ライフサイエンスコーパス: trauma

1 20% being reported for recurrent penetrating trauma.

2 the inability to account for combat-related trauma.

3 ntact, which may frequently result in ocular trauma.

4 rring through fascial defects, usually after trauma.

5 eat psychopathology emerging after childhood trauma.

6 y characterize the multicellular response to trauma.

7 responses to subsequent corneal infection or trauma.

8 without a reported history of preceding knee trauma.

9 noid hemorrhage, intracerebral hematoma, and trauma.

10 tion associated with peripheral infection or trauma.

11 t of posttraumatic stress disorder following trauma.

12 brain cortex in vivo after mild compressive trauma.

13 miting, unintentional weight loss, or recent trauma.

14 The reference group was women without trauma.

15 rge level I trauma center within 24 hours of trauma.

16 pt and predicted symptoms at 12 months after trauma.

17 olism (VTE) varies with blunt or penetrating trauma.

18 on or symptom prevention in the aftermath of trauma.

19 al sepsis to sterile syndromes such as major trauma.

20 ped into those with trauma and those without trauma.

21 , and decreases brain lesion size induced by trauma.

22 rvae exposed to drugs that mimic excitotoxic trauma.

23 se morbidity and mortality from abusive head trauma.

24 represents a promising therapy for cortical trauma.

25 nd cortical and hippocampal neurons in brain trauma.

26 uries of PLC in patients with spine cervical trauma.

27 to reduce fear when administered soon after trauma.

28 sk factors for VTE with blunt vs penetrating trauma.

29 ger arising from foreign invaders and tissue trauma.

30 cochlea, vulnerability to damage from noise trauma.

31 in infants at increased risk of abusive head trauma.

32 otective eyewear when they sustained the eye trauma.

33 ing the fracture with an additional thoracic trauma.

34 esonance (MR) imaging within 90 days of knee trauma.

35 lasting for days, months and even years post-trauma.

36 :RBC transfusion ratios for patients without trauma.

37 nd treat psychopathology linked to childhood trauma.

38 n tissues and is often preceded by injury or trauma.

39 ion of bone marrow lesions (BMLs) after knee trauma.

40 d approximately 1, 3, 6, and 12 months after trauma.

41 havior and substance disorders before random traumas.

42 nal (IP)-related traumas from combat-related traumas.

43 n identifiable cause of lens subluxation was trauma (11 eyes).

44 ansfusions were received by patients without trauma (767 [88.7%]), by men (582 [67.3%]), and for intr

45 4 of 1137) and similar between groups (blunt trauma, 9% [n = 73] vs penetrating trauma, 9.6% [n = 31]

46 ps (blunt trauma, 9% [n = 73] vs penetrating trauma, 9.6% [n = 31]; P = .76).

47 Within minutes of trauma, a comprehensive leukocytosis, elevated serum pro

48 ion may reduce the incidence of abusive head trauma (AHT) of infants and young children.

49 enia, such as social isolation and childhood trauma, also affect presynaptic dopaminergic function.

50 be exploited as a therapeutic target for CNS trauma and disease.SIGNIFICANCE STATEMENT Spinal cord in

51 cause blinding corneal infections following trauma and during contact lens wear.

52 fragility, frequent fractures in absence of trauma and growth deficiency.

53 tors of sterile inflammatory responses after trauma and injury.

54 a controlled cortical impact model of brain trauma and measured biochemical, anatomic, and behaviora

55 retinae degenerated slowly after the axonal trauma and neurons died.

56 ctures are common in high-energy blunt chest trauma and often occur with multiple consecutive rib fra

57 erable to a variety of insults like acoustic trauma and ototoxic drugs.

58 tra-early, within 1-hour, immune response to trauma and perform an exploratory analysis of its relati

59 ifibrinolytics reduce death from bleeding in trauma and post-partum haemorrhage.

