ライフサイエンスコーパス: trematode

1 hat Boudicca is actively transcribed in this trematode.

2 sed to isolate E. risticii from the infected trematode.

3 physiological susceptibility of fish to this trematode.

4 ng to their sources: horse blood or infected trematodes.

5 cariae and sporocysts of digenetic virgulate trematodes.

6 ratures than either cold- or warm-acclimated trematodes.

7 HDM-1 may be involved in heme homeostasis in trematodes.

8 gastropod immune molecule and resistance to trematodes.

9 a major group of snail pathogens, digenetic trematodes.

10 cts containing encysted N. risticii-infected trematodes.

11 following priming with attenuated digenetic trematodes (acquired resistance) in this model invertebr

12 terest for understanding heme homeostasis in trematodes and as potential targets for the development

13 s an obligate intracellular bacterium of the trematodes and mammals.

14 suggest that MF6p/HDMs can transport heme in trematodes and thereby shield the parasite from the harm

15 Digenean trematodes are a large, complex group of parasitic flatw

16 abundance of these often debilitating larval trematodes (atrazine alone accounted for 51%).

17 collectively accounted for 87% of the total trematode biomass within the three ponds.

18 cloned and characterised from the parasitic trematode blood fluke Schistosoma haematobium.

19 sterase (AChE) present on the surface of the trematode blood fluke Schistosoma has been implicated in

20 crease exposure and susceptibility to larval trematodes by augmenting snail intermediate hosts and su

21 s developing stages (metacercariae) of these trematodes cause reductions in minnow activity, growth a

22 tures above 22 degrees C the snails released trematode cercariae tentatively identified as virgulate

23 cretions was associated with the presence of trematode cercariae.

24 asitic roundworms (nematodes) and flatworms (trematodes), collectively known as helminths.

25 se cercariae for 3-4 months a year, the pond trematode communities produced an average of 153 mg m(-2

26 nd is frequently parasitized by the digenean trematode Cryptocotyle lingua.

27 Mid-summer trematode dry biomass averaged 0.10 g m(-2), which was e

28 two amphibian parasites (ranaviruses and the trematode Echinoparyphium sp.), we examined the influenc

29 After infection with the digenetic trematode Echinostoma paraensei, hemolymph of the snail

30 Opisthorchis viverrini is a fish-borne trematode endemic in East Asia.

31 Trematode exposure increased mortality and malformations

32 protein secreted by the parasitic flatworm (trematode) Fasciola hepatica that belongs to a broad fam

33 The aetiological agents of this disease are trematode flatworms (Schistosoma) that live and lay eggs

34 bilharzia--is a parasitic disease caused by trematode flukes of the genus Schistosoma.

35 Water borne cercaria(ae) of the trematode genus Schistosoma rapidly penetrate host skin.

36 e situation that the genomes of cestodes and trematodes have lost the piwi and vasa genes that are ha

37 Infection with the trematode helminth Schistosoma mansoni results in a para

38 In infection with the trematode helminth Schistosoma mansoni, the severity of

39 arvae, which are both important intermediate trematode hosts, dominated the dry biomass of free-livin

40 Common parasitic castrators include larval trematodes in snails, and isopod and barnacle parasites

41 e Fasciola MF6p/FhHDM-1 are present in other trematodes, including Clonorchis, Opistorchis, Paragonim

42 Lymnaea stagnalis, a cosmopolitan vector of trematodes infecting diverse vertebrates, whose ancestra

43 empted to examine the synergistic effects of trematode infection and exposure to chemical contaminant

44 nitrogen-poor litter species should decrease trematode infection in tadpoles via density- and trait-m

45 h polyphenolic levels, which should increase trematode infection via trait-mediated effects on tadpol

46 7: wording changed to 'which should increase trematode infection via trait-mediated effects on tadpol

47 s conclusively demonstrated that exposure to trematode infection was required for the development of

48 d laboratory experiments that link increased trematode infection, and increased limb deformities, to

49 une mechanisms to resist different stages of trematode infection, and that each set of mechanisms has

50 broad categories: chemical contaminants and trematode infection.

51 ng effects on tadpole per capita exposure to trematode infection.

52 g this outcome are in need of further study, trematode infections appear to benefit hosts that are ex

53 l relationship between atrazine and elevated trematode infections in amphibians.

54 urd uses examples of parasitized insects and trematode infections of snails to consider the evolution

55 known immunomodulators in human nematode and trematode infections, C. elegans is unique as a non-para

56 ng data from a study of Schistosoma mansoni (trematode) infections in Biomphalaria glabrata snails, w

57 s consistent with mechanical perturbation by trematode infestation but not with the effects of retino

58 ti-centennial scale changes in prevalence of trematodes infesting the bivalve host Abra segmentum thr

59 o digenetic trematode parasites, and bind to trematode larval surfaces, suggestive of a role in inter

60 e and snails, and had tadpoles with elevated trematode loads, further supporting a causal relationshi

61 Trematode.net is an independent component of Helminth.ne

62 d user-friendly manner and (ii) we introduce Trematode.net, a site dedicated to the dissemination of

63 ntroduces and presents its new sibling site, Trematode.net.

64 a collection of databases: Nematode.net and Trematode.net.

65 The trematodes obtained from each snail were pooled and test

66 Fasciola hepatica is a parasitic trematode of global importance in livestock.

67 ndemic parasitic infection caused by various trematodes of the genus Schistosoma.

