ライフサイエンスコーパス: transmitter

1 ic telemetry (10 years battery life acoustic transmitters).

2 questioned the importance of this intestinal transmitter.

3 olyte, neuro-modulator, and possibly a neuro-transmitter.

4 opulations and clearance/inactivation of the transmitter.

5 elease both a fast excitatory and inhibitory transmitter.

6 dulation and identify acetylcholine as a key transmitter.

7 elicited by submaximal concentrations of the transmitter.

8 tter, but not ATP, a Receptor (Type II) cell transmitter.

9 nal properties, including sensitivity to the transmitter.

10 vglut2 instead and use glutamate as a second transmitter.

11 groups may use GABA or glutamate as a second transmitter.

12 ignaling, of which serotonin (5-HT) is a key transmitter.

13 on cell require the probabilistic release of transmitter.

14 ons; this adult male is carrying a satellite transmitter.

15 enhances the AMPAR response to low levels of transmitter.

16 sites and that may themselves act as signal transmitters.

17 g autocrine actions that liberate retrograde transmitters.

18 ulate excitability and the release of neural transmitters.

19 we analyzed BP using radioactive telemetric transmitters.

20 signaling were nearly identical for the two transmitters.

21 ontribution of non-adrenergic sympathetic co-transmitters.

22 nteraction between the fast- and slow-acting transmitters.

23 e MF1 mice using intraperitoneally implanted transmitters.

24 teen fin whales were equipped with satellite transmitters, 8 in the Pelagos Sanctuary (although two c

25 The known transmitters accumulate in a cell type selective manner,

26 ADP at sites of vascular injury or synaptic transmitters acting at metabotropic Gq-coupled receptors

27 lies to mammals, a major mechanism for amine transmitter action is to raise astrocyte [Ca(2+)]i and r

28 he role of mucosal 5-HT and suggest that the transmitter acts as a key initiator of fecal pellet prop

29 and in the adult brain, with observations of transmitter addition, loss, or replacement of one transm

30 ic autonomy that enables a small-field, dual-transmitter amacrine cell to process diverse dendritic f

31 l (FSO) communications link between a ground transmitter and a ground receiver via a moving unmanned-

32 oil is driven by an alternating current as a transmitter and a vector of phase changes are measured f

33 ggests a dynamic physiological role for this transmitter and highlights the importance of determining

34 ansmission through reuptake of extracellular transmitter and is a target for addictive compounds such

35 o evaluate the performance of the injectable transmitter and its effect on the survival of implanted

36 scillators, each unified by its neuropeptide transmitter and mode of coupling to other units.

37 tion systems, accurate alignment between the transmitter and receiver is important to guarantee suffi

38 exing systems since misalignment between the transmitter and receiver may cause crosstalk among chann

39 coupling between conducting loops serving as transmitter and receiver.

40 dopamine from a slow transmitter into a fast transmitter and revealing new opportunities for studying

41 ctromagnetic fields (RF-EMFs) from broadcast transmitters and childhood cancer.

42 approach aimed at discovering new endogenous transmitters and hormones.

43 It is shown that graphene-based acoustic transmitters and receivers have a wide bandwidth, from t

44 ATP serves as a principal afferent transmitter, and also is the key trigger for autocrine p

45 NCs as sensitizers, bound organic ligands as transmitters, and molecular annihilators has the advanta

46 The size and performance of the transmitter are key limiting factors.

47 The respective transmitters are mostly spatially separated, that is, sy

48 Neuronal transmitters are released from nerve terminals via the f

49 t specialized junctions, the synapses, where transmitters are released from vesicles in a Ca(2+)-depe

50 However, most caged transmitters are, surprisingly, severe antagonists of io

51 Dale's principle of "one neuron releases one transmitter at all its synapses," a growing number of ex

52 ch constitutes the main source of releasable transmitter at glycinergic synapses.

53 rocytes from both regions responded to these transmitters at 1 day in culture.

54 itters that modify the effect of "classical" transmitters at the synapse.

55 opamine (DA) and is the major determinant of transmitter availability in the brain.

