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1 inoma, adult polycystic kidney disease or in transitional cell carcinoma.
2 atment of organ-confined upper urinary tract transitional cell carcinoma.
3 line therapy and emerging drugs for advanced transitional-cell carcinoma.
4 tment has an antitumor effect in superficial transitional-cell carcinoma.
5 in patients with unresectable or metastatic transitional-cell carcinoma.
6 microsatellite loci in 20 grade III invasive transitional cell carcinomas.
7 adder proliferative lesions, including three transitional cell carcinomas.
8 ssues and normal mammary tissues and bladder transitional cell carcinomas.
9 g water and the incidence of urinary bladder transitional cell carcinomas.
10 ickening in four patients (three of whom had transitional cell carcinoma), a renal abscess, a coloves
11 In the SCID-beige mice, T24 human bladder transitional cell carcinoma also was used as the tumor i
12 he current fund of knowledge about prostatic transitional cell carcinoma and the implications for dia
13 and nature of FGFR3 mutations in a panel of transitional cell carcinomas and cell lines and studied
14 ses, 24 were renal cell carcinoma, one was a transitional cell carcinoma, and one was an angiomyolipo
17 n of p27(Kip1) and cyclin E in human bladder transitional cell carcinoma cells correlates with advanc
18 sing an early-stage renal cell carcinoma and transitional cell carcinoma, chronic lymphocytic leukemi
19 PDE5 was overexpressed in human squamous and transitional cell carcinomas compared with normal urothe
20 chemotherapy in advanced upper urinary tract transitional cell carcinoma continue to evolve and remai
21 F-7, pancreatic carcinoma (CRL 1420, bladder transitional cell carcinoma EJ, and melanoma LOX) but no
23 onsidered related to the study drug (bladder transitional cell carcinoma in the ozanezumab group and
25 ancer, testis cancer and upper urinary tract transitional cell carcinoma, no recently published study
26 ase 2 trial, we enrolled patients with T2-4a transitional cell carcinoma of the bladder at 24 medical
28 Rb) expression was evaluated in 185 cases of transitional cell carcinoma of the bladder from patients
31 erin and interferon are clinically active in transitional cell carcinoma of the bladder, but their me
41 rectomy has been the treatment of choice for transitional cell carcinoma of the upper urinary tract.
42 lear cell) renal carcinoma (11 of 11 cases), transitional cell carcinoma of the urinary bladder (6 of
43 ssue sections of canine spontaneous invasive transitional cell carcinoma of the urinary bladder (a mo
44 s accrued from patients with muscle-invasive transitional cell carcinoma of the urinary bladder (TCC-
45 COX-1 and COX-2 expression in human invasive transitional cell carcinoma of the urinary bladder by im
46 of chromosome 3 was conducted in 72 cases of transitional cell carcinoma of the urinary bladder using
48 oving the precision of the estimated risk of transitional cell carcinoma of the urinary tract associa
49 ylation of four CpG islands in human urinary transitional cell carcinomas of different stages and gra
50 ed that around one-third (33/101) of primary transitional cell carcinomas of the bladder overexpress
52 ation of these genes by analyzing 69 primary transitional cell carcinomas of the bladder with a panel
53 but was detected in 25 of 29 (86%) invasive transitional cell carcinomas of the urinary bladder and
54 ity, we have studied a cohort of superficial transitional cell carcinomas of the urinary bladder by i
55 ed the pattern of allelic imbalance in human transitional cell carcinomas of the urinary bladder incl
56 ymphadenectomy, with the intent to cure, for transitional-cell carcinoma of the bladder between July
57 Patients with measurable, locally advanced transitional-cell carcinoma of the bladder who were not
58 STT1, GSTM3, and GSTP1 in 1150 patients with transitional-cell carcinoma of the urinary bladder and 1
61 ond-line treatment of patients with advanced transitional-cell carcinoma of the urothelial tract.
63 n of the bladder tumor and biopsy identified transitional cell carcinoma or urothelial carcinoma inva
64 sic fibroblast growth factor (bFGF) in human transitional cell carcinoma, reduce its angiogenesis, an
67 identified as a region of non-random LOH in transitional cell carcinoma, suggesting the presence of
68 y included 59 well-characterized superficial transitional cell carcinomas (Ta, n = 28; T1, n = 31) fo
71 y culture (P0) and after passage in vitro of transitional cell carcinoma (TCC) biopsies that represen
72 ghly tumorigenic and highly metastatic human transitional cell carcinoma (TCC) cell line 253J B-V ove
73 ltetrazolium bromide (MTT) assays in bladder transitional cell carcinoma (TCC) cell lines RT4 and SW7
74 and protein are observed among several human transitional cell carcinoma (TCC) cell lines, suggesting
75 GF) accumulates in the nucleus in aggressive transitional cell carcinoma (TCC) cells and this histolo
78 IFN-alpha therapy for clinical treatment of transitional cell carcinoma (TCC) has prompted us to inv
82 of noninvasive methods for the diagnosis of transitional cell carcinoma (TCC) of the bladder remains
84 invasive method for monitoring patients with transitional cell carcinoma (TCC) of the bladder would b
85 q is the most frequent genetic alteration in transitional cell carcinoma (TCC) of the bladder, implic
90 mine the relative levels of MSI and EMAST in transitional cell carcinoma (TCC) of the upper and lower
91 eleted in many human malignancies, including transitional cell carcinoma (TCC) of the urinary bladder
92 ations are known to be associated with human transitional cell carcinoma (TCC) of the urinary bladder
94 orifice positive for a high-grade papillary transitional cell carcinoma (TCC) with muscularis propri
95 on forms of bladder tumors: 69 of 97 (71.1%) transitional cell carcinoma (TCC), 38 of 53 (71.6%) squa
97 remains the standard of care for metastatic transitional cell carcinoma (TCC), but its limitations i
98 predominant component in the development of transitional cell carcinoma (TCC), but the particular pa
104 invasive or high-grade recurrent superficial transitional cell carcinoma (TCC; lymph node-negative, 1
105 pure squamous cell carcinomas (SCC), and in transitional cell carcinomas (TCC) of the bladder with S
106 five sarcomatoid RCC, two oncocytomas, three transitional cell carcinomas (TCC) of the renal pelvis a
108 elial carcinomas, including 28 pTa low-grade transitional cell carcinomas (TCC), 31 pTa high-grade TC
111 nd carboplatin for the treatment of advanced transitional-cell carcinoma (TCC) of the urothelium has
112 in previously treated patients with advanced transitional-cell carcinoma (TCC) of the urothelium.
113 previously untreated patients with advanced transitional-cell carcinoma (TCC) to assess its efficacy
114 eported survival of patients with metastatic transitional-cell carcinoma (TCC) treated with systemic
115 icacy and toxicity in patients with advanced transitional-cell carcinoma (TCC) who had failed to resp
116 b and p53 pathway alterations in 12 invasive transitional cell carcinomas (TCCs) and addressed the fu
122 % of the K5.COX-2 transgenic mice; no TCH or transitional cell carcinoma was observed in wild-type bl
124 NU plus H202 or H202 alone formed high-grade transitional cell carcinomas when injected into nude mic
125 st described in 1991 for upper urinary tract transitional cell carcinoma, with long-term data now eme
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