ライフサイエンスコーパス: trigeminal ganglion

1 l trigeminovascular neurons (recorded in the trigeminal ganglion).

2 responses in hundreds of neurons across the trigeminal ganglion.

3 placode-derived neurons of the fused amniote trigeminal ganglion.

4 number in either the dorsal root ganglion or trigeminal ganglion.

5 s its activity in the neocortex, retina, and trigeminal ganglion.

6 tosis and increases in neuron numbers in the trigeminal ganglion.

7 ain contribute to the ciliary as well as the trigeminal ganglion.

8 but increased viral titers in the stroma and trigeminal ganglion.

9 likely acquired from VZV reactivation in the trigeminal ganglion.

10 a and certain cranial ganglia, including the trigeminal ganglion.

11 presence of both cell types in the embryonic trigeminal ganglion.

12 us sensory neurons, their normal fate in the trigeminal ganglion.

13 lly regulated and different from that of the trigeminal ganglion.

14 e ophthalmic and mandibular divisions of the trigeminal ganglion.

15 lly (2 days), HSV-1 was observed only in the trigeminal ganglion.

16 two distinct roles during development of the trigeminal ganglion.

17 h in the retina or for PCD in the retina and trigeminal ganglion.

18 n of BDNF- and NGF-responsive neurons in the trigeminal ganglion.

19 bryos there were normal levels of PCD in the trigeminal ganglion.

20 ensory neurons in the ophthalmic lobe of the trigeminal ganglion.

21 e in blood flow following stimulation of the trigeminal ganglion.

22 and cytokine response gene-2 in the eye and trigeminal ganglion.

23 437-545/animal) were seen in the ipsilateral trigeminal ganglion.

24 xpression of CGRP and its receptor in rhesus trigeminal ganglion.

25 ng on the early immune events in the eye and trigeminal ganglion.

26 Brn-3.0 is abundantly expressed in the adult trigeminal ganglion.

27 cripts (LAT) in the latently infected rabbit trigeminal ganglion.

28 t precocious neuronal differentiation of the trigeminal ganglion.

29 pulations of corneal afferent neurons in the trigeminal ganglion.

30 ed for neuronal nitric oxide synthase in the trigeminal ganglion.

31 ate entirely from the ophthalmic lobe of the trigeminal ganglion.

32 phthalmic and maxillomandibular lobes of the trigeminal ganglion.

33 al crest cells to the ophthalmic lobe of the trigeminal ganglion.

34 ensory neurons in the ophthalmic lobe of the trigeminal ganglion.

35 acellular recording from single cells in the trigeminal ganglion.

36 Removal of one trigeminal ganglion allowed axons of the contralateral g

37 s in chick neural crest cells populating the trigeminal ganglion also reduced the frequency of neurog

38 f primary sensory neurons of the ipsilateral trigeminal ganglion also stained positive for the report

39 neurons also received a novel input from the trigeminal ganglion and an overlapping input from a late

40 wholemount explant cultures of embryonic rat trigeminal ganglion and brainstem or in dissociated cell

41 into diverse derivatives, including cornea, trigeminal ganglion and branchial arch cartilage.

42 on of calcitonin gene-related peptide in the trigeminal ganglion and c-Fos in the trigeminal nucleus

43 derived neural crest or placode cells during trigeminal ganglion and corneal development, and after a

44 estern immunoblot analyses were performed on trigeminal ganglion and corneal lysates to determine abu

45 uring the initial stages of advance from the trigeminal ganglion and do not have a net attractive inf

46 are expressed by sensory neurons of the rat trigeminal ganglion and in what combinations these gene

47 Throughout the E10.5-E13.5 interval, the trigeminal ganglion and its targets in the CNS do not ex

48 HSV-1 infection in an area innervated by the trigeminal ganglion and may explain why the presence of

49 Thus, somatotopy of trigeminal ganglion and nerve organization is only parti

50 e neural correlates of vibrissa resonance in trigeminal ganglion and primary somatosensory cortex (SI

51 lted in loss of the ophthalmic branch of the trigeminal ganglion and reduced corneal innervation.