60 for mediation of the relation between combat trauma and PTSD symptoms by longitudinal changes in DNA

61 cturing practice-artesunate in patients with trauma and severe hemorrhage is planned.

62 tween PTSD symptoms in the acute phase after trauma and the chronic phase.

63 Patients were grouped into those with trauma and those without trauma.

64 athways between different forms of childhood trauma and violence against women.

65 including neurodegenerative diseases, after trauma, and after stroke, and is characterized by increa

66 Medical, surgical, trauma, and cardiovascular ICUs of an academic medical c

67 genetic factors, masticatory hyperfunction, trauma, and continued growth, but the underlying mechani

68 eficits, restores motor function after brain trauma, and decreases brain lesion size induced by traum

69 ce or non-partner sexual violence, childhood trauma, and harsh parenting (smacking their children as

70 to the erosion of muscle mass following burn trauma, and we have previously shown concurrent activati

71 vironmental stimuli, becomes pathological in trauma- and stress-based psychiatric syndromes, such as

72 lso play a pathogenetic role by transmitting trauma- and stressor-related disorders (TSRD) across gen

73 Preventive interventions post trauma are lacking.

74 sed generalization of fear from a stress- or trauma-associated environment to a neutral context or en

75 as no trauma at either blood draw (n = 175), trauma at blood draw 1 but no PTSD at either draw (n = 1

76 red diagnostic interviews) was defined as no trauma at either blood draw (n = 175), trauma at blood d

77 quences of exposure to football-related head trauma at the high school level.

78 ounger than 18 years treated for blunt torso trauma at the University of California, Davis Medical Ce

79 Using the Trauma Audit and Research Network (TARN) database, we an

80 phy (CT) utilization in blunt abdominopelvic trauma (BAPT) over an 8-year period at an urban level 1

81 bowel perforation following blunt abdominal trauma (BAT) is an uncommon situation with high morbidit

82 evaluating the undertriage of patients with trauma because the anatomic triage criteria for patients

83 cancer treatment due to its smaller surgical trauma, better selectivity towards tumor cells, reduced

84 admissions to either the medical or surgical/trauma/burn ICU with available continuous electrocardiog

85 Monocytes showed robust expansion following trauma but displayed decreased stimulated proinflammator

86 pathophysiology of many neurologic diseases/trauma, but the effect of immune cells and factors on ne

87 for improved timeliness and access to major trauma care among patients injured in rural regions.

88 public institutions that provide most of the trauma care in Lima, Peru, and included urban resident a

89 Although the urgency of trauma care limits explicit discussion and consideration

90 luded urban resident and faculty surgery and trauma care physicians.

91 DC is an important development in trauma care that provides a valuable case study in surgi

92 Services providing trauma care were attacked more than other services.

93 recent conceptual and practical advances in trauma care, in both military and civilian settings, and

94 urvival of patients is crucial in evaluating trauma care, understanding recovery patterns and timefra

95 th 1 or more previous admissions to an urban trauma center (recidivists) were identified and compared

96 r idiopathic liver failure (DT) in a level 1 trauma center and large transplant center.

97 iving proximity to the nearest level I or II trauma center and/or rural designation in the Centers fo

98 nsertion at Boston Medical Center (a level I trauma center at Boston University School of Medicine) b

99 for major injury to a Canadian level I or II trauma center between April 1, 2006, and March 31, 2012.

100 merican College of Surgeons-verified level I trauma center between August 1, 2011, and January 1, 201

101 the intensive care unit of a single level I trauma center between January 2011 and May 2014 were ana

102 triage criteria for transfer to higher-level trauma center care.

103 The trauma center designation process should consider volume

104 fied 118 patients who presented to a level 1 trauma center emergency department and who underwent dua

105 Acute care level one trauma center in an inner city of the state of Connectic

106 We observed significant variation in trauma center mortality across Canadian provinces, speci

107 the emergency department of a large level I trauma center within 24 hours of trauma.

108 92 (10.4%) were transferred to a designated trauma center.