56 cifically, taste-modifying interactions, via transmitters, between gustatory and chemosensory afferen

57 n simultaneously occupy both the orthosteric transmitter binding site and the allosteric vestibule of

58 At each transmitter binding site three aromatic groups determine

59 nonical alpha-gamma interface containing the transmitter binding sites and at the noncanonical delta-

60 in of the subunit: the E loop (a loop of the transmitter-binding domain) and a region closer to the a

61 subunit does not participate in a canonical transmitter-binding site.

62 type mouse AChRs having a mutation(s) at the transmitter-binding sites.

63 The neuromodulatory transmitters, biogenic amines, have profound effects on

64 uttle in catecholamine vesicle membranes for transmitter biosynthesis.

65 GABA is an inhibitory transmitter but can sometimes produce paradoxical excita

66 otonin (5-HT), a Presynaptic (Type III) cell transmitter, but not ATP, a Receptor (Type II) cell tran

67 Actin bundles are therefore not simple force transmitters, but instead, complex mechano-transducers t

68 tamate, synaptic AMPARs were desensitized by transmitter by >90%.

69 the receptor function in the absence of the transmitter, can be changed either by mutation to increa

70 ur findings identify NA as a neuromodulatory transmitter capable of triggering epigenetic, transcript

71 clocked QKD operation of an indium phosphide transmitter chip and a silicon oxynitride receiver chip-

72 e show that synaptic inputs of two different transmitter classes locally direct dendrite growth in a

73 synaptic receptor sensitivity, and efficient transmitter clearance.

74 ays that provide RF references, and wireless transmitters clocked by the oscillators.

75 ve to any changes in the output power of the transmitter, compensates for the use of different data l

76 IEC) has been used to quantify the vesicular transmitter content in mammalian vesicles.

77 d by several features, including morphology, transmitter content, and synapse architecture [1].

78 to identify neurons, based on cell class and transmitter content, that express each receptor.

79 referential exocytosis of vesicles with high transmitter content.

80 de (VIP) to detect neural elements and their transmitter contents in wholemounts and sections of card

81 mines the temporal and spatial dispersion of transmitter, controls the extent of receptor activation,

82 y radiowave observations of the NAA 24.0 kHz transmitter, Cutler, Maine, made from Halley Station, An

83 al log2 sCD14 concentration was higher among transmitters (defined as pairs in which maternally trans

84 the first known implanted energy-harvesting transmitter demonstrated in vivo.

85 a non-traditional, photonic enabled, compact transmitter device for tunable, ultrawide band (UWB) rad

86 Nitric oxide (NO) functions widely as a transmitter/diffusible second messenger in the central n

87 However, parallel fibres can also release transmitter directly into the extracellular space, from

88 tionally antagonized by SytI, enabling rapid transmitter discharge from single vesicles.

89 mammalian systems, discuss the phenomenon of transmitter dualism in the context of developmental spec

90 When serving as a transmitter for photon upconversion, 2,3-PyAn yielded th

91 translational control at the CYB561 step of transmitter formation.

92 Although the release of peptide transmitters from POMC neurons is regulated by energy st

93 e of GABA release in addition to the peptide transmitters from POMC neurons.

94 n of the GABA type A (GABAA) receptor by the transmitter GABA and basal activity employing concatemer

95 pport the idea that interactions between the transmitter GABA and the allosteric agonists propofol, p

96 These neurons also release the amino acid transmitter GABA, which can inhibit downstream neurons.

97 urons can release either the amino acid (AA) transmitter gamma-aminobutyric acid (GABA) or glutamate.

98 Modulators of transmitter-gated ion channels have a wide range of maxi

99 le formulation for analyzing the behavior of transmitter-gated ion channels.

100 , increasing the supply of the physiological transmitter Glut increased the frequency and signaling c

101 icular transport of the principal excitatory transmitter glutamate depends primarily on membrane pote

102 following a reduction in the potency of the transmitter glycine; this resulted from a rapid deactiva

103 Successful development of this transmitter greatly expands the potential for long-term

104 nt sensing of endogenously generated gaseous transmitter H2S in its aqueous form (bisulfide or hydrog

105 salmon smolts implanted with the injectable transmitter had a higher survival probability from relea

106 phase transmission events occurred after the transmitter had interrupted ART.

