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1 action produces soluble endoglin, we treated trophoblastic BeWo cells with either a broad-spectrum MM
3 f giant syncytia, and that fusion of a human trophoblastic cell line expressing endogenous syncytin c
5 ellular Ca handling was investigated using a trophoblastic cell line, Rcho-1, derived from a rat chor
6 blastocysts to uterine epithelium and human trophoblastic cell lines to uterine epithelial cell line
8 n that mediates apical cell adhesion between trophoblastic cells and luminal epithelial cells of the
9 n of single chain urokinase binding to human trophoblastic cells by long chain fatty acids was dose-d
10 Our results demonstrate that transduction of trophoblastic cells by the herpes simplex virus vector,
12 high basal expression of CYP27B1 and VDR in trophoblastic cells from the placenta, we hypothesized t
16 h levels were observed only in proliferating trophoblastic cells of the placenta and in adrenal gland
18 ransfer of the lacZ reporter gene into human trophoblastic cells using herpes simplex virus and adeno
19 t inefficient transduction of differentiated trophoblastic cells with HSV.CMVlac is because of dimini
25 e placental syncytiotrophoblast and deported trophoblastic debris serve as sources of fetal mHAgs.
26 (TS) cell self-renewal, and promotes further trophoblastic differentiation both upstream and independ
29 otherapy to eliminate persistent gestational trophoblastic disease (pGTD) was required in three of 19
30 ise strategies for management of gestational trophoblastic disease and address some of the controvers
31 astic tumours are a rare form of gestational trophoblastic disease and consequently information about
33 of 631 patients who underwent treatment for trophoblastic disease had or developed evidence of brain
34 5 550 women were registered with gestational trophoblastic disease in the UK (1976-2006), of whom 62
35 Indications for chemotherapy in gestational trophoblastic disease include raised human chorionic gon
38 ly lower levels of expression in gestational trophoblastic disease tissues, particularly in complete
43 on can divert ES cells toward extraembryonic trophoblastic fates and implicate Ras-MAPK signaling in
44 ing the differentiation of Rcho-1 cells into trophoblastic giant cells supports the importance of CaB
45 thologic tissue-remodeling processes such as trophoblastic implantation, wound healing, and tumor inv
46 trium, whereas syncytiotrophoblasts covering trophoblastic lacunae or newly formed chorionic villi re
48 ife-threatening complications of gestational trophoblastic neoplasia (four of 15 vs none of 36; p=0.0
49 logy and Obstetrics score >/= 7) gestational trophoblastic neoplasia (GTN) frequently receive etoposi
51 All women should be screened for gestational trophoblastic neoplasia after termination of pregnancy.
53 eed, most patients with low-risk gestational trophoblastic neoplasia are treated with single agent ch
54 with a pathological diagnosis of gestational trophoblastic neoplasia at the time of their pregnancy t
55 Until the fundamental biology of gestational trophoblastic neoplasia becomes more clearly understood,
60 and molar placentas, as well as gestational trophoblastic neoplasia, and assessed the effects of a n
65 is expressed in the majority of gestational trophoblastic neoplasias, and is essential for choriocar
69 tified gene regulatory networks for a set of trophoblastic proteins found to be differentially expres
71 trophoblast, IDO is not expressed in murine trophoblastic tissue but instead is found in stromal cel
72 te hydatidiform mole in which extraembryonic trophoblastic tissue develops but the embryo itself suff
74 n neoplastic diseases that derive from fetal trophoblastic tissues and represent semiallografts in pa
77 c tumors (n = 20), and 60% of placental site trophoblastic tumors (n = 10) exhibited FASN positivity.
78 horiocarcinomas (n = 33), 90% of epithelioid trophoblastic tumors (n = 20), and 60% of placental site
81 first molecular evidence that placental site trophoblastic tumors and epithelioid trophoblastic tumor
82 al site trophoblastic tumors and epithelioid trophoblastic tumors are of fetal (trophoblastic) origin
83 al site trophoblastic tumors and epithelioid trophoblastic tumors are thought to arise from intermedi
86 ncer risks for patients cured of gestational trophoblastic tumors with modern chemotherapy appear sim
87 composed of choriocarcinoma, placental-site trophoblastic tumour, and epithelioid trophoblastic tumo
88 l-site trophoblastic tumour, and epithelioid trophoblastic tumour, and many forms are derived from th
90 f whom 62 were diagnosed with placental-site trophoblastic tumours and included, retrospectively, in
95 these separated blastomeres can become small trophoblastic vesicles, which in turn induce decidual re
96 and immunohistochemistry localized CYP2J2 in trophoblastic villi and deciduas at 12 weeks and term.
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