ライフサイエンスコーパス: triton

1 oglia and DI-TNC1 astrocytes were exposed to triton (1%) or CORM-3 (10-100 iM) and cytotoxicity (lact

2 With Triton added at an optimal level, close to critical mice

3 erived for other solar nebula bodies such as Triton and Pluto, but is very different from that of the

4 ing molecule in the atmospheres of Pluto and Triton and probably the main nitrogen reservoir from whi

5 aration of non-canonical histone variants by triton AU(TAU) and 2D TAU electrophoresis; and immunoblo

6 al, human pancreata were decellularized with Triton-based solution and thoroughly characterized.

7 3% p-formaldehyde and permeabilized with 1% Triton before immunocytochemical detection of ZO-1 and n

8 using the Cirrus 5000, RTVue XR Avanti, and Triton DRI OCT platforms with default layer segmentation

9 both the recombinant 27-kDa (r27) and native Triton-extracted 17-kDa (TX17) Cryptosporidium antigens.

10 We followed myosin classes V, VI, and X on triton-extracted actin cytoskeletons from Drosophila S2,

11 inhibitors decreased the level of cTnT-ND in Triton-extracted myofibrils.

12 These aggregates were hyperphosphorylated, Triton insoluble, and thioflavin-S positive, either comi

13 T) and phosphorylated (Ser129) hSYN(A53T) in Triton-insoluble aggregates.

14 A Triton-insoluble band ("raft fraction") at the 5%/30% su

15 of ezrin in undifferentiated cells showed a Triton-insoluble ezrin compartment negative for phospho-

16 states by cycling between Triton-soluble and Triton-insoluble forms.

17 smoplakin but not desmocollin (Dsc) 3 in the Triton-insoluble fraction of cell lysates within 2 hours

18 ted in the mutant oocytes and found that the Triton-insoluble fraction of tubulin was significantly d

19 Lipid analysis showed that, the Triton-insoluble fraction was highly enriched in cholest

20 m of MLK3 is inactive and redistributes to a Triton-insoluble fraction.

21 also abrogated interaction of F protein with Triton-insoluble lipid rafts.

22 ownregulation of the cytoskeletal-associated triton-insoluble pool of E-cadherin and the appearance o

23 h this effect and that Triton-soluble versus Triton-insoluble tau may be independently targeted by ki

24 Pulse-chase analysis showed that Triton-insoluble, newly synthesized ezrin transiently co

25 Triton is Neptune's principal satellite and is by far th

26 compounds was done by using an acute model (Triton model), in which compounds 3f and 3l showed signi

27 ual configuration has led to the belief that Triton originally orbited the Sun before being captured

28 mia and bleeding risk, a large proportion of TRITON participants (42%) were predicted to experience n

29 paired myofilament calcium responsiveness in Triton-permeabilized cardiomyocytes.

30 Neelsen stain involving cytospin slides with Triton processing, and an ESAT-6 immunocytochemical stai

31 lly inward on the silicon surface exposed to Triton((R)) X-100 containing HF based etchant solution.

32 when dimethyl sulfoxide was used instead of Triton((R)) X-100 in the etchant solution, no such morph

33 To transfer Triton-resistant chromatin binding to green fluorescent

34 ok domain from full-length LEDGF/p75 reduced Triton-resistant chromatin binding, while deletion of bo

35 Neptune is a far more likely explanation for Triton's capture.

36 Triton shows the pattern of a segregating surfactant in

37 he sarcomere shortening-Ca2+ relationship in Triton-skinned cardiomyocytes revealed a significant red

38 rce and relaxation rate were investigated in Triton-skinned rat caudal arterial smooth muscle strips.

39 were isolated by mechanical homogenization, Triton-skinned, and attached to micropipettes that proje

40 M myosin filament assembly, as assessed by a Triton solubility assay and a proximity ligation assay a

41 amentous actin (sF-actin) fractions based on Triton solubility of cell lysates.

42 uorescence recovery after photobleaching and Triton solubility.

