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1 raction after cycloplegia with 2 drops of 1% tropicamide.
2 rect MSNs mediate the antiakinetic action of tropicamide.
3 inducement of cycloplegia with 2 drops of 1% tropicamide.
4 octramine but not to 4-DAMP, pirenzepine, or tropicamide.
5 by the addition of 2.5% phenylephrine and 1% tropicamide.
7 .In all groups oxybuprocain 0.4%, cocain 4%, tropicamide 1%, phenylephrine 10%, diclophenac 0.1% alon
9 rinic antagonists atropine, scopolamine, and tropicamide (1-2 microM) caused substantial decreases of
14 Provocative testing with mydriatic agents (tropicamide and atropine 1%) caused significant increase
17 eye followed 1 minute later by 1 drop of 1% tropicamide and then a second drop of 1% tropicamide 4 t
20 topical, ocular, ophthalmic, phenylephrine, tropicamide, cardiovascular effect, side effect, blood p
25 ll subjects underwent pupil dilation with 1% tropicamide eye drops at the baseline visit, before any
27 lts from the open field assay indicated that tropicamide increases activity in both the WT and KO mic
28 , 8.1) > hexahydrosiladiphenidol (M3, 8.0) > tropicamide (M4, 6.4) > pirenzepine (M1, 6.1) > methoctr
29 upil dilation with 2.5% phenylephrine and 1% tropicamide, ocular alignment was remeasured in primary
30 mal human volunteers underwent dilation with tropicamide, phenylephrine, and a combination of the two
31 M1 and M4 mAChR antagonists telenzepine and tropicamide, respectively, were tested in the same model
32 than that of pirenzepine, methoctramine, and tropicamide to inhibit the contractile response to musca
33 ss this hypothesis we used an M4 antagonist, tropicamide, to reduce the activity through the M4 mAChR
34 rom the marble-burying assay have shown that tropicamide treatment resulted in a decreased number of
37 and its effects were compared with those of tropicamide, with and without dapiprazole in a double-ma
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