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1 tologous CD4 T cells and was attributed to a transfusion reaction.
2 Twenty-two (1.1%) had delayed serologic transfusion reaction.
3 n TRALI or control PCs that did not elicit a transfusion reaction.
4 lood itself, or the probability or extent of transfusion reaction.
5 can cause life-threatening delayed hemolytic transfusion reactions.
6 ating a risk for Vel blood typing errors and transfusion reactions.
7 eic plasma carries with it risks for adverse transfusion reactions.
8 sociated with laboratory evidence of delayed transfusion reactions.
9 ibodies to HNA-2a frequently cause pulmonary transfusion reactions.
10 177, and the role of antibodies to HNA-2a in transfusion reactions.
11 blood components frequently causes pulmonary transfusion reactions.
12 pansion as well as the risk of infection and transfusion reactions.
13 smission, and recurrent febrile nonhemolytic transfusion reactions.
14 that could lead to immune mediated hemolytic transfusion reactions.
15 ew opportunistic event/death or frequency of transfusion reactions.
16 e antigenic determinants, thereby minimizing transfusion reactions.
17 t time of transfusion and review of reported transfusion reactions.
18 ll as the signs, symptoms, and management of transfusion reactions.
20 prophylactic use and treatment of hemolytic transfusion reactions and complement-mediated hemolytic
21 including life-threatening delayed hemolytic transfusion reactions and difficulty in finding compatib
22 nically significant problem that can lead to transfusion reactions and difficulty in locating future
24 at block antibody binding in the settings of transfusion reactions and hemolytic disease of the newbo
25 against RBC non-ABO Ags can cause hemolytic transfusion reactions and limit availability of compatib
26 usion requirements, the incidence of delayed transfusion reactions, and de novo abs after transplant
27 for the specific management of anaphylactic transfusion reactions are contradictory as to the utilit
29 solution, designed to reduce plasma-related transfusion reactions, are also increasingly available b
31 p erythrocytes may pose a risk for hemolytic transfusion reactions due to their elevated PX2 levels.
35 he pathophysiology of IgG-mediated hemolytic transfusion reactions (HTRs), and deeper understanding i
36 related acute lung injury (TRALI), hemolytic transfusion reactions (HTRs), and transfusion-associated
37 echanisms that can trigger delayed hemolytic transfusion reactions in sickle cell disease, and we des
41 he probability of life-threatening hemolytic transfusion reactions, not all patients generate anti-RB
44 the impact on infectious complications, (2) transfusion reaction rate, (3) leukodepletion, (4) reduc
45 lood induce antibody responses and hemolytic transfusion reactions similar to those seen in patients.
47 ctoriness to transfusions, alloimmunization, transfusion reactions, the transmission of infectious ag
48 ion of incompatible blood, acute and delayed transfusion reactions, transfusion-related acute lung in
49 nd management of each diagnostic category of transfusion reaction using evidence-based recommendation
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