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1 -SNPH coupling inhibited the motor adenosine triphosphatase.
2 were shown to be activated by Ran guanosine triphosphatase.
3 he level of the vacuolar-type H(+)-adenosine triphosphatase.
4 pumping enzyme vacuolar-type H(+)-adenosine triphosphatase.
5 hich encodes a copper-transporting adenosine triphosphatase.
6 ral large structural proteins and nucleoside triphosphatases.
7 C is activated by Rho family small guanosine triphosphatases.
8 f cell surface-associated vacuolar adenosine triphosphatases.
9 sensitivity and drug efficacy in nucleotide triphosphatases.
10 he absence of its two different types of RNA triphosphatases.
11 nd physically interacts with small guanosine triphosphatases.
12 horylation of plasma membrane H(+)-adenosine triphosphatase 2 at Ser(899), mediating the inhibition o
14 s involved in the purine metabolism (inosine triphosphatase, 5'-nucleotidase cytosolic-II, purine nuc
15 at the Insert B domain of the Dnm1 guanosine triphosphatase (a DRP) contains a novel motif required f
16 es (AIs) that contain the cellular adenosine triphosphatase ABCE1 (ATP-binding cassette protein E1).
17 ing protein 1 (ELMOD1), a guanine nucleoside triphosphatase activating protein (GAP) for ARF6, as the
19 rsed at the lagging pole where the guanosine triphosphatase activating protein MglB disrupts the MglA
20 ce that neurofibromin, via its Ras guanosine triphosphatase -activating activity, controls ERK1/2-dep
21 -CYK4 centralspindlin complex is a guanosine triphosphatase-activating protein (GAP) for Rac1 and not
24 at encodes alpha2-chimaerin, a Rac guanosine triphosphatase-activating protein (RacGAP) signaling pro
27 es small ARF GAP1 (SMAP1), an ARF6 guanosine triphosphatase-activating protein that functions in clat
30 2/Bub2/Cdc16 domain-containing Rab guanosine triphosphatase-activating proteins (GAPs) and identified
32 signaling to engage small GTPase (guanosine triphosphatase) activation and AMPAR synaptic delivery d
34 r has ATPase and, more generally, nucleoside triphosphatase activities that are indistinguishable fro
37 the N-terminal part exhibits NTPase and RNA triphosphatase activity and is proposed to have helicase
38 s altered by inhibition of dynamin guanosine triphosphatase activity and is temporally distinct from
41 es contractility by decreasing the adenosine triphosphatase activity of the cardiac myosin heavy chai
42 , suppression of PDGF-induced Rac1 guanosine triphosphatase activity, and Akt and extracellular signa
43 this protein has been shown to possess dNTP triphosphatase activity, which is proposed to inhibit HI
48 MDCK) cells and identified the rho-guanosine triphosphatase adaptor and scaffolding protein IRSp53 as
49 , inhibition of filamentous septin guanosine triphosphatases alleviates constriction defects in Auror
50 ses expression of the gastric H, K-adenosine triphosphatase alpha-subunit (HKalpha), which could cont
51 terminal serine proteinase domain and an RNA triphosphatase, an NTPase domain, and an RNA helicase in
52 e folate pathway-related genes, deoxyuridine triphosphatase and dihydrofolate reductase, the silencin
53 ists of an N-terminal domain with nucleoside triphosphatase and helicase activities and a C-terminal
54 FtsZ is an essential bacterial guanosine triphosphatase and homolog of mammalian beta-tubulin tha
55 known exchange factor for the Ran guanosine triphosphatase and performs essential roles in nuclear t
57 strate- and product-bound mammalian thiamine triphosphatase and with previously reported structures o
58 oluble and stable, did not bind to guanosine triphosphatases and bound more tightly to the PAK1 kinas
59 equires the function of Rho family guanosine triphosphatases and reorganization of the actin cytoskel
60 on molecules that activate RAS/RHO guanosine triphosphatases and their effector mitogen-activated pro
61 e and activates mTORC1 via the Rag guanosine triphosphatases and their regulators GATOR1 and GATOR2.
