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1 ith the transmission of headache pain by the trigeminal nerve.
2 tina, that have no direct connections to the trigeminal nerve.
3 y unilateral or bilateral stimulation of the trigeminal nerve.
4 t changes in ADCs in either direction in the trigeminal nerve.
5 stimulus (US) was applied to the ipsilateral trigeminal nerve.
6 d cerebral, is innervated by fibers from the trigeminal nerve.
7 y branches of the ophthalmic division of the trigeminal nerve.
8 ed exposure to odors that also stimulate the trigeminal nerve.
9 stopping and the branching of the mandibular trigeminal nerve.
10 timulation of the supraorbital branch of the trigeminal nerve.
11 tion, to ablate the ophthalmic branch of the trigeminal nerve.
12 essure, as induced by occlusal loads, on the trigeminal nerve.
13 ocoagulation of the ophthalmic branch of the trigeminal nerve.
14 alization of DPANs in all 3 divisions of the trigeminal nerve.
15 erior colliculus to the motor nucleus of the trigeminal nerve.
16 TSE) to destroy the ophthalmic branch of the trigeminal nerve.
17 a significant contribution to the ophthalmic trigeminal nerve.
18 al and bilateral), arm and third division of trigeminal nerve.
19 reas innervated by the third division of the trigeminal nerve.
20 n unconditioned stimulus (US) applied to the trigeminal nerve.
21 ds on the head of the fish innervated by the trigeminal nerve.
22  peptide (CGRP)-immunoreactive fibers of the trigeminal nerve.
23 f the principal sensory nucleus (PrV) of the trigeminal nerve, a major relay station for somatotopic
24 distribution of the ophthalmic branch of the trigeminal nerve, abnormal capillary venous vessels in t
25                                    Following trigeminal nerve activation, afferent release of CGRP ca
26                    We propose that following trigeminal nerve activation, CGRP secretion from neurona
27 nnervation of the nasal mucosa by monitoring trigeminal nerve activity in patients with IR and health
28                Disordered sensory input from trigeminal nerve afferents, such as aberrant feedback fr
29                                          The trigeminal nerve, along with the cervical nerve roots, s
30 ture branching of the major divisions of the trigeminal nerve and a failure to correctly innervate wh
31 thalmic and maxillary divisions of the right trigeminal nerve and cervical spinal nerve afferents.
32 applied to the nasal epithelium activate the trigeminal nerve and evoke changes in respiratory rate.
33  of afferents from the three branches of the trigeminal nerve and from the lingual branch of the hypo
34  that spry3 is expressed specifically in the trigeminal nerve and in spinal motor and sensory neurons
35 leus, A1-5 and C1-3 nuclei, subnuclei of the trigeminal nerve and nucleus tractus solitarius.
36 ons such as the mesencephalic nucleus of the trigeminal nerve and the interomedial lateral cell colum
37  TSE was confirmed by gross histology of the trigeminal nerve and was considered effective if the cor
38                                Activation of trigeminal nerves and release of neuropeptides that prom
39 ommissure and the mesencephalic tract of the trigeminal nerve) and one ventral (the mammillotegmental
40 erves C1-C8, branches of C2, branches of the trigeminal nerve, and hindlimb nerves.
41 enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymeninges.
42 uclei, motor and mesencephalic nuclei of the trigeminal nerve, and some motor neurons in the spinal c
43 halon and pons, the principal nucleus of the trigeminal nerve, and the dorsal raphe.
44                 Two patients with unilateral trigeminal nerve anesthesia-one following basal skull fr
45 e activated by antidromic stimulation of the trigeminal nerve, as well as by acute immobilization str
46 the diameter and cross-sectional area of the trigeminal nerves at 5 mm from the entry point of the ne
47                    The results indicate that trigeminal nerve atrophy can be depicted noninvasively i
48 tering the response to A-fiber inputs to the trigeminal nerve because all stimuli were too weak to ac
49 e, the ophthalmic, maxillary, and mandibular trigeminal nerve branches maintain a somatotopic segrega
50 ists of a thin membrane, innervated by three trigeminal nerve branches that project to a specific nuc
51 al tracers to identified pit-organ-supplying trigeminal nerve branches.
52 reas innervated by cervical nerves or by the trigeminal nerve but do not cross a border defined by th
53 ated by the first and second division of the trigeminal nerve but in some cases also included areas i
54 ed by the first (ophthalmic) division of the trigeminal nerve, but also the back of the head, innerva
55 eted VR1 mRNA from the spinal nucleus of the trigeminal nerve, but not from other areas such as the i
56 es between the peripheral electrosensory and trigeminal nerves, but these senses remain separate in t
57                    Injury to branches of the trigeminal nerve can sometimes result in persistent dysa
58 at stimulation of another cranial nerve, the trigeminal nerve, can also cause cortical and thalamic d
59 he lingual nerve, a peripheral branch of the trigeminal nerve, can be damaged during the surgical rem
60                                              Trigeminal nerves collecting sensory information from th
61 nopus embryos growth cones of the mandibular trigeminal nerve contact cells located in the posterior
62 al ganglion nor the ophthalmic branch of the trigeminal nerve contained cholinergic elements.
