ライフサイエンスコーパス: transglutaminase

1 sign better substrates or inhibitors of this transglutaminase.

2 e fibrin network and cross-linking by plasma transglutaminase.

3 results from the polyamination of tubulin by transglutaminase.

4 that neuronal tubulin can be polyaminated by transglutaminase.

5 followed by repolymerization with microbial transglutaminase.

6 odification method involving thermolysin and transglutaminase.

7 lecular weight peptides were cross-linked by transglutaminase.

8 n of antibodies against epidermal and tissue transglutaminases.

9 ism of Nt(Q)-amidase are similar to those of transglutaminases.

10 b, -2f, and -2g proteins), the cross-linking transglutaminase 1 enzyme (Tg-1) and Muc5AC mRNA transcr

11 ucocerebrosidase, acid sphingomyelinase, and transglutaminase 1.

12 ldren tested positive for IgA against tissue transglutaminase (1.0%; 95% confidence interval, 0.4%-1.

13 Transglutaminase-1 (TGase-1) is a Ca(2+)-dependent enzym

14 into the pathophysiology of TGM1 ichthyoses, transglutaminase-1 enzymatic activity, and potential the

15 sive, temperature-sensitive mutations in the transglutaminase-1 gene (TGM1).

16 le gene mutated in lamellar ichthyosis (LI), transglutaminase-1, in rat keratinocytes, we created an

17 , and anti-IgA autoantibodies against tissue transglutaminase (12%).

18 celiac disease or antibodies against tissue transglutaminase 2 (controls), healthy family members of

19 to both human Factor XIII (FXIII) and tissue transglutaminase 2 (hTG2).

20 evious work in our laboratory has shown that transglutaminase 2 (TG2) acting as a coreceptor for inte

21 s of total IgA antibodies, IgA antibodies to transglutaminase 2 (TG2) and endomysium, as well as IgA

22 Antibodies to the autoantigen transglutaminase 2 (TG2) are a hallmark of celiac diseas

23 Autoantibodies specific for the enzyme transglutaminase 2 (TG2) are a hallmark of the gluten-se

24 A plasma cells (PCs) specific for the enzyme transglutaminase 2 (TG2) are abundant in the small intes

25 Transglutaminase 2 (TG2) catalyzes transamidation or dea

26 Transglutaminase 2 (TG2) expression is required for epid

27 Tissue transglutaminase 2 (TG2) has been of particular interest

28 Molecular deletion of transglutaminase 2 (TG2) has been shown to improve funct

29 Genetic ablation of enzyme transglutaminase 2 (TG2) in Mgp(-/-) mice dramatically r

30 The mechanism of activation of transglutaminase 2 (TG2) in the extracellular matrix rem

31 that it is a substrate for cross-linking by transglutaminase 2 (TG2) into higher-order species.

32 Transglutaminase 2 (TG2) is a Ca(2+)-dependent cross-lin

33 Tissue transglutaminase 2 (TG2) is a multifunctional protein pr

34 Transglutaminase 2 (TG2) is a multifunctional protein th

35 Transglutaminase 2 (TG2) is a ubiquitously expressed enz

36 Transglutaminase 2 (TG2) is a ubiquitously expressed enz

37 Tissue transglutaminase 2 (TG2) is overexpressed in epithelial

38 Transglutaminase 2 (TG2) is secreted by a non-classical

39 The multifunctional, protein cross-linking transglutaminase 2 (TG2) is the main autoantigen in celi

40 The multifunctional enzyme transglutaminase 2 (TG2) is the target of autoantibodies

41 l commercial assays that measure IgA against transglutaminase 2 (TG2) or IgG against DGP.