68 logenetic relatives of these helminths, also trematodes of the phylum Platyhelminthes and major human

69 Trematodes or tadpoles were independently acclimated to

70 riae) of two of their most common species of trematode, Ornithodiplostomum ptychocheilus and Posthodi

71 in the extremely O2-avid hemoglobin from the trematode Paramphistomum epiclitum have been investigate

72 ion-resistant oxy-myoglobin complex from the trematode Paramphistomum epiclitum, and the residues wer

73 Sequence alignment of hemoglobins of the trematodes Paramphistomum epiclitum and Gastrothylax cru

74 es of growth, development and survival), the trematode parasite (production of the cercaria infective

75 ogs (Hyla regilla) exposed to cercariae of a trematode parasite (Ribeiroia sp.).

76 Here, we combined field-derived estimates of trematode parasite infections in aquatic snails with mea

77 The trematode parasite Ribeiroia ondatrae sequentially infec

78 frog tadpoles (Lithobates clamitans) by the trematode parasite Ribeiroia ondatrae with fully replica

79 Aquatic larvae (cercariae) of the trematode parasite Schistosoma mansoni rapidly penetrate

80 Infection with the trematode parasite Schistosoma mansoni results in a dist

81 emarkable activity against the blood-feeding trematode parasite Schistosoma mansoni.

82 e major intermediate hosts for the digenetic trematode parasite Schistosoma mansoni.

83 ish were exposed to a fixed dose of a common trematode parasite.

84 Using a guild of larval trematode parasites (six species) and an amphibian host,

85 ecosystem-level biomass and productivity of trematode parasites alongside the biomass of free-living

86 workers on the role of NO in defense against trematode parasites and of Kanazawa et al. on cestode pa

87 was postulated that components derived from trematode parasites block receptors on the defence cells

88 and replication in their intermediate hosts, trematode parasites down regulate host defence responses

89 ylus variegatus and Aplodactylus punctatus), trematode parasites for a keyhole limpet (Fissurella lat

90 a significant amount of energy moves through trematode parasites in freshwater pond ecosystems, and t

91 up-regulated following exposure to digenetic trematode parasites, and bind to trematode larval surfac

92 involving freshwater snails, amphibians and trematode parasites, we conducted a year-long, outdoor e

93 s), that are secreted by medically important trematode parasites.

94 cept - subversion of host cell signalling by trematode parasites.

95 sible candidates) of the abundance of larval trematodes (parasitic flatworms) in the declining northe

96 will lead to significant intensification of trematode parasitism, suppressed fecundity of common ben

97 velopmental pathway in the life cycle of the trematode pathogen Schistosoma mansoni.

98 uence of the 16S rRNA gene identified in one trematode pool was identical to that of the type strain

99 Out of a total of 209 trematode pools, 50 pools were found to be positive by P

100 ographic regions and persistent infection of trematode populations with E. risticii during summer and

101 nly consume free-living cercariae (parasitic trematodes) reduced metacercarial infections in tadpoles

102 nts to test how transmission of the virulent trematode Ribeiroia ondatrae was affected by the diversi

103 ns: wetlands with a greater abundance of the trematode Ribeiroia ondatrae were more likely to have ma

104 trials with P. regilla and the most virulent trematode (Ribeiroia ondatrae), experimental reductions

105 dies has implicated infection by a digenetic trematode--Ribeiroia ondatrae--as an important cause of

106 tal schistosomiasis, caused by the parasitic trematode Schistosoma haematobium, affects over 112 mill

107 and chronic disease caused by the parasitic trematode Schistosoma mansoni after deposition of eggs i

108 while conferring immunity to the intestinal trematode Schistosoma mansoni Here, we report that abrog

109 The intravascular trematode Schistosoma mansoni is a causative agent of sc

110 The trematode Schistosoma mansoni is one of the etiological

111 The digenetic trematode Schistosoma mansoni that causes the form of sc

112 Biomphalaria glabrata to infection with the trematode Schistosoma mansoni.

113 e a population of neoblast-like cells in the trematode Schistosoma mansoni.

114 tein produced exclusively by the eggs of the trematode Schistosoma mansoni.

115 g lymphoid tissues of mice infected with the trematode Schistosoma mansoni.

116 oots with potent activity against pathogenic trematode Schistosoma mansoni.

117 One representative example is the trematode Schistosoma, which causes schistosomiasis, an

118 new NRs were identified in the Platyhelminth trematode, Schistosoma mansoni.

119 bout whether similar cell types exist in any trematode species.

120 ausative agent of Potomac horse fever, using trematode stages collected from Juga yrekaensis snails.

121 Upon exposure to infection with digenetic trematodes such as Echinostoma paraensei, the freshwater

122 ivity of heme, and (ii) MF6p/HDMs from other trematodes, such as Opisthorchis viverrini and Paragonim

123 rphosis) in our tadpole-parasitic cercarial (trematode) system, would be a negative and positive pred

124 mature snails were infected with one of six trematode taxa, amounting to a density of 13 infected sn

125 e effects of atrazine were consistent across trematode taxa.

126 On average, each trematode taxon produced between 14 and 1660 free-swimmi

127 Trematodes that were acclimated to intermediate temperat

128 thin each infected snail consisted of larval trematode tissue, which collectively accounted for 87% o

129 ived from gene duplication, as are all known trematode TKs.

130 risticii was found to be transmittable from trematodes to mice and was subsequently passaged from in

131 The annual production of free-swimming trematode transmission stages was greater than the combi

132 e exhibiting elevated abundance was the same trematode used in the laboratory infection.

133 The biomass of trematodes was particularly high, being comparable to th

134 ium, five nematodes, three cestodes, and one trematode) were included in the negative specimens.