43 in interaction network that remains after 1% Triton solubilization.

44 n and deactivation states by cycling between Triton-soluble and Triton-insoluble forms.

45 0 kDa ectodomain fragment in the cytoplasmic triton-soluble pool.

46 may be associated with this effect and that Triton-soluble versus Triton-insoluble tau may be indepe

47 cular events as compared with clopidogrel in TRITON-TIMI 38 (Trial to Assess Improvement in Therapeut

48 aintenance doses of prasugrel on events in a TRITON-TIMI 38 (TRial to Assess Improvement in Therapeut

49 e potent antiplatelet agent prasugrel in the TRITON-TIMI 38 (Trial to Assess Improvement in Therapeut

50 In the TRITON-TIMI 38 (TRial to Assess Improvement in Therapeut

51 red, and the data combined with the existing TRITON-TIMI 38 database.

52 Researchers in the TRITON-TIMI 38 randomized 13,608 subjects with acute cor

53 A subset of the TRITON-TIMI 38 study (Trial to Assess Improvement in The

54 prasugrel or clopidogrel with stents in the TRITON-TIMI 38 study.

55 The TRITON-TIMI 38 trial has shown that prasugrel-a novel, p

56 In the TRITON-TIMI 38 trial, 13,608 patients with an acute coro

57 In the TRITON-TIMI 38 trial, the primary endpoint was the compo

58 dogrel (n=1471) or prasugrel (n=1461) in the TRITON-TIMI 38 trial.

59 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38) demonstrated that treatment with prasugr

60 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38) showed an overall reduction in ischemic

61 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38), we fit risk models for ischemic events

62 l--Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38).

63 ugrel-Thrombolysis in Myocardial Infarction (TRITON-TIMI 38).

64 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38).

65 e index procedure following randomisation in TRITON-TIMI 38, and were further subdivided by type of s

66 Among clopidogrel-treated subjects in TRITON-TIMI 38, carriers had a relative increase of 53%

67 In TRITON-TIMI 38, the safety and efficacy outcomes of pras

68 In TRITON-TIMI 38, we classified 12,674 patients into low-d

69 nts treated with clopidogrel or prasugrel in TRITON-TIMI 38.

70 the US healthcare system by using data from TRITON-TIMI 38.

71 l with clopidogrel among subjects with DM in TRITON-TIMI 38.

72 ugrel-Thrombolysis in Myocardial Infarction (TRITON-TIMI) 38.

73 Our model predicts that Triton was once a member of a binary with a range of pla

74 ized indole-based fibrates were evaluated in Triton WR-1339 and high fat diet (HFD)-induced hyperlipi

75 assessed using [(35)S]methionine labeling in Triton WR1339-treated mice, was not increased in fasting

76 The Triton X (TX)-series are alkylphenol polyethoxylates -ty

77 r of FANCI foci form and become resistant to Triton X extraction in response to mitomycin C treatment

78 n Ultra, 2% sodium dodecyl sulfate (SDS)-10% Triton X with and without sonication, Infectio Diagnosti

79 All extractions using SDS/Triton X with or without sonication were inhibited.

80 ase activity required Mg(2+) ions (8 mm) and Triton X-100 (0.25 mm) at pH 5.0.

81 Maximum PAP activity was dependent on Triton X-100 (20 mm), PA (2 mm), Mg(2+) (0.5 mm), and 2-

82 mple dilution with an OIS and the surfactant Triton X-100 (inorganic media) or ethanol (organic media

83 o]-1-propanesulfonate (CHAPS, zwitterionic), Triton X-100 (nonionic), sodium dodecyl sulfate (SDS, an

84 derivatives in aqueous solutions of reduced Triton X-100 (RTX-100) were determined by measurements o

85 ort, we confirm that TRAF2 translocates to a Triton X-100 (TX)-insoluble compartment upon TNF-R2 enga

86 month-old mice by intranasal irrigation with Triton X-100 (TX).