42 e is presented in this study that shows that transglutaminase 2 (TG2) plays a critical role in the ad

43 For example, Trx activates extracellular transglutaminase 2 (TG2) via reduction of an intramolecu

44 n 1 (MTA1), a master chromatin modifier, and transglutaminase 2 (TG2), a multifunctional enzyme, are

45 cross-linked by transamidation catalyzed by transglutaminase 2 (TG2), itself an inflammation-regulat

46 aspase-3 activation through stabilization of transglutaminase 2 (TG2), which cross-links and inactiva

47 ion plasma cells (PCs), we have analyzed the transglutaminase 2 (TG2)-specific VH:VL autoantibody rep

48 odies reactive to gluten or the self-antigen transglutaminase 2 (TG2).

49 on of autoantibodies specific for the enzyme transglutaminase 2 (TG2).

50 isfolded keratins; 2) elevated levels of the transglutaminase 2 (TG2); 3) increased K8 phosphorylatio

51 Interactions among IgA, CD71, and transglutaminase 2 (Tgase2) were analyzed by flow cytome

52 es the AP-1 transcription factor Fos and the transglutaminase 2 (TGase2), a cross-linking enzyme with

53 We identified transglutaminase 2 (TGM2) as a putative tumor suppressor

54 up 10 secretory phospholipase A2, Wnt5a, and transglutaminase 2 (Tgm2).

55 EMA-positive sera were further tested for transglutaminase 2 antibodies (TG2-IgA).

56 type and detection of anti-gliadin and anti-transglutaminase 2 antibodies to identify a subgroup of

57 activation in different species and identify Transglutaminase 2 as a conserved M2 marker that is high

58 ERalpha as a direct regulator of macrophage transglutaminase 2 expression, a multifunctional atherop

59 Transglutaminase 2 has an affinity to the MUC2 CysD2 dom

60 Whereas the development of anti-transglutaminase 2 IgA is linked with gastrointestinal d

61 patients, who have antibodies against tissue transglutaminase 2 in the absence of villous atrophy.

62 Transforming growth factor-beta and transglutaminase 2 stimulate Snail-dependent invadosome

63 e deposits (sCD89, transferrin receptor, and transglutaminase 2) decreased markedly 1 week after trea

64 rypsin (modified or unmodified by the enzyme transglutaminase 2) or the peptic-tryptic digest of glia

65 uced mesangial surface expression of TGase2 (transglutaminase 2), which in turn up-regulated TfR1 exp

66 infiltrating T cells, HLA-DR expression, and transglutaminase 2-targeted IgA deposits.

67 002-1E03 from a digest of gliadin treated by transglutaminase 2.

68 transition gene signature characteristic of transglutaminase 2/transforming growth factor-beta activ

69 tein keratin 8 (K8) and its cross-linking by transglutaminase-2 (TG2) are essential for MDB formation

70 Transglutaminase-2 (TG2) is a new anti-fibrotic target f

71 Transglutaminase-2 (TGM-2) has been implicated in severa

72 ell culture models, and this requires active transglutaminase-2 (TGM2).

73 Pep1 did not inhibit transglutaminase-2 activity, and incorporation of biotin

74 lts (for levels of antibodies against tissue transglutaminase-2 and deamidated gliadin peptide), symp

75 nct from the two CD hallmark antigens tissue transglutaminase-2 and deamidated gliadin, exhibiting 71

76 uired for MDB formation, including increased transglutaminase-2 and keratin overexpression, with a gr

77 Additionally, transglutaminase-2 expression was determined after YAP s

78 en fibrillar collagens, introduced by tissue transglutaminase-2, are necessary for the self-assembly

79 /K18) and ubiquitin that are cross-linked by transglutaminase-2.

80 tabilized by the matrix cross-linking enzyme transglutaminase-2.

81 biochemically by blotting with antibodies to transglutaminase-2/p62 proteins and to K8/K18/ubiquitin

82 Recently, a transglutaminase 3 knockout (TGM3/KO) mouse was generate

83 PADI3 (peptidylarginine deiminase 3), TGM3 (transglutaminase 3), and TCHH (trichohyalin) in a total

84 Sardy et al. reported that transglutaminase-3 (TG3) colocalizes with the IgA.

85 ished from acral PSS, caused by mutations in transglutaminase 5.