52 irus replication in the eye and in the local trigeminal ganglion and reduced herpetic blepharitis and

53 icantly reduced CXCL10 levels in the eye and trigeminal ganglion and reduced mononuclear cell infiltr

54 w responses to electrical stimulation of the trigeminal ganglion and superior sagittal sinus.

55 and nociceptive brain pathways involving the trigeminal ganglion and thalamus.

56 A neural connection between the trigeminal ganglion and the auditory brainstem was inves

57 the dorsoventral axis of the embryonic mouse trigeminal ganglion and thus can be considered trigemina

58 CGRP immunoreactivity (IR) within the feline trigeminal ganglion and trigeminal nucleus of normal adu

59 t contribute to the ophthalmic branch of the trigeminal ganglion and use it, in conjunction with DiI

60 an-amine (BDA) were made into the guinea pig trigeminal ganglion, and anterograde labeling was examin

61 on and activation, extralymphoid sites (eye, trigeminal ganglion, and brain) contained only activated

62 ed and uninjured nerves in the skin, soma in trigeminal ganglion, and central terminals in the spinal

63 the developing retina, olfactory epithelium, trigeminal ganglion, and hair follicles.

64 wed quail nuclei in the proximal part of the trigeminal ganglion, and quail nerves in the pericorneal

65 The trigeminal ganglion, another potential source, had no NP

66 als that the regulatory role of Brn3a in the trigeminal ganglion appears to be conserved in more post

67 ensory neurons of the ophthalmic lobe of the trigeminal ganglion are derived from two embryonic cell

68 sumption that primary sensory neurons of the trigeminal ganglion are sensitive to various combination

69 tages of neurogenesis in the embryonic mouse trigeminal ganglion are supported by BDNF whereas most n

70 The sensory neurons of the embryonic mouse trigeminal ganglion are supported in culture by differen

71 genous Wnt signaling activity in the forming trigeminal ganglion, as indicated by Wnt reporter expres

72 ctron microscopy studies of the axons of the trigeminal ganglion at E13.5 revealed that in the p75 mu

73 of naturally occurring neuronal death in the trigeminal ganglion at E14, trigeminal neurons from bcl-

74 ttern of cells that infiltrate the A/J mouse trigeminal ganglion at various times after HSV-1 corneal

75 h in vitro, but thereafter permit or attract trigeminal ganglion axon outgrowth.

76 Thus, trigeminal ganglion axons always project to rhombomere (

77 ing embryo, in which the spinal ganglion and trigeminal ganglion-bound iodinated inhibin A.

78 howed quail nuclei in the distal part of the trigeminal ganglion, but no quail nerves in the cornea o

79 a transient rise in IL-6 mRNA levels in the trigeminal ganglion, but not other cytokine transcripts

80 A-, TrkB- and TrkC-expressing neurons in the trigeminal ganglion by activating directly each of these

81 and reduces the latent HSV-1 genome load per trigeminal ganglion by approximately 200-fold.

82 s type 1 (HSV) was introduced into the mouse trigeminal ganglion by stereotaxic injection.

83 , we could detect immunoreactive profiles of trigeminal ganglion cell axons that contained many vesic

84 s are able to form in the virtual absence of trigeminal ganglion cell death and suggest that mechanis

85 ptosis and subsequent ventricle enlargement, trigeminal ganglion cell loss, and abnormal hindbrain ar

86 inal complex established that these branched trigeminal ganglion cells also extended an axon into the

87 These trigeminal ganglion cells are unique among primary senso

88 Additionally, we cultured dissociated trigeminal ganglion cells in the presence of NGF, NT-3,

89 However, following infection of primary trigeminal ganglion cells in vitro with a recombinant HS

90 labeling techniques in rats to identify the trigeminal ganglion cells innervating each of these terr

91 can elicit persistent neural adaptations in trigeminal ganglion cells innervating the dura, prominen

92 ations from (1) cross-sectional areas of AEN trigeminal ganglion cells labeled with WGA-HRP, and (2)

93 e used compartmental cultures of dissociated trigeminal ganglion cells to determine the effect of Sem

94 These results indicate that some trigeminal ganglion cells with sensory endings in the na

95 d human embryonic kidney (HEK) cells and rat trigeminal ganglion cells) on the silica surface of a pl

96 nusual projections of primary afferents from trigeminal ganglion cells, and, subsequently, increased

97 The number of trigeminal ganglion cells, their size distribution under

98 ion to compartmental cultures of dissociated trigeminal ganglion cells.