87 s the tegument was resistant to removal with Triton X-100 (TX-100), whereas it was lost nearly comple

88 e demonstrated that Gs alpha migrates from a Triton X-100 (TX-100)-insoluble membrane domain (lipid r

89 otein expression by Western blot analysis of Triton X-100 (TX-100)-soluble and TX-100-insoluble cell

90 (erythro-GUVs) when exposed to the detergent Triton X-100 (TX-100).

91 ity, and viability caused by the presence of Triton X-100 (TX100), a nonionic surfactant, were studie

92 Triton X-100 also thickens Lo domains, but partitions to

93 The protein was solubilized with 1% Triton X-100 and 0.5 M sodium chloride and then purified

94 of these observations, we used a mixture of Triton X-100 and 1-butanol and observed that water-solub

95 % or 1% sodium dodecyl sulfate (SDS) or 0.1% Triton X-100 and assayed for clinical chemistry and mala

96 The subsequent introduction of Triton X-100 and CoQ10 causes the MLs lysis and the cres

97 amprenavir, and the Envs were solubilized in Triton X-100 and isolated by sedimentation in a sucrose

98 Milk sample slurries were prepared using Triton X-100 and nitric acid for direct analysis of Pb u

99 The protein was solubilized with Triton X-100 and purified to near-homogeneity.

100 en perfused with PBS, followed by successive Triton X-100 and SDS solutions in saline buffer.

101 In Triton X-100 and sodium cholate solutions, the aggregate

102 SDS, Brij-35, Brij-58, Triton X-100 and Span-40 were explored for the extractio

103 s dependent on the presence of the detergent Triton X-100 and the methyldonor S-adenosylmethionine.

104 d on a single cryo-cooled crystal grown with Triton X-100 and the structure was solved by molecular r

105 KCl solution containing nonionic surfactant Triton X-100 and their translocation was studied at diff

106 In aqueous solution of nonionic surfactants (Triton X-100 and Tween 20) arrays from the second series

107 gh concentrations of inorganic ions, using a Triton X-100 aqueous solution to dilute the sample durin

108 ap the interface of the Bax oligomer we used Triton X-100 as a membrane surrogate and performed site-

109 duction of micellar organised media by using Triton X-100 as an extraction solvent.

110 nd basolateral plasma membrane domains using Triton X-100 as detergent, and characterized their lipid

111 Bovine ROS membranes were incubated with 1% Triton X-100 at 4 degrees C and subjected to density gra

112 ondrial membranes contains up to 120 nmol of Triton X-100 bound per nanomole of the enzyme.

113 Removal of Triton X-100 bound to bovine cytochrome bc(1) was accomp

114 (pH 7.6) containing 0.1 M NaCl and 1% (v/v) Triton X-100 caused liberation of the Ru(bpy)32+ from th

115 e complexation and extraction (pH, DDTC, and Triton X-100 concentration, vortex agitation time and co

116 , these GPC microdomains are soluble in cold Triton X-100 detergent and are thus distinct from conven

117 WT beta2 subunits are resistant to live cell Triton X-100 detergent extraction from the hippocampal a

118 activity by approximately 70% and abolished Triton X-100 detergent inhibition of Ca-dependent nucleo

119 glycosylation status, expression level, and Triton X-100 detergent sensitivity.

120 ions containing BIG/TIR3 and partitions into Triton X-100 detergent-resistant membrane (DRM) fraction

121 We demonstrated that Triton X-100 dissociated the heptameric complex into thr

122 Treatment of blood samples with 0.1% SDS or Triton X-100 does not inactivate EBOV.

123 all TM layers, but not in tissues killed by Triton X-100 exposure.

124 Triton X-100 extracted approximately 10% of the cellular

125 phalloidin staining and Western blotting of Triton X-100 extracted cell lysate.

126 was found to be largely insoluble following Triton X-100 extraction and cofractionationed with bioch

127 this fraction was resistant to high salt and Triton X-100 extraction at pH 6.5.

128 Experiments using Triton X-100 extraction followed by OptiPrep density gra

129 is showed that full-length LEDGF/p75 resists Triton X-100 extraction from chromatin.

