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1 -SNPH coupling inhibited the motor adenosine triphosphatase.
2  were shown to be activated by Ran guanosine triphosphatase.
3 he level of the vacuolar-type H(+)-adenosine triphosphatase.
4  pumping enzyme vacuolar-type H(+)-adenosine triphosphatase.
5 hich encodes a copper-transporting adenosine triphosphatase.
6 ral large structural proteins and nucleoside triphosphatases.
7 C is activated by Rho family small guanosine triphosphatases.
8 f cell surface-associated vacuolar adenosine triphosphatases.
9  sensitivity and drug efficacy in nucleotide triphosphatases.
10 he absence of its two different types of RNA triphosphatases.
11 nd physically interacts with small guanosine triphosphatases.
12 horylation of plasma membrane H(+)-adenosine triphosphatase 2 at Ser(899), mediating the inhibition o
13 a critical proteasome subunit, non-adenosine triphosphatase 4 (PSMD4), was reduced in old mice.
14 s involved in the purine metabolism (inosine triphosphatase, 5'-nucleotidase cytosolic-II, purine nuc
15 at the Insert B domain of the Dnm1 guanosine triphosphatase (a DRP) contains a novel motif required f
16 es (AIs) that contain the cellular adenosine triphosphatase ABCE1 (ATP-binding cassette protein E1).
17 ing protein 1 (ELMOD1), a guanine nucleoside triphosphatase activating protein (GAP) for ARF6, as the
18           We have identified a Rho guanosine triphosphatase activating protein (RhoGAP), PAC-1, which
19 rsed at the lagging pole where the guanosine triphosphatase activating protein MglB disrupts the MglA
20 ce that neurofibromin, via its Ras guanosine triphosphatase -activating activity, controls ERK1/2-dep
21 -CYK4 centralspindlin complex is a guanosine triphosphatase-activating protein (GAP) for Rac1 and not
22                            p190Rho-guanosine triphosphatase-activating protein (GAP) is known to inhi
23       Here, we show that the Cdc42 guanosine triphosphatase-activating protein (GAP) Rga1 establishes
24 at encodes alpha2-chimaerin, a Rac guanosine triphosphatase-activating protein (RacGAP) signaling pro
25 binds simultaneously to the Cdc42p guanosine triphosphatase-activating protein Bem3p.
26 ure events, independent of the Rho guanosine triphosphatase-activating protein domain.
27 es small ARF GAP1 (SMAP1), an ARF6 guanosine triphosphatase-activating protein that functions in clat
28 lating the expression level of Ras guanosine triphosphatase-activating protein.
29 vity for p120RasGAP (RASA1), a Ras guanosine triphosphatase-activating protein.
30 2/Bub2/Cdc16 domain-containing Rab guanosine triphosphatase-activating proteins (GAPs) and identified
31                            Two Rab guanosine triphosphatases-activating proteins (GAPs) have been imp
32  signaling to engage small GTPase (guanosine triphosphatase) activation and AMPAR synaptic delivery d
33                       Both nuclease and dNTP triphosphatase activities of SAMHD1 are associated with
34 r has ATPase and, more generally, nucleoside triphosphatase activities that are indistinguishable fro
35 rmation, a mutant virus that lacked both RNA triphosphatase activities was constructed.
36 dependent protease, RNA helicase, and 5'-RNA triphosphatase activities.
37  the N-terminal part exhibits NTPase and RNA triphosphatase activity and is proposed to have helicase
38 s altered by inhibition of dynamin guanosine triphosphatase activity and is temporally distinct from
39                         Both Rab18 guanosine triphosphatase activity and isoprenylation are required
40                   Here, we show that the RNA triphosphatase activity of DUSP11 promotes the RNA silen
41 es contractility by decreasing the adenosine triphosphatase activity of the cardiac myosin heavy chai
42 , suppression of PDGF-induced Rac1 guanosine triphosphatase activity, and Akt and extracellular signa
43  this protein has been shown to possess dNTP triphosphatase activity, which is proposed to inhibit HI
44  its double-stranded DNA-dependent adenosine triphosphatase activity.
45 ng predominantly determined by its guanosine triphosphatase activity.
46 polyhedrovirus encodes two proteins with RNA triphosphatase activity.
