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1 synthetic membranes, and the accumulation of triacylglycerol.
2 l role in the synthesis of the storage lipid triacylglycerol.
3 placement followed by a late accumulation of triacylglycerol.
4  the esterification of free fatty acids into triacylglycerol.
5 ids, accumulation of oligogalactolipids, and triacylglycerol.
6 tions of CPS on phosphatidylcholine (PC) and triacylglycerol.
7 tion, coincident with increased synthesis of triacylglycerol.
8 osphatidylcholine, and ultimately enter seed triacylglycerol.
9 cles fail to release fatty acids from stored triacylglycerol.
10 free sugars on total and LDL cholesterol and triacylglycerols.
11 thesis and its regulation to the assembly of triacylglycerols.
12 ounced reallocation of lipidome peak area to triacylglycerols.
13 the essentiality of major classes, including triacylglycerols.
14 cetylation required the conversion of FAs to triacylglycerols.
15 linolein/oleodilinolein represented the main triacylglycerols.
16 ydrolyze omega-3 fatty acids from structured triacylglycerols.
17 and the pattern in NEFAs echoed that of VLDL-triacylglycerols.
18 , respectively], only whey protein decreased triacylglycerol (-0.23 mmol/L; P = 0.025) compared with
19 ealthy controls was dominated by lipoprotein triacylglycerol (1)H NMR resonances and isoleucine.
20 s in the intensities of selected lipoprotein triacylglycerol (1)H NMR signals over those of healthy c
21 g and detecting lipids, namely, cholesterol, triacylglycerols, 1,2-diol diesters, wax esters, cholest
22 /- 3.6%; n = 13) exhibited a 45% higher VLDL-triacylglycerol 16:1n-7 molar percentage (P < 0.01), 16%
23 data provide support for the use of the VLDL-triacylglycerol 16:1n-7 molar percentage as a biomarker
24                        In all subjects, VLDL-triacylglycerol 16:1n-7 was significantly (P < 0.01) rel
25               Phospholipids (40.2-43.6%) and triacylglycerols (31.8-34.7%) were the most represented
26  those of DHA supplementation (re-esterified triacylglycerol; 90% pure) on inflammation markers (prim
27  was modified with a decreased proportion of triacylglycerol accompanied by the increase of phospholi
28                       The resultant purified triacylglycerols accomplished with the oxidative state (
29 te also inhibits v-ATPase function, yielding triacylglycerol accumulation but not insulin resistance.
30 mice exhibited a marked reduction in hepatic triacylglycerol accumulation compared with wild type obe
31  lipid-producing strain (45-fold increase in triacylglycerol accumulation) through the disruption of
32  AtGPAT9 is required for wild-type levels of triacylglycerol accumulation, and the transcript level i
33 OLEOSIN1 in Nicotiana benthamiana stimulates triacylglycerol accumulation, but their coexpression wit
34 es, potentially reduce the uptake of dietary triacylglycerol aiding in weight management.
35 ses in HDL cholesterol, LDL cholesterol, and triacylglycerols, although for LDL cholesterol and triac
36 ocytes incorporated 33% less fatty acid into triacylglycerol and 46% more into the pathway of beta-ox
37 sition of sn-1,2-diacylglyerol, thus forming triacylglycerol and a lysophospholipid.
38 terol biosynthesis, whereas SREBP-1 controls triacylglycerol and glycerophospholipid biosynthesis.
39  missing is an SREBP-1 analog that regulates triacylglycerol and glycerophospholipid homeostasis in r
40 blish Mga2 as a transcriptional regulator of triacylglycerol and glycerophospholipid homeostasis in S
41  genes, and mga2Delta cells showed disrupted triacylglycerol and glycerophospholipid homeostasis, mos
42                    Mga2 and SREBP-1 regulate triacylglycerol and glycerophospholipid synthesis, where
43 he levels of PA and DAG for the synthesis of triacylglycerol and membrane phospholipids, the growth o
44                                              Triacylglycerol and phosphatidylcholine molecular specie
45  ER was able to incorporate fatty acids into triacylglycerol and phospholipids.
46     Later isolates displayed accumulation of triacylglycerol and reduced expression of fadD23, someti
47  the msn2Deltamsn4Delta strain had increased triacylglycerol and steryl ester levels.