130 Triton X-100 extraction of cardiac muscle fibers promote

131 teins CD4, CXCR4, and CCR5, as determined by Triton X-100 extraction.

132 We observed that Triton X-100 extracts of N. gonorrhoeae strain F62 conta

133 rthermore, GPC was more readily extracted by Triton X-100 from adenosine triphosphate (ATP)-depleted

134 A mixture of phospholipids and Triton X-100 in a molar ratio of 5:1 forms well-aligned

135 by sodium dodecyl sulfate, Nonidet P-40, or Triton X-100 in the mass spectrometry analysis.

136 e neuroglian is resistant to extraction with Triton X-100 in the sorting zone and nerve layer, possib

137 tion of cardiac myocytes with saponin and/or Triton X-100 increased NAADP synthesis, indicating that

138 otidase activities while greatly attenuating Triton X-100 inhibition of Mg-dependent nucleotidase act

139 with the actin cytoskeleton, we isolated the Triton X-100 insoluble actin cytoskeleton from platelets

140 cking the N-terminal domain, was detected in Triton X-100 insoluble fractions in Western blot analysi

141 was associated almost exclusively with 0.1% Triton X-100 insoluble material, consistent with its sig

142 ibited by DP-S2849G-GFP in the cytoskeletal (Triton X-100 insoluble) fraction, and keratin filament r

143 Triton X-100 is employed at finely tuned concentrations

144 t interact in the hetero-complex formed in a Triton X-100 micelle as a membrane surrogate.

145 ha (>15-fold) with a K(a) of 2.4 mol % C-1-P/Triton X-100 micelle.

146 We also show that smaller Triton X-100 micelles give a read length of 103 bases in

147 Arabidopsis SphK with Sph presented in mixed Triton X-100 micelles indicated that SphK associates wit

148 zyme activity in a cell-free system using PA/Triton X-100 mixed micelles as substrate, analyzing it i

149 centrations at all surface concentrations in Triton X-100 mixed micelles.

150 hange in the lipin 1beta affinity for the PA/Triton X-100 mixed micelles.

151 d point temperature of non-ionic surfactant, Triton X-100 occurred and complex was entrapped in surfa

152 We have studied the solubilization by Triton X-100 of binary mixtures composed of egg sphingom

153 ticity because the micelle-forming detergent Triton X-100 only minimally affects TRPV1 properties.

154 Confocal microscopy of Triton X-100 permeabilized neutrophils showed that a sma

155 lin than did the non-EGF receptor-containing Triton X-100 rafts.

156 c(1) is fully active; however, protein-bound Triton X-100 significantly interferes with structural st

157 Triton X-100 solubility assays demonstrate that mutant D

158 First, we did cold Triton X-100 solubility assays.

159 ve compound, did not significantly alter the Triton X-100 solubility properties of the membrane.

160 both by fluorescence microscopy and by actin Triton X-100 solubility.

161 -48 h) certain mixtures were not amenable to Triton X-100 solubilization at one or more temperatures.

162 quantitatively removed using injection of 5% Triton X-100 solution, generating a fresh surface for ea

163 ively removed using a cleaning method of 0.5%Triton X-100 sonication plus 1 N nitric acid sonication.

164 ional channels, even without the presence of Triton X-100 that has been found necessary for in vitro-

165 Some tissues were exposed to Triton X-100 to establish dead tissue controls.

166 ndorff-perfused mouse hearts were treated by triton X-100 to produce endothelial dysfunction and subs

167 can be stimulated 35-fold by the addition of Triton X-100 to the reaction mixture.

168 function during reperfusion was impaired in triton X-100 treated hearts compared with vehicle-treate

169 Triton X-100 treatment of transgenic mouse cardiac myofi

170 exhibit differential functional stability in Triton X-100 versus dodecyl maltoside.

