ライフサイエンスコーパス: tropicalis

1 distantly related anuran amphibian (Xenopus tropicalis).

2 tHEP2) from the Western clawed frog (Xenopus tropicalis).

3 mportant roles in eye development in Xenopus tropicalis.

4 , a major allergen from the dust mite Blomia tropicalis.

5 ource for genetic and genomic analyses in X. tropicalis.

6 ng transcriptome of the related frog Xenopus tropicalis.

7 e unique to one subgenome and absent from X. tropicalis.

8 s gene ablates forelimb formation in Xenopus tropicalis.

9 esent a draft genome sequence assembly of X. tropicalis.

10 the dorsal embryonic spinal cord of Xenopus tropicalis.

11 and soma of the African clawed frog Xenopus tropicalis.

12 and genome-wide sequence analysis in Xenopus tropicalis.

13 lis (Xt) allurin, a homologous protein in X. tropicalis.

14 ies in the rapidly breeding diploid frog, X. tropicalis.

15 . glabrata, C. krusei, C. lusitaniae, and C. tropicalis.

16 s and developmental stages of X.laevis and X.tropicalis.

17 NTPDase family in Xenopus laevis and Xenopus tropicalis.

18 chordin, noggin, and follistatin, in Xenopus tropicalis.

19 mportation of the West African frog, Xenopus tropicalis.

20 ts of the only diploid Xenopus frog, Xenopus tropicalis.

21 ral additional ones that are conserved in X. tropicalis.

22 reased proportion of Candida glabrata and C. tropicalis.

23 bicans, C. glabrata, C. parapsilosis, and C. tropicalis.

24 ion in WT mice enhanced susceptibility to C. tropicalis.

25 s in vitro enhanced their ability to kill C. tropicalis.

26 naling during early embryogenesis in Xenopus tropicalis.

27 esents a novel group of major allergen in B. tropicalis.

28 for C. parapsilosis, 0.06 and 0.007; for C. tropicalis, 0.03 and 0.015; for C. krusei, 0.25 and 0.12

29 06, 99.9; C. parapsilosis, 0.5/0.5, 99.0; C. tropicalis, 0.03/0.06, 99.7; C. krusei, 0.12/0.5, 99.0;

30 ns, 0.015/0.03; C. glabrata, 0.015/0.015; C. tropicalis, 0.03/0.06; C. krusei, 0.06/0.12; C. kefyr, 0

31 psilosis, 0.12 (97.6) and 0.06 (97.2) for C. tropicalis, 0.5 (99.8) and 0.5 (99.4) for C. krusei, 0.1

32 r C. parapsilosis, 0.0, 1.5, and 0.5; for C. tropicalis, 0.9, 0.7, and 0.9; and for C. krusei, 0.5, 6

33 8.9%), 0.12 (99.4%), and 0.12 (99.1%) for C. tropicalis; 0.25(100%), 0.03 (100%), and 0.12 (100%) for

34 C. lusitaniae, 10 C. parapsilosis, and 5 C. tropicalis (1 fluconazole-resistant isolate) isolates.

35 ia mucosa (1), Escherichia coli (3), Candida tropicalis (1), Propionibacterium (1), and Rothia (1).

36 the isolates of C. albicans (0.4%), Candida tropicalis (1.3%), and Candida parapsilosis (2.1%); howe

37 brata (7.9%), C. parapsilosis (1.7%), and C. tropicalis (1.4%).

38 9%), Candida parapsilosis (17%), and Candida tropicalis (10%).

39 labrata (18.0%), C. parapsilosis (17.2%), C. tropicalis (10.5%), and C. krusei (1.9%).

40 arapsilosis (17.3%), C. glabrata (17.2%), C. tropicalis (10.9%), C. krusei (1.9%), and other Candida

41 da albicans, 10 Candida glabrata, 10 Candida tropicalis, 10 Candida krusei, 10 Candida dubliniensis,

42 osis, 100%; C. glabrata/C. krusei, 92.3%; C. tropicalis, 100%) and specificity (C. albicans/C. paraps

43 osis, 100%; C. glabrata/C. krusei, 94.8%; C. tropicalis, 100%).

