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1 ation of acinar cells to the nestin-positive transitional cell.
2 superficial endocardial layer of atrial and transitional cells.
3 of large-sized cells somewhat similar to the transitional cells.
4 band of well-polarized but poorly excitable transitional cells.
5 artment, enriched for CXCL13-responsive late transitional cells.
6 is higher than that of mesothelial cells and transitional cells.
7 iated with the appearance of nestin-positive transitional cells.
8 native pathway, the survival of neonatal B-1 transitional cells and their maturation into B-1 B cells
9 lar B cells in splenic development, immature transitional cells are an essential component for unders
12 Fifteen patients with recurrent superficial transitional cell bladder carcinoma who experienced prio
13 ammary and prostate cancers, the majority of transitional cell bladder tumors showed Id1 protein expr
14 exhibits significant activity in metastatic transitional cell cancer with minimal toxicity, but surv
16 ed EN, (ii) these adducts can be detected in transitional cell cancers, and (iii) A:T --> T:A transve
18 y culture (P0) and after passage in vitro of transitional cell carcinoma (TCC) biopsies that represen
19 ghly tumorigenic and highly metastatic human transitional cell carcinoma (TCC) cell line 253J B-V ove
20 ltetrazolium bromide (MTT) assays in bladder transitional cell carcinoma (TCC) cell lines RT4 and SW7
21 and protein are observed among several human transitional cell carcinoma (TCC) cell lines, suggesting
22 GF) accumulates in the nucleus in aggressive transitional cell carcinoma (TCC) cells and this histolo
25 IFN-alpha therapy for clinical treatment of transitional cell carcinoma (TCC) has prompted us to inv
29 of noninvasive methods for the diagnosis of transitional cell carcinoma (TCC) of the bladder remains
31 invasive method for monitoring patients with transitional cell carcinoma (TCC) of the bladder would b
32 q is the most frequent genetic alteration in transitional cell carcinoma (TCC) of the bladder, implic
37 mine the relative levels of MSI and EMAST in transitional cell carcinoma (TCC) of the upper and lower
38 ations are known to be associated with human transitional cell carcinoma (TCC) of the urinary bladder
39 eleted in many human malignancies, including transitional cell carcinoma (TCC) of the urinary bladder
41 orifice positive for a high-grade papillary transitional cell carcinoma (TCC) with muscularis propri
42 on forms of bladder tumors: 69 of 97 (71.1%) transitional cell carcinoma (TCC), 38 of 53 (71.6%) squa
44 remains the standard of care for metastatic transitional cell carcinoma (TCC), but its limitations i
45 predominant component in the development of transitional cell carcinoma (TCC), but the particular pa
51 invasive or high-grade recurrent superficial transitional cell carcinoma (TCC; lymph node-negative, 1
52 In the SCID-beige mice, T24 human bladder transitional cell carcinoma also was used as the tumor i
53 he current fund of knowledge about prostatic transitional cell carcinoma and the implications for dia
55 n of p27(Kip1) and cyclin E in human bladder transitional cell carcinoma cells correlates with advanc
56 chemotherapy in advanced upper urinary tract transitional cell carcinoma continue to evolve and remai
57 F-7, pancreatic carcinoma (CRL 1420, bladder transitional cell carcinoma EJ, and melanoma LOX) but no
59 onsidered related to the study drug (bladder transitional cell carcinoma in the ozanezumab group and
60 ase 2 trial, we enrolled patients with T2-4a transitional cell carcinoma of the bladder at 24 medical
62 Rb) expression was evaluated in 185 cases of transitional cell carcinoma of the bladder from patients
65 erin and interferon are clinically active in transitional cell carcinoma of the bladder, but their me
75 rectomy has been the treatment of choice for transitional cell carcinoma of the upper urinary tract.
76 lear cell) renal carcinoma (11 of 11 cases), transitional cell carcinoma of the urinary bladder (6 of
77 ssue sections of canine spontaneous invasive transitional cell carcinoma of the urinary bladder (a mo
78 s accrued from patients with muscle-invasive transitional cell carcinoma of the urinary bladder (TCC-
79 COX-1 and COX-2 expression in human invasive transitional cell carcinoma of the urinary bladder by im
80 of chromosome 3 was conducted in 72 cases of transitional cell carcinoma of the urinary bladder using
82 oving the precision of the estimated risk of transitional cell carcinoma of the urinary tract associa
83 n of the bladder tumor and biopsy identified transitional cell carcinoma or urothelial carcinoma inva
89 % of the K5.COX-2 transgenic mice; no TCH or transitional cell carcinoma was observed in wild-type bl
92 ickening in four patients (three of whom had transitional cell carcinoma), a renal abscess, a coloves
93 ses, 24 were renal cell carcinoma, one was a transitional cell carcinoma, and one was an angiomyolipo
95 sing an early-stage renal cell carcinoma and transitional cell carcinoma, chronic lymphocytic leukemi
96 ancer, testis cancer and upper urinary tract transitional cell carcinoma, no recently published study
97 sic fibroblast growth factor (bFGF) in human transitional cell carcinoma, reduce its angiogenesis, an
98 identified as a region of non-random LOH in transitional cell carcinoma, suggesting the presence of
100 st described in 1991 for upper urinary tract transitional cell carcinoma, with long-term data now eme
106 nd carboplatin for the treatment of advanced transitional-cell carcinoma (TCC) of the urothelium has
107 in previously treated patients with advanced transitional-cell carcinoma (TCC) of the urothelium.