109 and, and Washington, DC, to a single level I trauma center.

110 California, Davis Medical Center, a level I trauma center.

111 was conducted at an urban, academic, level I trauma center.

112 T) over an 8-year period at an urban level 1 trauma center.

113 Single-institution level 1 trauma center.

114 of 424 trauma patients admitted to a level 1 Trauma Center.

115 Level I trauma center.

116 proportion of patients treated in designated trauma centers died during the first 24 hours of hospita

117 rease in risk-adjusted mortality in Canadian trauma centers during the study period, representing 248

118 The criteria were then sent to 384 verified trauma centers in the United States, Canada, Australia,

119 Nonetheless, variability might exist between trauma centers in timeliness of fixation.

120 g critical resources were cared for in major trauma centers vs 88.7% of urban patients.

121 greater among patients treated in designated trauma centers, these patients were substantially in wor

122 Although early mortality is low after severe trauma, chronic critical illness is a common trajectory

123 A retrospective trauma cohort study was conducted using data from the Pe

124 from 31.6% to 76.9% among residents for core trauma competency procedures.

125 We propose that early after trauma, complete dendritic stripping following rtACS pro

126 hold PTSD (N=19), or no PTSD (referred to as trauma control subjects) (N=17).

127 Relative to trauma control subjects, PTSD patients showed stronger c

128 urgeon technical and nontechnical skills for trauma core competencies before and after training and u

129 74 patients from 2003 to 2015), the National Trauma Data Bank (NTDB) (5755095 patients from 2003 to 2

130 a were extracted from the 2007-2012 National Trauma Data Bank.

131 Median LY30 was lower on post-trauma day (PTD)1 to PTD4 in patients with poor compared

132 and bibliographies for articles relating to trauma DC.

133 ) is a major cause of potentially survivable trauma death.

134 eiber, Guest Editors of the Special Issue on Trauma, describe a new era in exploration of the biology

135 itioned fear expression predicts anxiety and trauma disorder vulnerability, FGF2 may be a clinically

136 ent pathways for individuals who experienced trauma during childhood might usefully inform clinicians

137 clinically significant complication of major trauma (e.g., burn injury) and include various aspects o

138 n by the treating ED physician or a standard trauma evaluation alone.

139 atients were randomly assigned to a standard trauma evaluation with the FAST examination by the treat

140 in subjects with current PTSD compared with trauma-exposed control subjects (Cohen's d = -0.17, p =

141 aken together suggest that PTSD patients and trauma-exposed controls can be distinguished by enhanced

142 as altered in patients with PTSD relative to trauma-exposed matched controls who did not develop the

143 sounds (multivariate p = .007) compared with trauma-exposed participants without PTSD (n = 26).

144 ctional and does not separate the effects of trauma exposure from those of PTSD.

145 rtance of identifying patients at risk after trauma exposure is discussed.

146 Moreover, activation following trauma exposure reduced the susceptibility for PTSD-like

147 ers (ie, sex, co-morbid conditions, types of trauma exposure, and behavioral sources of inflammation)

148 of risk for maintaining PTSD symptoms after trauma exposure.

149 ith 34 676 respondents who reported lifetime trauma exposure.

150 the emergency department within 24 hours of trauma exposure.

151 specific PTSD induced by a single prolonged trauma exposure.

152 f the focused assessment with sonography for trauma (FAST) examination in children is unknown.

153 As time elapses after trauma, fear circuitry and dysphoric PTSD symptoms appea

154 es distinguishing interpersonal (IP)-related traumas from combat-related traumas.

155 dults with critical illness due to sepsis or trauma, ganciclovir did not reduce IL-6 levels and the c

156 In the blunt trauma group, more patients with VTE than without VTE ha

157 In the penetrating trauma group, more patients with VTE than without VTE ha

158 Abusive head trauma had a higher prevalence of seizures during resusc

159 ealing with mechanically induced periodontal trauma, has been available and potentially useful since

160 ute respiratory distress syndrome, and major trauma have been developed by a multispecialty task forc

161 les, called exosomes, secreted into ML after trauma/hemorrhagic shock (T/HS) have the potential to ac

162 lephone clinical assessments (including head trauma history) with informants were performed blinded.

