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1 so had less re-expression of fetal genes and transforming growth factor beta1.
2 portant platelet released cytokines, such as transforming growth factor beta1.
3 talloproteinase-2, alkaline phosphatase, and transforming growth factor beta1.
4 , including bone morphogenetic protein 2 and transforming growth factor beta1.
5  and blunting of profibrogenic activities of transforming growth factor beta1.
6 al and cellular events and the activation of transforming growth factor beta1.
7 egulation of profibrotic pathway mediated by transforming growth factor-beta1.
8  of fibroblasts to the stimulatory effect of transforming growth factor-beta1.
9 n-T-helper type 1-related signals, including transforming growth factor-beta1.
10 ed upregulation of CSF-1R expression via the transforming growth factor-beta1.
11 tissue inhibitor of metalloproteinase-1, and transforming growth factor-beta1.
12 roduction of both cytokines was inhibited by transforming growth factor-beta1.
13 beta6 integrin, an activator of latent local transforming growth factor-beta1.
14 roteinase-2, matrix metalloproteinase-9, and transforming growth factor-beta1/2 and the number of Mac
15        Alu RNA expression is also induced by transforming growth factor-beta1, a major driver of EMT.
16 on memory B cells, whereas interleukin-4 and transforming growth factor-beta1 act as negative regulat
17 haVbeta6 integrin, an important regulator of transforming growth factor-beta1 activation.
18 signaling pathway, resulting in decreased LV transforming growth factor-beta1 activity (P=0.03).
19 s ability to block myostatin, activin A, and transforming growth factor-beta1, all of which are negat
20 le to undergo EMT in vitro when treated with transforming growth factor-beta1 alone or in combination
21 th factor receptor 1, 2, and 3; osteopontin; transforming growth factor beta1 and beta2; thrombospond
22                          The balance between transforming growth factor beta1 and bone morphogenetic
23 nts with SCD and isolated cytokines, such as transforming growth factor beta1 and endothelin-1, enhan
24 s in CCD-18Co fibroblasts in the presence of transforming growth factor beta1 and insulin-like growth
25 llagen, vimentin, and the fibrogenic factors transforming growth factor beta1 and insulin-like growth
26 d collagen I; this was inhibited by blocking transforming growth factor beta1 and p38 mitogen-activat
27 ed products, and decreased after blockade of transforming growth factor beta1 and p38 mitogen-activat
28 es the release of active MMP2 in response to transforming growth factor beta1 and rescues tissue inte
29                 Furthermore, the increase in transforming growth factor beta1 and type I transforming
30 ced expression of the profibrotic mediators, transforming growth factor-beta1 and connective tissue g
31 brosis, as well as its interaction with both transforming growth factor-beta1 and decorin.
32 L-6, but not IL-8, induced the expression of transforming growth factor-beta1 and elastic fiber prote
33 le and the numbers of which are regulated by transforming growth factor-beta1 and hypoxia.
34 was up-regulated by the profibrotic cytokine transforming growth factor-beta1 and in PD effluent-deri
35                  Among the cytokines tested, transforming growth factor-beta1 and interleukin-1beta e
36             In wild-type mice, we found that transforming growth factor-beta1 and myostatin co-locali
37            Decorin prevented the increase in transforming growth factor-beta1 and phosphorylated Smad
38 d investigated the interrelationship between transforming growth factor-beta1 and secreted frizzled-r
39 any types of carcinoma where it may activate transforming growth factor-beta1 and transforming growth
40  necrosis factor-alpha and reduced levels of transforming growth factor-beta1 and vascular endothelia
41 o enhanced (by 20% to 40%) the expression of transforming growth factors beta1 and beta2, vascular en
42  renal and glomerular hypertrophy as well as transforming growth factor-beta1) and extracellular matr
43  whereas SNAI2, vimentin, S100A4, FN1, HRAS, transforming growth factor beta1, and CD44H were high.