86 ibodies toward the newly identified neuronal transglutaminase 6 (TG6).

87 mutations in the TGM6 gene, which codes for transglutaminase 6 (TG6).

88 Antibodies against transglutaminase 6 can serve as a marker in addition to

89 inked with gastrointestinal disease, an anti-transglutaminase 6 IgG and IgA response is prevalent in

90 howed cerebellar IgA deposits that contained transglutaminase 6.

91 skin barrier by regulating the activities of transglutaminases, a family of Ca(2+)-dependent crosslin

92 epidermal desmosomes, upregulated epidermal transglutaminase activity and heightened resistance to S

93 -out mice, led to a rapid rise in intestinal transglutaminase activity in a manner that could be inhi

94 Inhibiting polyamine synthesis or transglutaminase activity significantly decreases microt

95 f radioactive substrates and allows relative transglutaminase activity to be measured simultaneously

96 Transglutaminase activity was increased in fibrosis, and

97 Anbu's function appears to be related to transglutaminase activity, not the general stress respon

98 f a substrate, called Plugin, by the seminal transglutaminase AgTG3.

99 L.) (flour or seed) and cold gelling agents (transglutaminase, alginate or gelatin).

100 B (SS-A and SS-B, respectively), antitissue transglutaminase and antiendomysial antibodies, and para

101 LA-DQ8 genes) and autoantibodies (antitissue transglutaminase and antiendomysial).

102 erologic tests for antibodies against tissue transglutaminase and deamidated gliadin peptide, greatly

103 f the profibrotic cleavage substrates tissue transglutaminase and endoglin accompanied MMP-14 suppres

104 re tested for CD based on analysis of tissue transglutaminase and endomysial antibodies.

105 esults of serum tests for IgA against tissue transglutaminase and endomysium or on both a health care

106 as well as for IgA antibodies against tissue transglutaminase and endomysium.

107 lpha, histone deacetylase, overexpression of transglutaminase and iNOS) and cross talk between NF-kap

108 on-(gamma-glutamyl) lysine cross-linkages by transglutaminase and that enzymatic cross-linking increa

109 Sera were tested for tissue transglutaminase and, if abnormal, for endomysial antibo

110 our antibody tests including IgA anti-tissue transglutaminase and/or IgA anti- endomysium permitted d

111 ults (IgA/IgG gliadin, endomysium, or tissue transglutaminase) and compared them with 213,208 age- an

112 bodies against gliadin, 1.22% against tissue transglutaminase, and 0.61% against endomysium (P>.05 vs

113 idase, gastric parietal cells (PCAs), tissue transglutaminase, and 21-hydroxylase was tested using a

114 g of VEGF165, catalysed by the enzyme tissue transglutaminase, and associates with MVs through its in

115 Concentrations of IgA, IgA antitissue transglutaminase, and endomysial antibodies were measure

116 anti-dpgli, IgG anti-dpgli, IgA anti- tissue transglutaminase, and IgA anti-endomysium) yielded posit

117 +SPI, WPI+CN and SPI+CN, were produced using transglutaminase, and their in vitro IgE reactivity and

118 high levels of autoantibodies against tissue transglutaminase (anti-TG2) are strongly indicative of a

119 years and analyzed for antibodies to tissue transglutaminase (anti-TG2A) using a radiobinding assay,

120 Serum samples were analyzed for anti-tissue transglutaminase (anti-tTG) concentrations at age 6 y.

121 between levels of antibodies against tissue transglutaminase (anti-tTG, a marker of celiac disease)

122 Pooled estimates for IgA antitissue transglutaminase antibodies (7 studies) were 0.89 (95% C

123 , antinuclear antibodies (ANAs), anti-tissue transglutaminase antibodies (AGTAs), and anti-thyroid pe

124 d optimized for determination of anti-tissue transglutaminase antibodies (anti-tTG) in human serum.