163 mitted with traumatic injury to 1 of 4 major trauma hospitals across Australia from March 13, 2004, t

164 of resuscitated hemorrhagic shock and tissue trauma (HS/T).

165 All operating rooms and surgical and trauma ICUs at the institution.

166 ctural, and functional changes after aseptic trauma in mice related to astrocytes and later in neuron

167 l hemorrhage in infants without a history of trauma in whom trauma may not be part of the differentia

168 ood immune cell subsets that occur following trauma, including induction of Th17-type CD4 T cells, re

169 emerges as a promising avenue to combat head trauma-induced chronic cognitive deficits.

170 suggested AAV vectors persisted only in the trauma-induced corneas; however, a neutralizing antibody

171 Trauma-induced expansion of Th17-type CD4 T cells was se

172 microvascular barrier function to ameliorate trauma-induced hypotension, offering a novel therapeutic

173 nstrating prevention of neuropathology after trauma-induced nerve injury.

174 Trauma induces a complex immune response that requires a

175 Surgical trauma induces local and systemic inflammatory responses

176 ive error and amblyopia, globe damage due to trauma, infection and nutritional deficiency, retinal di

177 e of six human inflammatory diseases (burns, trauma, infection, sepsis, endotoxin and acute respirato

178 y neurological disorders, including strokes, trauma, inflammatory diseases, infectious diseases, and

179 rgent findings on a measure of clinical post-trauma intrusion symptoms at 1 week, but not on other sy

180 ported in a previous paper that four earlier traumas involving interpersonal violence significantly p

181 enign stimuli that "resemble" aspects of the trauma is a central feature of the disorder.

182 atio-based resuscitation in patients without trauma is associated with improved survival.

183 t for pulmonary embolism (PE) prophylaxis in trauma is controversial.

184 Immediate management of ocular trauma is critical in order to prevent blindness.

185 Abusive head trauma is the leading cause of death from physical abuse

186 tle is known about how exposure to childhood trauma is translated into biological risk for psychopath

187 y, such as lipoatrophy, lumpectomy or facial trauma, is a formidable challenge in reconstructive medi

188 wing body of evidence suggests that surgical trauma launches a systemic inflammatory response that re

189 urrence and a cycle of abuse, with childhood trauma leading to violence against women and further chi

190 When re-experiencing symptoms and trauma load were examined together in relation to right

191 ding the Nontechnical Skills assessment, the Trauma Management Skills scoring system, the Crisis Reso

192 tion across the period of exposure to combat trauma marks susceptibility for PTSD.

193 PREMISE: Although trauma may be considered a random act, geographical patt

194 infants without a history of trauma in whom trauma may not be part of the differential diagnosis has

195 tranasal oxytocin administration early after trauma may prevent PTSD, because oxytocin administration

196 19] years) and 324 patients with penetrating trauma (mean [SD] age, 35 [15] years), the rate of VTE w

197 In 813 patients with blunt trauma (mean [SD] age, 47 [19] years) and 324 patients w

198 GF2 priming might protect DPCs from the post-trauma microenvironment in which DPCs infiltrate and res

199 incidence of respiratory distress and ocular trauma observed in this class of dogs is highly correlat

200 antly predicted PTSD after subsequent random traumas (odds ratio (OR)=1.3-2.5).

201 d indicators of PTSD almost immediately post-trauma, often many months prior to clinical diagnosis.

202 tranasal oxytocin administration early after trauma on subsequent clinician-rated PTSD symptoms.

203 antly predicted PTSD after subsequent random traumas only among respondents with prior PTSD (OR=5.6).

204 issues, is a major complication after severe trauma or amputation.

