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1 suggest the "out-of-frame pairing" model of translational frameshifting.
2 ch as transcription errors, mRNA damage, and translational frameshifting.
3 is recognized as 'slippery' and promotes -1 translational frameshifting.
4 ion of eukaryotic antizyme genes requires +1 translational frameshifting.
5 ion of IS6110 is thought to be controlled by translational frameshifting.
6 issociation causing premature termination or translational frameshifting.
7 protein coding genes inferred as sites of +1 translational frameshifting.
8 ks known structural information required for translational frameshifting.
9 s tau or as the shorter gamma that arises by translational frameshifting.
10 as a uniquely extreme example of programmed translational frameshifting.
11 orms: tau and the shorter gamma, produced by translational frameshifting.
12 ovided evidence diagnostic for +1 programmed translational frameshifting, a phenomenon disparately re
13 fting in vivo, we show that the induction of translational frameshifting also occurs under stressful
14 uggest that expression of uncB is reduced by translational frameshifting and/or a translational false
15 coronaviruses, with polyprotein processing, translational frameshifting, and multiple sgRNA formatio
16 onal Lsi2 protein via transcriptional and/or translational frameshifting, and this limited amount of
17 t (gamma) forms generated through programmed translational frameshifting, but the need for both forms
18 omplex negative feedback system in which the translational frameshifting event may be viewed in engin
20 logenetically diverse and extensive usage of translational frameshifting in animal mitochondrial codi
21 t there is a high frequency of programmed +1 translational frameshifting in ciliates of the Euplotes
23 sm of gene regulation, the utilization of -1 translational frameshifting in regulating mammalian gene
24 tem-loop structure can significantly enhance translational frameshifting in the presence of the pepti
25 capsid-polymerase fusion protein produced by translational frameshifting in vivo may be due to specif
26 f HmbR in DNM2 was found to be controlled by translational frameshifting involving a polyguanine (G)
31 effort to reconcile in vitro observations of translational frameshifting on Leishmania RNA virus 1-4
32 ple GGU-CAG-A, are also prone to significant translational frameshifting, suggesting the possibility
33 e II (HTLV-2) use a similar mechanism for -1 translational frameshifting to overcome the termination
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