125 gneto immunosensor for the detection of anti-transglutaminase antibodies (ATG2) in celiac disease was

126 other unselected populations, IgA antitissue transglutaminase antibodies and IgA antiendomysial antib

127 ific screening serologic tests (anti- tissue transglutaminase antibodies IgA [anti-TTG] and ednomysea

128 Positivity for IgA tissue transglutaminase antibodies was detected in 97%.

129 itivities of Simtomax, endomysial and tissue-transglutaminase antibodies were compared in 133 prospec

130 It captures the target tissue transglutaminase antibody (anti-tTG), and finally allows

131 ning for celiac disease using the IgA tissue transglutaminase antibody (IgA tTG) and, if positive, te

132 ounger (P = .03), had lower titers of tissue transglutaminase antibody (P = .001), and less frequentl

133 The majority (98%) of FPs requested anti-transglutaminase antibody (tTG-Ab) titres for CD diagnos

134 ions revealed a slightly elevated IgA tissue transglutaminase antibody level in the setting of serum

135 We find that these two transglutaminases are activated by local acidification a

136 Transglutaminases are also involved in fibrin(ogen) rete

137 independent of coagulation factor 13 (FXIII) transglutaminase, as ANIT challenge in FXIII-deficient m

138 for up to 20 years for development of tissue transglutaminase autoantibodies (tTGA).

139 from birth were screened annually for tissue transglutaminase autoantibodies (tTGAs).

140 efined as being positive for islet or tissue transglutaminase autoantibodies at 2 consecutive clinic

141 Y study who were tested for islet and tissue transglutaminase autoantibodies, respectively.

142 bodies (HR, 0.99; 95% CI, 0.95-1.03), or the transglutaminase autoantibody (HR, 1.00; 95% CI, 0.98-1.

143 sults suggest that a risk of islet or tissue transglutaminase autoimmunity need not influence the rec

144 Together, these results emphasize how transglutaminases can coordinate the activities of other

145 Transglutaminases catalyze the covalent linkage of prote

146 cation of a disulfide bond and pH-controlled transglutaminase-catalyzed modification of lysine, respe

147 Endogenous brain transglutaminase-catalyzed polyaminated tubulins have th

148 Together, these findings suggest that transglutaminase-catalyzed polyamination of tubulins sta

149 Transglutaminase-catalyzed posttranslational incorporati

150 Moreover, we observed that the transglutaminase coagulation factor XIIIA (FXIIIA) was o

151 The cornified envelope is assembled from transglutaminase cross-linked proteins and lipids in the

152 uggest that the proposed evolution of animal transglutaminase cross-linking activity from ancestral b

153 Dynamic rheometry indicated that transglutaminase cross-linking and niosomes charging of

154 Transglutaminase cross-linking prior to emulsification s

155 techniques for monitoring the intramolecular transglutaminase cross-links of pea proteins, based on p

156 f 5.7 mum were fabricated and charged into a transglutaminase-cross-linked whey protein solution that

157 Transglutaminase crosslinked faba bean protein extensive

158 Transglutaminase-crosslinked type II collagen showed inc

159 preparations, perhaps mediated by increased transglutaminase crosslinking, may contribute to the dec

160 atelet aggregation, by receptor-independent, transglutaminase-dependent covalent linkage to cellular

161 elets stabilized FXIII-depleted thrombi in a transglutaminase-dependent manner.

162 ted by a mutant containing an insertion in a transglutaminase domain protein (GSU3361), which suddenl

163 Cyk3p possesses a transglutaminase domain that is essential for function,

164 Cross-linking the dialyzed emulsion with transglutaminase eliminated the detection of free lactas

165 beads (MBs) as a solid support in which the transglutaminase enzyme (TG2) is covalently immobilized

166 sed on the covalent immobilization of tissue transglutaminase enzyme in its open conformation (open-t

167 cornified envelope precursors and the tissue transglutaminase enzyme that cross-links them.