48 t on the NP-HPTLC plates, whereas individual triacylglycerol and wax ester species were separated on
49                             Plasma levels of triacylglycerols and diacylglycerols, the lipoproteins t
50  High resolution (13)C NMR spectra of plasma triacylglycerols and glucose provided new insights into
51 n the endoplasmic reticulum in cells lacking triacylglycerols and localize exclusively to the endopla
52    For example, mixed cis and trans forms of triacylglycerols and phosphatidylcholines were identifie
53 ial distribution of two major lipid species, triacylglycerols and phosphatidylcholines.
54          Very-low-density lipoprotein (VLDL)-triacylglycerols and plasma free FA [nonesterified fatty
55 s are specific organelles for the storage of triacylglycerols and steryl esters.
56 hosphatidylethanolamine, phosphatidylserine, triacylglycerol, and cholesteryl ester.
57 erol, hypertension, high blood glucose, high triacylglycerol, and insulin resistance.
58 ral important lipids, including cholesterol, triacylglycerol, and phospholipids.
59  reduced plasma levels of apoC-II, apoC-III, triacylglycerols, and diacylglycerols, and increased apo
60 es from baseline in fasting and postprandial triacylglycerol, apolipoprotein B-48 (apoB-48; reflectin
61  high-density lipoprotein (HDL) cholesterol, triacylglycerols, apolipoproteins A-I and B, or very low
62 iogenesis and conversion of phospholipids to triacylglycerol are required for restoring some phosphol
63 itative data are reported on fatty acids and triacylglycerols as relative percentage of total fractio
64 algae that accumulate significant amounts of triacylglycerols as storage lipids when their growth is
65 ained oleosin and several other proteins and triacylglycerols as the main lipids.
66                  Focusing on engineering the triacylglycerol assembly mechanisms led to modest increa
67 d polar compounds formation in sunflower oil triacylglycerols at 120 degrees C were investigated in t
68 he position of the omega-3 fatty acid on the triacylglycerol backbone influences how digestion occurs
69 incided with elevated gene expression of key triacylglycerol biosynthesis components.
70           Although the importance of PDAT in triacylglycerol biosynthesis has been illustrated in som
71 nging together gene expression levels of the triacylglycerol biosynthesis pathway and oil composition
72 th the others, including diacylglycerols and triacylglycerols, branched-chain amino acids, and marker
73 cing pancreatic lipase activity would reduce triacylglycerol breakdown resulting in lower amounts bei
74    Pancreatic lipase is important in dietary triacylglycerol breakdown; reducing pancreatic lipase ac
75 abolism and accumulate more intramyocellular triacylglycerol but have normal glucose and insulin tole
76 ns of the environment by the accumulation of triacylglycerol, but the relative changes occurring in m
77 either nutrient triggered an accumulation of triacylglycerol, but with different time scales and magn
78 adiposity index, whole body weight, glucose, triacylglycerol, cholesterol and blood pressure, without
79  production; stored within lipid droplets as triacylglycerol, cholesterol esters, and retinol esters;
80      Six lipid species (all belonging to the triacylglycerol class and containing palmitate at the fi
81          The HF+PO diet reduced the level of triacylglycerols compared with the control.
82  a more direct instrumental determination of triacylglycerol composition along with sn-2 positional i
83 erification process for their fatty acid and triacylglycerol composition, free fatty acid (FFA) conte
84 choline, leading to a proportional change in triacylglycerol composition.
85 on the quality parameters and fatty acid and triacylglycerol compositions of their oils.
86                                Elevations in triacylglycerol concentration relative to baseline were
87                              The lower serum triacylglycerol concentration was associated with reduce
88                        Insulin, glucose, and triacylglycerol concentrations as well as blood pressure
89 ementation attenuated the increase in plasma triacylglycerol concentrations during the HFMM test that
90 acuoles suggested vacuolar storage of NO3(-) Triacylglycerol concentrations in the NR-KO cells increa
91 urces, which coincided with increased plasma triacylglycerol concentrations.
