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1 in the dorsal striatum die within 24 h after transient cerebral ischemia.
2 be a substrate for functional recovery after transient cerebral ischemia.
3 e markedly modified in the hippocampus after transient cerebral ischemia.
4 rate) after traumatic brain injury (TBI) and transient cerebral ischemia.
5 s for preventing delayed neuronal loss after transient cerebral ischemia.
6 halographic (EEG) silence in anticipation of transient cerebral ischemia.
7 intraperitoneally), given within 2 hours of transient cerebral ischemia, acutely increased endotheli
8 eatures of neuronal apoptosis resulting from transient cerebral ischemia and related brain insults fo
9 ypertension, cardiovascular disease, stroke, transient cerebral ischemia, and end-stage renal disease
10 ay contribute to the brain damage induced by transient cerebral ischemia, and may be involved in the
11 ns could attenuate BBB disruption induced by transient cerebral ischemia, by inhibition of MMP2 and M
15 In the present studies, we demonstrate that transient cerebral ischemia in female rats caused a 30%
17 ration induces acute preconditioning against transient cerebral ischemia in rats, the potential for d
18 tery (MCA) occlusion, and observed that this transient cerebral ischemia induced substantial apoptosi
19 his study, we report for the first time that transient cerebral ischemia induces aberrant mitotic pro
21 atio [OR], 7.73; CI, 2.78-21.52; P < 0.001), transient cerebral ischemia (OR, 7.67; CI, 5.31-11.07; P
25 utamate concentrations in an animal model of transient cerebral ischemia using in vivo cerebral micro
28 glycoprotein is differentially influenced by transient cerebral ischemia when carried out under normo
29 n tissue NOx levels immediately after 10 min transient cerebral ischemia, which gradually returned to
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