108 previously untreated patients with advanced transitional-cell carcinoma (TCC) to assess its efficacy
109 eported survival of patients with metastatic transitional-cell carcinoma (TCC) treated with systemic
110 icacy and toxicity in patients with advanced transitional-cell carcinoma (TCC) who had failed to resp
111 ymphadenectomy, with the intent to cure, for transitional-cell carcinoma of the bladder between July
112 Patients with measurable, locally advanced transitional-cell carcinoma of the bladder who were not
113 STT1, GSTM3, and GSTP1 in 1150 patients with transitional-cell carcinoma of the urinary bladder and 1
114 al urotoxicity, including the development of transitional-cell carcinoma of the urinary bladder.
116 ond-line treatment of patients with advanced transitional-cell carcinoma of the urothelial tract.
121 y included 59 well-characterized superficial transitional cell carcinomas (Ta, n = 28; T1, n = 31) fo
122 pure squamous cell carcinomas (SCC), and in transitional cell carcinomas (TCC) of the bladder with S
123 five sarcomatoid RCC, two oncocytomas, three transitional cell carcinomas (TCC) of the renal pelvis a
125 elial carcinomas, including 28 pTa low-grade transitional cell carcinomas (TCC), 31 pTa high-grade TC
126 b and p53 pathway alterations in 12 invasive transitional cell carcinomas (TCCs) and addressed the fu
128 and nature of FGFR3 mutations in a panel of transitional cell carcinomas and cell lines and studied
129 PDE5 was overexpressed in human squamous and transitional cell carcinomas compared with normal urothe
130 ylation of four CpG islands in human urinary transitional cell carcinomas of different stages and gra
131 ed that around one-third (33/101) of primary transitional cell carcinomas of the bladder overexpress
133 ation of these genes by analyzing 69 primary transitional cell carcinomas of the bladder with a panel
134 but was detected in 25 of 29 (86%) invasive transitional cell carcinomas of the urinary bladder and
135 ity, we have studied a cohort of superficial transitional cell carcinomas of the urinary bladder by i
136 ed the pattern of allelic imbalance in human transitional cell carcinomas of the urinary bladder incl
137 NU plus H202 or H202 alone formed high-grade transitional cell carcinomas when injected into nude mic
143 as defined by expression of alpha-SM actin) "transitional" cells, coexpressing both endothelial and S
147 d thymocytes paradoxically appear as CD4+8lo transitional cells during their differentiation into CD8
148 specimens, as well as 11 cases of low-grade transitional cell dysplasia, 21 cases of carcinoma in si
150 mRNA was observed in proliferating oval and transitional cells, forming duct-like structures of cyto
153 a keratin 5 promoter, is sufficient to cause transitional cell hyperplasia (TCH) in 17% and 75% of th
155 een proposed that these oval cells represent transitional cells in a nonhepatocytic liver facultative
156 the risk allele, due to an expansion of late transitional cells in a stage targeted by selection mech
157 markers, suggest that CK19+/NCAM+ cells are transitional cells in the biliary lineage and that rare
159 onstrate that B-1 B cells are generated from transitional cell intermediates that emerge in a distinc
160 Akap12-/- prostate lobes suggests that these transitional cells may be the source of the lymph node m
161 Severe B-cell deficiency affects mature and transitional cells, mimicking the action of rituximab.
162 or, proHB-EGF, localizes to the cytoplasm of transitional cells of the human bladder urothelium and t
163 hway and allowing circus movement within the transitional cells of the posterior AV nodal connection.
164 w that, in contrast to the dependence of B-2 transitional cells on the alternative pathway, the survi
167 gest that hFLMPCs are mesenchymal-epithelial transitional cells, probably derived from mesendoderm.
170 ed, among the oval cells, a subpopulation of transitional cells showing features of maturing hepatocy
172 e bacterial QIRs were harbored in underlying transitional cells, stimulation of epithelial turnover t
173 isthmal) zone of the gastric unit and become transitional cells (TCs) with molecular and ultrastructu
174 auses loss of parietal cells, development of transitional cells that express markers of mucus neck an
176 s of carcinoma in situ (CIS), 38 superficial transitional cell tumors (STCC, stages T(a)-T(1)), 65 mu
177 NA expression in two of five muscle-invasive transitional cell tumors when compared with normal sampl
178 that SHPCs may represent an intermediate or transitional cell type between oval cells and mature hep
181 renal failure and a strong association with transitional cell (urothelial) carcinoma of the upper ur
182 lear outer sulcus cells (OSC) and vestibular transitional cells (VTC) are part of the parasensory epi
183 acterized by a preponderance of immature and transitional cells, whose persistence was associated wit
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