44 talloproteinase-9, B-cell activating factor, transforming growth factor-beta1, and elastase were high
45 cytokines IL-1beta, IL-18, interferon-gamma, transforming growth factor-beta1, and IL-4 as well as hi
46 ession levels of fibroblast growth factor-2, transforming growth factor-beta1, and platelet-derived g
47 ponse to myelin perturbations, we identified transforming growth factor-beta1 as a partial mediator o
48 gnaling pathways, including Snail, Twist and transforming growth factor-beta1 as well as the mesenchy
49 ated by tissue stiffness and the profibrotic transforming growth factor-beta1, but its role in cell f
50 pression was modulated by gut microbiota and transforming growth factor-beta1, but not by retinoic ac
51                                              Transforming growth factor-beta1, but not endoplasmic re
52 tatin, growth differentiation factor 11, and transforming growth factor beta1, by cleaving extracellu
53 hemokine ligand 5 [CCL5]) and profibrogenic (transforming growth factor beta1, collagen type 4 alpha
54 ce, MUC5AC and matrix remodeling parameters (transforming growth factor-beta1, collagen I, and fibron
55 nd membrane-bound latency-associated peptide/transforming growth factor beta1, compared with uninfect
56 ry occlusion and were divided into 3 groups: transforming growth factor beta1-conditioned human MSC-l
57                                              Transforming growth factor beta1-conditioned human MSC-l
58 al expression of fibrogenic genes, including transforming growth factor beta1, connective tissue grow
59 vels of key mediators of fibrosis, including transforming growth factor beta1, connective tissue grow
60  HA-binding protein TSG-6 in maintaining the transforming growth factor beta1-dependent HA coat, TSG-
61 it as a key difference between IL-1beta- and transforming growth factor beta1-dependent HA matrices.
62  sequencing (RNA-seq) analysis revealed that transforming growth factor beta1 (encoded by Tgfb1) is e
63 tu by antagonizing E-cadherin, combined with transforming growth factor-beta1, epidermal growth facto
64           On the molecular level, microglial transforming growth factor-beta1 expression is down-regu
65 hickening, podocyte foot-process fusion, and transforming growth factor-beta1 expression were amelior
66 , pancreatic duodenal homeobox-1, and active transforming growth factor-beta1 expression were increas
67 so resulted in fibrosis, which was linked to transforming growth factor-beta1 expression.
68     Here, we show that the expression of the transforming growth factor beta1 gene (Tgfb1) affects th
69 association with CAA of polymorphisms in the transforming growth factor beta1 gene (two studies, 449
70 tides such as alpha-smooth muscle actin, and transforming growth factor-beta1 had no effect on FGFR1O
71 LGs had higher Th-17- (interleukin [IL]-23R, transforming growth factor-beta1, IL-17A, CC chemokine a
72     We determined that genetic deficiency in transforming growth factor beta1 impairs endometriosis-l
73                                   TGF-beta1 (transforming growth factor-beta1) importantly contribute
74 ntly reduced thrombin activity and levels of transforming growth factor beta1 in BAL fluid, while sim
75 D25(+) T regulatory cells, and production of transforming growth factor beta1 in the spleen.
76 ced marked accumulation of activin A but not transforming growth factor-beta1 in conditioned media.
77                                              Transforming growth factor-beta1 in PICF and GCF exhibit
78 l population and increased the expression of transforming growth factor-beta1 in the liver.
79 Extracellular TG2 was sufficient to activate transforming growth factor-beta1 in tubular epithelial c
80 es indicate that the upregulation of Nox4 by transforming growth factor-beta1 in ZO SMCs is responsib
81                                  The myokine transforming growth factor-beta1 increases during inflam
82 ombination of fibroblast growth factor 2 and transforming growth factor beta1 induce profound morphol
83 In isolated adult mouse cardiac fibroblasts, transforming growth factor-beta1 induced cardiac fibrobl
84 owever, activation of AhR in the presence of transforming growth factor-beta1 induced Foxp3(+) iT(reg
85 Furthermore, tumor necrosis factor-alpha and transforming growth factor-beta1 induced the expression
86             Overexpression of Epac1 inhibits transforming growth factor beta1-induced collagen synthe
87 ty of this technology to prevent and inhibit transforming growth factor beta1-induced myodifferentiat
88 ecreted frizzled-related protein 2 prevented transforming growth factor-beta1-induced atrophy in C2C1
89                           Berberine reversed transforming growth factor-beta1-induced EMT and caused
90 ansplant model, and in the in vitro model of transforming growth factor-beta1-induced EMT in normal r
91 , Nox-2, phospho-smad2, and alpha-SMA during transforming growth factor-beta1-induced EMT of NRK52E c
92 ound that the miR-200 family members inhibit transforming growth factor-beta1-induced epithelial-mese
93                    PAR-1 activation enhanced transforming growth factor-beta1-induced integrin beta6
94 synthetic and contractile phenotype, in both transforming growth factor-beta1-induced myofibroblast d
95 fibrosis and cardiac dysfunction by blocking transforming growth factor-beta1-induced phospho-Smad2/3
