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1 current-use additive flame retardants (FRs), tris (1,3-dichloro-isopropyl) phosphate (TDCPP) and Fire
2 tial and temporal distribution are given for Tris-(1-chloro-2-propyl) phosphate (TCiPP), EHDPP, tri-n
3                                              Tris-(1-chloro-2-propyl)phosphate (TCPP) was the most ab
4    The evidence includes structures with Bis-Tris (2,2-bis(hydroxymethyl)-2,2',2''-nitrilotriethanol)
5 t m/z 399, 421, 609.4, and 819.9 as those of tris (2-butoxyethyl) phosphate, originating in the gray
6 ein solutions (pH 7.4) were reduced by using Tris (2-carboxyethyl) phosphine HCl and irradiated with
7 ction with complete and partial reduction by tris (2-carboxyethyl) phosphine.
8                                 Furthermore, tris (2-chloroethyl) phosphate (TCEP), a known carcinoge
9 boxymethyl)-L-lysine amide (NTA-pyrene) and [tris-(2,2'-bipyridine) (4,4'-bis(4-pyrenyl-1-ylbutyloxy)
10  associated with excited-state relaxation in tris-(2,2'-bipyridine)ruthenium(II).
11        Reduction of fully active enzyme with tris-(2-carboxyethyl) phosphine converted it from a low
12                                          The tris-(2-carboxyethyl) phosphine-reduced enzyme was oxyge
13 ted KD values in the range 10-25 mm, whereas tris-(2-carboxyethyl)-phosphine showed stronger affinity
14  exogenous ligands (GSH, dithiothreitol, and tris-(2-carboxyethyl)-phosphine) to the Cu(I) was invest
15 k(cat) is approximately 10-fold greater when tris-(2-carboxyethyl)phosphine (TCEP), the most efficien
16 odiglycol) or the non-sulphur reducing agent Tris-(2-carboxylethyl)phosphine (TCEP).
17 [3,2-d:2',3'-f]-quinoxaline (dpq), and PCN = tris-(2-cyanoethyl)phosphine, have been synthetized and
18 s achieved by using 2-nitrophenyloctylether, tris-(2-ethylhexyl) phosphate (10%) and di-(2-ethylhexyl
19 me, 40 s; SLM, 2-nitrophenyl octyl ether+10% tris-(2-ethylhexyl) phosphate+10% di-(2-ethylhexyl) phos
20 yl)-N,N'-dimethyl-1,2-ethylenediamine; TPA = tris-(2-pyridylmethyl)amine] catalyze the oxidation of o
21 nt with those previously observed for simple tris -2,2'-bipyridyl Ru(II) complexes.
22 rt complex based upon the well-characterized tris -2,2'-bipyridyl Ru(II).
23 ical reaction of Tp'Rh(L)(eta2-PhN=L) [Tp' = tris-(3,5-dimethyl pyrazolyl)borate, L = CNCH(2)CMe(3)]
24  method is based on the use of two cellulose-tris-(3,5-dimethylphenylcarbamate) columns connected in
25 ing Tp'Re(O)(diolato) complex (Tp' = hydrido-tris-(3,5-dimethylpyrazolyl)borate) was measured competi
26 ] reactive fragment have been studied (Tp' = tris-(3,5-dimethylpyrazolyl)borate; L = CNCH2CMe3).
27 th chiral shift reagents, Eu(hfc)3 [europium tris [3-[(heptafluoropropyl)hydroxymethylene]-(+)-campho
28 te, the ERalpha selective agonist PPT (1,3,5-tris (4-hydroxyphenyl)-4-propyl-1H-pyrazole) and the ERb
29 n acoustically levitated droplets of aqueous tris (50 mM) at pH 8.5 at 22 +/- 2 degrees C and in supe
30 survival rates (50.9%) than patients without TRIs (74.3%; P=0.03).
31 sed to interrogate interfaces formed between tris-(8-hydroxyquinoline) aluminum (Alq(3)) and vapor-de
32 omposed of a thin-film organic semiconductor tris-(8-hydroxyquinoline) aluminum (Alq3) doped with DCM
33 inescence from an Alq(3)/TPD heterojunction (tris-(8-hydroxyquinoline)aluminum/N,N'-bis(3-methylpheny
34                  Exhaustive reduction of the tris-[8]annulenyl isocyanurate with potassium in THF gen
35 duct reveal the actual reduced species to be tris-[8]annulenyl isocyanurate.
36 ed Arabidopsis 5PTases, which also hydrolyze tris- and bis inositol phosphate molecules.