62 tein levels of MIG-2, a Rho family guanosine triphosphatase, and/or down-regulate INA-1, an integrin
63 ility signaling proteins (kinases, guanosine triphosphatases, and guanine exchange factors) controlle
64 gella when exogenous ubiquitin and adenosine triphosphatase are added, suggesting that the ubiquitin
65 bers of the RAS subfamily of small guanosine triphosphatases are found in > 30% of all human cancers.
67 We found that HIV-1 Nef and the guanosine triphosphatase Arf1 induced trimerization and activation
68 Rafiq et al. reveal that the small guanosine triphosphatase ARF1, a well-known orchestrator of membra
69 served members of the AAA+ family (adenosine triphosphatases associated with diverse cellular activit
70 6 (which carries activators of Rho guanosine triphosphatase) at the cell cortex using total internal
72 clohexylurea group, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) channels fo
73 retion by the nongastric H(+)/K(+) adenosine triphosphatase (ATP12A) acidified airway surface liquid,
74 We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary for amino
76 ed of the Mre11 nuclease and Rad50 adenosine triphosphatase (ATPase) active sites dimerizes through a
77 tive orientation of the N-terminal adenosine triphosphatase (ATPase) and C-terminal nuclease domains.
78 nd 130 nM in microtubule-dependent adenosine triphosphatase (ATPase) and cell-based cytotoxicity assa
79 ties related to inhibition of Na/K adenosine triphosphatase (ATPase) and stimulation of sarcoplasmic
80 Here we present evidence that the adenosine triphosphatase (ATPase) cycle of the SF1 helicase Upf1 i
81 ent in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at which the
82 nally characterized the DExD/H box adenosine triphosphatase (ATPase) Dhh1, a critical regulator of th
83 eparated from its ring-shaped AAA+ adenosine triphosphatase (ATPase) domain by a 15-nanometer coiled-
84 the conserved Microrchidia (MORC) adenosine triphosphatase (ATPase) family, which are predicted to c
85 xamined whether ITP can be used by adenosine triphosphatase (ATPase) in human erythrocytes or recombi
86 species and cell types, Na(+),K(+)-adenosine triphosphatase (ATPase) is arguably the most powerful co
87 of MetNI reveals two copies of the adenosine triphosphatase (ATPase) MetN in complex with two copies
88 t of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, unfolds, an
89 otein (Prp5p) is an RNA-stimulated adenosine triphosphatase (ATPase) required for prespliceosome form
91 for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA translocation an
92 acilitates both recruitment of the adenosine triphosphatase (ATPase)-activating cochaperone Aha1 and,
93 symporter, and hydrogen potassium adenosine triphosphatase [ATPase]) showed reduced expression or mi
94 croscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral staircase
97 m ring-shaped hexameric AAA-family adenosine triphosphatases (ATPases), dynein's large size and compl
98 the microrchidia (MORC) family of adenosine triphosphatases (ATPases), has been shown to be involved
102 Protein release factor 3 (RF3), a guanosine triphosphatase, binds to ribosome after release of the n
104 witch by sensing mitochondrial Rho guanosine triphosphatase-Ca(2+) and as a brake by anchoring mitoch
106 at in living cells the cytoplasmic adenosine triphosphatase called ClpV specifically recognizes the c
107 re, we examine the function of Rho guanosine triphosphatase CDC-42 in AJ formation and regulation dur
108 PAR proteins (including the small guanosine triphosphatase CDC-42) have an active role in regulating
109 zation of the conserved Rho-family guanosine triphosphatase, Cdc42, to the cortical region destined t
112 embrane by the p97/Cdc48-Ufd1-Npl4 adenosine triphosphatase complex is essential for mitochondria-ass
114 of BRG1-also known as SMARCA4, an adenosine triphosphatase-containing chromatin remodeler-and SMAD3
116 mer composed of a conserved 54-kDa guanosine triphosphatase (cpSRP54) and a unique 43-kDa subunit (cp
118 tor of the vacuolar proton-pumping adenosine triphosphatase, cytosolic calcein fluorescence became qu
119 how that genetic variants leading to inosine triphosphatase deficiency, a condition not thought to be