107 Acetylcholine, the primary neuromuscular transmitter, has long been presumed to mediate this acti

108 the ability of a neuron to release multiple transmitters, has long been recognized in selected circu

109 ggers vesicle fusion and release of neuronal transmitters, however, the dynamics of this process is n

110 In addition to peptide transmitters, hypothalamic neurons, including proopiomel

111 ABSTRACT: In addition to peptide transmitters, hypothalamic neurons, including proopiomel

112 ll, these factors and genital factors of the transmitter (i.e., male circumcision, bacterial vaginosi

113 Of 36 transmitters identified, 9 were determined on the basis

114 Natural stimuli can drive these changes in transmitter identity, with matching changes in postsynap

115 ic precursors to control acquisition of 5-HT transmitter identity.

116 Blood pressure transmitter-implanted female C57BL/6J mice.

117 NO also acts as a gaseous transmitter in a variety of biological processes.

118 ants decreased activation of Src, an initial transmitter in Na/K-ATPase signal transduction, and of C

119 o be unaffected by the low levels of ambient transmitter in the brain.

120 obutyric acid (GABA) is the major inhibitory transmitter in the mature brain but is excitatory in the

121 t there are few auditory synapses using both transmitters in maturity.

122 rtant role for non-adrenergic sympathetic co-transmitters in mediating the vasoconstrictor response t

123 rom ArPPLNP1 act as inhibitory neuromuscular transmitters in starfish, which contrasts with the myoex

124 he bottlenecks, emphasis is on the design of transmitters in terms of molecular energetics, photophys

125 Y (NPY) is one of the most abundant protein transmitters in the central nervous system with roles in

126 th circulating hormones and locally released transmitters in the last common ancestor of Onychophora

127 ibitory ascending inputs, and the inhibitory transmitters include both gamma-aminobutyric acid (GABA)

128 of HIV-positive mothers (nontransmitters and transmitters) inhibits HIV transmission.

129 L1, acting as a Ca(2+) channel in TVs, links transmitter-initiated cyclic nucleotide signaling with C

130 n, thereby transforming dopamine from a slow transmitter into a fast transmitter and revealing new op

131 d, the vesicular transport of most classical transmitters involves a mechanism of H(+) exchange, whic

132 ce fringe pattern about the mid-latitude NAA transmitter is due to a 3 km reduction in the effective

133 The newly developed injectable transmitter is the first acoustic transmitter that can b

134 y transfer (TET) from NC donors to molecular transmitters is one of the bottlenecks, emphasis is on t

135 o serving as an energy source and purinergic transmitter, is an essential element in the concentratio

136 Neuropeptide Y (NPY), a stress modulatory transmitter, is associated with posttraumatic stress dis

137 H2S, a recently discovered gaso-transmitter, is endogenously generated by many cell type

138 ich serve as inter- and intracellular signal transmitters, is key in understanding a variety of funct

139 LC evokes arousal similar to the excitatory transmitter, L-glutamate.

140 cell bodies, increased DAcyt was not due to transmitter leakage from synaptic vesicles but rather to

141 Transmitter levels of serotonin, dopamine and norepineph

142 and pi-conjugated molecules, focusing on the transmitter ligand at the organic-inorganic interface.

143 rage capacities of the batteries used in the transmitters limit the time that the implanted animals c

144 rs (GPCRs) including receptors for classical transmitters, lipid signals, and peptides as well as doz

145 ly modulated light sources positioned at the transmitter location.

146 of parasympathetic fibres and their distinct transmitter mechanisms and how these vary with age, dise

147 .84); however, non-adrenergic sympathetic co-transmitters mediated a significant portion of the vasoc

148 pamine receptors, while another unidentified transmitter mediates the delayed excitation.