171 An average LC50 value of 138 microM for Triton X-100 was obtained for an incubation period of 7-

172 Combining the use of Triton X-100 with the newly introduced UPLC method, we w

173 ted as detergent concentrations (Tween 80 or Triton X-100) are increased up to their critical micelle

174 les (e.g., diheptanoylphosphatidylcholine or Triton X-100) or 0.1-0.2 M inorganic salts.

175 to nonionic micelles in the running buffer (Triton X-100), linking the tagged DNA to the micellar dr

176 pon the addition of nonionic detergent (0.1% Triton X-100).

177 of Jurkat cells in the presence of the agent Triton X-100).

178 r the homogenization of the oil samples with Triton X-100, 200 muL of methanol was added to facilitat

179 Conversely pre-extraction in Triton X-100, a treatment that promotes SNARE complexes

180 onorhamnolipid was compared to that of SDBS, Triton X-100, and ethanol.

181 ocked with cis-Golgi, were solubilized in 2% Triton X-100, and proteins were immunoprecipitated using

182 e are solubilized by lower concentrations of Triton X-100, at least within certain temperature ranges

183 rmediates are disrupted by solubilization in Triton X-100, but chemical cross-linking stabilizes a pu

184 s was more easily perturbed by the detergent Triton X-100, but not other non-ionic detergents.

185 ECM by detergent decellularization methods (Triton X-100, DOC and SDS are compared).

186 ta activity was abolished by the denaturants triton X-100, Gua-HCl, Gua-thiocyanate, SDS and urea in

187 ed IC50 values of three cytotoxic chemicals, Triton X-100, H2O2, and cadmium chloride, as model compo

188 mphiphiles, sodium dodecyl sulfate (SDS) and Triton X-100, in addition to a similar synthesized dendr

189 TcGP63 is soluble in cold Triton X-100, in contrast to Leishmania GP63, which is d

190 erythrocytes were more easily extractable by Triton X-100, indicating weaker association to the cytos

191 in a highly purified plant PSII preparation (Triton X-100, octylthioglucoside).

192 LpxE activity is dependent upon Triton X-100, optimal near pH 6.5, and Mg2+-independent.

193 When lipid vesicles were disrupted by Triton X-100, PrP aggregation was necessary to maintain

194 ation of transfected cells with digitonin or Triton X-100, respectively.

195 7-s incubation times were 290 and 250 microM Triton X-100, respectively.

196 was diluted and homogenised by n-hexane and Triton X-100, respectively.

197 e inner membrane fraction was solubilized by Triton X-100, suggesting that GerD is a lipoprotein, and

198 Interestingly, CHAPS and Triton X-100, thanks to similar surface binding preferen

199 When extracted with Triton X-100, the isolated Ndi1 did not contain Q.

200 l sulfate (SDS), sodium deoxycholate, Chaps, Triton X-100, Triton X-114, NP-40, Brij-35, octyl glucos

201 llows: n-octyl glucoside, dodecyl maltoside, Triton X-100, Tween 20, 3-[(3-cholamidopropyl)dimethylam

202 xperiments in which neutral additives (e.g., Triton X-100, Tween 20, poly(ethylene glycol)) are remov

203 from beta(2)m upon exposure to the detergent Triton X-100, whereas a mutant expressing only glycan 2

204 nol gave slightly better results than when a Triton X-100-ethanol solution was used for dilution.

205 O flipping in proteoliposomes generated from Triton X-100-extracted Saccharomyces cerevisiae microsom

206 The resulting Triton X-100-free cytochrome bc(1) retained nearly full

207 of USA300 was found to be more resistant to Triton X-100-induced autolysis and also to lysis by lyso

208 on of gcp expression can effectively inhibit Triton X-100-induced lysis, eliminate penicillin- and va

209 lated (activated) EGFR was found only in the Triton X-100-insoluble (lipid raft) fraction, whereas to

210 ither WTsyn or A53Tsyn led to a reduction in Triton X-100-insoluble aggregates and an increase in pro

211 localized in Triton X-100-soluble as well as Triton X-100-insoluble cell fractions.