92  P = 0.03) and postprandial (P = 0.01) serum triacylglycerol concentrations.
93                           Natural sources of triacylglycerols containing omega-3 fatty acids are of p
94 38 lipases with a large number of structured triacylglycerols containing omega-3 fatty acids.
95 lation by Pho85-Pho80, caused an increase in triacylglycerol content and lipid droplet number in cell
96            All mutants analyzed had a higher triacylglycerol content and perturbed whole-cell fatty a
97                         Although an elevated triacylglycerol content in non-adipose tissues is often
98 ted high liver glycogen content, lower liver triacylglycerol content, and lower serum concentrations
99 evidenced by the 2.25-fold increase in liver triacylglycerol content, but did not induce advanced liv
100 insulin resistance, and elevation in hepatic triacylglycerol content.
101                            Variations in the triacylglycerol contents and melting and crystallization
102                                        Liver triacylglycerol contents were reduced by both protein so
103 t, the low amount of dietary 16:1n-7 in VLDL-triacylglycerols corresponded to a stronger signal of el
104 t the time evolution of the concentration of triacylglycerols, DAG, MAG and free fatty acids (FFA) an
105 roper neutral lipid compartmentalization and triacylglycerol degradation during postgerminative growt
106 striking, consisting of completely saturated triacylglycerol, diacylglycerol, and monoacylglycerol wi
107 concentrations, and decreased ectopic lipid (triacylglycerol/diacylglycerol) content in liver and mus
108 ly into the stomach, we show that inhibiting triacylglycerol digestion disrupts normal behaviour of s
109        In this study we examined the role of triacylglycerol digestion for intestinal fat sensing, an
110 fat sensing, and compared the effects of the triacylglycerol digestion products, fatty acids and 2-mo
111  monomers at IP, min/%; time reaching 10% of triacylglycerol dimers and polymers, min) in general bel
112  the cessation of growth and accumulation of triacylglycerols during nitrogen starvation, and the tem
113 io of 16:1n-7 to 16:0 [SCD1(1)(6)] in plasma triacylglycerol FA has been used as an index to reflect
114       We found no difference in postprandial triacylglycerol, FFA, insulin, glucose, glucagon, or GIP
115 nitrogen deprivation, were unable to degrade triacylglycerols following nitrogen resupply.
116 membrane separation produces products in the triacylglycerol form which possess better oxidative stab
117 HFA), only accumulates to high levels in the triacylglycerol fraction of castor (Ricinus communis) en
118 cyl-CoA synthetase LCS2 in the production of triacylglycerol from de novo-synthesized fatty acids.
119                                 Furthermore, triacylglycerols from HepG2-SMS1 cells are enriched in p
120 sterol, low-density lipoprotein cholesterol, triacylglycerol, glucose, insulin, C-peptide, homeostasi
121 ects on fasting or postprandial cholesterol, triacylglycerol, glucose, or hepatic insulin clearance i
122                                 Postprandial triacylglycerol, HDL-cholesterol, HDL(3)-cholesterol, an
123 acids up to 17% of total fatty acids in seed triacylglycerols; however, total seed oil is also reduce
124    No difference in effects was observed for triacylglycerol, hsCRP, insulin, and glucose concentrati
125 selective inhibitor of the key intracellular triacylglycerol hydrolase, adipose triglyceride lipase.
126                          Lipases, a class of triacylglycerol hydrolases, have been extensively tested
127 zes the penultimate step in the synthesis of triacylglycerol (i.e. the production of diacylglycerol b
128 lgae oils have been studied in detail, their triacylglycerols identified and sn-2 positional arrangem
129 ntration was associated with reduced fasting triacylglycerol in chylomicrons and very-low-density lip
130 nd of major membrane lipids in seedlings and triacylglycerol in mature siliques.