205 ns only to massive osteochondral surgery for trauma or malignancy, and is confounded by potential con

206 om surgical resection or injuries induced by trauma or toxins.

207 hyaluronan metabolism in response to tissue trauma or ultraviolet radiation.

208 as a noninvasive biomarker for resilience to trauma or, conversely, to the potential development of p

209 78% (22 of 460) of other patients with chest trauma (OR, 1.50; 95% CI: 0.65, 3.47; P = .3371).

210 fections, solid or hematologic malignancies, trauma, or obstetric calamities.

211 s conducted using data from the Pennsylvania Trauma Outcome Study (PTOS) (461974 patients from 2003 t

212 - 18.94 control versus 1,092 x 106/l +/- 165 trauma, p < 0.0005) and CD14+HLA-DRlow/- monocytes (34.9

213 /- 4.48 control versus 95.72 x 106/l +/- 8.0 trauma, p < 0.05) and reduced leukocyte cytokine secreti

214 Missing the diagnosis of abusive head trauma, particularly in its mild form, is common and con

215 in response to an increased understanding of trauma patient physiology and changing injury patterns a

216 Severely injured human blunt trauma patients (n = 472, average injury severity score

217 Prospective observational study of 424 trauma patients admitted to a level 1 Trauma Center.

218 Trauma patients admitted to the hospital are at increase

219 Assessment of physical frailty in older trauma patients admitted to the intensive care unit is o

220 To determine if IVC filter insertion in trauma patients affects overall mortality.

221 In addition, many trauma patients are also intoxicated, which generally wo

222 However, the use of opioid prescriptions in trauma patients at hospital discharge has not been explo

223 hnicians arriving with intentionally injured trauma patients from January 1 to December 31, 2013 at a

224 ns from a cohort of 280 consecutive civilian trauma patients from University Hospitals of Lyon, Franc

225 on of the ESA epoetin alfa to critically ill trauma patients has been associated with a reduction in

226 More than half of older trauma patients in this study had sarcopenia, osteopenia

227 acute lung injury that could be targeted in trauma patients prone to early lung dysfunction.

228 telets-to-packed RBCs described in trials of trauma patients were not observed in this analysis of a

229 e NEXUS Head CT DI reliably identifies blunt trauma patients who require head CT imaging and could si

230 Trauma patients with alcohol withdrawal syndrome experie

231 Among 451 trauma patients with an IVC filter and 1343 matched cont

232 One hundred thirty-five adult blunt trauma patients with hemorrhagic shock who survived beyo

233 PATIENTS OR Trauma patients with injury severity scores greater than

234 affects how patients with traumatic injury (trauma patients) interact with locoregional health care

235 populations, such as pediatric and pregnant trauma patients, and the potential for future research,

236 puted tomographic (CT) head imaging in blunt trauma patients.

237 were independent predictors of mortality in trauma patients.

238 ion of damage control (DC) for management of trauma patients.

239 ore effective than UH for VTE prophylaxis in trauma patients.

240 Trauma recidivists were 7% of the total trauma population from 1997 to 2008, representing 3147 p

241 dered a random act, geographical patterns of trauma potentially emerge.

242 on-related hippocampal activation soon after trauma predicted future PTSD symptom severity.

243 Acoustic enrichment immediately after noise trauma prevents circuit reorganizations and gap detectio

244 om blood of female participants in the Grady Trauma Project and found that serum estradiol levels ass

245 Trauma/PTSD status (based on structured diagnostic inter

246 ar mixed models, we examined associations of trauma/PTSD status with biomarkers measured twice, 10 to

247 tive research as part of a larger project on trauma quality improvement practices in Peru.

248 CM exposure was assessed using the Childhood Trauma Questionnaire.

249 associated with exposure to repetitive brain trauma (RBT) continues to strengthen.

250 The Trauma Readiness Index for experts (mean [SD], 74 [4]) w

251 alth need to develop interventions to reduce trauma recidivism and preventable death.