212 Flightless-I targets caspase-11 to the Triton X-100-insoluble cytoskeleton fraction and the cel

213 tin and moesin and was found enriched in the Triton X-100-insoluble fraction along with p67(phox) and

214 searched for Dyrk1A binding proteins in the Triton X-100-insoluble fraction extracted with urea and

215 ome isoforms being enriched primarily in the Triton X-100-insoluble fraction in lens tissue.

216 The Triton X-100-insoluble fractions of M. penetrans and M.

217 c tail abrogated the association of BHA with Triton X-100-insoluble lipid rafts.

218 fts, Triton X-100-soluble fractions, and the Triton X-100-insoluble pellet following apical infection

219 post-translationally converted into a 20-kDa Triton X-100-insoluble proteasome substrate.

220 The amount of endogenous p130Cas in the Triton X-100-insoluble protein fraction, and fibronectin

221 redistribution of Kir2.1 and Kv2.1 from the Triton X-100-insoluble to the Triton X-100-soluble membr

222 radient analysis of plasma membrane-labeled, Triton X-100-lysed cells shows that proximity measured b

223 Triton X-100-permeabilized proplatelets containing dynei

224 t with EGCG caused a marked reduction in the Triton X-100-resistant membrane fraction.

225 class V myosin isoforms are associated with Triton X-100-resistant membranes isolated from mouse for

226 uced using nonspecific alkaline phosphatase, Triton X-100-solubilized membranes from DGK1-overexpress

227 Cytochrome bc(1) isolated from Triton X-100-solubilized mitochondrial membranes contain

228 Using proteoliposomes reconstituted from Triton X-100-solubilized rat liver ER membrane proteins,

229 osomes prepared from phosphatidylcholine and Triton X-100-solubilized rat liver ER-membrane proteins.

230 TJ proteins were redistributed/localized in Triton X-100-soluble as well as Triton X-100-insoluble c

231 ereas total cellular EGFR was present in the Triton X-100-soluble fraction.

232 hyperphosphorylated occludin in lipid rafts, Triton X-100-soluble fractions, and the Triton X-100-ins

233 Claudin-4 was localized to Triton X-100-soluble gradient fractions of control or CP

234 Kv2.1 from the Triton X-100-insoluble to the Triton X-100-soluble membrane fraction.

235 Ubr1 function leads slGFP to accumulate in a Triton X-100-soluble state with slGFP degradation interm

236 y by affinity isolation using native but not Triton X-100-treated budded vesicles.

237 ed iron oxidation, and was also sensitive to Triton X-100.

238 ut are disrupted in harsh detergents such as Triton X-100.

239 teinase K only when exosomes were exposed to Triton X-100.

240 e removed effectively by nonionic surfactant Triton X-100.

241 olubilized from frozen RBC by addition of 1% Triton X-100.

242 +), O 2, alpha-ketoglutarate, ascorbate, and Triton X-100.

243 ective to facilitate CeO2-NPs transport than Triton X-100.

244 t is extracted from low density fractions by Triton X-100.

245 nel is reversibly inhibited by the detergent Triton X-100.

246 acrylate combined with a nonionic surfactant Triton X-100.

247 Analysis of surface dilution kinetics with Triton X-100/PA-mixed micelles yielded constants for sur

248 of 4.2, 4.5, and 4.3 mol %, respectively) in Triton X-100/PA-mixed micelles.

249 In this study, a Triton X-100/phosphatidylcholine (PC) mixed micelle assa

250 t when the apposed bilayers are dissolved in Triton X-100; it is also observed during fusion of isola

251 Circular dichroism (CD) spectroscopy, Triton X-114 (TX-114) phase partitioning, and liposome i

252 Five CPE factors, surfactant (Triton X-114 (TX-114)) concentration, pH, ionic strength

253 arabinogalactan-peptidoglycan complex, and a Triton X-114 (Tx114)-solubilized protein pool were effec

254 ive assays were performed in AMTPS formed by Triton X-114 and sodium tartrate.

255 PrcB localizes to the detergent phase of Triton X-114 cell surface extracts and migrates as a 22-

256 teins because of their aberrant behaviour by Triton X-114 detergent fractionation.