96             Previous studies have shown that transforming growth factor-beta1-induced transcript 1 (T
97                    These phenomena depend on transforming growth factor-beta1 induction.
98 type with patches of collagen deposition and transforming growth factor-beta1 induction.
99                         We observed that the transforming growth factor-beta1 inhibitor, secreted fri
100 nclusion of Rho-associated kinase (ROCK) and transforming growth factor-beta1 inhibitors.
101 or 1 (VEGFR1), VEGFR2, Tie2, erythropoietin, transforming growth factor beta1, insulinlike growth fac
102                                 We show that transforming growth factor-beta1 is responsible for the
103                           Vaginal pH and the transforming growth factor beta1 level increased, but hu
104 nfiltration, proinflammatory mRNAs, and TGF-(Transforming Growth Factor-)beta1 levels.
105 nsverse aortic constriction increased active transforming growth factor-beta1 levels and phosphorylat
106                                        Serum transforming growth factor-beta1 levels were significant
107 if) ligand 1; stromal cell-derived factor 1; transforming growth factor-beta1; matrix metallopeptidas
108          Additionally, our data suggest that transforming growth factor-beta1 may be a factor in indu
109                              Id2 also blocks transforming growth factor beta1-mediated expression of
110 tablishes a positive feedback loop enhancing transforming growth factor-beta1-mediated atrophic effec
111 rizzled-related protein 2 reduction enhances transforming growth factor-beta1-mediated effects and in
112 n of a constitutively active FOXO3a overrode transforming growth factor-beta1-mediated invasive pheno
113 3), CD11b-F4/80+Gr1+ monocytes (P < 0.0001), transforming growth factor beta1 mRNA (P = 0.04), and al
114 although anti-inflammatory IL-10, IL-13, and transforming growth factor beta1 mRNA levels were not co
115 d WT mice, but Col1 alpha1, Col3 alpha1, and transforming growth factor-beta1 mRNA increased much mor
116  observed in tumor necrosis factor-alpha and transforming growth factor-beta1 mRNA levels and intrace
117 ional repressors, snail and slug, induced by transforming growth factor-beta1 or extracellular matrix
118 lasminogen activator inhibitor 1 (P=0.0002), transforming growth factor beta1 (P=0.0004), tissue inhi
119 yme B (P<0.002), CD103 (P<0.005) but not for transforming growth factor-beta1 (P>0.05).
120 expression levels of fibrogenic markers (eg, transforming growth factor-beta1, platelet-derived growt
121                         In activation of the transforming growth factor-beta1 precursor (pro-TGF-beta
122 ith previous reports, Extract PBS suppressed transforming growth factor-beta1 promoter activation in
123    Mechanistically, this process engages the transforming growth factor beta1 protein and Notch signa
124 ransforming growth factor-beta1 (TGF-beta1), transforming growth factor-beta1 receptor (TGF-beta1R),
125  Nrf2 suppressed the activity of a synthetic transforming growth factor-beta1-responsive CAGA-directe
126 ndogenous TLR4 ligands, results in augmented transforming growth factor-beta1 sensitivity with increa
127 hepatic macrophage aggregation, resulting in transforming growth factor beta1-signaled collagen depos
128       Gene set enrichment analysis uncovered transforming growth factor-beta1 signaling activation in
129 ates in a negative feedback mechanism on the transforming growth factor-beta1 signaling and downregul
130 eas the sik1(-/-) mice exhibited upregulated transforming growth factor-beta1 signaling and increased
131 arly- and late-stage cardiac remodeling with transforming growth factor-beta1 signaling at the center
132 odulation of activin-A, activin-B, Nodal and transforming growth factor-beta1 signaling.