37                                    The rigid tris- and bis(catecholamide) ligands H(6)A, H(4)B and H(
38 ctive 5PTase whose activity is restricted to tris- and bis-, but not mono-phosphorylated phosphatidyl
39                     Agonist induced inositol tris- and bis-phosphate production and calcium release r
40 ectroscopic characterization of Cs-symmetric tris- and C2v-symmetric tetra-adducts of C70, which are
41                                     Inositol tris- and tetrakisphosphates interact via three phosphat
42 investigations of a series of 53 novel bis-, tris-, and tetrakis(carboxylato)platinum(IV) complexes,
43                     A series of mono-, bis-, tris-, and tetrakis(porphinato)zinc(II) (PZn)-elaborated
44                                        Bis-, tris-, and tetrakisuracil-substituted 12-, 13-, 17-, and
45 abeled at the single cysteine residue with a tris (bipyridyl)ruthenium(II) reagent to form Ru-39-Cc.
46 ay 3), injected CA II protein + Tris or just Tris (Day 3), measured I(NBC) or the initial rate at whi
47 l) spermidine (1.1-2.6), and N(1),N(5),N(14)-tris-(dihydrocaffeoyl) spermine (trace - 11.1).
48 aining the supF gene, was treated with 2,3,5-tris-(glutathion-S-yl)hydroquinone and replicated in bot
49                               However, 2,3,5-tris-(glutathion-S-yl)hydroquinone, a potent nephrotoxic
50               In support of this view, 2,3,5-tris-(glutathione-S-yl)hydroquinone (TGHQ)-induced phosp
51 electivity of K approximately equal to Na >> Tris > N-methyl-D-glucamine (NMDG), and is blocked by hi
52 onstrated using hydroxyethylcellulose and 3-[tris-(hydroxymethyl) methylamino]-1-propanesulfonic acid
53       Changing the pH buffer from HEPES to N-tris-(hydroxymethyl)methyl-2-aminoethanesulfonic acid di
54  resting state that consists of a mixture of tris-(kappa2-amidate)aluminum(III) complexes.
55                            Enterobactin, the tris-(N-(2,3-dihydroxybenzoyl)serine) trilactone siderop
56 were performed using a relatively accessible tris-(N-phenyl)urea.
57 CoBr(2) and Co metal in the presence of TPB (tris-(o-diisopropylphophinophenyl)borane) gives (TPB)CoB
58 ported [SiP(3)]Co(N(2)) (1-N(2)) ([SiP(3)] = tris-(o-diisopropylphosphinophenyl)silyl).
59    The photosensitized oxidation of 2',3',5'-tris-(O-tert-butyldimethylsilyl)-8-oxo-7,8-dihydroguanos
60 complex supported by a bridging deca-anionic tris-(OMe)salophen(10-) ligand where three (OMe)salophen
61 uction affording the Co(I)-Co(II)-Co(I) [Co3(tris-(OMe)salophen)Na6(THF)6], 6 complex supported by a
62  hepatitis C associated with the presence of TRIs (P=0.04) along with donor-specific antibodies (P=0.
63                     Upon incubation in 50 mM Tris (pH 7.5), 0.1 M NaCl, and 5 mM EDTA at 37 degrees C
64 om virions disrupted by treatment with 50 mM Tris (pH 7.5), 0.5 M NaCl, 0.5% NP-40, and 10 mM dithiot
65  k1 = approximately 2 x 10(6) M-1 s-1 [20 mM Tris (pH 7.5), 6 mM NaCl, 5 mM MgCl2, 5 mM 2-mercaptoeth
66 onditions [350 mM KCl, 8 mM MgCl2, and 30 mM Tris (pH 7.5)], a complex with an association constant o
67 ation constant in our standard buffer (40 mm Tris (pH 7.9), 10 mm MgCl(2), 0.1 mm dithiothreitol, 5%
68 e addition of glycerol to 500 mM NaCl, 20 mM Tris (pH 8.4), 2 mM beta-mercaptoethanol significantly e
69 , 27% sucrose (wt/vol), 2 mM EDTA, and 10 mM Tris (pH 9), were required for efficient OM release, as
70 A low-ionic-strength solution, 6.1muS/cm 1mM Tris (pH 9.3), was used to produce ACEO and proved the f
71 f a sample that contains a weak base such as Tris (pK(a) = 8.2) is presented here for the first time.
72 ligands in the meridianal isomer of aluminum tris-(quinoline-8-olate) (Alq(3)), a widely used electro
73 aphically distinct myo-inositol mono-, bis-, tris-, tetrakis-, pentakis-, and hexakisphosphates have
74 e found to bind with exceptional affinity to tris-(triazole ethylamine) cryptophane, a previously uns
75  results from hydrogen peroxide oxidation of Tris [tris(hydroxymethyl)aminomethane] present in transc

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