120 ed Trio-dependent Rac1 activation, adenosine triphosphatase-deficient Hsc70 (D10N) abrogated Trio Rac
121 n a Rag-, Ragulator-, and vacuolar adenosine triphosphatase-dependent fashion, the translocation of m
122 the TASCC was amino acid- and Rag guanosine triphosphatase-dependent, and disruption of mTOR localiz
123 d assembly of some dynamin-related guanosine triphosphatases depends on adaptor proteins restricted t
124 is essential for stimulating HSP70 adenosine triphosphatase diverges in candidate orthologues, includ
125 iral replication on an intact nucleotide/RNA triphosphatase domain and an N-terminal cluster of basic
126 conserved Swc2/YL1 subunit and the adenosine triphosphatase domain of Swr1 are mainly responsible for
127 ns within Hebo: a TUDOR domain, an adenosine triphosphatase domain, and a new domain, HEBO, specifica
128 ositions (p.G488R, p.A495V) in the guanosine triphosphatase domain, each segregating with affected in
132 we provide evidence that the large guanosine triphosphatase dynamin2 and its partner, endothelial nit
133 actor Tif6 by the translocase-like guanosine triphosphatase Efl1 is a critical late maturation step.
134 42 (Cdc42) is a member of the Rho guanosine triphosphatase family and has pivotal functions in actin
135 ppears to load/unload RuvBL AAA(+) adenosine triphosphatase from pre-snoRNPs; and (d) a potential mec
136 adherens junction proteins and Rho guanosine triphosphatase from the cell periphery to the lagging ta
138 tional antiterminator, an inosine/xanthosine triphosphatase, GidA, a methyl-accepting chemotaxis prot
139 e factor (GEF) for Tem1, the small guanosine triphosphatase governing activity of the Saccharomyces c
141 SRP and SR, which stimulates their guanosine triphosphatase (GTPase) activities, leading to dissociat
143 this study, we show that the small guanosine triphosphatase (GTPase) adenosine diphosphate ribosylati
144 ect interaction between a small guanosine 5'-triphosphatase (GTPase) and a transmembrane receptor.
146 kinase 2 (LRRK2) protein has both guanosine triphosphatase (GTPase) and kinase activities, and mutat
148 -2, a protein that binds the small guanosine triphosphatase (GTPase) Cdc42 and a negative regulator o
152 t rotation and is catalyzed by the guanosine triphosphatase (GTPase) elongation factor G (EF-G).
153 Protein synthesis requires several guanosine triphosphatase (GTPase) factors, including elongation fa
154 by amino acids is mediated by Rag guanosine triphosphatase (GTPase) heterodimers on the lysosome.
161 bioprobe), we revealed that Cdc42 guanosine triphosphatase (GTPase) remains inactive within Drosophi
162 and animal cytokinesis, the small guanosine triphosphatase (GTPase) Rho1/RhoA has an established rol
165 of a 43-member IFN-gamma-inducible guanosine triphosphatase (GTPase) superfamily in mouse and human g
166 Elongation factor G (EF-G) is a guanosine triphosphatase (GTPase) that plays a crucial role in the
167 (PKG), protein kinase A (PKA), Rho guanosine triphosphatase (GTPase), and MLC phosphatase was monitor
168 sis, elongation factor G (EF-G), a guanosine triphosphatase (GTPase), binds to the ribosomal PRE-tran
169 ion, we identified the RAB35 small guanosine triphosphatase (GTPase)-a protein previously implicated
172 membrane-associated ROPs [Rho-like guanosine triphosphatases (GTPase) from plants], leading to change
173 witch region I of immunity-related guanosine triphosphatases (GTPases) (IRGs), a family of host defen
175 rotein that interacts with the Rag guanosine triphosphatases (GTPases) and Ragulator in an amino acid
176 s recruited to the lysosome by Rag guanosine triphosphatases (GTPases) and regulates anabolic pathway
177 ositol 4-kinases (PI4Ks) and small guanosine triphosphatases (GTPases) are essential for processes th
181 rocess is mediated by dynamin-like guanosine triphosphatases (GTPases) called atlastins (ATLs), which
184 uring the cellular activity of small guanine triphosphatases (GTPases) in response to a specific stim
187 he activity of the recycling small guanosine triphosphatases (GTPases) Rab4 or Rab11 was sufficient t
188 signaling pathways involving small guanosine triphosphatases (GTPases) regulate cell polarization.