133 on of the contractile phenotype of VSMCs via transforming growth factor-beta1-signaling inhibition.
134 hanistically, GSK-3beta inhibits profibrotic transforming growth factor-beta1/SMAD-3 signaling via in
135                         Knockdown of YAP1 in transforming growth factor-beta1-stimulated dermal fibro
136 X5 SNP-expressing cells had higher basal and transforming growth factor-beta1-stimulated expression a
137 nd interleukin 1 receptor antagonist but not transforming growth factor beta1, suggesting that alkylg
138 ce similar phenotypes, as well as of several transforming growth factor-beta1 target genes, such as m
139                                              Transforming growth factor beta1 (TGF-beta) promotes ren
140                                              Transforming growth factor beta1 (TGF-beta), enriched in
141 models of fibrotic renal disease by opposing transforming growth factor beta1 (TGF-beta)-dependent fi
142 d that IL-17A mediates epithelial injury via transforming growth factor beta1 (TGF-beta1) and down-re
143 es of orally tolerized mice showed increased transforming growth factor beta1 (TGF-beta1) and interle
144 ves increased migration and up-regulation of transforming growth factor beta1 (TGF-beta1) and its dow
145                                              Transforming growth factor beta1 (TGF-beta1) and osteopo
146 kophilin-2 (PKP2) in cardiomyocytes elevates transforming growth factor beta1 (TGF-beta1) and p38 mit
147                                 We evaluated transforming growth factor beta1 (TGF-beta1) as a key up
148 eal injury typically involves fibrosis, with transforming growth factor beta1 (TGF-beta1) as one of i
149                                 We show that transforming growth factor beta1 (TGF-beta1) can induce
150 lower expression of the profibrogenic factor transforming growth factor beta1 (TGF-beta1) compared to
151 al immunosensor for the determination of the transforming growth factor beta1 (TGF-beta1) cytokine co
152 d activity of the immunosuppressive cytokine transforming growth factor beta1 (TGF-beta1) due to high
153       Understanding inhibitory mechanisms of transforming growth factor beta1 (TGF-beta1) has provide
154                                    Increased transforming growth factor beta1 (TGF-beta1) in mammary
155                                              Transforming growth factor beta1 (TGF-beta1) is a master
156                                 We show that transforming growth factor beta1 (TGF-beta1) is activate
157                                              Transforming growth factor beta1 (TGF-beta1) is not only
158 of proinflammatory mediators while restoring transforming growth factor beta1 (TGF-beta1) levels as e
159 CsA treatment and to investigate the role of transforming growth factor beta1 (TGF-beta1) on this CsA
160 rming cartilage in the presence of exogenous transforming growth factor beta1 (TGF-beta1) or with TGF
161                      We demonstrate that the transforming growth factor beta1 (TGF-beta1) pathway up-
162       The generation of oxidative stress and transforming growth factor beta1 (TGF-beta1) production
163 .5 kPa) polyacrylamide gels (with or without transforming growth factor beta1 (TGF-beta1)) and low pr
164      Addition of blocking antibodies against transforming growth factor beta1 (TGF-beta1), -2, and -3
165                          Here, we found that transforming growth factor beta1 (TGF-beta1), a critical
166  by serotonin, which activates expression of transforming growth factor beta1 (TGF-beta1), a powerful
167 r messenger RNA and/or protein expression of transforming growth factor beta1 (Tgf-beta1), collagen 1
168          It has been shown that pro-fibrotic transforming growth factor beta1 (TGF-beta1)-driven epit
169           A critical step in this process is transforming growth factor beta1 (TGF-beta1)-mediated tr
170                In spite of a large number of transforming growth factor beta1 (TGF-beta1)-regulated g
171 fibroblasts to myofibroblasts in response to transforming growth factor beta1 (TGF-beta1).
172 ofibroblast phenotype under the influence of transforming growth factor beta1 (TGF-beta1).
173 KRAS-variant with p16 status and blood-based transforming growth factor beta1 (TGF-beta1).
174 n hepatogenic profibrotic cells activated by transforming growth factor beta1 (TGF-beta1).
