ライフサイエンスコーパス: trichrome stain

1 scle actin), and collagen deposition (Masson trichrome stain).

2 and collagen preservation (demonstrated with trichrome staining).

3 volume fraction was quantified, using Masson trichrome stain.

4 collagen deposition as evaluated by Masson's trichrome stain.

5 onserved for modified acid-fast and modified trichrome stains.

6 geal fibrosis on pretreatment biopsies using trichrome staining.

7 d left ventricular (LV) fibrosis as shown by trichrome staining.

8 Plaque composition was characterized by trichrome staining.

9 (in square millimeters) were identified with trichrome staining.

10 or delineation of myocardial structure after trichrome staining.

11 We examined with a modified trichrome stain 124 stool specimens collected over a 2-y

12 es in feces were detected microscopically by trichrome stain and by PCR and within enterocytes by in

13 Analysis of Masson trichrome staining and fibrotic marker protein and mRNA

14 oups, ITB SP-D (-/-) survivors had increased Trichrome staining and tissue hydroxyproline levels.

15 ermate controls as assessed by peribronchial trichrome staining and total lung collagen content.

16 n through the anastomoses were examined with trichrome-staining and immunohistochemistry (IHC) for ac

17 Histologic appearance (hematoxylin-eosin and trichrome staining) and presence of cell inflammation (C

18 (by echocardiography), fibrosis (with Masson Trichrome staining), and apoptosis (with TUNEL staining)

19 ung fibrosis as evidenced by histopathology, trichrome staining, and biochemical analysis for collage

20 nase levels, hematoxylin and eosin, Masson's trichrome staining, and collagen I expression.

21 ssed by lung collagen content, peribronchial trichrome staining, and immunostaining with anticollagen

22 llagen deposition, as visualized by Masson's trichrome staining, and up-regulated mRNA expression of

23 oscopy using hematoxylin and eosin Goldner's trichrome stains, and polarized light and evaluated for

24 Trichrome staining confirmed that the scalpel incisions

25 Masson's trichrome stain demonstrated dense scar tissue, signalin

26 ne levels as well as Sirius red and Masson's Trichrome staining demonstrated advanced portal collagen

27 . duodenalis and C. parvum or C. hominis) or trichrome stain (E. histolytica).

28 The sections were treated with Masson's trichrome stain for light microscopic examination of mus

29 ted vital HSCC; b) serial sections of fixed, trichrome-stained HSCC; and c) scanning electron microsc

30 nd inflammation by hematoxylin and eosin and trichrome staining, IHC, and in situ hybridization (ISH)

31 per total tissue area was assessed in Masson trichrome stained kidney tissues.

32 Analysis of renal scarring by Masson trichrome staining, kidney hydroxyproline levels, and co

33 taining of wet mounts of stool sediments and trichrome staining, lack the sensitivity to detect Crypt

34 cetate concentration to stool specimens, and trichrome staining method in suspicious cases.

35 processed for histochemical studies (Masson trichrome stain), molecular markers of fibrosis (collage

36 Stool specimens were next analyzed by trichrome staining of both unconcentrated and concentrat

37 Masson's trichrome staining of histological sections demonstrated

38 Masson trichrome staining of liver tissue was used to identify

39 subepithelial fibrosis as assessed by either trichrome staining or lung collagen levels.

40 and in methylene blue/azure II/basic fuchsin trichrome-stained plastic sections, suggesting the prese

41 However, modifications to the trichrome staining procedures will be necessary to impro

42 9038 and Spearman rho = 0.8107 [P = .0002 vs trichrome staining]; r = 0.9540 and rho = 0.8821 [P < .0

43 al fibrosis (total lung collagen content and trichrome staining), reduced thickness of the peribronch

44 ount of collagen fibers (P=0.01), and Masson trichrome staining revealed a greater proportion of dens

45 Masson's trichrome staining revealed increased deposition of coll

46 Mason's trichrome staining revealed intense signal in centrilobu

47 fferent (P=0.8, not significant), and Masson trichrome staining revealed no significant change in fib

48 bnormal muscle fibres are best identified in trichrome-stained sections as harbouring amorphous, gran

49 Total and segmental areas were quantified on trichrome-stained sections of coronary atherectomy tissu

50 ession is known to cause pulmonary fibrosis, trichrome-stained sections of lung tissues were analyzed

51 te, an interpretation supported by images of trichrome-stained sections.

52 with two visual fibrosis-scoring methods on trichrome-stained slides.

53 gen III morphometry and visual assessment of trichrome-stained slides.

54 patient population, examination of a single trichrome-stained smear of a concentrated stool specimen

55 asites, of which 1,011 (99.2%) were found in trichrome-stained smears of unconcentrated specimens whi

56 was determined with colorimetric analysis of trichrome-stained specimens.

57 Total and segmental areas were quantified on trichrome-stained tissue of hypercellular tissue, collag

58 mean percentage of positive pixels at Masson trichrome staining was 7.28% vs 5.57%, respectively (P =

59 nd eosin, elastin van Gieson's, and Masson's trichrome staining was performed 6 and 12 months later a

60 Using immunohistochemistry and trichrome staining, we also observed elevated levels of

61 .); both Wheatley's modification of Gomori's trichrome stain (WT) and EcoStain (ES) were used to stai