131 mmonly is used to induce the accumulation of triacylglycerol in microalgae, leads to a state of cellu
132 yzes the committed step for the synthesis of triacylglycerol in Saccharomyces cerevisiae, exerts a ne
133                           Sensing of dietary triacylglycerol in the proximal small intestine results
134  we report that resting levels of long-chain triacylglycerols in mitochondrial myopathy correlate wit
135 ch is known about the role of LDs in storing triacylglycerols in seeds, their biogenesis and function
136 es of cholesterol esters, phospholipids, and triacylglycerols in the context of lesion characteristic
137 all plant membrane phospholipids and storage triacylglycerols is catalyzed by a glycerol-3-phosphate
138                Arabidopsis SEIPINs increased triacylglycerol levels and altered composition.
139 hese mice had reductions in both circulating triacylglycerol levels and the mRNA levels of lipogenic
140                     Reduced plasma and liver triacylglycerol levels in E1 and E4-mice were linked to
141 ol promotes the reduction of cholesterol and triacylglycerol levels.
142  associated with decreased serum glucose and triacylglycerol levels.
143 s on ECHIDNA (ECH), a plant homolog of yeast Triacylglycerol lipase (TLG2/SYP4) interacting protein T
144  we studied the topology of Tgl3p, the major triacylglycerol lipase of the yeast Saccharomyces cerevi
145 mutants defective in SUGAR-DEPENDENT1 (SDP1) triacylglycerol lipase or PEROXISOMAL ABC TRANSPORTER 1
146 differentially expressed bacterial proteins (triacylglycerol lipase, N-acetylmuramoyl-L-alanine amida
147  investigate the regulatory network of yeast triacylglycerol lipases in more detail, we also examined
148        Tgl3p, Tgl4p, and Tgl5p are the major triacylglycerol lipases of the yeast Saccharomyces cerev
149 showed increased expression of mRNAs for the triacylglycerol lipases PPA2105 and PPA1796 and the hyal
150  these investigations to the two other yeast triacylglycerol lipases, Tgl4p and Tgl5p.
151     Total cholesterol (TC), LDL cholesterol, triacylglycerol, lipoprotein(a), and apolipoprotein B we
152 ls contain subcellular lipid droplets with a triacylglycerol matrix enclosed by a layer of phospholip
153 wed that Ime4 epitranscriptionally regulates triacylglycerol metabolism and vacuolar morphology throu
154     A number of individual genes involved in triacylglycerol metabolism have previously been reported
155 exposure are the phospholipid and fatty acid triacylglycerol metabolism pathways.
156 dentified here between m(6)A methylation and triacylglycerol metabolism via the Ime4 protein provides
157 get for the engineering of high-biomass/high-triacylglycerol microalgae.
158  basis of their structure or position in the triacylglycerol molecule.
159 ant performance (the ratio of IP to oxidized triacylglycerol monomers at IP, min/%; time reaching 10%
160 zation of phosphatidate for the synthesis of triacylglycerol or membrane phospholipids.
161 e any systematic effect on fatty acids (FA), triacylglycerols or nutritional fat subclasses but signi
162 er that DDHD2 regulates brain triglycerides (triacylglycerols, or TAGs).
163 s or any secondary outcomes including plasma triacylglycerols, oxidized LDL, and LDL cholesterol.
164                           c9,t11-CLA lowered triacylglycerol (P </= 0.01) and had no effect on other
165 0(-7)), fasting insulin (P=5.4x10(-32)), and triacylglycerols (P=1.4x10(-29)).
166 , blood pressure (P=7.7x10(-5)), and fasting triacylglycerols (P=9.0x10(-5)), and PC14:1/0:0 was posi
167                           As a percentage of triacylglycerol palmitate, de novo lipogenesis was 2-fol
168 glycerol between the phosphatidylcholine and triacylglycerol pathways, to the benefit of the former.
169 ains across lipid species, including di- and triacylglycerols, phospholipids, cholesteryl esters, and
170 mmon Kilka (Clupeonella cultiventris caspia) triacylglycerols (PKO) as affected by 1-1.5% (w/w) of un
171  the highly regulated storage and release of triacylglycerol primarily in adipose tissue, and excessi
172 to gain a basic mechanistic understanding of triacylglycerol production in photosynthetic cells.
173 Apart from these methods, the specificity of triacylglycerol profiles has previously been detected in
174 s the effect of irradiation treatment in the triacylglycerol profiles of chestnut.