47 d N7 positions, and also displays nucleotide triphosphatase activity.
48 MDCK) cells and identified the rho-guanosine triphosphatase adaptor and scaffolding protein IRSp53 as
49 , inhibition of filamentous septin guanosine triphosphatases alleviates constriction defects in Auror
50 ses expression of the gastric H, K-adenosine triphosphatase alpha-subunit (HKalpha), which could cont
51 terminal serine proteinase domain and an RNA triphosphatase, an NTPase domain, and an RNA helicase in
52 e folate pathway-related genes, deoxyuridine triphosphatase and dihydrofolate reductase, the silencin
53 ists of an N-terminal domain with nucleoside triphosphatase and helicase activities and a C-terminal
54     FtsZ is an essential bacterial guanosine triphosphatase and homolog of mammalian beta-tubulin tha
55  known exchange factor for the Ran guanosine triphosphatase and performs essential roles in nuclear t
56 by overexpression of the capping enzymes RNA triphosphatase and RNA guanylyltransferase.
57 strate- and product-bound mammalian thiamine triphosphatase and with previously reported structures o
58 oluble and stable, did not bind to guanosine triphosphatases and bound more tightly to the PAK1 kinas
59 equires the function of Rho family guanosine triphosphatases and reorganization of the actin cytoskel
60 on molecules that activate RAS/RHO guanosine triphosphatases and their effector mitogen-activated pro
61 e and activates mTORC1 via the Rag guanosine triphosphatases and their regulators GATOR1 and GATOR2.
62 tein levels of MIG-2, a Rho family guanosine triphosphatase, and/or down-regulate INA-1, an integrin
63 ility signaling proteins (kinases, guanosine triphosphatases, and guanine exchange factors) controlle
64 gella when exogenous ubiquitin and adenosine triphosphatase are added, suggesting that the ubiquitin
65 bers of the RAS subfamily of small guanosine triphosphatases are found in > 30% of all human cancers.
66                            The Rho guanosine triphosphatases are well known regulators of cell juncti
67    We found that HIV-1 Nef and the guanosine triphosphatase Arf1 induced trimerization and activation
68 Rafiq et al. reveal that the small guanosine triphosphatase ARF1, a well-known orchestrator of membra
69 served members of the AAA+ family (adenosine triphosphatases associated with diverse cellular activit
70 6 (which carries activators of Rho guanosine triphosphatase) at the cell cortex using total internal
71  an ER network is dependent on the guanosine triphosphatase atlastin (ATL).
72 clohexylurea group, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) channels fo
73 retion by the nongastric H(+)/K(+) adenosine triphosphatase (ATP12A) acidified airway surface liquid,
74    We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary for amino
75                      The vesicular adenosine triphosphatase (ATPase) acidifies intracellular compartm
76 ed of the Mre11 nuclease and Rad50 adenosine triphosphatase (ATPase) active sites dimerizes through a
77 tive orientation of the N-terminal adenosine triphosphatase (ATPase) and C-terminal nuclease domains.
78 nd 130 nM in microtubule-dependent adenosine triphosphatase (ATPase) and cell-based cytotoxicity assa
79 ties related to inhibition of Na/K adenosine triphosphatase (ATPase) and stimulation of sarcoplasmic
80  Here we present evidence that the adenosine triphosphatase (ATPase) cycle of the SF1 helicase Upf1 i
81 ent in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at which the
82 nally characterized the DExD/H box adenosine triphosphatase (ATPase) Dhh1, a critical regulator of th
83 eparated from its ring-shaped AAA+ adenosine triphosphatase (ATPase) domain by a 15-nanometer coiled-
84  the conserved Microrchidia (MORC) adenosine triphosphatase (ATPase) family, which are predicted to c
85 xamined whether ITP can be used by adenosine triphosphatase (ATPase) in human erythrocytes or recombi
86 species and cell types, Na(+),K(+)-adenosine triphosphatase (ATPase) is arguably the most powerful co
87 of MetNI reveals two copies of the adenosine triphosphatase (ATPase) MetN in complex with two copies
88 t of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, unfolds, an
89 otein (Prp5p) is an RNA-stimulated adenosine triphosphatase (ATPase) required for prespliceosome form
90            p97 is a hexameric AAA+ adenosine triphosphatase (ATPase) that is an attractive target for
91 for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA translocation an
92 acilitates both recruitment of the adenosine triphosphatase (ATPase)-activating cochaperone Aha1 and,
93  symporter, and hydrogen potassium adenosine triphosphatase [ATPase]) showed reduced expression or mi
94 croscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral staircase
95              In eukaryotes, P-type adenosine triphosphatases (ATPases) generate the plasma membrane p
96                      DNA-dependent adenosine triphosphatases (ATPases) participate in a broad range o
97 m ring-shaped hexameric AAA-family adenosine triphosphatases (ATPases), dynein's large size and compl
98  the microrchidia (MORC) family of adenosine triphosphatases (ATPases), has been shown to be involved
99 es belonging to the AAA+ family of adenosine triphosphatases (ATPases).