257 oteins were also found to partition into the Triton X-114 detergent phase and were sensitive to prote

258 o reduction by extracellularly added DTT and Triton X-114 detergent phase partitioning.

259 Triton X-114 extraction of outer membrane vesicle prepar

260 TULP1 partitions to the aqueous phase during Triton X-114 extraction.

261 Here we compare the biological properties of Triton X-114 extracts derived from avirulent and virulen

262 Recently, Triton X-114 extracts of a virulent strain of M. arthrit

263 eared to be genuine lipoproteins as shown by Triton X-114 fractionation and sensitivity to globomycin

264 eed, experiments in this work show that upon Triton X-114 fractionation of thylakoid membranes, PsbQ

265 onstrated endogenous Rac1 in the nucleus and Triton X-114 partition revealed that this pool is prenyl

266 P66 was examined by both Triton X-114 phase partitioning and circular dichroism,

267 r dichroism, heat modifiability by SDS-PAGE, Triton X-114 phase partitioning and liposome incorporati

268 proteomic protocols: solvent extraction and Triton X-114 phase partitioning method.

269 r dichroism, heat modifiability by SDS-PAGE, Triton X-114 phase partitioning, and liposome incorporat

270 Triton X-114 phase partitioning, liposome floatation ass

271 Mycoplasma membrane fractionation and Triton X-114 phase separation showed that MG_186 was a m

272 etween the detergent and aqueous phases upon Triton X-114 phase separation, demonstrating an intrinsi

273 d on the basis of a combination of selective Triton X-114 solubilization, radiolabeling with [(3)H]pa

274 Such derivatization of the Triton X-114 surfactant following CPE was found to provi

275 Triton X-114 was chosen as the surfactant for the extrac

276 Cloud point extraction (CPE) using Triton X-114 was successfully applied as an extractive p

277 Triton X-114 was used for sample clean-up and as a fluor

278 polyethylene glycol tert-octylphenyl ether (Triton X-114) as a surfactant prior to its detection by

279 polyethylene glycol tert-octylphenyl ether (Triton X-114) at pH 5.0.

280 e was extracted using a non-ionic detergent (Triton X-114), followed by temperature-induced phase par

281 he surfactant octylphenoxypolyethoxyethanol (Triton X-114).

282 ), sodium deoxycholate, Chaps, Triton X-100, Triton X-114, NP-40, Brij-35, octyl glucoside, octyl thi

283 Imaging and Triton X-114-extraction confirm TFPI and ADTRP associati

284 h-salt treatment of mycoplasma membranes and Triton X-114-partitioned mycoplasma fractions confirmed

285 terol and simple sphingolipids, thus forming Triton X-114-resistant DRMs.

286 hment for hydrophobic membrane proteins with Triton X-114.

287 nto the surfactant-rich phase of 0.06% (w/v) Triton X-114.

288 hexafluorophosphate phase in the presence of Triton X-114.

289 er proportion of total GFAP was found in the Triton X-insoluble fraction of plectin-deficient fibrobl

290 three nonlipid amphiphiles, vitamin E (VE), Triton-X (TX)-100, and benzyl alcohol (BA).

291 We utilize purified Triton-X 100, a nonionic surfactant, to make soap bubble

292 H 7), aqueous, micellar solutions of reduced Triton-X 100.

293 larized by successive sodium dodecyl sulfate/Triton-X cycles.

294 nts to study spectrin aggregate formation by Triton-X extraction and immunocytochemistry followed by

295 nding molecules partition into a low density Triton X100 resistant phase suggesting their association

296 Trace amounts of Triton X100, a nonionic surfactant, release the trapped

297 Using Triton X100, insoluble raft membranes contained homomeri

298 ine carbonate or different concentrations of Triton X100, Nonidet P40 and Brij-58 nonionic detergents

299 eric (curry) extract curcumin, and detergent Triton X100.

300 es, but its effect is different from that of Triton X100.