175 ds, liposoluble antioxidants, fatty acid and triacylglycerol profiles, and oxidative status of oil ob
176 ly associated with the level of intrahepatic triacylglycerol (r = 0.53; P = .007).
177 2% and approximately 14% variance for FA and triacylglycerols, respectively) to processing.
178 ol (vitamin A), esterified and packaged into triacylglycerol-rich chylomicrons for bodily distributio
179 ation rewires cellular metabolism to adopt a triacylglycerol-rich lifestyle reliant on the Kennedy pa
180 Blood was collected hourly for 10 h, and the triacylglycerol-rich lipoprotein (TRL) fraction was isol
181        ApoC-II is provided as a component of triacylglycerol-rich lipoproteins and is the co-factor f
182 l community support the concept of targeting triacylglycerol-rich lipoproteins to reduce risk of CVD.
183 r lipoprotein lipase (LPL) at the surface of triacylglycerol-rich lipoproteins.
184 lar diacylglycerol acyltransferase activity, triacylglycerol secretion, and mitochondrial function.
185 ining linoleic acid (LA), and more saturated triacylglycerol species than control eggs.
186 tes within holobionts featured predominantly triacylglycerols, sterol esters, and free fatty acids.
187  generation, insulin resistance, and reduced triacylglycerol storage.
188 PL) activity and stimulates the lipolysis of triacylglycerol stored by adipocytes in the white adipos
189 tinued to run, liver and muscle glycogen and triacylglycerol stores were similar in both genotypes; h
190                  We found that the uptake of triacylglycerol substrates via the scavenger receptor CD
191 atase that catalyzes the penultimate step in triacylglycerol synthesis and plays a role in the transc
192 rsion of phosphatidate to diacylglycerol for triacylglycerol synthesis and simultaneously controls ph
193     These include transcripts for starch and triacylglycerol synthesis but also transcripts for photo
194            Intriguingly, the coexpression of triacylglycerol synthesis isozymes from castor along wit
195 te that these unusual structures recruit the triacylglycerol synthesis machinery and grow by expansio
196 f Pah1 and its physiological functions (e.g. triacylglycerol synthesis).
197 arker of pentose phosphate pathway activity, triacylglycerol synthesis, and flux through anaplerotic
198 hatases and catalyze the penultimate step of triacylglycerol synthesis.
199 h is the penultimate step in one pathway for triacylglycerol synthesis.
200 iates in the glycerol-3-phosphate pathway of triacylglycerol synthesis: diacylglycerol (DAG), which m
201 green alga C. reinhardtii showed substantial triacylglycerol (TAG) accumulation and up-regulation of
202 actylum NO inhibits cell growth and triggers triacylglycerol (TAG) accumulation.
203  (66% of 1,3-DAG, 18% of 1,2-DAG) and 16% of triacylglycerol (TAG) along with micro nutrients like ga
204           WD-induced accumulation of hepatic triacylglycerol (TAG) and diacylglycerol (DAG) content w
205  the accumulation of high cellular levels of triacylglycerol (TAG) and starch are variants of what ma
206                Neutral lipids, predominantly triacylglycerol (TAG) and sterol ester, are stored withi
207          The neutral lipids steryl ester and triacylglycerol (TAG) are stored in the membrane-bound o
208 ses (DGATs) catalyze a rate-limiting step of triacylglycerol (TAG) biosynthesis in higher plants and
209 s between milk FA composition and genes from triacylglycerol (TAG) biosynthesis pathway.
210                                Inhibition of triacylglycerol (TAG) biosynthetic enzymes has been sugg
211 oleaginous plant species is the formation of triacylglycerol (TAG) by the acyl-CoA-dependent acylatio
212  and K270, purity properties; fatty acid and triacylglycerol (TAG) composition and antioxidant compou
213 dy demonstrates a strong interaction between triacylglycerol (TAG) composition and effects of shear r
214 tudy was to gain knowledge about the role of triacylglycerol (TAG) composition in fatty acids (FA) of
215 ncomitantly with MGDG decrease, the level of triacylglycerol (TAG) containing medium chain FAs increa
216                                          The triacylglycerol (TAG) crystal structures and morphologie
217 on of desnutrin/ATGL at S406 to decrease its triacylglycerol (TAG) hydrolase activity, lowering basal
218  triglyceride lipase (ATGL), a major hepatic triacylglycerol (TAG) hydrolase, were inversely regulate
219 ty, lipid synthesis, and the accumulation of triacylglycerol (TAG) in leaf tissue.