44 C. glabrata, 99.9%, 99.9%, and 100%; for C. tropicalis, 100%, 99.8%, and 100%; for C. krusei, 100%,

45 labrata (24%), C. parapsilosis (13%), and C. tropicalis (12%).

46 owed by Candida glabrata (25.6%) and Candida tropicalis (16.3%).

47 (56.9%), with C. parapsilosis (25.6%) and C. tropicalis (17.0%) being more prominent in LAM.

48 apsilosis, 19 of C. guilliermondii, 12 of C. tropicalis (2 mutant strains), and 11 of C. krusei.

49 losis, 13.4% Candida glabrata, 10.1% Candida tropicalis, 2.4% Candida krusei, 1.7% Candida guilliermo

50 )/1 (90.5), and 0.5 (97.8)/0.5 (93.9) for C. tropicalis; 2 (99.3)/4 (100.0), 32 (99.4)/32 (99.3), and

51 6 C. glabrata, 1,238 C. parapsilosis, 996 C. tropicalis, 270 C. krusei, 99 C. lusitaniae, 88 C. guill

52 14% C. parapsilosis, 14% C. glabrata, 12% C. tropicalis, 3% C. krusei, 1% C. guilliermondii, and 2% o

53 14% C. glabrata, 14% C. parapsilosis, 11% C. tropicalis, 3% C. krusei, and 4% other Candida spp.

54 ns of Candida albicans (11 mutants), Candida tropicalis (4 mutants), Candida krusei (3 mutants), and

55 60 isolates of C. albicans (9 isolates), C. tropicalis (5 isolates), C. krusei (2 isolates), and C.

56 solates of Candida albicans (4 isolates), C. tropicalis (5 isolates), C. krusei (4 isolates), C. kefy

57 ans species C. glabrata (10.2% to 11.7%), C. tropicalis (5.4% to 8.0%), and C. parapsilosis (4.8% to

58 s (23.7%), Candida glabrata (12.7%), Candida tropicalis (5.8%), Candida krusei (4%), and others (1.8%

59 tes of C. parapsilosis, 1,895 isolates of C. tropicalis, 508 isolates of C. krusei, 205 isolates of C

60 C. parapsilosis, 1,048 (17.8%) isolates; C. tropicalis, 527 (8.9%) isolates; C. krusei, 109 (1.9%) i

61 or C. parapsilosis; 2, 0.12, and 0.06 for C. tropicalis; 64, 0.5, and 0.5 for C. krusei.

62 rusei, 91.6%; C. parapsilosis, 86.6%; and C. tropicalis, 86.4%.

63 rata and Candida parapsilosis (15%), Candida tropicalis (9%), and miscellaneous Candida spp. (6%).

64 labrata (14.8%), C. parapsilosis (12.5%), C. tropicalis (9.4%), C. krusei (2.7%), and C. lusitaniae (

65 dida albicans (2.7%), C. glabrata (4.1%), C. tropicalis (9.7%), and other less common yeast species (

66 .3% (95% CI, 85.4%-96.6%) for C. albicans/C. tropicalis, 94.2% (95% CI, 84.1%-98.8%) for C. parapsilo

67 llows: C. krusei, 100%; C. albicans, 98%; C. tropicalis, 97%; C. glabrata, 93%; C. parapsilosis, 85%;

68 parapsilosis; 99.2%, 99.2%, and 96.8% for C. tropicalis; 97.1%, 97.1%, and 97.1% for C. krusei.

