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3 ical heart failure had Doppler assessment of transvalvular flow and right heart catheterization perfo
4 al entities might be identified according to transvalvular flow rates and pressure gradients, resulti
8 (</=40%), low-gradient aortic stenosis (mean transvalvular gradient <40 mm Hg and indexed aortic valv
10 95% CI, 1.21-2.26; P = .001), pre-TAVR mean transvalvular gradient (0.98; 95% CI, 0.97-0.99; P = .00
13 n=144) and PLG-SAS (n=205) according to mean transvalvular gradient (mean gradient >40 or </=40 mm Hg
14 left ventricular (LV) dysfunction and a low transvalvular gradient (TVG) is associated with improved
16 cellent prosthetic valve function with a low transvalvular gradient and no left ventricular outflow t
17 Doppler-derived measures of peak and mean transvalvular gradient correlated well with reference st
19 valve area <0.6 cm(2)/m(2)) present with low transvalvular gradient despite a normal left ventricular
20 Correlation between aortic valve weight and transvalvular gradient improved further when gender was
22 V) outflow, LV ejection fraction (LVEF), and transvalvular gradient on outcomes following transcathet
23 nce was uniformly good after redo TAVR (mean transvalvular gradient post redo TAVR: 12.5+/-6.1 mm Hg)
26 in patients with severe aortic stenosis, low transvalvular gradient, and severe left ventricular dysf
27 has superb hemodynamics in terms of residual transvalvular gradient, effective orifice area, and regr
28 de the following: central flow capacity, low transvalvular gradient, low thrombogenicity, durability,
29 a more physiological flow pattern and lower transvalvular gradient, which may have an important bear
34 ht, in general, the women had higher average transvalvular gradients (p </= 0.005) and lower average
36 with the ACURATE neo valve resulted in lower transvalvular gradients and consequently less prosthesis
37 heter valves and results in reduced residual transvalvular gradients and increased valve effective or
38 a (EOA) of a prosthetic valve is superior to transvalvular gradients as a measure of valve function,
40 the weights of stenotic aortic valves to the transvalvular gradients or to the calculated aortic valv
45 prosthetic TR in patients with and without a transvalvular lead more commonly occurred 2 years or lat
46 w-up of 25 months, in 5 patients (9%) with a transvalvular lead significant (moderate or greater) pro
49 avalvular leakage was observed in 113 (32%), transvalvular leakage in 47 (13%), and both in 12 (3%).
50 tion (EF) < or =35% and aortic stenosis with transvalvular mean gradient <30 mm Hg underwent aortic v
51 ite severe left ventricular dysfunction, low transvalvular mean gradient, and increased operative mor
54 valve weight, age at operation, preoperative transvalvular peak pressure gradient, calculated aortic
55 lder age, valves of lighter weight and lower transvalvular peak pressure gradients, and more often si
56 cised stenotic aortic valves to preoperative transvalvular peak systolic gradients and to calculated
58 s increased (from <1 g to >6 g), the average transvalvular peak systolic pressure gradients progressi
59 onstrated a consistent overestimation of the transvalvular pressure (average of 54%, range 5%-136%) r
60 tretching of valve tissue caused by elevated transvalvular pressure can activate valvular interstitia
62 esophageal echocardiography (TEE) as well as transvalvular pressure measurements during cardiac cathe
63 erate prosthetic valve dysfunction (moderate transvalvular regurgitation in 1, moderate stenosis in 1
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