99 igher levels of IFN-alphabeta (533 U) in the trigeminal ganglion compared with nontransgenic mice (70

100 Following a survival period of 3-7 days, the trigeminal ganglion contained double-labeled, small (11.

101 h neural crest and placode contribute to the trigeminal ganglion, corneal innervation is entirely neu

102 We hypothesized that the trigeminal ganglion could be one possible site.

103 which resides in the sensory neurons of the trigeminal ganglion, could be stress reactivated to prod

104 iability of the labeled DPANs in dissociated trigeminal ganglion cultures using calcium microfluorome

105 Here, we show that treatment of primary rat trigeminal ganglion cultures with WIN led to dephosphory

106 k HSV-1 reactivation from latency in ex vivo trigeminal ganglion cultures.

107 ral crest or placodal origin, neurons of the trigeminal ganglion derive from both populations.

108 neurons from the dorsomedial portion of the trigeminal ganglion (DM-TG), which are largely cutaneous

109 ptor Neuropilin-1 (Npn1) is expressed in the trigeminal ganglion during cornea development.

110 thin the brainstem trigeminal nuclei and the trigeminal ganglion during elongation and branching-arbo

111 e imaging (fMRI) to assess activation in the trigeminal ganglion during innocuous mechanical (brush)

112 V-1-infected cell polypeptide 27 mRNA in the trigeminal ganglion during the acute (day 6 postinfectio

113 nt role in limiting HSV-1 replication in the trigeminal ganglion during the acute stage of infection.

114 Trigeminal ganglion electrophysiologic single unit recor

115 nstem or in dissociated cell cultures of the trigeminal ganglion, exogenous supply of NGF leads to ax

116 We have used the mouse trigeminal ganglion explant model and reverse transcript

117 ctivation phenotype was determined using the trigeminal ganglion explant model.

118 term dissociated cultures, we cultured early trigeminal ganglion explants with and without their targ

119 ex vivo neurite outgrowth and myelination of trigeminal ganglion explants.

120 L-6 expression and HSV-1 reactivation in the trigeminal ganglion following hyperthermic stress of mic

121 ttern of HSV-1 latent infection of the mouse trigeminal ganglion following ocular inoculation with vi

122 he development of neural crest cells and for trigeminal ganglion formation, however, migrating neural

123 tonomously on neural crest cells and promote trigeminal ganglion formation.

124 Single unit activity was recorded in the trigeminal ganglion from 40 mechanosensitive dural affer

125 citonin gene-related peptide (CGRP) from the trigeminal ganglion has been established as a key player

126 ite stage, by which time condensation of the trigeminal ganglion has begun.

127 levels of infectious virus were detected in trigeminal ganglion homogenates from calves infected wit

128 ill be valuable for measuring changes in the trigeminal ganglion in human models of neuropathic pain

129 ter unilateral electrical stimulation of the trigeminal ganglion in mice lacking serotonin 5-HT1B (5-

130 h near complete loss of the ophthalmic (OpV) trigeminal ganglion in the most severe cases after overe

131 med cell death (apoptosis) in neurons of the trigeminal ganglion in vivo and in tissue culture cells

132 resents VZV reactivation, most likely in the trigeminal ganglion, in the absence of clinical herpes z

133 ensity of the inflammatory infiltrate in the trigeminal ganglion increased until days 12 to 21 p.i.,

134 HSV antigen expression in the trigeminal ganglion (indicative of the viral lytic cycle

135 s study provides the first evidence that the trigeminal ganglion innervates the cochlear nucleus and

136 High-intensity electrical stimulation of the trigeminal ganglion is accompanied by mast cell degranul

137 al pulp innervation by nerve fibers from the trigeminal ganglion is an excellent example of nerve-tar

138 Among these, the trigeminal ganglion is missing 70% of the normal number

139 Furthermore, we examined if the trigeminal ganglion is protected by the blood-brain barr

140 We also show that Kir4.1 in the trigeminal ganglion is reduced after chronic constrictio

141 The trigeminal ganglion is reduced in size and lacks project

142 ensory neurons of the ophthalmic lobe of the trigeminal ganglion, is a useful model of sensory neuron

143 The organization of the trigeminal ganglion, its somatotopic projections upon th

144 e in IL-6 mRNA and protein expression in the trigeminal ganglion latently infected with HSV-1.