69 I, 98.3%-99.4%) for Candida albicans/Candida tropicalis, 99.3% (95% CI, 98.7%-99.6%) for Candida para

70 e evaluated a systemic infection model of C. tropicalis, a clinically relevant, but poorly understood

71 ase to knockdown endogenous Dot1L in Xenopus tropicalis, a diploid species highly related to the well

72 n, we studied the oocyte of the frog Xenopus tropicalis, a giant cell with an equally giant nucleus.

73 duce TNF-alpha following stimulation with C. tropicalis Ags.

74 transgenic mouse, specific for the major B. tropicalis allergen Blo t 5, that targets the lung rathe

75 of the C. albicans, C. dubliniensis, and C. tropicalis ALS genes indicated that, within each species

76 This difficulty exists in Xenopus tropicalis, an anuran quickly becoming a relevant model

77 In this study we investigate Candida tropicalis, an important human fungal pathogen that has

78 uced susceptibility to amphotericin B for C. tropicalis and C. glabrata.

79 albicans mutants, and one mutant each of C. tropicalis and C. krusei were classified as susceptible

80 rata, two of C. albicans, and one each of C. tropicalis and C. krusei were classified as susceptible

81 The limit of detection was 1 CFU/mL for C. tropicalis and C. krusei, 2 CFU/mL for C. albicans and C

82 accompanied by a concomitant increase in C. tropicalis and C. parapsilosis.

83 GPR12; GPRx orthologs are present in Xenopus tropicalis and Danio rerio, but apparently not in birds

84 underlying deep mesenchymal layer in Xenopus tropicalis and extend our previous findings for Xenopus

85 on profiles in developing zebrafish, Xenopus tropicalis and mice and suggests roles for Tet proteins

86 the phylotypic period in zebrafish, Xenopus tropicalis and mouse.

87 gE to Dermatophagoides pteronyssinus, Blomia tropicalis and their tropomyosins Der p 10 and Blo t 10

88 Comparison of X. tropicalis and X. laevis blots revealed comparable expre

89 ression of many identified miRNAs in both X. tropicalis and X. laevis.

90 g sequence from western clawed frog (Xenopus tropicalis) and zebrafish (Danio rerio).

91 glabrata, 20 Candida parapsilosis, 9 Candida tropicalis, and 1 each of Candida krusei and Candida lus

92 a, 46 C. albicans, 36 C. parapsilosis, 19 C. tropicalis, and 20 other species).

93 glabrata, 162 of C. parapsilosis, 124 of C. tropicalis, and 35 of C. krusei.

94 .3% of isolates for C. albicans, 6.2% for C. tropicalis, and 4.1% for C. parapsilosis).

95 a, 79 C. albicans, 23 C. parapsilosis, 18 C. tropicalis, and 49 other species) and 161 contrived samp

96 tes of C. parapsilosis, 1,841 isolates of C. tropicalis, and 503 isolates of C. krusei.

97 s, 38 C. glabrata, 10 C. parapsilosis, 12 C. tropicalis, and 7 C. krusei) against seven antifungal ag

98 labrata, 22 Candida parapsilosis, 14 Candida tropicalis, and 8 Candida krusei isolates), as determine

99 dida glabrata, Candida parapsilosis, Candida tropicalis, and Aspergillus fumigatus.

100 Fluconazole resistance in C. albicans, C. tropicalis, and C. parapsilosis isolates was low (1%), b

101 ant isolates of C. albicans, C. glabrata, C. tropicalis, and C. rugosa remained S to voriconazole.

102 ted of TNF-alpha were more susceptible to C. tropicalis, and CARD9-deficient neutrophils and monocyte

103 usitaniae, 4 were C. albicans, and 3 were C. tropicalis, and five isolates belonged to other Candida

104 pping studies in the related diploid frog X. tropicalis, and for other reasons.

105 e early development of the amphibian Xenopus tropicalis, and found that n1-src expression is regulate

106 C. albicans, two Candida krusei, two Candida tropicalis, and one C. parapsilosis isolate; P > 0.05 ve

107 of Candida albicans, Candida krusei, Candida tropicalis, and perhaps Candida glabrata.

