ライフサイエンスコーパス: transmission disequilibrium test

1 associated with symptomatic VL (P = 0.0265, transmission disequilibrium test).

2 ele among cases and their parents (e.g., the transmission/disequilibrium test).

3 rated in this cohort using extensions of the transmission disequilibrium test.

4 /mutations for association with DN using the transmission disequilibrium test.

5 sky adolescent drinking was tested using the transmission disequilibrium test.

6 We analyzed the data using the transmission disequilibrium test.

7 ngs were confirmed by using the family-based transmission disequilibrium test.

8 using various modifications of the extended transmission disequilibrium test.

9 test is more flexible and powerful than the transmission-disequilibrium test.

10 o (LR) chi(2), and of disequilibrium, by the transmission/disequilibrium test.

11 s real promise lies in generalization of the transmission/disequilibrium test.

12 markers on chromosome 12q15-q24.1 using the transmission/disequilibrium test.

13 , but less significantly (P=.0014), with the transmission/disequilibrium test.

14 n addition, association was assessed via the transmission/disequilibrium test.

15 tors for spina bifida by use of the two-step transmission/disequilibrium test.

16 ore approach, affected sibpair analyses, and transmission disequilibrium tests.

17 arkers) from sliding windows of all sizes to transmission disequilibrium tests.

18 was achieved using modified case-control and transmission-disequilibrium tests.

19 can also perform single-locus and multilocus transmission/disequilibrium tests.

20 hort of JIA families from 3 countries, using transmission disequilibrium testing.

21 Using the transmission disequilibrium test, a trend toward overtra

22 Using sex and genotype-conditioned transmission disequilibrium test analyses, we found that

23 orts (P = 4.4 x 10(-16)) and by family-based transmission disequilibrium test analysis (P = 0.0006).

24 A transmission disequilibrium test analysis was significan

25 With transmission disequilibrium test analysis, cleft lip wit

26 PL score 2.235, ASM LOD score 1.939) and, on transmission disequilibrium test analysis, D11S4090 at 1

27 Multimarker transmission disequilibrium testing analysis was negativ

28 for association, the latter by means of the transmission disequilibrium test and by a case-control a

29 by using both an affected-only family-based transmission disequilibrium test and case-control method

30 Using a novel approach called the polygenic transmission disequilibrium test and data from 6,454 fam

31 Using transmission disequilibrium test and haplotype analyses,

32 A locus (odds ratio [OR] 2.18, P = 0.0005 by transmission disequilibrium test and P = 0.002, by pedig

33 Conditional extended transmission disequilibrium test and pairwise extended t

34 overtransmission in NSOFC cases through the transmission disequilibrium test and through analyses of

35 ncrease power, as compared with the standard transmission/disequilibrium test and equivalent quantita

36 e transmitted versus nontransmitted alleles (transmission disequilibrium test) and both maternally ve

37 ium test, AAO effects using the quantitative transmission disequilibrium test, and interactions using

38 , including the haplotype relative risk, the transmission-disequilibrium test, and affected family-ba

39 ium test procedure, the conditional extended transmission/disequilibrium test, and the stepwise logis

40 the HLA-DRB1*15 allele using a family-based transmission disequilibrium test approach in 1690 indivi

41 e polymorphisms array and analyzed using the transmission disequilibrium test as implemented in PLINK

42 on, with the use of both frequency-based and transmission/disequilibrium test-based designs.

43 for population association with PCOS in the transmission/disequilibrium test (chi(2) >/= 3.84; nomin

44 sease-gene discovery in the case-control and transmission/disequilibrium test designs.

45 sequilibrium was assessed using the extended transmission disequilibrium test (ETDT) program.

46 e of association with RA (using the extended transmission disequilibrium test [ETDT]).

47 With the use of the transmission disequilibrium test, evidence for linkage d

48 nce for excess transmission was found by the Transmission Disequilibrium Test for rs2056202, rs229281

49 We also carried out transmission disequilibrium tests for 135 European Ameri

50 We carried out transmission-disequilibrium testing for V274I in 8003 in

51 The transmission/disequilibrium test for a three-site haplot

52 based linkage-disequilibrium mapping and the transmission/disequilibrium test, for both biallelic SNP

53 s of 194 nuclear families through use of the transmission/disequilibrium test gives P = .001.