100           Here we show that rotary adenosine triphosphatases (ATPases)/synthases from Thermus thermop
101  the BAG1 and bradyzoite-specific nucleoside triphosphatase (B-NTPase) promoters.
102  Protein release factor 3 (RF3), a guanosine triphosphatase, binds to ribosome after release of the n
103                       PTP/BVP is also an RNA triphosphatase, but is not essential for viral replicati
104 witch by sensing mitochondrial Rho guanosine triphosphatase-Ca(2+) and as a brake by anchoring mitoch
105 e cleaved from a2 isoform vacuolar adenosine triphosphatase called a2NTD.
106 at in living cells the cytoplasmic adenosine triphosphatase called ClpV specifically recognizes the c
107 re, we examine the function of Rho guanosine triphosphatase CDC-42 in AJ formation and regulation dur
108  PAR proteins (including the small guanosine triphosphatase CDC-42) have an active role in regulating
109 zation of the conserved Rho-family guanosine triphosphatase, Cdc42, to the cortical region destined t
110 etween the decoding center and the guanosine triphosphatase center of EF-Tu.
111                                      The RNA triphosphatases characteristically hydrolyze nucleoside
112 embrane by the p97/Cdc48-Ufd1-Npl4 adenosine triphosphatase complex is essential for mitochondria-ass
113 neuron-specific proton pump and V0 adenosine triphosphatase component V100.
114  of BRG1-also known as SMARCA4, an adenosine triphosphatase-containing chromatin remodeler-and SMAD3
115         The Rab subfamily of small guanosine triphosphatases controls these processes by acting as a
116 mer composed of a conserved 54-kDa guanosine triphosphatase (cpSRP54) and a unique 43-kDa subunit (cp
117 s on membranes and suppress futile adenosine triphosphatase cycles.
118 tor of the vacuolar proton-pumping adenosine triphosphatase, cytosolic calcein fluorescence became qu
119 how that genetic variants leading to inosine triphosphatase deficiency, a condition not thought to be
120 ed Trio-dependent Rac1 activation, adenosine triphosphatase-deficient Hsc70 (D10N) abrogated Trio Rac
121 n a Rag-, Ragulator-, and vacuolar adenosine triphosphatase-dependent fashion, the translocation of m
122  the TASCC was amino acid- and Rag guanosine triphosphatase-dependent, and disruption of mTOR localiz
123 d assembly of some dynamin-related guanosine triphosphatases depends on adaptor proteins restricted t
124 is essential for stimulating HSP70 adenosine triphosphatase diverges in candidate orthologues, includ
125 iral replication on an intact nucleotide/RNA triphosphatase domain and an N-terminal cluster of basic
126 conserved Swc2/YL1 subunit and the adenosine triphosphatase domain of Swr1 are mainly responsible for
127 ns within Hebo: a TUDOR domain, an adenosine triphosphatase domain, and a new domain, HEBO, specifica
128 ositions (p.G488R, p.A495V) in the guanosine triphosphatase domain, each segregating with affected in
129  a C-terminal guanylyltransferase and RNA 5'-triphosphatase domain.
130                The dynamin-related guanosine triphosphatase Drp1 mediates the division of mitochondri
131                       The large guanosine 5'-triphosphatase dynamin (Dyn) plays an important role in
132 we provide evidence that the large guanosine triphosphatase dynamin2 and its partner, endothelial nit
133 actor Tif6 by the translocase-like guanosine triphosphatase Efl1 is a critical late maturation step.
