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1 patients for troponin I and 25 patients for troponin T).
2 relate with log(concomitant high-sensitivity troponin-T).
3 syndrome (NSTE-ACS) and an elevated cardiac troponin T.
4 tients with NSTE-ACS and an elevated cardiac troponin T.
5 by a positive test for cardiac troponin I or troponin T.
6 ned changes in pre-dialysis highly sensitive troponin T.
7 asmic reticulum Ca(2+) ATPase 1, and cardiac troponin T.
8 an Nkx2.5-LacZ reporter and upregulation of troponin T.
9 tients with NSTE-ACS and an elevated cardiac troponin T.
10 tality, better than high-sensitivity cardiac troponin T.
11 terminal pro-B-type natriuretic peptide, and troponin T.
12 detecting low concentrations of the protein troponin-T.
13 ated by the significantly reduced release of troponin-T.
14 system was developed that used thresholds of troponin T (0.05 ng/ml) and N-terminal pro-B-type natriu
15 se without (n = 43) (mean +/- SD increase in troponin T, 0.011 +/- 0.009 vs. 0.003 +/- 0.006 mug/L, P
18 ity C-reactive protein, and high-sensitivity troponin T, Abeta40 independently predicted CV death and
23 expression of cardiomyogenic markers cardiac troponin T and alpha-smooth muscle actin in CPCedeltaB c
24 ins, including myosin light chain-2 slow and troponin T and carbonylation of myosin heavy chains.
25 easurement of the cardiac troponins, cardiac troponin T and cardiac troponin I, using sensitive metho
26 the duration of such therapy, the levels of troponin T and creatine kinase, and the rates of bleedin
28 is of two meat tenderisation protein markers troponin T and desmin by the four proteases was detected
30 e 2222 patients with serial high-sensitivity troponin T and high-sensitivity troponin I measurements.
31 and serial plasma levels of high-sensitivity troponin T and midregional proatrial natriuretic peptide
32 l-known biomarkers (high-sensitivity cardiac troponin T and N-terminal pro-brain natriuretic peptide)
33 s ECG for 48 hours perioperatively, measured troponin T and N-terminal prohormone of brain natriureti
34 pomyosin alpha-1 chain, fast myotomal muscle troponin T and parvalbumin beta 2 increased their intens
35 beating sheets of cells that express cardiac troponin T and show a full range of action potential mor
36 th chest pain, evaluating them with standard troponin T and testing for hs-cTnT (Roche Diagnostics, B
40 ssociated with high plasma concentrations of troponin-T and N-terminal brain natriuretic propeptide,
43 late resulted in a lower heart rate, a lower troponin T, and a greater urine output as compared with
44 enaline, lipopolysaccharide binding protein, troponin T, and brain natriuretic peptide levels were me
45 d on clinical presentation, high-sensitivity troponin T, and cardiac magnetic resonance imaging, afte
47 N-terminal pro-brain natriuretic peptide and Troponin T, and functional assessment comprising the 6-m
48 atriuretic peptide, high-sensitivity cardiac troponin T, and high-sensitivity C-reactive protein sign
49 atriuretic peptide, high-sensitivity cardiac troponin T, and high-sensitivity C-reactive protein.
50 nalyses, including C-reactive protein, IL-6, troponin T, and N-terminal pro-B-type natriuretic peptid
52 Elevated levels of NT-proBNP, high-sensitive troponin-T, and growth-differentiation factor 15 identif
53 riuretic peptide (NT-proBNP), high-sensitive troponin-T, and growth-differentiation factor 15 with ca
54 arker measurement (NT-proBNP, high-sensitive troponin-T, and growth-differentiation factor 15) at the
56 f N-terminal pro-B-type natriuretic peptide, troponin-T, and urinary albumin excretion, increasing mo
58 Very low but detectable levels of cardiac troponin T are associated with total CVD and CVD death i
59 iuretic peptide and high-sensitivity cardiac troponin T are frequently elevated in severe sepsis or s
62 rence limit for the high-sensitivity cardiac troponin T assay (hs-cTnT) in 3 large independent cohort
63 tested with a precommercial high-sensitivity troponin T assay (hsTnT) and compared with a conventiona
64 The TRAPID-AMI trial study (High-Sensitivity Troponin-T Assay for Rapid Rule-Out of Acute Myocardial
65 the widespread use of high-sensitive cardiac troponin T assays, positive tests become frequent, but t
66 urve for the high-sensitivity assay of serum troponin T at 72 hours), inotrope score (calculated from
67 stable angina (28% of patients) and negative Troponin T at baseline to 3 groups: 2 groups received RI