175 ponse to the differentiation-inducing factor transforming growth factor beta1 (TGF-beta1).
176 nse to infection, and enhanced expression of transforming growth factor beta1 (TGF-beta1).
177 e increase in collagen I and III expression, transforming growth factor-beta1 (TGF- beta1) expression
178 dels of DN, and mesangial cells treated with transforming growth factor-beta1 (TGF- beta1) or high gl
179 nt memory T cells (TRM cells) require active transforming growth factor-beta1 (TGF-beta) for epiderma
180 r matrix proteins and hypertrophy induced by transforming growth factor-beta1 (TGF-beta) in renal mes
181                      Akt kinase activated by transforming growth factor-beta1 (TGF-beta) plays an imp
182          We report that expression levels of transforming growth factor-beta1 (TGF-beta), p53, and mi
183      Racial differences in the expression of transforming growth factor-beta1 (TGF-beta1) and caveoli
184 pression of Hmga2 enhances the activation of transforming growth factor-beta1 (TGF-beta1) and Cxcl12
185                     A model system combining transforming growth factor-beta1 (TGF-beta1) and EGF was
186 ogenic; in fact, T(H)17 cells generated with transforming growth factor-beta1 (TGF-beta1) and IL-6 pr
187 s of BM CD45(+)Col(+) cells was regulated by transforming growth factor-beta1 (TGF-beta1) and liposac
188  interactions with soluble mediators such as transforming growth factor-beta1 (TGF-beta1) and mechani
189 s, at least, partially through inhibition of transforming growth factor-beta1 (TGF-beta1) and reducti
190          CAF express elevated levels of both transforming growth factor-beta1 (TGF-beta1) and stromal
191  in both animal models led to a reduction of transforming growth factor-beta1 (TGF-beta1) and TGF-bet
192    In the present study, we evaluate whether transforming growth factor-beta1 (TGF-beta1) and tumor n
193                     Because the signaling of transforming growth factor-beta1 (TGF-beta1) and tumor n
194                     Both a neutralizing anti-transforming growth factor-beta1 (TGF-beta1) antibody an
195 which autocrine production and activation of transforming growth factor-beta1 (TGF-beta1) are require
196                        TSP-1 converts latent transforming growth factor-beta1 (TGF-beta1) complexes i
197 ed the rate of wound closure and reduced the transforming growth factor-beta1 (TGF-beta1) expression
198 ed systemic hypertension, suppressed Agt and transforming growth factor-beta1 (TGF-beta1) gene expres
199                                              Transforming growth factor-beta1 (TGF-beta1) has potent
200 s inhibited by increased accumulation of the transforming growth factor-beta1 (TGF-beta1) in skeletal
201 after stimulation with the profibrotic agent transforming growth factor-beta1 (TGF-beta1) in vitro.
202 ssion correlated with impaired activation of transforming growth factor-beta1 (TGF-beta1) in vivo and
203                                              Transforming growth factor-beta1 (TGF-beta1) induced end
204                            Here we show that transforming growth factor-beta1 (TGF-beta1) induces MMP
205                                              Transforming growth factor-beta1 (TGF-beta1) induces NOX
206         We have previously demonstrated that transforming growth factor-beta1 (TGF-beta1) induces ple
207 se PKA increased, autoinductive signaling by transforming growth factor-beta1 (TGF-beta1) initiated a
208   We investigated beta-catenin-dependent and transforming growth factor-beta1 (TGF-beta1) interaction
209 ractile protein-expressing myofibroblasts by transforming growth factor-beta1 (TGF-beta1) is a critic
210                                              Transforming growth factor-beta1 (TGF-beta1) is a fundam
211                                              Transforming growth factor-beta1 (TGF-beta1) is a multif
212                                              Transforming growth factor-beta1 (TGF-beta1) is a multif
213                                              Transforming growth factor-beta1 (TGF-beta1) is a potent
214 cellular carcinoma cells and identified that transforming growth factor-beta1 (TGF-beta1) is induced
215                                              Transforming growth factor-beta1 (TGF-beta1) is released
216  a concomitant increase in total circulating transforming growth factor-beta1 (TGF-beta1) levels (P =