134  42 (Cdc42) is a member of the Rho guanosine triphosphatase family and has pivotal functions in actin
135 ppears to load/unload RuvBL AAA(+) adenosine triphosphatase from pre-snoRNPs; and (d) a potential mec
136 adherens junction proteins and Rho guanosine triphosphatase from the cell periphery to the lagging ta
137 mutations in a copper-transporting adenosine triphosphatase gene, ATP7A.
138 tional antiterminator, an inosine/xanthosine triphosphatase, GidA, a methyl-accepting chemotaxis prot
139 e factor (GEF) for Tem1, the small guanosine triphosphatase governing activity of the Saccharomyces c
140                         Rho family guanosine triphosphatase (GTPase) 3 (Rnd3), a member of the small
141 SRP and SR, which stimulates their guanosine triphosphatase (GTPase) activities, leading to dissociat
142 onstitutively active because their guanosine triphosphatase (GTPase) activity is disabled.
143 this study, we show that the small guanosine triphosphatase (GTPase) adenosine diphosphate ribosylati
144 ect interaction between a small guanosine 5'-triphosphatase (GTPase) and a transmembrane receptor.
145                          LRRK2 has guanosine triphosphatase (GTPase) and kinase activities, and mutat
146  kinase 2 (LRRK2) protein has both guanosine triphosphatase (GTPase) and kinase activities, and mutat
147         This process requires Rab9 guanosine triphosphatase (GTPase) and the putative tether GCC185.
148 -2, a protein that binds the small guanosine triphosphatase (GTPase) Cdc42 and a negative regulator o
149                            The Rho guanosine triphosphatase (GTPase) Cdc42 regulates sequential molec
150  the exocyst complex, a common Ral guanosine triphosphatase (GTPase) effector.
151                                The guanosine triphosphatase (GTPase) elongation factor G (EF-G) catal
152 t rotation and is catalyzed by the guanosine triphosphatase (GTPase) elongation factor G (EF-G).
153 Protein synthesis requires several guanosine triphosphatase (GTPase) factors, including elongation fa
154  by amino acids is mediated by Rag guanosine triphosphatase (GTPase) heterodimers on the lysosome.
155 ion in the gene encoding the small guanosine triphosphatase (GTPase) NRAS.
156                Deregulation of Rho guanosine triphosphatase (GTPase) pathways plays an important role
157                            The small guanine triphosphatase (GTPase) proteins RhoA and RhoC are essen
158 pulating the activity of the small guanosine triphosphatase (GTPase) Rab1.
159                          The small guanosine triphosphatase (GTPase) Rab7 has been implicated in the
160                          The small guanosine triphosphatase (GTPase) Rap1 is an important regulator o
161  bioprobe), we revealed that Cdc42 guanosine triphosphatase (GTPase) remains inactive within Drosophi
162  and animal cytokinesis, the small guanosine triphosphatase (GTPase) Rho1/RhoA has an established rol
163            During cytokinesis, the guanosine triphosphatase (GTPase) RhoA orchestrates contractile ri
164                 Etd1 activates the guanosine triphosphatase (GTPase) Spg1 to trigger signaling throug
165 of a 43-member IFN-gamma-inducible guanosine triphosphatase (GTPase) superfamily in mouse and human g
166    Elongation factor G (EF-G) is a guanosine triphosphatase (GTPase) that plays a crucial role in the
167 (PKG), protein kinase A (PKA), Rho guanosine triphosphatase (GTPase), and MLC phosphatase was monitor
168 sis, elongation factor G (EF-G), a guanosine triphosphatase (GTPase), binds to the ribosomal PRE-tran
169 ion, we identified the RAB35 small guanosine triphosphatase (GTPase)-a protein previously implicated
170 les mediated by the atlastin (ATL) guanosine triphosphatase (GTPase).
171 ase-associated RAS subfamily small guanosine triphosphatase (GTPase).