108 s of X. laevis oocytes holds for those of X. tropicalis, and suggest that X. tropicalis oocytes offer

109 a spp., such as C. albicans, C. glabrata, C. tropicalis, and the C. parapsilosis group.

110 determining gene, DM-W, does not exist in X. tropicalis, and the sex chromosomes in the two species a

111 Candida albicans and Candida tropicalis are opportunistic fungal pathogens that can t

112 system, including the development of Xenopus tropicalis as a genetically tractable complement to the

113 ng the T3-dependent metamorphosis in Xenopus tropicalis as a model, we show here that high levels of

114 parapsilosis, Candida glabrata, and Candida tropicalis as well as other clinical species.

115 etraploid Xenopus laevis and diploid Xenopus tropicalis, as a model for postembryonic development, a

116 kifugu rubripes, Xenopus laevis, and Xenopus tropicalis, as well as subpockets involved in protein in

117 krusei, Candida guilliermondii, and Candida tropicalis), Aspergillus fumigatus, Scedosporium spp., F

118 C. parapsilosis ATCC 22019 (25 to 36 mm), C. tropicalis ATCC 750 (23 to 33 mm), and C. krusei ATCC 62

119 da parapsilosis ATCC 22019 (22 to 33 mm), C. tropicalis ATCC 750 (26 to 37 mm), and C. albicans ATCC

120 Candida tropicalis ATCC 750 was not useful for this purpose.

121 bicans ATCC 24333 and ATCC 76615 and Candida tropicalis ATCC 750, showed a less sharp fluconazole end

122 ocytes were necessary for defense against C. tropicalis, because their depletion in WT mice enhanced

123 C. tropicalis biofilm formation was dependent on the pherom

124 The Blomia tropicalis (Blo t) mite species is considered a storage

125 t sexual biofilm formation also occurs in C. tropicalis but, unlike C. albicans, biofilms are formed

126 Candida albicans, C. glabrata, C. tropicalis, C. krusei, and C. kefyr were the most suscep

127 a albicans, C. glabrata, C. parapsilosis, C. tropicalis, C. krusei, and C. lusitaniae; these account

128 . albicans, C. glabrata, C. parapsilosis, C. tropicalis, C. krusei, C. lusitaniae, and C. guilliermon

129 nt among the methods for all C. albicans, C. tropicalis, C. lusitaniae, and C. krusei isolates.

130 strate that C. albicans, C. dubliniensis, C. tropicalis, C. parapsilosis, and C. glabrata release bon

131 andida species (C. albicans, C. glabrata, C. tropicalis, C. parapsilosis, and C. krusei) account for

132 nged from 91% for C. glabrata to 100% for C. tropicalis, C. parapsilosis, C. guilliermondii, C. kruse

133 e-R isolates of C. albicans, C. glabrata, C. tropicalis, C. rugosa, C. lipolytica, C. pelliculosa, C.

134 nd in C. albicans serotypes A and B, Candida tropicalis, Candida guilliermondii, Candida glabrata, an

135 occus sanguis, Streptococcus mutans, Candida tropicalis, Candida parapsilosis, Candida krusei, Candid

136 (Candida albicans, Candida glabrata, Candida tropicalis, Candida parapsilosis, Candida lusitaniae, Ca

137 eronyssinus, Dermatophagoides farina, Blomia tropicalis, cat, German cockroach, Oriental cockroach, c

138 ted high-affinity telomere DNA binding by C. tropicalis Cdc13 (CtCdc13) and found that dimerization o

139 rican (Xenopus laevis) and Western (Silurana tropicalis) clawed frogs.

140 at interaction of FoxG1 with TLE2, a Xenopus tropicalis co-repressor of the Groucho/TLE family, is cr

141 rter spindles observed in egg extracts of X. tropicalis compared to X. laevis.

