potential of this platform for: i) transforming growth factor (TGF)-beta1-induced spatial d
298 JAK-STAT3 axis, Wnt-GSK3 signalling, and the transforming growth factor (TGF)beta family.
299 tokines (tumour necrosis factor [TNF]-alpha, transforming growth factor [TGF]-beta1, interleukin [IL]
300 e cells (SMCs) were stimulated with elevated transforming growth factor (TGFbeta) and its signaling p

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