172 membrane-associated ROPs [Rho-like guanosine triphosphatases (GTPase) from plants], leading to change
173 witch region I of immunity-related guanosine triphosphatases (GTPases) (IRGs), a family of host defen
174                            The Rag guanosine triphosphatases (GTPases) activate mTORC1 in response to
175 rotein that interacts with the Rag guanosine triphosphatases (GTPases) and Ragulator in an amino acid
176 s recruited to the lysosome by Rag guanosine triphosphatases (GTPases) and regulates anabolic pathway
177 ositol 4-kinases (PI4Ks) and small guanosine triphosphatases (GTPases) are essential for processes th
178                                Rho guanosine triphosphatases (GTPases) are implicated in TEM, but the
179                                Rab guanosine triphosphatases (GTPases) are pivotal regulators of memb
180                                The guanosine triphosphatases (GTPases) Arf, Sar1, and dynamin are cor
181 rocess is mediated by dynamin-like guanosine triphosphatases (GTPases) called atlastins (ATLs), which
182                                Rab guanosine triphosphatases (GTPases) control cellular trafficking p
183                                Rho guanosine triphosphatases (GTPases) control the cytoskeletal dynam
184 uring the cellular activity of small guanine triphosphatases (GTPases) in response to a specific stim
185                  The Rho family of guanosine triphosphatases (GTPases) is composed of members of the
186                                The guanosine triphosphatases (GTPases) of the immunity-associated pro
187 he activity of the recycling small guanosine triphosphatases (GTPases) Rab4 or Rab11 was sufficient t
188 signaling pathways involving small guanosine triphosphatases (GTPases) regulate cell polarization.
189                                Ras guanosine triphosphatases (GTPases) regulate signaling pathways on
190              Septins are filamentous guanine triphosphatases (GTPases) that associate with MTs, but t
191 o acids, which act through the Rag guanosine triphosphatases (GTPases) to promote mTORC1 translocatio
192       Nutrients signal via the Rag guanosine triphosphatases (GTPases) to promote the localization of
193                  Activation of Rho guanosine triphosphatases (GTPases) to the guanine triphosphate (G
194 feron (IFN)-inducible subfamily of guanosine triphosphatases (GTPases) with well-established activity
195  the Ragulator complex and the Rag guanosine triphosphatases (GTPases), causing release of the inacti
196  proteins, the Rho family of small guanosine triphosphatases (GTPases), is critical for this actin an
197          Many of those factors are guanosine triphosphatases (GTPases), proteins that catalyze the hy
198 t to be dependent on the Rag small guanosine triphosphatases (GTPases), the Ragulator complex, and th
199 esses all involve Rho family small guanosine triphosphatases (GTPases), which are regulated by the op
200       Amino acids activate the Rag guanosine triphosphatases (GTPases), which promote the translocati
201 vation of Ras homolog (Rho) family guanosine triphosphatases (GTPases), which regulate the formation
202 ns--a family of four related small guanosine triphosphatases (GTPases)--interact with mTORC1 in an am
203 tin cytoskeleton by inhibiting Rho guanosine triphosphatases (GTPases).
204 s signal to mTORC1 through the Rag guanosine triphosphatases (GTPases).
205 is catalyzed by the sequential action of RNA triphosphatase, guanylyltransferase, and (guanine-N-7)me
206            Activation of the small guanosine triphosphatase H-Ras by the exchange factor Son of Seven
207 e coiled coil emerging from Smc3's adenosine triphosphatase head.
208  the Caenorhabditis elegans P-type adenosine triphosphatase homolog, TAT-1, is critical for maintaini
209 e through the action of Na(+)/K(+) adenosine triphosphatases in an integrated in vitro lipid bilayer
210 ocus on the role of the Rho family guanosine triphosphatases in endothelial function and vasoconstric
211 hagy, we silenced VCP or expressed adenosine triphosphatase-inactive VCP.