149 ed along three dimensions (input divergence, transmitter mobilization, adenosine modulation) that pot

150 Variations in transmitter mobilization, CD73 levels, and afferent dive

151 ), an effect associated with distinctions in transmitter mobilization.

152 g to respiratory effects of Nalcn mutations; transmitter modulation of Nalcn may underlie state-depen

153 families, including cell-adhesion molecules, transmitter-modulator receptors, ion channels, signaling

154 te revealed that ATP serves as the mediating transmitter molecule released from skin cells after odor

155 We measured transmitter molecules released from vesicles in NGF-diff

156 hanism that involves glial release of active transmitter molecules, is its manifestation as N-methyl-

157 -protease from 53 nontransmitter mothers, 48 transmitter mothers, and 47 infected infants were assaye

158 ties between the nontransmitter (n = 53) and transmitter (n = 44) mothers (P = 0.48).

159 e functional importance of dual- (or multi-) transmitter neurons extends beyond actions on postsynapt

160 A 10% reduction in the scale size of the transmitter nighttime interference fringe pattern has be

161 eceptor function and liberate the retrograde transmitter, nitric oxide (NO).

162 The neuropeptide transmitter nociceptin, which binds to the nociceptin/or

163 perate in three different regimes--spin wave transmitter, non-volatile memory and spin wave detector,

164 such signals are modulated by other gaseous transmitters - notably hydrogen disulfide and carbon mon

165 Focal adhesion kinase (FAK), a key transmitter of growth factor and anchorage stimulation,

166 acetylcholine is most likely the excitatory transmitter of nonspiking type IIa1 local interneurons.

167 calcitriol represents a possible endogenous transmitter of Ptch/Smo interaction.

168 been identified as the potential inhibitory transmitter of spiking type I local interneurons, wherea

169 with wide-range responses, that are reliable transmitters of the incoming frequency, and mature neuro

170 rm that a single motoneuron can release both transmitters on a single post-synaptic Renshaw cell.

171 te the involvement of cannabinoid and opioid transmitters on the formation of placebo effects.

172 d acetylcholine bioavailability, measured as transmitter overflow into the lumen, increased purinergi

173 Each of these transmitters participates in local negative or positive

174 uced frequency and released lower amounts of transmitter per vesicle (i.e., reduced quantal size).

175 The injectable transmitter performed well and similarly to the proceedi

176 Recent evidence also suggests that the dual-transmitter phenotype can be dynamically regulated durin

177 To compare the organization and transmitter phenotype of LHb projections to the DR, dire

178 ons or the MnR and additionally examined the transmitter phenotype of major IP and MnR afferents by c

179 markers may be indicative of a switch in AA transmitter phenotype, fluorescent in situ hybridization

180 These glutamatergic dual transmitter phenotypes are the Orthopedia transcription

181 lectrodes placed over the motor cortex and a transmitter placed subcutaneously in the left side of th

182 pathetic nerve fibers that contain classical transmitters plus an array of neuropeptides and enzymes

183 , including presynaptic vesicular release of transmitter, postsynaptic receptor populations and clear

184 neurons, controlling presynaptic release of transmitter, postsynaptic signaling, and synaptic integr

185 Dendritic spines are the major transmitter reception compartments of glutamatergic syna

186 n Pet-1 targets from 5-HT synthesis genes to transmitter receptor genes required for afferent modulat

187 rgic receptors at the synaptic site requires transmitter-receptor interaction, receptor internalizati

188 ut signals using a sophisticated assembly of transmitter receptors and voltage-sensitive ion channel

189 ntity, with matching changes in postsynaptic transmitter receptors.

190 alignment of transmitter release sites with transmitter receptors.

191 studying the characteristics of a sample of transmitters, recruited through newly diagnosed, recentl

192 ents are weakly activated by outer hair cell transmitter release and are insensitive to sound.

193 2+) influx modulates action potential evoked transmitter release and directly drives asynchronous rel

194 Evoked excitatory transmitter release and excitatory postsynaptic currents

195 thelial cell function, muscle contractility, transmitter release and gene and protein expression were

196 in old mice exhibited increased asynchronous transmitter release and reduced synchronous release.