54 Transmission/disequilibrium tests have attracted much at

55 n to the 5 htSNPs was investigated using the transmission disequilibrium test in RA simplex families

56 gression in the case-control data and by the transmission disequilibrium test in the families.

57 By using transmission disequilibrium testing in a cohort containi

58 urtly missed statistical significance in the transmission-disequilibrium test in the full cohort (rs1

59 Use of the multiallelic transmission-disequilibrium test (MTDT), for nine loci o

60 ith type 2 diabetes (P = 0.008), but neither transmission disequilibrium test nor family-based associ

61 A transmission disequilibrium test of 334,923 single-nucle

62 Quantitative transmission disequilibrium testing of 472 CF patient-pa

63 Transmission disequilibrium testing of the microsatellit

64 time in plants by applying the quantitative transmission disequilibrium test on an association popul

65 None of the transmission disequilibrium test or case-control results

66 Data may be family-based, as in the transmission disequilibrium test, or from a case-control

67 7178130 variant was found to be supported by transmission disequilibrium testing (P = 0.006).

68 by SNPlex for linkage analysis and parental transmission disequilibrium testing (ParenTDT).

69 on disequilibrium test and pairwise extended transmission disequilibrium test predicted functional in

70 es have been described, such as the modified transmission/disequilibrium test procedure, the conditio

71 tions, the max_Zeta(2) test and the max_TDT (transmission/disequilibrium test) proposed by McIntyre e

72 based tests, two marker-based tests, and the Transmission Disequilibrium Test-Q5) to detect single nu

73 ression (CATHGEN) and the quantitative trait transmission disequilibrium test (QTDT; GENECARD).

74 A subsequent transmission disequilibrium test replication study in an

75 If, for example, the transmission/disequilibrium test restricted to fathers i

76 Analysis of a family study by transmission disequilibrium testing revealed a similar r

77 Using family-based tests of association (transmission disequilibrium test), rs878906, in intron 3

78 es are then used to construct a rare-variant transmission disequilibrium test (rvTDT) in the case-par

79 vidual and devised a new test called the sib transmission/disequilibrium test (S-TDT).

80 The transmission disequilibrium test showed significant over

81 equilibrium test (SDT, p = 0.68) and the sib transmission/disequilibrium test (Sib-TDT, p = 0.81).

82 Using the transmission disequilibrium test, significant transmissi

83 A new transmission/disequilibrium-test statistic is proposed f

84 Linkage (sib pair), association, and transmission disequilibrium testing studies on 942 Afric

85 A multiallelic transmission disequilibrium test suggested an associatio

86 Refinement of the region by transmission disequilibrium testing suggests that a cand

87 The transmission disequilibrium test (TDT) analyses of our d

88 for each gene were performed and analyzed by transmission disequilibrium test (TDT) analysis (FBAT so

89 Transmission disequilibrium test (TDT) and quantitative

90 The transmission disequilibrium test (TDT) and the pedigree

91 ed a genome-wide scan for TD by applying the transmission disequilibrium test (TDT) genome-wide to th

92 Analysis by the transmission disequilibrium test (TDT) in 1159 families

93 n the region for linkage with disease by the transmission disequilibrium test (TDT) in a UK data set

94 The transmission disequilibrium test (TDT) is a powerful fam

95 The transmission disequilibrium test (TDT) is a useful metho

96 terozygous C430T mutation in patients, and a transmission disequilibrium test (TDT) showed strong ove

97 l solutions have been proposed to extend the transmission disequilibrium test (TDT) to include cases

98 We used the transmission disequilibrium test (TDT) to investigate fa

99 velopment of a test that extends the classic transmission disequilibrium test (TDT) to one that accou

100 The transmission disequilibrium test (TDT) was computed to t

101 The transmission disequilibrium test (TDT) was used to deter

102 Family-based procedures such as the transmission disequilibrium test (TDT) were motivated by

103 nalyses of linkage disequilibrium, using the transmission disequilibrium test (TDT), confirmed the 48

104 Like the transmission disequilibrium test (TDT), the likelihood-r

105 analyze parent-child trio data, by using the transmission disequilibrium test (TDT), which is robust

106 families, were genotyped and analyzed using transmission disequilibrium testing (TDT) and multitest

107 kage analysis and association analysis using transmission disequilibrium testing (TDT) were also used

108 Haplotype construction and transmission disequilibrium testing (TDT) were performed

109 The transmission-disequilibrium test (TDT) is a robust test

110 We used the transmission-disequilibrium test (TDT) method to test fo

111 The transmission-disequilibrium test (TDT) of Spielman et al

112 Using a multiallelic, transmission-disequilibrium test (TDT), we found overall

113 age with a disease, using tests based on the transmission-disequilibrium test (TDT).

