1 sposase resulted in an 11.6-fold increase in
transpositional activity compared to controls, with 67%
2 ding domain is shown to greatly enhance SB's
transpositional activity in mammalian cells.
3 e argue that a large number of TEs and their
transpositional activity may be linked to differential r
4 There was a cessation in the
transpositional activity of DNA transposons during the l
5 The
transpositional activity of the Gal4-Mos1 transposase wa
6 The
transpositional activity of the piggyBac transposable el
7 ed regulatory mechanisms aimed at repressing
transpositional activity.
8 nstrated the transcriptional and presumptive
transpositional competency of the element.
9 f themselves as a common by-product of their
transpositional cycle.
10 ts that Ty1 and yeast have coevolved to link
transpositional dormancy to the integrity of the genome.
11 yces cerevisiae are maintained in a state of
transpositional dormancy.
12 n to be capable of producing large, unlinked
transpositional duplications in Drosophila.
13 Bound proteins also caused strong
transpositional enhancements near each end of HIV-I.
14 A sites for transposase binding, including a
transpositional enhancer called the internal activation
15 The
transpositional enhancer IAS appears to have little impa
16 -affinity transposase binding site, an 11-bp
transpositional enhancer, and, at the highest concentrat
17 (1) reflects global emanation after a single
transpositional event in recent evolutionary time.
18 These
transpositional fusions are defined by insertion of a 32
19 effects of a moderate dissociation rate and
transpositional handicap.
20 tent (approximately 20 to 27,000 copies) and
transpositional history of this family across the genus
21 preted as evidence for selection against the
transpositional increase of TEs.
22 ally abolish nuclear localization inactivate
transpositional integration but do not affect reverse tr
23 that it is silenced at the translational or
transpositional level.
24 We propose that dissimilar
transpositional mechanisms differentiate the TE classes
25 We have used
transpositional mutagenesis of a proline auxotroph (PAO9
26 s in chromosome condition strongly influence
transpositional outcome.
27 priate diversion of V(D)J rearrangement to a
transpositional pathway may also help to explain certain
28 ecombination signals into new DNA sites in a
transpositional reaction.
29 fication, very similar to the early steps of
transpositional recombination and retroviral integration
30 Central to the Mu
transpositional recombination are the two chemical steps
31 Using an extra-chromosomal
transpositional recombination assay, we show that Hermes
32 K serves an important protective role during
transpositional recombination in mammals.
33 eaction is closely related to known types of
transpositional recombination, such as that of HIV integ
34 cleavage and joining reactions essential for
transpositional recombination.
35 that encode an 87-kDa transposase enzyme and
transpositional repressor proteins.
36 presence of coding DNA may aid in preventing
transpositional resolution of V(D)J recombination interm
37 RAG1/2 has also been shown to catalyze
transpositional strand transfer of RSS-containing substr
38 sposition activity or capture of nonspecific
transpositional target DNA, suggesting this DNA occupies