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1 agnosis, and similarities between sepsis and transfusion-related acute lung injury.
2 ken pre- and postoperatively and at onset of transfusion-related acute lung injury.
3 were prospectively screened for the onset of transfusion-related acute lung injury.
4 easily missed and may mimic or coexist with transfusion-related acute lung injury.
5 product may meet the clinical definition of transfusion-related acute lung injury.
6 oagulation pathways involved in the onset of transfusion-related acute lung injury.
7 d fever are noted in patients with suspected transfusion-related acute lung injury.
8 s initially consistent with and diagnosed as transfusion-related acute lung injury.
9 rs2288904 previously shown to associate with transfusion-related acute lung injury.
12 ed, whereas 61 of 145 patients with possible transfusion-related acute lung injury (42%) died and 7 o
14 usion-related acute lung injury and possible transfusion-related acute lung injury also had a statist
15 he clinical syndrome is attributed solely to transfusion-related acute lung injury and bacterial seps
20 to transfusion-related complications such as transfusion-related acute lung injury and transfusion-as
22 ding to ABO-incompatible blood transfusion), transfusion-related acute lung injury, and bacterial con
25 ilant investigation in patients suspected of transfusion-related acute lung injury, as septic transfu
29 mbin-antithrombin complexes were enhanced in transfusion-related acute lung injury cases compared wit
30 plasminogen activator inhibitor-1 levels in transfusion-related acute lung injury cases compared wit
31 and one randomized controlled trial in which transfusion-related acute lung injury cases only involve
34 tal morbidity and mortality in patients with transfusion-related acute lung injury compared with tran
35 ury (acute respiratory distress syndrome and transfusion-related acute lung injury), for assessment o
36 usion-related acute lung injury and possible transfusion-related acute lung injury had an increased d
40 studies have shown that aged RBCs can induce transfusion-related acute lung injury in the presence of
41 ts only to HLA-I HCs, is unlikely to produce transfusion-related acute lung injury, in contrast to an
42 transfusion strategy reduces plasma-related transfusion-related acute lung injury incidence and poss
55 literature that impact our understanding of transfusion related acute lung injury (TRALI) and transf
70 has attained significant knowledge regarding transfusion-related acute lung injury (TRALI) through th
73 review is to present the two-event model of transfusion-related acute lung injury (TRALI), a life-th
75 reduced to exceedingly low levels in the US, transfusion-related acute lung injury (TRALI), hemolytic
76 od, acute and delayed transfusion reactions, transfusion-related acute lung injury (TRALI), transfusi
77 of neutrophil extracellular traps (NETs) in transfusion-related acute lung injury (TRALI), which is
84 lished estimates of known transfusion risks: transfusion-related acute lung injury, transfusion-relat
85 ifferential for any patient believed to have transfusion-related acute lung injury with clinical feat
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