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1 raction after cycloplegia with 2 drops of 1% tropicamide.
2 rect MSNs mediate the antiakinetic action of tropicamide.
3 inducement of cycloplegia with 2 drops of 1% tropicamide.
4 octramine but not to 4-DAMP, pirenzepine, or tropicamide.
5 by the addition of 2.5% phenylephrine and 1% tropicamide.
6 h gradually releases the active ingredients: tropicamide (0.25 mg) and phenylephrine (5.38 mg).
7 .In all groups oxybuprocain 0.4%, cocain 4%, tropicamide 1%, phenylephrine 10%, diclophenac 0.1% alon
8                                              Tropicamide (1%) is an effective cycloplegic agent in my
9 rinic antagonists atropine, scopolamine, and tropicamide (1-2 microM) caused substantial decreases of
10  Mydriasert compared to mydriatic eye drops (tropicamide [1%] plus phenylephrine [10%]).
11  1% tropicamide and then a second drop of 1% tropicamide 4 to 6 minutes later.
12                   Dilation of the pupil with tropicamide alone had no effect on the rate of clearance
13                After pupil dilation with 1 % tropicamide and 10 % phenylephrine for retinal examinati
14   Provocative testing with mydriatic agents (tropicamide and atropine 1%) caused significant increase
15 angles was low after pupillary dilation with tropicamide and oral acetazolamide prophylaxis.
16               Dilation with a combination of tropicamide and phenylephrine caused clearance of fluore
17  eye followed 1 minute later by 1 drop of 1% tropicamide and then a second drop of 1% tropicamide 4 t
18 raction after cycloplegia with 2 drops of 1% tropicamide at each annual visit.
19 roM muscarinic antagonists methoctramine and tropicamide but not by pirenzepine.
20  topical, ocular, ophthalmic, phenylephrine, tropicamide, cardiovascular effect, side effect, blood p
21                                              Tropicamide caused large decreases in firing after DFP,
22                     Lastly, we observed that tropicamide causes a significant decrease in the percent
23           Compared with placebo and with the tropicamide control, ibopamine decreased intraocular pre
24 mber, an effect that was not associated with tropicamide during its peak mydriasis.
25 ll subjects underwent pupil dilation with 1% tropicamide eye drops at the baseline visit, before any
26     To evaluate the cycloplegic effect of 1% tropicamide in myopic children and to determine whether
27 lts from the open field assay indicated that tropicamide increases activity in both the WT and KO mic
28 , 8.1) > hexahydrosiladiphenidol (M3, 8.0) > tropicamide (M4, 6.4) > pirenzepine (M1, 6.1) > methoctr
29 upil dilation with 2.5% phenylephrine and 1% tropicamide, ocular alignment was remeasured in primary
30 mal human volunteers underwent dilation with tropicamide, phenylephrine, and a combination of the two
31  M1 and M4 mAChR antagonists telenzepine and tropicamide, respectively, were tested in the same model
32 than that of pirenzepine, methoctramine, and tropicamide to inhibit the contractile response to musca
33 ss this hypothesis we used an M4 antagonist, tropicamide, to reduce the activity through the M4 mAChR
34 rom the marble-burying assay have shown that tropicamide treatment resulted in a decreased number of
35              In the passive avoidance assay, tropicamide treatment resulted in the improvement of per
36             The anti-parkinsonian effects of tropicamide were almost completely abolished in mutant l
37  and its effects were compared with those of tropicamide, with and without dapiprazole in a double-ma

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