142 We present a genetic map for Xenopus tropicalis, consisting of 2886 Simple Sequence Length Po

143 ke that of other tetrapods, the genome of X. tropicalis contains gene deserts enriched for conserved

144 glabrata, C. krusei, C. parapsilosis, and C. tropicalis), CSH status correlated with coaggregation wi

145 divergence increases between X.laevis and X.tropicalis differences in mRNA expression levels also in

146 king CARD9 were profoundly susceptible to C. tropicalis, displaying elevated fungal burdens in viscer

147 he C. dubliniensis probe, differentiating C. tropicalis DNA from C. albicans and C. dubliniensis.

148 Strikingly, young X. tropicalis DNA transposons are derepressed and recruit p

149 Both probes reacted with Candida tropicalis DNA, but the T(m) was 51.85 +/- 0.05 degrees

150 Xenopus tropicalis dril1 morphants also exhibit impaired gastrul

151 (H) chain (delta) from the amphibian Xenopus tropicalis during examination of the IgH locus.

152 The Blomia tropicalis dust mite is prevalent in tropical and subtro

153 GF8 performs a dual role in X. laevis and X. tropicalis early development.

154 We show that X. tropicalis egg extracts reconstitute the fundamental cel

155 ial of MMI and PTU using a validated Xenopus tropicalis embryo model.

156 Na(+), K(+)-ATPase beta3 subunit in Xenopus tropicalis embryos and show that its levels are downregu

157 ntal Cell, Akkers et al. report that Xenopus tropicalis embryos transition through early development

158 Superficially, Xenopus tropicalis embryos with reduced levels of XEgr-1 resembl

159 In both cultured cells and X. tropicalis embryos, membrane-bound Ephrins (Efns) B1 and

160 on, and Nodal signal transduction in Xenopus tropicalis embryos.

161 ding' strategy for the collection of Xenopus tropicalis embryos.

162 28% C. albicans, 27% C. parapsilosis, 26% C. tropicalis, etc.) were evaluated between January 2012 an

163 Here we describe the analysis of 219,270 X. tropicalis expressed sequence tags (ESTs) from four earl

164 cent sequencing of a large number of Xenopus tropicalis expressed sequences has allowed development o

165 that a single intranasal sensitization to B. tropicalis extract induces strong Th2 priming in the lun

166 In both X. tropicalis extracts and the spindle simulation, a balanc

167 d that microtubules polymerized slower in X. tropicalis extracts compared to X. laevis, but that this

168 deed, TPX2 was threefold more abundant in X. tropicalis extracts, and elevated TPX2 levels in X. laev

169 edicted transcription start sites in Xenopus tropicalis for genome wide identification of TR binding

170 Isolates of C. tropicalis from patients < or =1 year old were more susc

171 y ChIP-seq and RNA-seq approaches in Xenopus tropicalis gastrulae and find that occupancy of the core

172 that tsg acts as a BMP antagonist during X. tropicalis gastrulation since the tsg depletion phenotyp

173 Scaffolds from X. tropicalis genome assembly 2.0 (JGI) were scanned for Si

174 Comparisons of this map with the X. tropicalis genome Assembly 4.1 (JGI) indicate that the m

175 ude the Xenopus laevis genome, a new Xenopus tropicalis genome build, epigenomic data, collections of

176 id in completing the assembly of the Xenopus tropicalis genome but will also serve as a valuable reso

177 With the advent of the Xenopus tropicalis genome project, we analyzed scaffolds contain

178 representation of a minimum of 66% of the X. tropicalis genome, incorporating 758 of the approximatel

179 RNAs and large piRNA clusters in the Xenopus tropicalis genome, some of which resemble the Drosophila

180 gy, we identified unique SSLPs within the X. tropicalis genome.

181 a newly recognized "Crisp A" gene in the X. tropicalis genome.

182 me and compared it to the related diploid X. tropicalis genome.