191 o acids, which act through the Rag guanosine triphosphatases (GTPases) to promote mTORC1 translocatio
194 feron (IFN)-inducible subfamily of guanosine triphosphatases (GTPases) with well-established activity
195 the Ragulator complex and the Rag guanosine triphosphatases (GTPases), causing release of the inacti
196 proteins, the Rho family of small guanosine triphosphatases (GTPases), is critical for this actin an
198 t to be dependent on the Rag small guanosine triphosphatases (GTPases), the Ragulator complex, and th
199 esses all involve Rho family small guanosine triphosphatases (GTPases), which are regulated by the op
201 vation of Ras homolog (Rho) family guanosine triphosphatases (GTPases), which regulate the formation
202 ns--a family of four related small guanosine triphosphatases (GTPases)--interact with mTORC1 in an am
205 is catalyzed by the sequential action of RNA triphosphatase, guanylyltransferase, and (guanine-N-7)me
208 the Caenorhabditis elegans P-type adenosine triphosphatase homolog, TAT-1, is critical for maintaini
209 e through the action of Na(+)/K(+) adenosine triphosphatases in an integrated in vitro lipid bilayer
210 ocus on the role of the Rho family guanosine triphosphatases in endothelial function and vasoconstric
212 tes the activity of the Rho family guanosine triphosphatases (including RhoA and Rac1) in an adhesion
213 as combined with the vacuolar H(+)-adenosine triphosphatase inhibitor bafilomycin A1 or with cathepsi
214 hydrazide (Dynasore), a dynamin guanosine 5'-triphosphatase inhibitor, protected stromal cells agains
216 42 (Cdc42Hs) is a small, Rho-type guanosine triphosphatase involved in multiple cellular processes t
219 ally encoded, photoactivatable Rac guanosine triphosphatase is sufficient to direct migration of neut
221 ow that dynamin1 (Dyn1), the large guanosine triphosphatase, is an interacting partner of IRSp53 thro
223 estingly, Rap1, but not Rho family guanosine triphosphatases, is required for the response to collage
227 e triphosphatase (ITPA) gene causing inosine triphosphatase (ITPase) deficiency protect against ribav
228 nctional variants in ITPA that cause inosine triphosphatase (ITPase) deficiency were shown to protect
229 nhibition of Cdc42 and related Rho guanosine triphosphatases may be a general feature of cytokinesis
231 The mammalian dynamin-related guanosine triphosphatases Mfn1,2 and Opa1 are required for mitocho
232 t the mitochondrial outer membrane guanosine triphosphatase mitofusin (Mfn) 2 mediates Parkin recruit
233 tic properties of recombinant mouse thiamine triphosphatase (mThTPase) and determined its solution st
234 reported that the sodium potassium adenosine triphosphatase (Na/K-ATPase) can effect the amplificatio
235 alpha1 subunit of sodium potassium adenosine triphosphatase (Na/K-ATPase), acts as a receptor and an
238 its monomer-associated nucleoside and 5' RNA triphosphatase (NTPase/RTPase) activities that are media
239 ed as putative nucleotide binding nucleoside triphosphatases (NTPases) or nucleoside triphosphate (NT
240 lytic cleavage of the dynamin-like guanosine triphosphatase OPA1 in mitochondria is emerging as a cen
241 c oxidase, inhibition of F(1)F(0) adenosine triphosphatase, or replacement of all mtDNA-encoded gene
242 1, its partner Sel1, the cytosolic adenosine triphosphatase p97, and degradation by the proteasome.