68 val incremental change of the ln-transformed troponin-T at 72 h was 0.79 +/- 1.54 in the xenon group
70 scularization (P<0.01), presence of elevated troponin T before the procedure (P<0.01), and peripheral
71 as by detecting different concentrations of Troponin T biomarkers (cTnT) through antibody-functional
72 L and a perioperative high-sensitive cardiac troponin T change greater than or equal to 6.3 ng/L are
73 k predictive power of high-sensitive cardiac troponin T change in addition to the Revised Cardiac Ris
74 osite end point in patients with an abnormal troponin T concentration (>/=14 ng per liter) as compare
76 We tested for an association between the troponin T concentration and a composite end point of de
79 7 (99.6%) had detectable (>/=3 ng per liter) troponin T concentrations and 897 (39.3%) had abnormal t
80 7.1% among the patients who had had abnormal troponin T concentrations at baseline, as compared with
82 osite end point among patients with abnormal troponin T concentrations was 1.85 (95% confidence inter
83 riuretic peptide or high-sensitivity cardiac troponin T concentrations were independently associated
87 log-transformed concomitant high-sensitivity troponin-T concentrations (mixed linear model: t = 3.8,
90 o perform multiplexed detection of 100 fg/ml troponin T, creatine kinase MM, and creatine kinase MB i
91 flammatory markers included high-sensitivity troponin T, creatine kinase, myoglobin, N-terminal B-typ
92 igh-sensitivity C-reactive protein (hs-CRP), Troponin-T, creatine kinase-MB, fibrinogen, and D-Dimer
93 ty and changes in levels of highly sensitive troponin T (cTnT) and N-terminal pro-B-type natriuretic
96 mine whether there is immunoreactive cardiac troponin T (cTnT) expression in diseased skeletal muscle
98 biological significance of elevated cardiac troponin T (cTnT) in patients with neuromuscular disease
101 assumption that chronically elevated cardiac troponin T (cTnT) levels fluctuate randomly around a hom
103 Serum creatine kinase-MB (CK-MB) and cardiac troponin T (cTnT) levels were measured before and after
105 p device provides the capability for cardiac-troponin T (cTnT) measurements with co-existed 10 microg
110 udy was to evaluate whether elevated cardiac troponin T (cTnT) was independently associated with an i
111 epeated measures of highly sensitive cardiac troponin T (cTnT) were obtained in participants of the C
112 ry are cardiac troponin I (cTnI) and cardiac troponin T (cTnT) which have been considered as 'gold st
113 detector for an on-chip detection of cardiac troponin T (cTnT) with a detection limit of 12 pg/mL.
115 of TNNT2, the gene that encodes for cardiac troponin T (cTnT), a biomarker of myocardial injury.
116 tion between LVH, low but detectable cardiac troponin T (cTnT), and elevated N-terminal pro-B-type na
118 eric/cooperative mechanism is cardiac muscle troponin T (cTnT), the central region (CR) and the T2 re
123 factor-15 (GDF-15), high-sensitivity cardiac troponin T (cTnT-hs) and haemoglobin, age, and previous
124 ams and serial serum measurements of cardiac troponin T (cTnT; cardiac injury biomarker), N-terminal
125 ologic free light chains (p < 0.05), cardiac troponin-T (cTnT) (p < 0.01), and the Karnofsky index (p
126 lassification is based on cardiac biomarkers troponin-T (cTnT) and N-terminal pro-B-type natriuretic
127 ing of cardiac Troponin-I (cTnI) and cardiac-Troponin-T (cTnT) in a point-of-care sensor format.
130 whereas the dilated cardiomyopathy mutation, troponin T DeltaK210, decreased the Ca(2+) sensitivity o
132 r reference limit and in NSTEMI as a pre-PCI troponin T fall, followed by post-PCI troponin T rise >2
135 gh impedance spectroscopy was used to detect troponin-T functionalized immunoassays on nanotextured Z
136 ity C-reactive protein, and high-sensitivity troponin T, GDF-15 remained independently associated wit
137 omyopathy caused by mutations in the cardiac troponin T gene (TNNT2) has been associated with a high
138 d syndrome and long-QT syndrome, and cardiac troponin T gene, tnnt2, affected in human cardiomyopathi
140 proBNP (>14 pmol/L), elevated high-sensitive troponin-T (>14 ng/L), and elevated growth-differentiati
142 uretic peptide, and high-sensitivity cardiac troponin T, higher levels of Cp were associated with HF
143 r circulation and a high-sensitivity cardiac troponin T (hs-cTnT) acquired on the day of admission.