145 st follow-up questionnaire, were higher if a trigeminal-ganglion lesion had been created with radiofr

146 eyed to the somatosensory cortex through the trigeminal ganglion, medullary dorsal horn, and thalamus

147 sumptions: (1) electrical stimulation of the trigeminal ganglion mimics the neurogenic inflammatory p

148 However, in the trigeminal ganglion, naturally occurring neuronal death

149 a latently infected with VZV and one control trigeminal ganglion negative for VZV DNA that were remov

150 of NGF/TrkA signalling in vivo, the death of trigeminal ganglion neurones occurs independently of BAX

151 o2 expression occurs in approximately 26% of trigeminal ganglion neurons and 30% of corneal afferent

152 er with earlier work demonstrating a loss of trigeminal ganglion neurons and retinal ganglion cells i

153 elated with Cavalpha2delta1 up-regulation in trigeminal ganglion neurons and Vc/C2.

154 del in which dissociated wild-type embryonic trigeminal ganglion neurons are cultured on longitudinal

155 ulture showed that 65% of DiIC18-labeled rat trigeminal ganglion neurons are excited by capsaicin.

156 ing CGRP expression in the migraine-relevant trigeminal ganglion neurons are unknown.

157 rons at early embryonic (E) stages (E6-E12), trigeminal ganglion neurons at midstages (E9-E16), and v

158 er transgene was differentially regulated in trigeminal ganglion neurons depending upon age.

159 Trigeminal ganglion neurons from adult female Sprague Da

160 and show that this mutation renders DRG and trigeminal ganglion neurons hyperexcitable.

161 1632E renders dorsal root ganglion (DRG) and trigeminal ganglion neurons hyperexcitable.

162 VEGF enhanced neurite elongation in isolated trigeminal ganglion neurons in a dose-dependent manner.

163 made from dissociated guinea-pig nodose and trigeminal ganglion neurons in culture to study second m

164 (HMGA1), was readily detected in a subset of trigeminal ganglion neurons in latently infected calves

165 as no significant change in proliferation of trigeminal ganglion neurons in the noggin transgenic ani

166 hat activation of CGRP receptors on cultured trigeminal ganglion neurons increased endogenous CGRP mR

167 TRPA1 recorded from cell bodies of trigeminal ganglion neurons showed similar behavior with

168 ive-cycle genes in greater numbers of murine trigeminal ganglion neurons than LAT+ HSV type 1 at earl

169 Capsaicin evokes a membrane current in trigeminal ganglion neurons that is increased substantia

170 es have demonstrated that tonic responses of trigeminal ganglion neurons to maintained whisker deflec

171 e expression of key inflammatory proteins in trigeminal ganglion neurons under basal and inflammatory

172 Compartmental culture of trigeminal ganglion neurons was performed in Campenot de

173 Compartmental culture of trigeminal ganglion neurons was performed in Campenot de

174 Furthermore, no HSV strain 17-infected trigeminal ganglion neurons were apoptotic, but 0.087% o

175 Rohon-Beard, commissural, primary motor, and trigeminal ganglion neurons were distinctly stained in t

176 Trigeminal ganglion neurons were positive for beta-galac

177 selectively expressed in spinal sensory and trigeminal ganglion neurons within the peripheral nervou

178 In cultured rat trigeminal ganglion neurons, knockdown of either USF1 or

179 l, by whole-cell patch clamping cultured rat trigeminal ganglion neurons.

180 mp recordings were performed on cultured rat trigeminal ganglion neurons.

181 cocoa can prevent inflammatory responses in trigeminal ganglion neurons.

182 motes export of endogenous deltaR in primary trigeminal ganglion neurons.

183 rotein kinase regulation of the CGRP gene in trigeminal ganglion neurons.

184 unctional activities of the TRPA1 channel in trigeminal ganglion neurons.