168 ion tomography/magnetic resonance imaging of transglutaminase factor XIII (FXIII) and myeloperoxidase

169 Coagulation transglutaminase factor XIII (FXIII) exists in circulati

170 nt study, we determined that activity of the transglutaminase factor XIII (FXIII) is critical for rbc

171 d with and without ligation by the activated transglutaminase factor XIII (FXIIIa).

172 r matrix turnover, such as production of the transglutaminase factor XIII A subunit.

173 Here, we demonstrate that the coagulation transglutaminase, factor XIII (fXIII), drives arthritis

174 und to fibrin polymers to produce the active transglutaminase, factor XIIIa.

175 TG2 appears to be the member of the transglutaminase family primarily contributing to this p

176 ogical importance of inactive members of the transglutaminase family, which are found throughout euka

177 Crosslinking with transglutaminase followed by freeze drying resulted in a

178 factor of 10 below the recommended amount of transglutaminase for raw as well as heated restructured

179 Transglutaminase from Streptomyces mobaraensis (MTG) is

180 faba bean protein isolates were treated with transglutaminase from Streptomyces mobaraensis and tyros

181 ide bond, thrombin assists in activating the transglutaminase FXIIIa that incorporates cross-links in

182 Using a gelatin microbial transglutaminase (gelatin-mTG) cell culture platform tun

183 ity directed toward different members of the transglutaminase gene family could offer an explanation

184 Abeta(1-42) with three cross-linking agents: transglutaminase, glutaraldehyde, and Cu(II) with peroxi

185 hypothesised that the dosing of xylanase and transglutaminase has a positive impact on rye dough and

186 l cycle regulated and we unearth a bacterial transglutaminase homolog, HvyA, as restriction factor th

187 potency, and their selectivity toward other transglutaminase homologues is largely unknown.

188 Human tissue transglutaminase (hTG2) is a multifunctional enzyme.

189 ts with concentrations of IgA against tissue transglutaminase (IgA-TTG) >10-fold the upper limit of n

190 However, using the crosslinking enzyme Transglutaminase II to alter microstructure independentl

191 s have circulating antibodies against tissue transglutaminase; in children, European guidelines allow

192 assay using an assay for IgA against tissue transglutaminase; in subjects with positive test results

193 Transglutaminase increased the absolute zeta-potential v

194 All transglutaminases increased post-SNx peaking at loss of

195 vitro antioxidative hydrolysate, followed by transglutaminase-induced cross-linking and microemulsifi

196 Furthermore, treatment of mice with the pan-transglutaminase inhibitor cystamine resulted in signifi

197 I deficiency had normal retention, and a pan-transglutaminase inhibitor T101 had only a modest inhibi

198 or, inhibition of intestinal TG2 activity by transglutaminase inhibitors, inhibition of gluten peptid

199 here is no confirmation that TG2 is the only transglutaminase involved, neither there are strategies

200 We have identified a novel neuronal transglutaminase isozyme and investigated whether this e

201 In this study, we have profiled transglutaminase isozymes in the rat subtotal nephrectom

202 ody populations react with the two different transglutaminase isozymes.

203 ins are previously undetected members of the transglutaminase-like cysteine protease superfamily, and

204 e diphosphate via its catalytically inactive transglutaminase-like domain.

205 We have identified a transglutaminase-like protein (Cyk3p) that functions in

206 spholipid-binding protein annexin-1 and then transglutaminase-mediated crosslinking of annexin-1 thro

207 observed ratio allowed the identification of transglutaminase-mediated gamma-glutamyl isomers as inte

208 rgenic protein products could be produced by transglutaminase-mediated heterologous polymerization of

209 The transglutaminase-mediated, covalent cross-linking of pro

210 Impacts of microbial transglutaminase (MTGase) (0-0.6 units/g sample) on gel

211 Key modifying roles of microbial transglutaminase (MTGase) in the development of innovati

212 Microbial transglutaminase (MTGase) is an enzyme of the class of t

213 ia sp. C1112 for the production of microbial transglutaminase (MTGase, EC 2.3.2.13).