217                                              Transforming growth factor-beta1 (TGF-beta1) may stimula
218 to NK cell killing, which can be reversed by transforming growth factor-beta1 (TGF-beta1) neutralizin
219 his study addressed if the activation of the transforming growth factor-beta1 (TGF-beta1) pathway is,
220 P2 (S-phase kinase-associated protein 2) and transforming growth factor-beta1 (TGF-beta1) play import
221                                              Transforming growth factor-beta1 (TGF-beta1) plays essen
222 aim of our study was to evaluate the role of transforming growth factor-beta1 (TGF-beta1) polymorphis
223                             Control-released transforming growth factor-beta1 (TGF-beta1) promoted th
224                                              Transforming growth factor-beta1 (TGF-beta1) protects ag
225                                              Transforming growth factor-beta1 (TGF-beta1) protein exp
226                                              Transforming growth factor-beta1 (TGF-beta1) regulates i
227 , fibroblast growth factor-2 (FGF-2), and/or transforming growth factor-beta1 (TGF-beta1) to adult bo
228                      The induction of EMT by transforming growth factor-beta1 (TGF-beta1) was analyze
229 21, IL-1beta, IL-6, IL-17, IL-23, IL-10, and transforming growth factor-beta1 (TGF-beta1) were quanti
230 investigated the interaction between LOX and transforming growth factor-beta1 (TGF-beta1), a potent g
231 on of alpha-smooth muscle actin (alpha-SMA), transforming growth factor-beta1 (TGF-beta1), and matrix
232 ibrogenic phenotype through the induction of transforming growth factor-beta1 (TGF-beta1), collagen d
233 pression variations (versus normal aorta) of transforming growth factor-beta1 (TGF-beta1), connective
234 n expression of the Nrf2 pathway, as well as transforming growth factor-beta1 (TGF-beta1), fibronecti
235  astrocyte functions, we examined effects of transforming growth factor-beta1 (TGF-beta1), lipopolysa
236              We show that in the presence of transforming growth factor-beta1 (TGF-beta1), LRG1 is mi
237                           Gene expression of transforming growth factor-beta1 (TGF-beta1), nuclear fa
238 lial-mesenchymal transition (EMT) induced by transforming growth factor-beta1 (TGF-beta1), papillary
239     In addition, COPD cultures released more transforming growth factor-beta1 (TGF-beta1), reflecting
240 S cells), expression of markers of fibrosis, transforming growth factor-beta1 (TGF-beta1), transformi
241                              We identify two transforming growth factor-beta1 (TGF-beta1)-dependent m
242 ast growth factor (FGF-2) effectively blocks transforming growth factor-beta1 (TGF-beta1)-mediated my
243 ry stiffening with ageing is associated with transforming growth factor-beta1 (TGF-beta1)-related inc
244 ing and is largely regulated by the cytokine transforming growth factor-beta1 (TGF-beta1).
245 ey to this process is the mammalian cytokine transforming growth factor-beta1 (TGF-beta1).
246 sis and is largely regulated by the cytokine transforming growth factor-beta1 (TGF-beta1).
247 sion of bone morphogenic proteins (BMPs) and transforming growth factor-beta1 (TGF-beta1).
248 e loss induced by AbetaOs, via production of transforming growth factor-beta1 (TGF-beta1).
249 (Mmp2) and Mmp9, which could activate latent transforming growth factor-beta1 (TGF-beta1).
250 ce had impaired mitophagy and reduced active transforming growth factor-beta1 (TGF-beta1).
251 mice, and expression of calpain-1/2 and MMP2/transforming growth factor-beta1 (TGF-beta1).
252 in adipose tissue macrophages highlights the transforming growth factor beta1 (TGFB1) gene itself as
253                                          The transforming growth factor beta1 (TGFB1) polymorphism -5
254          Mice deficient in the gene encoding transforming growth factor-beta1 (Tgfb1(-/-) mice) acute
255                                              Transforming growth factor-beta1 (TGFB1) is a multifunct
256 d rs1982073, 'codon 10') in the 5' region of transforming growth factor-beta1 (TGFB1), a putative CF
257                               Treatment with transforming growth factor beta1 (TGFbeta), which is imp
258 sembly/absence of intercellular contacts and transforming growth factor-beta1 (TGFbeta) exposure.