212 tes the activity of the Rho family guanosine triphosphatases (including RhoA and Rac1) in an adhesion
213 as combined with the vacuolar H(+)-adenosine triphosphatase inhibitor bafilomycin A1 or with cathepsi
214 hydrazide (Dynasore), a dynamin guanosine 5'-triphosphatase inhibitor, protected stromal cells agains
215             Proton pump (H(+)/K(+)-adenosine triphosphatase) inhibitors (PPIs) are widely used to tre
216  42 (Cdc42Hs) is a small, Rho-type guanosine triphosphatase involved in multiple cellular processes t
217                          The Cdc42 guanosine triphosphatase is essential for cell polarization in sev
218            The a2 isoform vacuolar adenosine triphosphatase is found on the surface on many solid tum
219 ally encoded, photoactivatable Rac guanosine triphosphatase is sufficient to direct migration of neut
220            The Rab family of small guanosine triphosphatases is evolutionarily conserved and mediates
221 ow that dynamin1 (Dyn1), the large guanosine triphosphatase, is an interacting partner of IRSp53 thro
222                       Cdc42, a Rho guanosine triphosphatase, is thought to orchestrate critical actin
223 estingly, Rap1, but not Rho family guanosine triphosphatases, is required for the response to collage
224                 Genetic variation of inosine triphosphatase (ITPA) causing an accumulation of inosine
225       Two functional variants in the inosine triphosphatase (ITPA) gene causing inosine triphosphatas
226                                      Inosine triphosphatase (ITPA) variants causing ITPase deficiency
227 e triphosphatase (ITPA) gene causing inosine triphosphatase (ITPase) deficiency protect against ribav
228 nctional variants in ITPA that cause inosine triphosphatase (ITPase) deficiency were shown to protect
229 nhibition of Cdc42 and related Rho guanosine triphosphatases may be a general feature of cytokinesis
230                       An intrinsic guanosine triphosphatase mediates a contact between the head and c
231      The mammalian dynamin-related guanosine triphosphatases Mfn1,2 and Opa1 are required for mitocho
232 t the mitochondrial outer membrane guanosine triphosphatase mitofusin (Mfn) 2 mediates Parkin recruit
233 tic properties of recombinant mouse thiamine triphosphatase (mThTPase) and determined its solution st
234 reported that the sodium potassium adenosine triphosphatase (Na/K-ATPase) can effect the amplificatio
235 alpha1 subunit of sodium potassium adenosine triphosphatase (Na/K-ATPase), acts as a receptor and an
236 nits and possesses 40S-stimulated nucleoside triphosphatase (NTPase) activity.
237                          Here, by nucleoside triphosphatase (NTPase) assay and comparisons of six hig
238 its monomer-associated nucleoside and 5' RNA triphosphatase (NTPase/RTPase) activities that are media
239 ed as putative nucleotide binding nucleoside triphosphatases (NTPases) or nucleoside triphosphate (NT
240 lytic cleavage of the dynamin-like guanosine triphosphatase OPA1 in mitochondria is emerging as a cen
241  c oxidase, inhibition of F(1)F(0) adenosine triphosphatase, or replacement of all mtDNA-encoded gene
242 1, its partner Sel1, the cytosolic adenosine triphosphatase p97, and degradation by the proteasome.
243    Cdc42, a member of Rho GTPases (guanosine triphosphatases), participates in cytokine- and growth f
244 tructures through a unidirectional adenosine triphosphatase polymerization, primed with a single PrgJ
245 However, constitutively active Rag guanosine triphosphatases prevented TFEB translocation during mito
246 C3 pathways downstream of the ROP1 guanosine triphosphatase promoting actin assembly and disassembly,
247 disassembly of both ezrin and Na/K adenosine triphosphatase proteins from their cytoskeletal attachme
248  the sarcoplasmic reticulum Ca(2+)-adenosine triphosphatase pump and by augmented levels of the Na(+)
249                          The small guanosine triphosphatase Rab13 functions in exocytic vesicle traff
250 s homologue of the mammalian small guanosine triphosphatase Rab2.
251 gulated by Wnt5a through the small guanosine triphosphatases Rab4 and RhoB.
252 his study, we demonstrate that the guanosine triphosphatase Rab7 contributes to this recruitment.
253 tide exchange factor for the small guanosine triphosphatase Rab8, to promote recruitment of Rab8 and
254  downstream effectors of the small guanosine triphosphatases Rac and Cdc42, biochemically couple lead
255 mics and the activity of the small guanosine triphosphatases Rac and Cdc42.
256 jor effectors of the Rho guanosine 5'\x{2011}triphosphatase, Rac, and Cdc42.