183 Here we use a library of Xenopus tropicalis genomic sequences in bacterial artificial chr

184 developing aquatic species, such as Xenopus tropicalis, goldfish, and zebrafish, and in Arabidopsis

185 en together, our results demonstrate that C. tropicalis has a unique sexual program, and that entry t

186 of N-ethyl-N-nitrosourea mutagenized Xenopus tropicalis has identified an inner ear mutant named ecli

187 Forward genetic screens in Xenopus tropicalis have identified more than 80 mutations affect

188 kinase pathway to activate sperm, whereas C. tropicalis hermaphrodites use a TRY-5 serine protease pa

189 On the other hand, X. tropicalis, highly related to X. laevis, offers a number

190 ce RNAs (sisRNAs) have been found in Xenopus tropicalis, human cell lines, and Epstein-Barr virus; ho

191 seq) to explore the transcriptome of Xenopus tropicalis in 23 distinct developmental stages.

192 s in 65% of patients, C. glabrata in 21%, C. tropicalis in 9%, C. parapsilosis in 3%, and C. guillier

193 is the first report of Inonotus (Phellinus) tropicalis inciting human disease and describes the meth

194 2001; and the percentage of S isolates of C. tropicalis increased slightly, from 95.7% in 1997 to 96.

195 irected against an eGFP transgene in Xenopus tropicalis induced mutations consistent with nonhomologo

196 re was no difference in susceptibility to C. tropicalis infection between WT and IL-23p19(-/-), IL-17

197 Thus, protection against systemic C. tropicalis infection requires CARD9 and TNF-alpha, but n

198 miting fungal disease during disseminated C. tropicalis infection.

199 ced normally in CARD9(-/-) mice following C. tropicalis infection.

200 p region as bottle cells whereas those in X. tropicalis ingress by "relamination".

201 To establish X. tropicalis intestinal metamorphosis as a model for adult

202 morphological and cytological changes in X. tropicalis intestine during metamorphosis.

203 m Dermatophagoides mites, confirming that B. tropicalis is a major and distinct source of dust mite a

204 Hence, X. tropicalis is a useful model for the study of molecular

205 The western clawed frog Xenopus tropicalis is an important model for vertebrate developm

206 standing the sex-determination systems in X. tropicalis is critical for developing this flexible anim

207 nopus, and in particular the diploid Xenopus tropicalis, is also ideal for functional genomics.

208 Candida parapsilosis, and 5 (11.9%) Candida tropicalis isolates and 1 (2.4%) Cryptococcus neoformans

209 , compared to < 1% of C. parapsilosis and C. tropicalis isolates and no C. glabrata isolates.

210 of C. parapsilosis isolates, and 0.4% of C. tropicalis isolates.

211 nce breakpoint were also not observed for C. tropicalis isolates.

212 d to 7% of C. glabrata isolates and 6% of C. tropicalis isolates.

213 o 19.5% of C. glabrata isolates and 6% of C. tropicalis isolates.

214 tations were found in 5 C. glabrata and 2 C. tropicalis isolates; of these, 5 (including all C. glabr

215 In X. tropicalis, k-fiber MT bundles that connect to chromosom

216 ilar metamorphic changes in X. laevis and X. tropicalis, making it possible to use the large amount o

217 In contrast, C. tropicalis mating occurs efficiently at both 25 degrees

218 calis model system and assessed whether an X.tropicalis microarray platform can be used for X.laevis.

219 Analysis of Xenopus tropicalis miRNA genes revealed a predominate positionin

220 es, more than 300 genes encoding 142 Xenopus tropicalis miRNAs were identified.

221 vis model system and the increasingly used X.tropicalis model system and assessed whether an X.tropic

222 icans (n = 22), C. parapsilosis (n = 10), C. tropicalis (n = 1) C. glabrata (n = 22), C. krusei (n =

223 All C. albicans (n = 12), C. tropicalis (n = 12), C. glabrata (n = 9), and C. krusei

224 sei (n = 2), C. parapsilosis (n = 4), and C. tropicalis (n = 3).