185 acts with these repeats in latently infected trigeminal ganglion neurons.

186 Neither the trigeminal ganglion nor the ophthalmic branch of the tri

187 esonate, conferring frequency specificity to trigeminal ganglion (NV) and primary somatosensory corte

188 ial dura, using single-unit recording in the trigeminal ganglion of anesthetized male rats.

189 There was also a significant increase in the trigeminal ganglion of cells expressing the gamma delta

190 nd IFN-alpha mRNA levels were reduced in the trigeminal ganglion of GFAP-IFN-alpha mice compared with

191 CD8+, and DX5+ (NK cell marker) cells in the trigeminal ganglion of latent HSV-1-infected mice 24 h a

192 Within the trigeminal ganglion of normal subjects, cell bodies and

193 Single-unit recordings made in the trigeminal ganglion of rats were used to test changes in

194 s, higher levels of infectious virus in eye, trigeminal ganglion, or brain, and virtually complete de

195 g chicken embryo, nerves from the ophthalmic trigeminal ganglion (OTG) innervate the cornea in a seri

196 In the trigeminal ganglion, P2X(3) receptors are often co-expre

197 el of chronic orofacial pain; in this model, trigeminal ganglion Panx1 expression and function are ma

198 ressed in the surface ectoderm overlying the trigeminal ganglion, play a role in promoting the assemb

199 f phenotypic, target, and sex differences in trigeminal ganglion primary afferent neurons.

200 Electrical stimulation of the right trigeminal ganglion produced a unilateral expression of

201 The trigeminal ganglion provides the somatosensory innervati

202 the cornea to the periocular tissue via the trigeminal ganglion rather than by direct spread from co

203 ed that in mouse, presumptive targets of the trigeminal ganglion, rather than intermediate structures

204 The largest of the cranial ganglia, the trigeminal ganglion, relays cutaneous sensations of the

205 arly striking was restoration of near-normal trigeminal ganglion replication and neurovirulence of an

206 Stimulation of the trigeminal ganglion resulted in a frequency-dependent re

207 ents or whisker stimulation, because neither trigeminal ganglion sensory-evoked responses nor EMG act

208 at CD8(+) T cell control is expressed in the trigeminal ganglion, serving to curtail a source of viru

209 Orthodromic latencies to trigeminal ganglion shocks and antidromic activation fro

210 central root that was still attached to the trigeminal ganglion showed an increase in CGRP-IR within

211 In the largest of these, the trigeminal ganglion, Slit1-Robo2 signaling is essential

212 essed in skin, tongue, dorsal root ganglion, trigeminal ganglion, spinal cord and brain.

213 After 2-6 days, their trigeminal ganglions, spinal cords and brainstems were c

214 avasation within dura mater after electrical trigeminal ganglion stimulation in both wild-type and kn

215 the neurogenic dural inflammation following trigeminal ganglion stimulation in the guinea pig after

216 achykinin (the substance P precursor) in the trigeminal ganglion, suggesting that increased NGF was d

217 y, transgenic overexpression of Brn3a in the trigeminal ganglion suppresses the expression of the end

218 infection, CD8(+) T cells accumulate in the trigeminal ganglion (TG) and participate in the maintena

219 Both sensory trigeminal ganglion (TG) and sympathetic superior cervic

220 thy1-YFP mouse and determine if they promote trigeminal ganglion (TG) cell neurite growth.

221 Primary cultures of trigeminal ganglion (TG) cells from herpes simplex virus

222 -1) in primary cultures of latently infected trigeminal ganglion (TG) cells.

223 (HSV-1) reactivation from latency in ex vivo trigeminal ganglion (TG) cultures without destroying the

224 characterize this mutant in murine embryonic trigeminal ganglion (TG) cultures.

225 Axonal branches of the trigeminal ganglion (TG) display characteristic growth a

226 virus 1 (HSV-1) infection in the tree shrew trigeminal ganglion (TG) following ocular inoculation.