220 ycerolipid synthesis and the accumulation of triacylglycerol (TAG) in response to nutrient starvation
221                                              Triacylglycerol (TAG) is a storage lipid used for food p
222                                              Triacylglycerol (TAG) is the main storage lipid in plant
223 lorella and resulted in close agreement with triacylglycerol (TAG) levels determined by thin layer ch
224 triglyceride lipase (ATGL) serves as a major triacylglycerol (TAG) lipase and controls the bulk of in
225 vation in yeast and mammals, but the role of triacylglycerol (TAG) metabolism in plant stress respons
226                                              Triacylglycerol (TAG) metabolism is a key aspect of intr
227                       Enantiomers of racemic triacylglycerol (TAG) mixtures were separated using two
228 1MS2 used for (1)D allowed quantification of triacylglycerol (TAG) molecular species of Parinari cura
229 f how these PUFAs are channeled from PC into triacylglycerol (TAG) needs to be further explored.
230 tudied how pHo influences the stimulation of triacylglycerol (TAG) storage by low pO2 and LPS.
231 of PC that is required for LD biogenesis and triacylglycerol (TAG) storage.
232 in rapid proliferation but are redirected to triacylglycerol (TAG) stored in lipid droplets during st
233   The anabolism and catabolism of myocardial triacylglycerol (TAG) stores are important processes for
234                                        Liver triacylglycerol (TAG) synthesis and secretion are closel
235     Acyltransferases are key contributors to triacylglycerol (TAG) synthesis and, thus, are of great
236 omitantly, increased conversion of MGDG into triacylglycerol (TAG) was observed.
237 ned 69% PL, 9% diacylglycerol (DAG), and 23% triacylglycerol (TAG) with a stoichiometry of 46 PL, 6 D
238                                   Microalgal triacylglycerol (TAG), a promising source of biofuel, is
239 thogen accumulates lipid droplets containing triacylglycerol (TAG).
240 er 75% of fatty acids in white adipose (WAT) triacylglycerol (TAG).
241 l-coenzyme A (CoA)-dependent biosynthesis of triacylglycerol (TAG).
242  underlie a rigid SFA:MUFA:PUFA hierarchy in triacylglycerol (TAG).
243 xpressed in tissues that predominantly store triacylglycerol (TAG).
244  to the diacylglycerol (DAG, 17 species) and triacylglycerol (TAG, 17 species) classes.
245        Plant seeds are the primary source of triacylglycerols (TAG) for food, feed, fuel, and industr
246  two-steps enzymatic esterification and (iv) triacylglycerols (TAG) purification (liquid column chrom
247  (PL), diacylglycerols, free fatty acids and triacylglycerols (TAG) using thin layer chromatography.
248  store excess lipids as two major compounds, triacylglycerols (TAGs) and cholesteryl esters (CEs), in
249                                              Triacylglycerols (TAGs) and steryl esters, which are sto
250 ether relative concentrations of circulating triacylglycerols (TAGs) between carriers compared with n
251                                              Triacylglycerols (TAGs) displayed the complete panel of
252 o be a power combination for the analysis of triacylglycerols (TAGs) from tissue sections by laser de
253 mutants also constitutively over-accumulated triacylglycerols (TAGs) in a manner that was synergistic
254 validated to analyze ratios of regioisomeric triacylglycerols (TAGs) in fats and oils.
255  resulting in an accumulation of unsaturated triacylglycerols (TAGs) in the cytosol.