197 atergic synapses, indicating a difference in transmitter release and/or refilling mechanisms.

198 erminals in the saccule contain vesicles but transmitter release appears paracrine in nature, due to

199 The fast kinetics of transmitter release are determined by transient Ca(2+) e

200 icle fusion (the SNARE complex) and modulate transmitter release at conventional synapses, we examine

201 CaV2.2 channels regulate transmitter release at inhibitory and excitatory synapse

202 w that Syt1 and Syt2 can redundantly control transmitter release at specific brain synapses.

203 n the mammalian CNS are specialized for fast transmitter release at their output synapses.

204 presynaptic molecular mechanisms regulating transmitter release at these different sensory ribbon sy

205 changes in detrusor function and urothelial transmitter release but few studies have investigated ho

206 dynamic regulation of neuronal activity and transmitter release by activating inhibitory ATP-sensiti

207 ators have previously been shown to regulate transmitter release by inhibiting presynaptic Ca(2+) inf

208 of Munc18-1 drives short-term enhancement of transmitter release during PTP.

209 sson stochastic point process of excitation (transmitter release events from the IHC).

210 e (RIM) proteins are important regulators of transmitter release from active zones.

211 onto retinal bipolar cells is influenced by transmitter release from neighboring bipolar cells, impl

212 ool enabling manipulation of activity and/or transmitter release from targeted cell populations.

213 during membrane repolarization, and triggers transmitter release from the cell.

214 Action potential firing triggers transmitter release from the immature IHC that in turn g

215 d, we find unexpected involvement of altered transmitter release from the motor neuron.

216 ight the need to consider different types of transmitter release in circuit mapping and physiologic r

217 inhibition of presynaptic Ca(2+) channel and transmitter release in rat retinal rod bipolar cells dep

218 rstand how differential VTA connectivity and transmitter release in these LHA neurons influences this

219 uscular junction, is that the time course of transmitter release is independent of the extracellular

220 Exocytic transmitter release is regulated by the SNARE complex, w

221 change in dynamic properties of presynaptic transmitter release may underlie compromised synaptic pr

222 Ca(2+)-dependent transmitter release occurs in a fast and in a slow phase

223 The gain-of-function of transmitter release of the S218L mutant was reproduced i

224 mutants lacking ultraviolet cones, and when transmitter release or visual stimulation of ultraviolet

225 TPpre), which is expressed as an increase in transmitter release probability (Pr).

226 H neurons increased food intake; attenuating transmitter release reduced body weight.

227 he ribbon-type CAZ to achieve the continuous transmitter release required by synapses of neurons that

228 activity and suggest that different types of transmitter release should be considered when circuit ma

229 Synaptic communication requires alignment of transmitter release sites with transmitter receptors.

230 presynaptic ribbon-like structures at their transmitter release sites.

231 Modeling of Ca(2+)-dependent transmitter release suggests that the invariant time cou

232 n with impairments in the ability to sustain transmitter release that resulted in muscular weakness.

233 fferents can regulate neuronal excitability, transmitter release, and firing patterns in thalamic net

234 n uses an Epac2-dependent pathway to promote transmitter release, and that Epac2 is required to maint

235 nAChRs have an important role in regulating transmitter release, cell excitability, and neuronal int

236 Spike width helps to control transmitter release, conduction velocity, and firing pat

237 Syt2 alone had only minor effects on evoked transmitter release, despite the clear presence of the p

238 resynaptic action potential is essential for transmitter release, excitability and energy expenditure

239 ges at the synapse which result in increased transmitter release, failure of synaptic plasticity, and

240 The computational benefits of dual transmitter release, however, remain poorly understood.

241 ort the notion that APP and APLP2 facilitate transmitter release, likely through the interaction with

242 s that are essentially devoid of presynaptic transmitter release, we demonstrate that the formation a

243 transmitting sound information into synaptic transmitter release.

244 take-competent VMAT2 vesicles are capable of transmitter release.

245 tensity of stimulation or the probability of transmitter release.

246 rive from the characteristics of presynaptic transmitter release.