114 hat data are errorless, and introduces a new transmission/disequilibrium test (TDT(ae)) that allows f

115 ors on the type I error rate of a particular transmission/disequilibrium test (TDT(std)), which assum

116 his proposed method differs from the popular transmission/disequilibrium test (TDT) approach for fine

117 The transmission/disequilibrium test (TDT) developed by Spie

118 e ratio of sample sizes) between SDT and the transmission/disequilibrium test (TDT) for special famil

119 been demonstrated in the literature that the transmission/disequilibrium test (TDT) has higher power

120 Family-based association methods such as the transmission/disequilibrium test (TDT) have become very

121 When the transmission/disequilibrium test (TDT) is applied to mul

122 of the traditional family-based studies, the Transmission/Disequilibrium Test (TDT) measures the over

123 The transmission/disequilibrium test (TDT) originally was in

124 The transmission/disequilibrium test (TDT) proposed by Spiel

125 , Mexican-American, Chinese and Korean), the transmission/disequilibrium test (TDT) revealed a highly

126 The transmission/disequilibrium test (TDT) was also performe

127 riasis and this haplotype as assessed by the transmission/disequilibrium test (TDT) was of borderline

128 The transmission/disequilibrium test (TDT), a family-based t

129 and Sommer and that both can outperform the transmission/disequilibrium test (TDT), although the TDT

130 mily-based tests of association, such as the transmission/disequilibrium test (TDT), but this comes a

131 As detected by use of the transmission/disequilibrium test (TDT), individual marke

132 llele and use the conventional single-marker transmission/disequilibrium test (TDT), then the rapid i

133 The transmission/disequilibrium test (TDT), which detects li

134 Family data teamed with the transmission/disequilibrium test (TDT), which simultaneo

135 and a family-based study design known as the transmission/disequilibrium test (TDT).

136 This advantage is illustrated by the transmission/disequilibrium test (TDT).

137 en marker and disease was assessed using the transmission/disequilibrium test (TDT).

138 We performed transmission/disequilibrium testing (TDT) and haplotype

139 8 German trios with CD and demonstrating, by transmission/disequilibrium testing (TDT), that the same

140 Transmission/disequilibrium tests (TDT) based on family

141 such as case/control, measured genotype, and transmission/disequilibrium test [TDT]) will have no pow

142 and used unconditioned and DRB1-conditioned transmission disequilibrium tests (TDTs).

143 Transmission/disequilibrium tests (TDTs) were also perfo

144 f genotype and exposure, an extension of the transmission/disequilibrium test, tests for differences

145 We used the transmission disequilibrium test to analyze associations

146 Using the transmission-disequilibrium test to search the genome fo

147 istic, the nonfounder statistic, extends the transmission/disequilibrium test to accommodate affected

148 approach provides a natural extension of the transmission/disequilibrium test to any phenotype and to

149 applied to nuclear families, generalizes the transmission/disequilibrium test to arbitrary numbers of

150 and employed a liberalization of the sibling transmission/disequilibrium test to calculate odds ratio

151 A generalization of the transmission/disequilibrium test to detect association b

152 The transmission disequilibrium test was performed using TRA

153 The two-step transmission disequilibrium test was the first, family-b

154 Transmission disequilibrium testing was performed for af

155 The strongest effect in the transmission/disequilibrium test was observed in the INS

156 Using pedigree-based transmission disequilibrium tests, we observed modest ev

157 Using the transmission-disequilibrium test, we found no evidence o

158 Using the transmission-disequilibrium test, we found significant e

159 arametric likelihood ratio chi2 test and the transmission-disequilibrium test, we identified 12 marke

160 Using single-SNP and haplotype-based transmission/disequilibrium tests, we ranked genes on th

161 The quantitative transmission disequilibrium tests were done using 31,312

162 The chi-square and extended transmission disequilibrium tests were used to test for

163 The transmission disequilibrium test with use of trios (an a

164 ntrol study, a separate analysis that used a transmission-disequilibrium test with 76 informative fam

165 The X-linked sibling transmission/disequilibrium test (XS-TDT) and the recons