63                                              Trigeminal nerves displayed straight parallel courses at
64 ly 100% of patients experience trauma to the trigeminal nerve during orthognathic surgery, impairing
65 tro evidence that Robo-Slit signaling guides trigeminal nerves during cornea innervation.
66                                        Nasal trigeminal nerve endings are particularly sensitive to o
67 timulation of the supraorbital branch of the trigeminal nerve evoked relatively larger amplitude blin
68           Detection of such irritants by the trigeminal nerve evokes protective reflexes, including s
69           SCCs are innervated by peptidergic trigeminal nerve fibers but it is currently unknown if i
70 ndence of SCCs on innervation by eliminating trigeminal nerve fibers during development with neurogen
71 cleus also showed substantial innervation by trigeminal nerve fibers immunoreactive for calcitonin ge
72  outlier, with more than twice the number of trigeminal nerve fibers than any other species.
73 ese proteins might be involved in supporting trigeminal nerve fibers that innervate the dental pulp.
74 e to direct depolarization of acid-sensitive trigeminal nerve fibers, for example, polymodal nocicept
75  reversible subcutaneous block of peripheral trigeminal nerve fibers.
76  each case, infrared signals are detected by trigeminal nerve fibres that innervate specialized pit o
77 underwent microvascular decompression of the trigeminal nerve for medically intractable trigeminal ne
78  marked reduction in the calibre of the left trigeminal nerve from the nerve root exit zone in the po
79          Gem was also observed in developing trigeminal nerve ganglia in 12.5 day mouse embryos, demo
80 ry nerves are all derived as branches of the trigeminal nerve/ganglion similar to the situation encou
81                Effects of polySia removal on trigeminal nerve growth behavior were determined in vivo
82                  Effects of purified GAGs on trigeminal nerve growth cone behavior were tested using
83                                      Sensory trigeminal nerve growth cones innervate the cornea in a
84 suggest that GAGs may direct the movement of trigeminal nerve growth cones innervating the cornea.
85             During cornea development, chick trigeminal nerve growth cones reach the cornea margin at
86 e and the upper end of the size range in the trigeminal nerve (i.e., > 5 microm).
87 fields of each of the three divisions of the trigeminal nerve in healthy volunteers.
88 n involving the maxillary region (V2) of the trigeminal nerve in patients with spontaneous pain and e
89 n the principal sensory nucleus (PrV) of the trigeminal nerve in the brainstem.
90 timulation of the supraorbital branch of the trigeminal nerve in the dark and in the light.
91 the emerging fibers of the motor root of the trigeminal nerve in the mouse, which we have called the
92 ffusion coefficients (ADCs) of the optic and trigeminal nerves in 2-10-week-old rats were measured wi
93                             The sizes of the trigeminal nerves in 31 patients (18 men and 13 women; m
94                               Stimulation of trigeminal nerve induces pressor response and improves c
95 investigate a potential mechanism underlying trigeminal nerve injury-induced orofacial hypersensitivi
96 development of ectopic activity at a site of trigeminal nerve injury.
97                                 Although the trigeminal nerve innervates the meninges and participate
98 lforhodamine 101, peripheral hypoglossal and trigeminal nerves involved with tongue and jaw movements
99     The area innervated by the contralateral trigeminal nerve is represented in an 8-mm mediolateral
100   Tissue phantoms made of normal fixed mouse trigeminal nerves juxtaposed with and without gel were e
101 y to the infraorbital nerve, a branch of the trigeminal nerves, led to synaptic ultrastructural chang
102                                              Trigeminal nerve lesions at differing levels can result
103 marily targeted by the lingual branch of the trigeminal nerve (LV).
104 rovascular canals, that include parts of the trigeminal nerve; many branches of this complex terminat
105 lts indicate that viral dissemination in the trigeminal nerve may occur both within the axon and in t
106                                Injury to the trigeminal nerves may cause maladaptive changes in synap
107 nt stress, local constriction, and injury in trigeminal nerves may contribute to the pathogenesis of
108 ry nerve-mediated and combined olfactory and trigeminal nerve-mediated odorants activate different re
109 n restricted to the region innervated by the trigeminal nerves (n = 37); (2) pain in the trigeminal d
110 sensory cortex, in the representation of the trigeminal nerve, near cells with a tactile representati
111 o reported in the vertex, second division of trigeminal nerve, neck, nose, jaw, parietal region, ear,
112  nuclei, cranial nerve motor nuclei, sensory trigeminal nerve nuclei, inferior and superior colliculi
113  nucleus (SpM), the principle nucleus of the trigeminal nerve, nucleus laminaris and scattered cell g
114                                              Trigeminal nerve number was by far the largest and also
115               The present study examined the trigeminal nerve of a teleost fish, the rainbow trout (O
116                     The mean diameter of the trigeminal nerve on the symptomatic side was significant
117 infraorbital nerve (a major component of the trigeminal nerve) on protein expression was examined.