247 els augments dynamic range without affecting transmitter release.

248 s, suggesting a mechanism by which APP tunes transmitter release.

249 ys an important in vivo role and facilitates transmitter release.

250 ontributes to cAMP-dependent potentiation of transmitter release.

251 be involved in the modulation of presynaptic transmitter release.

252 cellular biosensors to monitor taste-evoked transmitter release.

253 os, consistent with a reduced probability of transmitter release.

254 ) channels, or synaptic proteins that affect transmitter release.

255 -channel trafficking, but is dispensable for transmitter release.

256 downregulation and decreased probability of transmitter release.

257 peptide (CGRP), which may be as an efferent transmitter released from peripheral axon terminals.

258 ssed in the spinal cord during the period of transmitter respecification and that spike activity caus

259 Transmitter respecification did not occur, suggesting th

260 Furthermore, transmitter secretion of cochlear inner hair cells is co

261 The transmitter sends out a unique identification code with

262 ssion within the DRN is thought to occur via transmitter spillover and paracrine activation of extras

263 otostimulation of their axons independent of transmitter status, we virally transfected VTA neurons w

264 made in identifying the factors that induce transmitter switching and in understanding the molecular

265 accounting for calcium-spike BDNF-dependent transmitter switching.

266 witching engages genetic programs regulating transmitter synthesis, but the mechanism by which activi

267 l epithelium) show a remarkable diversity of transmitters synthesized and secreted locally.

268 However, the noradrenergic transmitter system may be affected on a different level,

269 The ECS is a widely distributed transmitter system that controls gut functions periphera

270 To confirm that the noradrenergic transmitter system was responsible for the enhanced memo

271 and by which synaptic inputs from different transmitter systems are correctly partitioned onto a pos

272 ack the activity in multiple neuromodulatory transmitter systems as they control the state of the wak

273 asic DA provides a novel dynamic link of two transmitter systems central to the detection and selecti

274 on by both dopaminergic and non-dopaminergic transmitter systems in the brain.

275 clude, but are not limited to, monoaminergic transmitter systems, the hypothalamic-pituitary-adrenal

276 is subject to modulation by slow modulatory transmitter systems.

277 obula plate tangential cells by antagonistic transmitter systems.

278 g generation of commercially-available JSATS transmitters tested concurrently.

279 injectable transmitter is the first acoustic transmitter that can be implanted via injection instead

280 Acetylcholine is a neuromodulatory transmitter that controls synaptic plasticity and sensor

281 istent with a role for CGRP as an inhibitory transmitter that shapes peripheral taste signals via ser

282 oped and implemented a battery-free acoustic transmitter that uses a flexible piezoelectric beam to h

283 tide (AgRP) neurons, also release amino acid transmitters that can alter energy balance regulation.

284 tide (AgRP) neurons, also release amino acid transmitters that can alter energy balance regulation.

285 mice were implanted with wireless telemetry transmitters that enabled continuous measurements of ele

286 tissue region, NO can act more like a volume transmitter to influence, and perhaps coordinate, the be

287 For suspended films, the transition from transmitter to reflector occurs when the sheet resistanc

288 male mice implanted with wireless telemetry transmitters to a 10 day CSDS regimen known to produce a

289 The contribution of modulatory transmitters to anti-Hebbian LTP induction remains to be

290 An electromagnetic transmitter typically consists of individual components

291 ction in size and ability to implant the new transmitter via injection has reduced the tag or tagging

292 RF-EMF exposure from broadcast transmitters was modeled.

293 e inhibited by scattering by electromagnetic transmitter wave fields.

294 vefront of the optical field incident on the transmitter, we achieve directional control of the trans

295 Using on-bird microphone transmitters, we recorded the vocalisations of individua

296 Wireless electroencephalogram transmitters were implanted into 23 C57BL/6 mice before

297 The external transmitter wirelessly communicates with and powers the

298 mitter addition, loss, or replacement of one transmitter with another.

299 the selectivity of our detection method for transmitters with positive charge.

300 te that 5-HT is acting, in part, as a volume transmitter within the NTS.