256 dation is achieved by lipases that hydrolyze triacylglycerols (TAGs) into free fatty acids and glycer
257 de values (PVs) were determined and oxidised triacylglycerols (TAGs) measured by UHPLC-ESI/MS at 0, 7
258 o fingerprint the primary crystallisation of triacylglycerols (TAGs) molecules and their transition b
259                    This CSO contained twelve triacylglycerols (TAGs) out of which trilinolenin (alpha
260 rated and unsaturated FA distribution of the triacylglycerols (TAGs) present in high oleic sunflower
261 idylethanolamines (PEs), sphingomyelins, and triacylglycerols (TAGs) were associated with cardiovascu
262 LC-MS was employed for the identification of triacylglycerols (TAGs), carotenoids and phospholipids;
263 s lose their lipid droplets (LDs) containing triacylglycerols (TAGs), cholesteryl esters, and retinyl
264                                        Plant triacylglycerols (TAGs), or vegetable oils, provide appr
265                           Here, we show that triacylglycerols (TAGs), present in Arabidopsis guard ce
266 NPs that are mainly composed of high melting triacylglycerols (TAGs).
267 ic acid precursor of polar glycerolipids and triacylglycerols (TAGs).
268 ets (LDs) coincides with the accumulation of triacylglycerols (TAGs).
269 sterol and campesterol was incorporated into triacylglycerols (TAGs).
270 acids in the sn-2 and sn-3 positions of seed triacylglycerols (TAGs).
271 sion are redirected toward carbohydrates and triacylglycerols (TAGs).
272 smic lipid droplets (LDs) of neutral lipids (triacylglycerols [TAGs], sterylesters, etc.) are reserve
273  an integral membrane protein that catalyzes triacylglycerol (TG) synthesis using diacylglycerol and
274 rganelles that store neutral lipids, such as triacylglycerol (TG), as reservoirs of metabolic energy
275  to and is stabilized by droplets containing triacylglycerol (TG).
276 r laboratory has shown that the breakdown of triacylglycerols (TGs) is regulated in a cell-cycle-depe
277                                              Triacylglycerols (TGs) stored in lipid droplets (LDs) ar
278            Although mostly non-polar lipids (triacylglycerols, TGs) were present in the fish tissue,
279                  HepG2-SMS1 cells have fewer triacylglycerols than controls but similar diacylglycero
280 els of mycolic acid wax ester and long-chain triacylglycerols than those for wild-type bacteria.
281 the diacylglycerol used for the synthesis of triacylglycerol that accumulates in the stationary phase
282 lglycerols, although for LDL cholesterol and triacylglycerols there was significant heterogeneity bet
283 us quantifying individual fatty acids within triacylglycerols through multivariate linear regression
284  facilitate the movement of CPA from PC into triacylglycerol to produce viable seeds with additional
285 f diacylglyceryl-N,N,N-trimethylhomo-Ser and triacylglycerols, together with the up-regulation of gen
286 oisomer characterisation of fatty acids in a triacylglycerol usually requires the use of stereospecif
287 The release of the first fatty acid from the triacylglycerol was independent on the unsaturation degr
288                              The increase of triacylglycerols was no longer significant when studies
289 fferential localization of phospholipids and triacylglycerols was observed within the embryo and radi
290                      ApoC-II adsorption to a triacylglycerol/water interface resulted in large increa
291     We characterized apoC-II at phospholipid/triacylglycerol/water interfaces, which more closely mim
292 tions between eicosanoids, phospholipids and triacylglycerols, we provide evidence that these lipids
293 e, diacylglycerol (DAG) and intramyocellular triacylglycerol were increased.
294 otal cholesterol, LDL cholesterol, apoB, and triacylglycerol were similar with the 3 diets.
295                               Interestingly, triacylglycerols were found in areas surrounding neovess
296                                  Polymers of triacylglycerols were found only in the polar fraction o
297 e hydrolyzed to free DHA and are absorbed as triacylglycerol, whereas the transporter at blood brain
298 hingolipids, and nonpolar lipids (diacyl and triacylglycerols), which are the main lipid classes dete
299                               LPL hydrolyzes triacylglycerol, which increases local surface pressure
300 etween the theoretical and actual amounts of triacylglycerols with partition number 42 (DeltaECN42 |0

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