214 Microbial transglutaminases (MTGs) catalyze the formation of Gln-L

215 general role for the catalytically inactive transglutaminases of fungi and animals, some of which ha

216 ) during storage in emulsion gels containing transglutaminase or alginate.

217 has been indicated previously that during a transglutaminase reaction activated factor XIII (FXIIIa)

218 ng only the final deacylation portion of the transglutaminase reaction.

219 Analysis of heparin binding of the main transglutaminases revealed that although the interaction

220 The binding of SLPI with Cementoin to transglutaminase seems to be an effective strategy to tr

221 ic ATPase, anti-thyroid peroxidase, and anti-transglutaminase seropositivity, and these autoantibodie

222 use a proteomic approach in combination with transglutaminase-specific labeling to identify FXIIIa pl

223 lar methodologies to demonstrate that tissue transglutaminase (TG) activates downstream NF-kappaB sig

224 Aberrant transglutaminase (TG) activity has been implicated in th

225 The popularity of transglutaminase (TG) by the food industry and the varia

226 -MS/MS-method for the detection of microbial transglutaminase (TG) from Streptomyces mobaraensis in d

227 for the simultaneous detection of microbial transglutaminase (TG) from Streptomyces mobaraensis, and

228 A2), cyclooxygenase 2 (COX-2), thrombin, and transglutaminase (TG).

229 Transglutaminase (TG)2 release modulates tissue repair,

230 Type I transglutaminase (TG1) is an enzyme that is responsible

231 Expression of the transglutaminase TG2 has been linked to constitutive act

232 The ubiquitous cross-linking enzyme tissue transglutaminase (TG2) has been implicated in irreversib

233 Gluten lacks such peptides, but tissue transglutaminase (TG2) introduces negatively charged res

234 Tissue transglutaminase (TG2) is a multifunctional Ca(2+)-activ

235 Tissue transglutaminase (TG2) is a multifunctional enzyme invol

236 Tissue type transglutaminase (TG2) is a unique multifunctional prote

237 Type 2 transglutaminase (TG2) is an important cancer stem cell

238 ssion of the pro-inflammatory protein tissue transglutaminase (TG2) reprograms the transcription regu

239 ity of the matrix crosslinking enzyme tissue transglutaminase (TG2) via S-nitrosylation in young vess

240 inhibits melanoma growth and binds to tissue transglutaminase (TG2), a major crosslinking enzyme in E

241 Tissue transglutaminase (TG2), an enzyme involved in cell proli

242 Tissue transglutaminase (TG2), an enzyme that catalyzes Ca(2+)-

243 performance of antibody tests against tissue transglutaminase (TG2), deamidated gliadin peptide (DGP)

244 ated with the sulfhydryl-rich protein tissue transglutaminase (TG2), thereby endowing the membrane su

245 culating autoantibodies to the enzyme tissue transglutaminase (TG2).

246 ignificant increases in the levels of type-2 transglutaminase (TG2, which is implicated in transamida

247 nd levels of immunoglobulin A against tissue-transglutaminase (TGA-IgA) 10-fold or more the upper lim

248 nomer sample that had been cross-linked with transglutaminase (TGase) and digested with pepsin.

249 Tissue transglutaminase (TGase) has been implicated in neurodeg

250 Extracellular transglutaminase (TGase) has been shown previously to pl

251 Increasing evidence suggests that transglutaminase (TGase) plays a critical role in the pa

252 a milk base in the absence or presence of a transglutaminase (TGase) protein cross-linking step on t

253 system for investigating the role of tissue transglutaminase (TGase), a protein that has been linked

254 atin hydrolysates and glucosamine (GlcN) via transglutaminase (TGase), as well as glycation between f

255 designed to detect the catalytic activity of transglutaminase (TGase), which creates a covalent bond

256 ll epitope through treatment with the enzyme transglutaminase (TGase).

257 /50 degrees C) in the presence or absence of transglutaminase (TGase).