225 albicans (n = 174), C. glabrata (n = 57), C. tropicalis (n = 31), C. parapsilosis (n = 39), C. krusei

226 29.8%), C. parapsilosis (n = 59; 14.1%), C. tropicalis (n = 37; 8.8%), and C. krusei (n = 17; 4.1%).

227 cans (n = 124), C. parapsilosis (n = 44), C. tropicalis (n = 41), C. glabrata (n = 36), C. krusei (n

228 n = 486), Candida glabrata (n = 96), Candida tropicalis (n = 51), Candida parapsilosis (n = 47), Cand

229 ata (n = 722), C. parapsilosis (n = 666), C. tropicalis (n = 528), C. krusei (n = 143), C. lusitaniae

230 , Saccharomyces cerevisiae ( n = 9), Candida tropicalis (n = 8), Candida lusitaniae (n = 1), and Tric

231 an essential regulator of human and Xenopus tropicalis neural crest specification.

232 Oocytes of Xenopus tropicalis offer several practical advantages over those

233 for embryogenesis and premetamorphosis in X. tropicalis On the other hand, knocking out EVI and MDS/E

234 were C. glabrata, two isolates were Candida tropicalis, one isolate was Candida albicans, and one is

235 toplasm ("MPF activity") into G2-arrested X. tropicalis oocytes induces entry into meiosis I even whe

236 those of X. tropicalis, and suggest that X. tropicalis oocytes offer a good experimental system for

237 ocalizing RNAs in Xenopus laevis and Xenopus tropicalis oocytes, revealing a surprisingly weak degree

238 order for egg cytoplasm to induce GVBD in X. tropicalis oocytes.

239 Together, these studies reveal that C. tropicalis opaque cells form sexual biofilms with a comp

240 -2.44; P = .001), and infection with Candida tropicalis (OR, 1.64; 95% CI, 1.11-2.39; P = .01) as pre

241 Approximately 30% (in Xenopus tropicalis) or 20% (in Xenopus laevis) of injected embry

242 mia caused by a DA producer, C. albicans, C. tropicalis, or C. parapsilosis.

243 C. glabrata, C. parapsilosis, C. rugosa, C. tropicalis, or Saccharomyces cerevisiae grown under cond

244 gene and its unequivocal Silurana (Xenopus) tropicalis orthologue, SNC10.

245 We (i) show that the X.tropicalis probes provide an efficacious microarray plat

246 Candida tropicalis produced colonies similar to those of rare Cr

247 d cell biological experiments, the use of X. tropicalis provides novel insight into the complex mecha

248 We describe here a Xenopus tropicalis rax mutant, the first mutant analyzed in deta

249 Resumption of meiosis in oocytes of Xenopus tropicalis required translation but not transcription, a

250 psilosis, C. rugosa, C. stellatoidea, and C. tropicalis resulted in oligomannoside gel banding patter

251 Here we identify X. tropicalis' sex chromosome system by integrating data fr

252 Structural analysis of C. tropicalis sexual biofilms revealed stratified communiti

253 Small Xenopus tropicalis spindles resisted inhibition of two factors e

254 Interestingly, X. tropicalis spindles were approximately 30% shorter than

255 The crystal structure of the Candida tropicalis Stn1N complexed with Ten1 demonstrates an Rpa

256 a virtually identical architecture as the C. tropicalis Stn1N-Ten1.

257 cribe the expression and activity of Xenopus tropicalis Sulf2 (XtSulf2), which like XtSulf1, can act

258 or cervical ganglion and the tail of Xenopus tropicalis tadpoles are remodeled.

259 s supported by injecting the tail of Xenopus tropicalis tadpoles with peptide 4.2, a 20-aa sequence d

260 enes in C. albicans, C. dubliniensis, and C. tropicalis tend to be located on chromosomes that also e

261 heterotaxy, and now demonstrate, in Xenopus tropicalis, that galnt11 activates Notch signalling.