252 Trauma recidivists were 7% of the total trauma populatio

253 ctive cohort study using the Victorian State Trauma Registry (VSTR) was undertaken.

254 The trauma registry was linked to the National Death Index t

255 etrospective cohort constructed from a state trauma registry was linked to the statewide death regist

256 Fifteen trauma-related deaths occurred during the bike segment.

257 modulate longer-lasting fear in anxiety- and trauma-related disorders.

258 ing to reduce the development of stress- and trauma-related disorders.

259 d individuals who are at risk for developing trauma-related disorders.

260 recurring avoidance of situations that evoke trauma-related fears.

261 The prevalence of any trauma-related hospital admission was 10% (105 753 per 1

262 More frequent trauma-related hospital admissions in childhood, and bei

263 ing of a central yet understudied symptom of trauma-related psychopathology.

264 The burden of ocular trauma resulting from the recent earthquakes in Nepal ha

265 nglish-speaking patients who had experienced trauma resuscitation and were clinically stable with no

266 physiology and changing injury patterns and trauma resuscitation practices.

267 al physiology as defined by a triage-Revised Trauma Score of 12.

268 omarkers for resilience, or lack thereof, to trauma.SIGNIFICANCE STATEMENT Numerous studies over the

269 nrelated to genetic manipulation or to brain trauma.SIGNIFICANCE STATEMENT Our evidence that inbred a

270 s used to support patients with severe liver trauma (SLT), in ALF patients as a bridge to transplanta

271 Stroke and head trauma stimulate proliferation of endogenous neural stem

272 Bleeding complications arising from trauma, surgery, and as congenital, disease-associated,

273 Provinces with more recommended trauma system components had better patient survival.

274 These findings have implications for trauma system design.

275 reased with increasing number of recommended trauma system elements; adjusted odds ratio = 0.93 (0.87

276 Regionalization of trauma systems assumes a volume-outcome relationship for

277 on is an important component of high-quality trauma systems with access heavily influenced by insuran

278 that patient concerns corresponded well with trauma team goals.

279 Patients perceived trauma team members as competent, efficient, and caring.

280 Participants drew satisfaction from trauma team members' demeanor, expertise, and efficiency

281 optic nerve treatment methods for disease or trauma that result in permanent vision loss.

282 ldren treated in an ED following blunt torso trauma, the use of FAST compared with standard care only

283 For trauma, this drop took place well before DHR.

284 luminal sections without potentially causing trauma to tissue with a traditional OCT endoscope of a 1

285 Analyses by trauma type revealed a clear pattern of PTSD gene expres

286 identified as being significantly altered by trauma using cytometry by time-of-flight, RNAseq technol

287 Bedrock general surgery cases-trauma, vascular, pediatrics, and breast-decreased.

288 tal Disorders, 4th Edition) PTSD due to that trauma was assessed.

289 surgery without a history of preceding knee trauma was associated with radiographic progression of J

290 ty, but not habituation, 5 to 12 weeks after trauma was positively associated with the PTSD symptom i

291 One lifetime trauma was selected randomly for each respondent.

292 For men, all forms of childhood trauma were associated with all forms of intimate partne

293 For women, all forms of childhood trauma were associated with physical intimate partner vi

294 Other etiologies such as hypertension and trauma were less frequent, and in no case was an isolate

295 d 97 consecutive patients with reported knee trauma who underwent 1.5-T magnetic resonance (MR) imagi

296 atios are applied mostly to patients without trauma, who account for nearly 90% of all massive transf

297 ries were more common in patients with chest trauma with CC fractures (13%, 15 of 114) versus patient

298 and complex nature of the immune response to trauma, with immune alterations consistent with both act

299 (13%, 15 of 114) versus patients with chest trauma without CC fractures (4%, 18 of 460) (OR, 3.72; 9

300 cted significantly with three of the earlier traumas (witnessing atrocities, physical violence victim