257 creasing the activity of the small guanosine triphosphatase Rac1 and Cdc42.
258 hanced by signaling from the small guanosine triphosphatase, Rac1.
259 iated by chromosomes and the small guanosine triphosphatase Ran in a process requiring ~16 hours.
260                          The small guanosine triphosphatase Rap1 regulates LFA-1 adhesiveness through
261 ivation of the integrin regulatory guanosine triphosphatase Rap1 via the recruitment and activation o
262 to image the activity of the small guanosine triphosphatase Ras after NMDA receptor activation at ind
263 , such as RPGR (retinitis pigmentosa guanine triphosphatase regulator) that are expressed in both rod
264       In many organisms, the small guanosine triphosphatase RhoA controls assembly and contraction of
265 ase c-Src and stimulated the small guanosine triphosphatase RhoA, consequently inhibiting cell spread
266 FoxF directly upregulate the small guanosine triphosphatase RhoDF, which synergizes with Cdc42 to con
267  a mimic of the human Ras-like Rho guanosine triphosphatase RhoG.
268  mouse embryos that lack the small guanosine triphosphatase RSG1 die at embryonic day 12.5, with deve
269                                The guanosine triphosphatase Sar1 controls the assembly and fission of
270 lum (SR) and reuptake by the Ca(2+)adenosine triphosphatase SERCA.
271 asmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator of calciu
272 rcoplasmic reticulum Ca(2+) uptake adenosine triphosphatase (SERCA2a), phospholamban (PLB), and incre
273 tropic perturbations to Rho family guanosine triphosphatase signaling and myosin II activity.
274 receptor for DRP1, the cytoplasmic guanosine triphosphatase that catalyzes mitochondrial fission.
275 Mtr4 is an essential RNA-dependent adenosine triphosphatase that is required for all of the nuclear f
276 ated protein 1 (Drp1), a cytosolic guanosine triphosphatase that polymerizes and constricts the organ
277 ncodes a multitransmembrane domain adenosine triphosphatase that traffics from the trans-Golgi networ
278  the activity of Rho and Rap small guanosine triphosphatases that control integrin activation by chem
279 , a member of the septin family of guanosine triphosphatases that form a diffusion barrier in budding
280                 Rabs are monomeric guanosine triphosphatases that regulate membrane trafficking and a
281        Fzo1 and Mgm1 are conserved guanosine triphosphatases that reside in the outer and inner membr
282                   Mammalian soluble thiamine triphosphatase (ThTPase) is a 25-kDa cytosolic enzyme th
283       Competitive binding of small guanosine triphosphatases to the IS domain disrupts the autoinhibi
284 4 (EF4/LepA) is a highly conserved guanosine triphosphatase translation factor.
285 ogether with reduced vacuolar H(+)-adenosine triphosphatase (V-ATPase) activity, accounts for the red
286 leading to the removal of vacuolar adenosine triphosphatase (V-ATPase) and the neutralization of lyso
287 iched for subunits of the vacuolar adenosine triphosphatase (V-ATPase) complex, a proton pump require
288 gh inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the luminal concentr
289                      The vesicular adenosine triphosphatase (v-ATPase) is a proton pump that acidifie
290 0 domain of the vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) is directly implicated in secr
291 s never recycle vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) or neutralize to form postlyso
292 tor complex, and the vacuolar H(+)-adenosine triphosphatase (v-ATPase).
293 at the neuronal vacuolar-type H(+)-adenosine triphosphatase V0 subunit a1 (V100) can regulate the for
294                 Binding to the AAA adenosine triphosphatase valosin-containing protein (VCP)/p97 is t
295                            The AAA-adenosine triphosphatase Vps4 disassembles and recycles the ESCRT-
296 derstood family members are the eukaryal RNA triphosphatases, which catalyze the initial step in mRNA
297 pressorium by means of four septin guanosine triphosphatases, which polymerize into a dynamic, hetero
298 ter clusters may be universal for nucleotide triphosphatases with conserved active sites, such as myo
299              The activation of Rho guanosine triphosphatases within the extending growth cone facilit
300  organelles, requires a Rab-family guanosine triphosphatase (Ypt7p), a Rab effector and Sec1/Munc18 (

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