262 stickleback and fugu, the amphibian Xenopus tropicalis, the monotreme platypus and the marsupial opo

263 In B. tropicalis, the most prevalent and allergenic allergens

264 Y/PRINCIPAL FINDINGS: We observed that in X. tropicalis, the premetamorphic intestine was made of mai

265 model of respiratory sensitization to Blomia tropicalis, the principal asthma allergen in the tropics

266 We show that in Xenopus tropicalis, these processes are connected to the outer-s

267 ALS gene families in C. dubliniensis and C. tropicalis; three partial ALS genes were isolated from e

268 to knockdown expression of xtBcor in Xenopus tropicalis, thus creating an animal model for OFCD syndr

269 nce information and genetic advantages of X. tropicalis to dissect the pathways governing adult intes

270 different vertebrates, ranging from Xenopus tropicalis to Homo sapiens, demonstrating that there is

271 Significantly, we demonstrate that C. tropicalis uses a phenotypic switch to regulate a crypti

272 S. aureus coinoculated with C. krusei or C. tropicalis was highly lethal, similar to C. albicans, wh

273 Xenopus tropicalis was used to make GFP reporter lines with (gam

274 ated knockdown in Xenopus laevis and Xenopus tropicalis we show that Nkx6.1 knockdown results in para

275 enesis in X. laevis and gene knockdown in X. tropicalis, we demonstrate that endogenous Dot1L is crit

276 Using Xenopus tropicalis, we have undertaken the first analysis of the

277 a model induced by intranasal exposure to B. tropicalis, we observed that a single intranasal sensiti

278 Tested using Xenopus tropicalis, we show that founders containing transplants

279 first positional cloning of a mutation in X. tropicalis, we show that non-contractile hearts in muzak

280 Using morpholino oligonucleotides in Xenopus tropicalis, we show that reducing tsg gene product resul

281 ates of C. albicans, C. parapsilosis, and C. tropicalis were all highly susceptible to fluconazole (9

282 ates of C. albicans, C. parapsilosis, and C. tropicalis were all highly susceptible to fluconazole (f

283 l strains of C. albicans, C. glabrata and C. tropicalis were evaluated.

284 Larvae of Silurana tropicalis (Western clawed frog) were exposed to DY7-con

285 early nervous system development in Xenopus tropicalis, where ZIP12 antisense morpholino knockdown i

286 ant-based protocols, and the diploid Xenopus tropicalis which is used for genetics and gene targeting

287 osis (which caused 21 to 24% of BSIs) and C. tropicalis (which caused 7 to 10% of BSIs) were more com

288 cluding C. glabrata, C. dubliniensis, and C. tropicalis, which are frequently more resistant to antif

289 vis and its smaller diploid relative Xenopus tropicalis, which contains smaller cells and nuclei.

290 Xenopus tropicalis, which is a small, faster-breeding relative o

291 in-dependent MT severing was increased in X. tropicalis, which, unlike X. laevis, lacks an inhibitory

292 xposure favored growth of C. glabrata and C. tropicalis, while caspofungin generally favored signific

293 was lowest for Candida glabrata and Candida tropicalis with both test systems.

294 Here, we coupled the frog Xenopus tropicalis with Optical Coherence Tomography (OCT) to cr

295 against C. albicans, C. parapsilosis, and C. tropicalis, with both former agents being more potent (M

296 mly in embryos of Xenopus laevis and Xenopus tropicalis, with prominent expression in the notochord,

297 e epigenome and the enhancer landscape in X. tropicalis x X. laevis hybrid embryos.

298 We have identified a 7.28 kb Xenopus tropicalis Xmyf-5 (Xtmyf-5) genomic DNA fragment that ac

299 urification, and characterization of Xenopus tropicalis (Xt) allurin, a homologous protein in X. trop

300 ntified two genes for each family in Xenopus tropicalis: Xtsprouty1, Xtsprouty2, Xtspred1, and Xtspre