259                                              Transforming growth factor beta1 (TGFbeta1) and bone mor
260                      The interaction between transforming growth factor beta1 (TGFbeta1) and hedgehog
261                           Here, we show that transforming growth factor beta1 (TGFbeta1) and interleu
262 emical inhibitor of Notch signaling and anti-transforming growth factor beta1 (TGFbeta1) antibody.
263 r effects of human polymorphisms influencing transforming growth factor beta1 (TGFbeta1) expression,
264 inhibitor of metalloproteinase-1 (TIMP1) and transforming growth factor beta1 (TGFbeta1) in activated
265                                              Transforming growth factor beta1 (TGFbeta1) induced alph
266                                 We show that transforming growth factor beta1 (TGFbeta1) is a potent
267                                              Transforming growth factor beta1 (TGFbeta1) is required
268                                              Transforming growth factor beta1 (TGFbeta1) is the princ
269                                              Transforming growth factor beta1 (TGFbeta1) plays a cruc
270 that the integrin alphavbeta8 and its latent transforming growth factor beta1 (TGFbeta1) protein liga
271                               High levels of transforming growth factor beta1 (TGFbeta1) were detecte
272 ide SLC4A2 expression under stimulation with transforming growth factor beta1 (TGFbeta1), a cytokine
273 ne human renal epithelial cells treated with transforming growth factor beta1 (TGFbeta1), a model of
274 gressive changes in renal mRNA expression of transforming growth factor beta1 (TGFbeta1), endothelin-
275  of bone morphogenetic protein 7 (BMP-7) and transforming growth factor beta1 (TGFbeta1)-induced huma
276 e medium on VN or collagen (CL) with 1 ng/mL transforming growth factor beta1 (TGFbeta1).
277 onor corneas with interleukin-10 (IL-10) and transforming growth factor-beta1 (TGFbeta1) altered the
278                            Overexpression of transforming growth factor-beta1 (TGFbeta1) in the norma
279          The ubiquitously expressed cytokine transforming growth factor-beta1 (TGFbeta1) promotes car
280 factors (IRFs), IFN-stimulated genes (ISGs), transforming growth factor-beta1 (TGFbeta1), and several
281 and Cdc42 are the principal mediators of the transforming growth factor-beta1 (TGFbeta1)-stimulated e
282 s myofibroblast differentiation triggered by transforming growth factor-beta1 (TGFbeta1).
283 2 physically interacted and colocalized with transforming growth factor-beta1 through its cysteine-ri
284                SD-4 on SS cells also trapped transforming growth factor-beta1 to their cell surface,
285 nd latent paracrine signaling cytokines (eg, transforming growth factor-beta1) to promote maladaptive
286 ere, we show that the profibrogenic agonist, transforming growth factor beta1, transcriptionally decr
287 ditioned medium resulted in up-regulation of transforming growth factor-beta1, transforming growth fa
288 ed transcriptional profiling of kidneys from transforming growth factor-beta1 transgenic (Tg) mice, c
289  and interferon-gamma but increased IL-2 and transforming growth factor-beta1, two cytokines required
290 s had increased expression of the cytokines, transforming growth factor-beta1, vascular endothelial g
291                                 In addition, transforming growth factor-beta1 via Smad2 was necessary
292 egulation by endoplasmic reticulum stress or transforming growth factor-beta1, was analyzed by small
293                alpha-Smooth muscle actin and transforming growth factor beta1 were increased in both
294 ses in levels of periostin, collagen VI, and transforming growth factor beta1 were linearly correlate
295 urinary cell mRNA for granzyme B, CD103, and transforming growth factor-beta1 were measured to ascert
296 f CCL2, CCL3, interleukin (IL)-6, IL-10, and transforming growth factor-beta1 were significantly elev
297  factor-alpha, hepatocyte growth factor, and transforming growth factor-beta1 were significantly lowe
298 terferon-gamma, tumor necrosis factor-alpha, transforming growth factor-beta1) were evaluated.
299 ed mesenchymal cells secreted high levels of transforming growth factor-beta1, which down-regulates m
300 cient skin and wounds had elevated levels of transforming growth factor-beta1, which have been shown

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