227 KLF15 were frequently expressed in the same trigeminal ganglion (TG) neuron during reactivation and

228 Ectopic discharges from injured trigeminal ganglion (TG) neurons and thalamocortical reo

229 latency-associated transcript (LAT)-positive trigeminal ganglion (TG) neurons coexpressed SSEA3, 71%

230 solated mouse dorsal root ganglion (DRG) and trigeminal ganglion (TG) neurons expressing the cold-sen

231 We report that ~50% of trigeminal ganglion (TG) neurons retrogradely labeled fr

232 CD8(+) T cells provide immunosurveillance of trigeminal ganglion (TG) neurons that harbor latent HSV-

233 in nonneuronal cells (MRC5) and adult murine trigeminal ganglion (TG) neurons using the Illumina plat

234 ordings were performed on cultured adult rat trigeminal ganglion (TG) neurons voltage-clamped near th

235 The number of latent sites established in trigeminal ganglion (TG) neurons was determined using a

236 f neurons, cultured dissociated adult murine trigeminal ganglion (TG) neurons were assessed for relat

237 The ability to genetically manipulate trigeminal ganglion (TG) neurons would be useful in the

238 virus type 1 (HSV-1) establishes latency in trigeminal ganglion (TG) neurons.

239 nt TRESK subunits in HEK293T cells and mouse trigeminal ganglion (TG) neurons.

240 gf8 as a novel modifier of BMP4 signaling in trigeminal ganglion (TG) neurons.

241 fter reactivation of latent infection in the trigeminal ganglion (TG) was examined in the mouse using

242 f herpes simplex virus type 1 (HSV-1) in the trigeminal ganglion (TG) was induced by UV irradiation o

243 ger RNA (mRNA) in the sensory neurons of the trigeminal ganglion (TG) which innervate the nasal epith

244 ades sensory neurons with cell bodies in the trigeminal ganglion (TG), replicates briefly, and then e

245 ent protein kinase R (PKR) in the cornea and trigeminal ganglion (TG).

246 demonstrate receptor binding sites in rhesus trigeminal ganglion (TG).

247 ction in viral Ag expression in the eyes and trigeminal ganglion that correlated with a reduction in

248 ivity of primary afferent neurons in the rat trigeminal ganglion that innervate the dural venous sinu

249 Here, we show that proper formation of the trigeminal ganglion, the largest of the cranial ganglia,

250 the present study, we show that, in the rat trigeminal ganglion, the location of the primary sensory

251 e transport from the application site to the trigeminal ganglion, the numbers of stained DPANs, and t

252 somatosensory neurons of the dorsal root and trigeminal ganglion, the transient receptor potential me

253 ing analysis indicated that in the adult rat trigeminal ganglion there exist at least two nicotinic r

254 Trigeminal ganglion tissue sections were processed for s

255 (5-HT1D-IR) peripheral afferents within the trigeminal ganglion (TRG) and lumbar dorsal root ganglio

256 egulatory targets of Brn3a in the developing trigeminal ganglion using microarray analysis of Brn3a m

257 information, primary sensory neurons in the trigeminal ganglion (Vg) have often been described as en

258 n via afferents whose cell bodies lie in the trigeminal ganglion (Vg).

259 HSV was introduced into the murine trigeminal ganglion via stereotaxic guided injection.

260 noketone treatment on IL-6 expression in the trigeminal ganglion was also measured.

261 Briefly, the trigeminal ganglion was electrically stimulated for 30 s

262 l crest to form somatosensory neurons in the trigeminal ganglion was significantly reduced compared w

263 no change in the viral load from the eye or trigeminal ganglion when comparing the AED-treated with

264 the cornea, the virus is transmitted to the trigeminal ganglion, where a brief period of virus repli

265 e ophthalmic branch of the latently infected trigeminal ganglion, where they acquire and maintain an

266 The trigeminal ganglion, which arises lateral to the midbrai

267 In contrast, the trigeminal ganglion, which innervates papillae but not t

268 They originate in the trigeminal ganglion, which is derived from two embryonic

269 The trigeminal ganglion, which provides sensory input to the

270 e limiting for CGRP receptor activity in the trigeminal ganglion, which raises the possibility that e

271 A 3-dimensional reconstruction of an entire trigeminal ganglion with 2-photon laser scanning fluores

272 for the identification of cell bodies in the trigeminal ganglion with axons in the IAN.