ライフサイエンスコーパス: transverse myelitis

1 arre syndrome (GBS), meningoencephalitis, or transverse myelitis.

2 marker NMO-IgG and its application to acute transverse myelitis.

3 lase, may be useful prognostic indicators in transverse myelitis.

4 ibutes to an evolving understanding of acute transverse myelitis.

5 ers in the setting of a paraneoplastic acute transverse myelitis.

6 cent advances in inflammatory non-infectious transverse myelitis.

7 e initially diagnosed as NMOSD or idiopathic transverse myelitis.

8 e ataxia (4), and other sub-acute syndromes (transverse myelitis (1), opsoclonus myoclonus (1)).

9 be answered before we truly understand acute transverse myelitis: (1) What are the various triggers f

10 acute disseminated encephalomyelitis (12%), transverse myelitis (12%), and cerebellar ataxia (10%).

11 ad worse outcomes than typical patients with transverse myelitis/acute disseminated encephalomyelitis

12 cond event: 4 of 9 (44%) developed recurrent transverse myelitis and 1 of 9 (11%) developed optic neu

13 ncreased frequency of longitudinal extensive transverse myelitis and a favourable clinical outcome in

14 She then presented with recurrent transverse myelitis and a vesicobullous rash over her ar

15 Classically, transverse myelitis and acute disseminated encephalomyel

16 for approaching how we classify subtypes of transverse myelitis and acute disseminated encephalomyel

17 ation from hysterical paraplegia, the second transverse myelitis and its consequences for bladder fun

18 emity paralysis, and was diagnosed as having transverse myelitis and membranous glomerulonephritis se

19 Two patients had initial CNS manifestations: transverse myelitis and meningoencephalitis.

20 ome; 0.1 for anaphylaxis and death; 0.04 for transverse myelitis and pancreatitis; and 0.009 for moto

21 tential immunopathogenic mechanisms in acute transverse myelitis and related inflammatory disorders o

22 nty), 7 (18%) with encephalitis, 3 (8%) with transverse myelitis, and 1 (3%) with newly diagnosed chr

23 comitant acute neuromuscular disease), 2 had transverse myelitis, and 1 had chronic inflammatory demy

24 itial diagnoses included multiple sclerosis, transverse myelitis, and unspecified neurodegenerative m

25 -old woman who had relapsing optic neuritis, transverse myelitis, AQP4-IgG seropositivity, and recurr

26 Prognostic factors in pediatric transverse myelitis are reviewed.

27 um of disorders associated with inflammatory transverse myelitis, as well as a greater appreciation o

28 For example, in acute transverse myelitis associated with systemic disease (i.

29 re were no differences in the frequencies of transverse myelitis, brainstem involvement, cerebellar s

30 isorders (including longitudinally extensive transverse myelitis), contributes to an evolving underst

31 who had an initial longitudinally extensive transverse myelitis, delay to diagnosis/treatment was gr

32 Optic neuritis or transverse myelitis developed after vomiting onset in 11

33 Short transverse myelitis does not exclude consideration of AQ

34 on of novel biomarkers associated with acute transverse myelitis has led to a better understanding of

35 Acute transverse myelitis in children, in contrast to adults,

36 ignificant neurologic toxicity consisting of transverse myelitis in five patients, CNS toxicity in th

37 Acute transverse myelitis is a group of disorders characterize

38 Acute transverse myelitis is a pathogenetically heterogeneous

39 Short transverse myelitis is not uncommon in NMOSD and, when i

40 ative patients with longitudinally extensive transverse myelitis (LETM) behave differently from those

41 Longitudinally extensive transverse myelitis (LETM) is a frequently devastating c

42 ial presentation of longitudinally extensive transverse myelitis (LETM) predicts relapse of myelitis

43 litis optica (NMO), longitudinally extensive transverse myelitis (LETM), multiple sclerosis with a hi

44 Acute transverse myelitis may be an isolated entity or may occ

45 ected, included radiation recall (n = 2) and transverse myelitis (n = 1).

46 taxia (n = 4), and other subacute syndromes (transverse myelitis [n = 1], opsoclonus myoclonus [n = 1

47 No transverse myelitis or late toxicities of grade 4 or gre

48 ted encephalomyelitis (ADEM), one episode of transverse myelitis or optic neuritis, multiple sclerosi

49 patterns of weakness and sensory loss due to transverse myelitis or peripheral nerve disease.

50 yelinating event (optic neuritis, incomplete transverse myelitis, or brain-stem or cerebellar syndrom

51 of demyelination (optic neuritis, incomplete transverse myelitis, or brain-stem/cerebellar syndrome)

52 monofocal syndromes such as optic neuritis, transverse myelitis, or isolated brainstem syndromes in

53 characterised by a longitudinally extensive transverse myelitis (p=0.003), more often a complete res

54 on length, total lesion burden and number of transverse myelitis relapses did not correlate with pain

55 r month to 1.4 per month, and admissions for transverse myelitis remained constant at 0.6 per month.

56 optic neuritis and longitudinally extensive transverse myelitis [reported on at least 1 magnetic res

57 ocused on recurrent longitudinally extensive transverse myelitis (rLETM) are lacking.

58 of 20 initially presented with 2 attacks of transverse myelitis (rLETM-onset NMO).

59 Short transverse myelitis (STM; <3 vertebral segments) is cons

60 s to be elucidated about the causes of acute transverse myelitis, tantalizing clues as to the potenti

61 In idiopathic acute transverse myelitis, there is an intraparenchymal or per

62 ephalomyelitis (ADEM), two with ON, one with transverse myelitis (TM) and two with clinically isolate

63 Abs and present with optic neuritis (ON) and transverse myelitis (TM) are diagnosed with NMO and thos

64 Transverse myelitis (TM) is an immune-mediated spinal co

65 rts have suggested that vaccines may trigger transverse myelitis (TM) or acute disseminated encephalo

66 y isolated syndromes (CIS), 27 patients with transverse myelitis (TM), 50 patients with human immunod

67 racterised by severe optic neuritis (ON) and transverse myelitis (TM).

68 l optic neuritis (ON); 20% bilateral ON; 15% transverse myelitis (TM); 15% simultaneous TM&ON; 10% Ac

69 Transverse myelitis was the initial diagnosis.

70 nts with an initial longitudinally extensive transverse myelitis were excluded (n = 151).