118 ion of somatotopic organization, we compared trigeminal nerve organization in turtle, chick, and mous
119 s innervated by the ophthalmic branch of the trigeminal nerve originate contralaterally from insular
120            Odorants that also stimulated the trigeminal nerve produced additional cingulate, temporal
121 otine to the various epithelia served by the trigeminal nerve produces irritation and/or pain by acti
122 ipsilateral principal sensory nucleus of the trigeminal nerve (PrV) correspond to the whiskers.
123         The principal sensory nucleus of the trigeminal nerve (PrV) relays the facial sensations to t
124 target, the principal sensory nucleus of the trigeminal nerve (PSN).
125     Continuous unilateral stimulation of the trigeminal nerve reduced electrographic seizure activity
126 ges in subjects with neuropathic pain of the trigeminal nerve resulting in most cases (20 of 23) from
127                           In most cases, the trigeminal nerve root demyelination involves the proxima
128                               Examination of trigeminal nerve roots from patients with compression of
129                   We report a patient with a trigeminal nerve section who continued to have attacks.
130 timulation of the supraorbital branch of the trigeminal nerve (SO) ipsilateral to the upper eyelid wi
131 muli (HFS) to the supraorbital branch of the trigeminal nerve (SO) modified subsequent reflex blinks
132 ble of increasing cerebral perfusion, making trigeminal nerve stimulation (TNS) a promising strategy
133 s such as deep brain stimulation, vagus, and trigeminal nerve stimulation are effective only in a fra
134                                              Trigeminal nerve stimulation had been reported to activa
135                  These findings suggest that trigeminal nerve stimulation is safe in awake rats and s
136 e results demonstrate that seizure-triggered trigeminal nerve stimulation is technically feasible and
137 rval (ISI) between the onset of amygdala and trigeminal nerve stimulation.
138 nks and reflex blinks evoked by supraorbital trigeminal nerve stimulation.
139 ectopic activity in adjacent branches of the trigeminal nerve suggest that the fibre types or anatomi
140 tion through a critical interaction with the trigeminal nerve, supporting the concept that the integr
141 inputs, it is hypothesized that auditory and trigeminal nerve synaptic inputs onto abducens motor neu
142  use this novel in vivo assay to isolate the trigeminal nerve target-recognition molecule(s).
143 ntia nigra, the mesencephalic nucleus of the trigeminal nerve, the cochlear nucleus and the superior
144 ssociated with sensory-nerve branches of the trigeminal nerve, the orbital vasculature, and the perio
145 ity properties in the root entry zone of the trigeminal nerve, the spinal trigeminal tract, or the ve
146 terization of the infraorbital branch of the trigeminal nerve, the tactile map in the granule cell la
147 ensitive fibers of the lingual branch of the trigeminal nerve to cooling from 35 degrees to 10 degree
148 f injury to the maxillary branch (V2) of the trigeminal nerve to produce constant and long-lasting pr
149  showed direct projections from auditory and trigeminal nerves to abducens motor neurons.
150                      The overall response of trigeminal nerves to peripheral inflammatory stimuli inv
151 ferior alveolar nerve, another branch of the trigeminal nerve, to determine whether differences in ne
152 tating substances leads to activation of the trigeminal nerve, triggering protective reflexes that in
153 n at sites within the three divisions of the trigeminal nerve (V1, V2, and V3) and also to the ipsila
154                   Motor function of the left trigeminal nerve was preserved.
155 ke rats while the infraorbital branch of the trigeminal nerve was stimulated via a chronically implan
156 esentation of body regions innervated by the trigeminal nerve were elucidated in monkey cerebral cort
157 nd odorants that stimulate the olfactory and trigeminal nerves were compared by using repetitive imag
158    The sensory fibers of the hypoglossal and trigeminal nerves were found projecting to the Purkinje
159 verse and longitudinal sections of optic and trigeminal nerves were studied with electron microscopy.
160 lation included face areas innervated by the trigeminal nerve, whereas somatic fields of 89% of neuro
161              KS was localized in the path of trigeminal nerves, whereas DS and CSA/C-rich areas were
162 c reflex (TCR) occurs upon excitation of the trigeminal nerve with a resulting bradycardia and hypote
163 ct blockade of CGRP release by inhibition of trigeminal nerves would be similarly effective in both m

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