243 Cdc42, a member of Rho GTPases (guanosine triphosphatases), participates in cytokine- and growth f
244 tructures through a unidirectional adenosine triphosphatase polymerization, primed with a single PrgJ
245 However, constitutively active Rag guanosine triphosphatases prevented TFEB translocation during mito
246 C3 pathways downstream of the ROP1 guanosine triphosphatase promoting actin assembly and disassembly,
247 disassembly of both ezrin and Na/K adenosine triphosphatase proteins from their cytoskeletal attachme
248 the sarcoplasmic reticulum Ca(2+)-adenosine triphosphatase pump and by augmented levels of the Na(+)
252 his study, we demonstrate that the guanosine triphosphatase Rab7 contributes to this recruitment.
253 tide exchange factor for the small guanosine triphosphatase Rab8, to promote recruitment of Rab8 and
254 downstream effectors of the small guanosine triphosphatases Rac and Cdc42, biochemically couple lead
259 iated by chromosomes and the small guanosine triphosphatase Ran in a process requiring ~16 hours.
261 ivation of the integrin regulatory guanosine triphosphatase Rap1 via the recruitment and activation o
262 to image the activity of the small guanosine triphosphatase Ras after NMDA receptor activation at ind
263 , such as RPGR (retinitis pigmentosa guanine triphosphatase regulator) that are expressed in both rod
265 ase c-Src and stimulated the small guanosine triphosphatase RhoA, consequently inhibiting cell spread
266 FoxF directly upregulate the small guanosine triphosphatase RhoDF, which synergizes with Cdc42 to con
268 mouse embryos that lack the small guanosine triphosphatase RSG1 die at embryonic day 12.5, with deve
271 asmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator of calciu
272 rcoplasmic reticulum Ca(2+) uptake adenosine triphosphatase (SERCA2a), phospholamban (PLB), and incre
274 receptor for DRP1, the cytoplasmic guanosine triphosphatase that catalyzes mitochondrial fission.
275 Mtr4 is an essential RNA-dependent adenosine triphosphatase that is required for all of the nuclear f
276 ated protein 1 (Drp1), a cytosolic guanosine triphosphatase that polymerizes and constricts the organ
277 ncodes a multitransmembrane domain adenosine triphosphatase that traffics from the trans-Golgi networ
278 the activity of Rho and Rap small guanosine triphosphatases that control integrin activation by chem
279 , a member of the septin family of guanosine triphosphatases that form a diffusion barrier in budding
285 ogether with reduced vacuolar H(+)-adenosine triphosphatase (V-ATPase) activity, accounts for the red
286 leading to the removal of vacuolar adenosine triphosphatase (V-ATPase) and the neutralization of lyso
287 iched for subunits of the vacuolar adenosine triphosphatase (V-ATPase) complex, a proton pump require
288 gh inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the luminal concentr
290 0 domain of the vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) is directly implicated in secr
291 s never recycle vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) or neutralize to form postlyso
293 at the neuronal vacuolar-type H(+)-adenosine triphosphatase V0 subunit a1 (V100) can regulate the for
296 derstood family members are the eukaryal RNA triphosphatases, which catalyze the initial step in mRNA
297 pressorium by means of four septin guanosine triphosphatases, which polymerize into a dynamic, hetero
298 ter clusters may be universal for nucleotide triphosphatases with conserved active sites, such as myo
300 organelles, requires a Rab-family guanosine triphosphatase (Ypt7p), a Rab effector and Sec1/Munc18 (
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