258 Transglutaminases (TGases) are ubiquitous enzymes that t

259 evident 2 d after birth, followed by reduced transglutaminase (TGM) activity, transepidermal water lo

260 Transglutaminase (TGM)-2 has been shown to contribute to

261 in, angiopoietin-like factor (ANGPTL)-7, and transglutaminase (TGM)-2.

262 Tissue transglutaminase (TGM2) belongs to a family of calcium-d

263 In vitro tissue transglutaminase (Tgm2) cross-linking was monitored and

264 By gene expression screening, tissue transglutaminase (TGM2) was identified as one of the gen

265 t extents on stiffer substrates; TAZ, tissue transglutaminase (TGM2), and soluble frizzled-related pr

266 ins (Krts), keratin-associated proteins, and transglutaminases (Tgms) and their substrates were signi

267 Transglutaminases (TGs) are known to exhibit remarkable

268 ormed by extensive cross-linking activity of transglutaminases (TGs) during terminal epidermal differ

269 Coagulation factor XIIIa (FXIIIa) is a transglutaminase that covalently cross-links fibrin and

270 Plasma coagulation factor XIII (FXIII) is a transglutaminase that promotes cross-linking of the extr

271 Thus, unlike the active transglutaminases that activate RhoA, the multidomain pr

272 edicted after GC-1g, and the mean IgA-tissue transglutaminase titers declined, contrary to the predic

273 ne, TIG3 interacts with and activates type I transglutaminase to enhance keratinocyte terminal differ

274 lyacrylamide gel electrophoresis profiles of transglutaminase-treated low concentration (0.01% w/w) p

275 milar with known CD-specific antigens tissue transglutaminase (tTG) and deamidated gliadin, and the c

276 Immunoglobulin A (IgA) antibodies to tissue transglutaminase (tTG) are the serologic test of choice

277 Tissue transglutaminase (tTG) assay was available, but one-thir

278 Tissue transglutaminase (tTG) functions as a GTPase and an acyl

279 tracerebral hemorrhage (ICH) and tissue-type transglutaminase (tTG) has a role in neurodegenerative d

280 often relies on the presence of anti-tissue transglutaminase (tTG) IgA autoantibodies.

281 revious work showed that cell surface tissue transglutaminase (tTG) induces clustering of integrins a

282 Tissue transglutaminase (tTG) is an acyltransferase/GTP-binding

283 ecognition scaffold is the following: tissue transglutaminase (tTG) is immobilized as a capturing age

284 Here, we show that tissue transglutaminase (tTG) is one such protein.

285 nal biopsy, and serologic analysis of tissue transglutaminase (tTg) levels; endomysial antibody (EMA)

286 glycated haemoglobin (HbA1c) and IgA tissue transglutaminase (tTg) were collected prior to, and foll

287 al antibodies (EMA) and antibodies to tissue transglutaminase (tTG) were developed to screen for celi

288 to different antigens, in particular tissue transglutaminase (tTG), gliadin (Glia), and endomysium.

289 ind the protein cross-linking enzyme, tissue transglutaminase (tTG), in cancer cell migration.

290 Tissue transglutaminase (tTG), involved in PTM of gluten antige

291 disease have serum antibodies against tissue transglutaminase (tTG).

292 r of the protein cross-linking enzyme tissue transglutaminase (tTG).

293 Transglutaminase type 2 (TG2) is an extracellular matrix

294 The positive predictive value of tissue transglutaminase type 2 (tTG) antibodies performed with

295 Rates of elevated levels of anti-transglutaminase type 2 and antigliadin antibodies were

296 Anti-transglutaminase type 2 and antigliadin antibodies were

297 re than 12000 participants) found IgA tissue transglutaminase was associated with high accuracy (sens

298 Using recombinant human transglutaminases, we developed enzyme-linked immunosorb

299 HLA DQ2/DQ8 and serum antibodies against transglutaminase were analysed.

300 Coagulation factor XIII (FXIII) is a transglutaminase with a well defined role in the final s

301 protein was crosslinked to some extent with transglutaminase with higher dosages (100 and 1000nkat/g