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1                                                             Treatment with 20% beeswax was the most suitable for industri
2                                                             Treatment with 3P10 reverses excessive lipid oxidation in tum
3                                                             Treatment with adipogenic medium induced a significant increm
4                                                             Treatment with aminoglycosides such as streptomycin or gentam
5                                                             Treatment with anti-vascular endothelial growth factor agents
6                                                             Treatment with icatibant (bradykinin-B2-antagonist) or anakin
7                                                             Treatment with lovastatin, a mevalonate biosynthesis inhibito
8                                                             Treatment with naked anti-CD26 mAb KU44.13A did not have any
9                                                             Treatment with Tat-A and Tat-B caused membrane disruption and
10                                                             Treatment with the CSF1R inhibitor GW2580 significantly atten
11                                                             Treatment with TNF inhibitors in RA patients reverses the exp
12                                                In addition, treatment with cGMP significantly enhanced Ryr2 expression in
13 wer maximum decrease in plasma viremia, respectively, after treatment with a V3 loop bnAb.
14 ementia, but these changes are attenuated or reversed after treatment with the DDR1 inhibitor, nilotinib.
15                 Rab27b KD also decreased LC3 turnover after treatment with an autophagosome-lysosome fusion inhibitor, ch
16                                  Both depletion of Pyk2 and treatment with a Pyk2 kinase inhibitor increased viral DNA co
17  tissues and in response to environmental stressors such as treatment with heat, chemicals, or virus elicitors of UPR.
18 ing of reduced child mortality in communities with biannual treatment with azithromycin remain unclear.
19  monthly) from 12 cAMR patients who failed standard of care treatment with intravenous immune globulin + rituximab with o
20 ced GSL and monosialotetrahexosylganglioside levels, and co-treatment with standard chemotherapeutics sensitized cells to
21                                                Conventional treatment with an anti-PD-L1 mAb had no significant antitumor
22 sistent benefits for subgroups based on age, sex, diabetes, treatment with an ARNI and baseline eGFR, but suggested treat
23                                      Finite, short-duration treatment with a clinically relevant dose of GS-9688 is well
24 and STAT3 might overcome drug resistance encountered during treatment with single agent alone.
25 sms involved in PDAC metastases were investigated following treatment with P-AscH(-).
26 e extent of surgery, and lately also to select patients for treatment with type 2 biologics.
27                                                Furthermore, treatment with CI- and SEC-exosomes leads to differential bio
28 g/L Zn and 2.81 g/L Cu, which was subjected to hydrothermal treatment with the addition of levulinic acid (LA).
29                              In alpha2delta-1 gene KO mice, treatment with FK506 failed to increase the frequency of NMDA
30 V infections, it has been hypothesized that the benefits of treatment with renin-angiotensin system inhibitors in SARS-Co
31 e best of our knowledge, this is the first reported case of treatment with retrobulbar triamcinolone injections.
32                                                  One day of treatment with tropisetron improved both cognitive and P50 in
33 IV infection, as these cells can persist despite decades of treatment with antiretroviral therapy (ART).
34                                                      Add-on treatment with BIO for only 4 weeks attenuates progression of
35 ed high-dose melphalan and stem cell transplantation and/or treatment with a proteasome inhibitor.
36  or PH is unknown.Objectives: To evaluate whether postnatal treatment with rhIGF-1 (recombinant human IGF-1)/BP3 (binding
37                                                         Pre-treatment with SKF-83566, a D1 receptor antagonist, attenuate
38 homa, who relapsed or had refractory disease after previous treatment with one to three systemic regimens (with at least
39                             In clinical trials, HCV salvage treatment with Sofosbuvir/Velpatasvir/Voxilaprevir (SOF/VEL/V
40                                                  Subsequent treatment with low-dose EPO triggered robust RBC production i
41 ient with schwannomatosis-associated N/S HNST, and targeted treatment with the small-molecule ERBB inhibitor lapatinib le
42                           These data suggest that long-term treatment with gemcitabine leads to extensive reprogramming o
43 ve and P50 inhibition deficits, suggesting that longer term treatment with alpha7 nAChR agonists for these deficits in SC
44 alized origami slows bacterial growth more effectively than treatment with free lysozyme.
45                                 We further demonstrate that treatment with radiation and polyinosinic-polycytidylic acid
46  histologic assessment of retinal morphology, we found that treatment with these flavonoids prior to light insult remarka
47                                                We show that treatment with lysozyme-functionalized origami slows bacteria
48                                                         The treatment with FDC of sofosbuvir and daclatasvir achieved hig
49 suffering from ulcerative colitis (UC), in which first-time treatment with vedolizumab was initiated.
50 ssel and/or left main coronary artery disease randomized to treatment with PCI or CABG in the SYNTAX trial.