145 h small elevations of high-sensitive cardiac troponin T (hs-cTnT) are associated with incident heart
147 evel, with elevated high-sensitivity cardiac troponin T (hs-cTnT) concentrations (>/=14 ng/L) using P
148 ide (NT-proBNP) and high-sensitivity cardiac troponin T (hs-cTnT) concentrations were measured by ele
149 c value of baseline high-sensitivity cardiac troponin T (hs-cTnT) elevation in SCAD patients undergoi
150 oglobin (HbA1c) and high-sensitivity cardiac troponin T (hs-cTnT) in 9,661 participants free of CHD a
151 o mental stress and high-sensitivity cardiac troponin T (hs-cTnT) in healthy older individuals withou
152 cremental prognostic value of high-sensitive troponin T (hs-cTnT) in heart failure (HF) beyond that o
153 ions of introducing high-sensitivity cardiac troponin T (hs-cTnT) into clinical practice and to defin
155 etectable (<5 ng/l) high-sensitivity cardiac troponin T (hs-cTnT) level and an electrocardiogram (ECG
156 that any detectable high-sensitivity cardiac troponin T (hs-cTnT) level is associated with adverse ou
157 eferable when using high-sensitivity cardiac troponin T (hs-cTnT) levels in the diagnosis of acute my
159 endpoints included high-sensitivity cardiac troponin T (hs-cTnT) on day 4, left ventricular (LV) rem
160 algorithm based on high-sensitivity cardiac troponin T (hs-cTnT) testing at presentation and again 1
161 we assessed whether high-sensitivity cardiac troponin T (hs-cTnT), a marker of subclinical myocardial
162 atriuretic peptide, high-sensitivity cardiac troponin T (hs-cTnT), and high-sensitivity cardiac tropo
163 eptide (NT-proBNP), high-sensitivity cardiac troponin T (hs-cTnT), and low-density lipoprotein choles
164 eptide (NT-proBNP), high-sensitivity cardiac troponin T (hs-cTnT), Cystatin-C (Cys-C), high-sensitivi
165 , measured by a new highly sensitive cardiac troponin T (hs-cTnT), may reflect ischemia without necro
166 nography and measurement of high-sensitivity troponin T (hs-TnT) and N-terminal pro B-type natriureti
169 tic peptide (NT-proBNP) and high-sensitivity troponin T (hs-TnT) can be used as surrogate markers and
170 The prognostic value of high-sensitivity troponin T (hs-TnT) elevation after elective percutaneou
171 the prevalence of elevated high-sensitivity troponin T (hs-TnT) in 298 patients with heart failure w
172 ate the prognostic value of high-sensitivity troponin T (hs-TnT) in addition to clinical risk factors
173 trasensitive tests to measure high-sensitive troponin T (hs-TnT) serum levels revealed the presence o
174 elationship between OSA and high-sensitivity troponin T (hs-TnT), cardiac structure, and CV outcomes
175 he prognostic importance of high-sensitivity troponin T (hs-TnT), N-terminal pro-brain natriuretic pe
177 farction) evaluated high-sensitivity cardiac troponin-T (hs-cTnT) in a 1-hour acute myocardial infarc
178 h a biomarker (e.g. high sensitivity cardiac troponin T [hs-cTnT] or B-type natriuretic peptide [BNP]
179 rdial damage (using high-sensitivity cardiac troponin-T [hs-cTnT]) and with coronary heart disease (C
182 nknown if evaluation with a high-sensitivity troponin T (hsTnT) assay affects prognosis in this large
184 of the patients and high-sensitivity cardiac troponin T in 84.5%, with higher concentrations in those
186 mited to the use of natriuretic peptides and troponin-T in patients with increased cardiovascular ris
187 ined as an absolute high-sensitivity cardiac troponin T increase of >/=14 ng/L from preoperative to p
189 ease significantly over time, troponin I and troponin T increased moderately, and no consistent clini
192 ntractile regulatory proteins troponin I and troponin T, is reduced during the first 3 weeks after bi
195 confidence limits (CL): 1.52 to 12.30), peak troponin T level (OR: 1.20; 95% CL: 1.08 to 1.34), and u
197 vel, hemoglobin A1c level, phosphorus level, troponin T level, log N-terminal pro-B-type natriuretic
198 nd-point was the proportion of patients with Troponin T levels >3xULN postprocedure (at 6 or 18-24 ho
202 study was to examine the extent of change in troponin T levels in patients with non-ST-segment elevat
203 es, almost complete normalization of cardiac troponin T levels in serum and of left ventricular end d
206 patients with NSTE-ACS and elevated cardiac troponin T levels, an early invasive strategy has no ben
207 nfarct size (P<0.005) and a 61% reduction in troponin-T levels (P<0.05) in comparison with saline con
210 edural increases of high-sensitivity cardiac troponin T (mean: 9.9 ng/ml, range: 2.7 to 19.0 ng/ml vs
212 o assess whether: 1) very small increases in troponin T, measured by a new highly sensitive cardiac t
213 rdiography, and concomitant high-sensitivity troponin-T measurement in patients with severe sepsis or
214 24-hour postoperative high-sensitive cardiac troponin T measurements and the respective changes were
215 re- and postoperative high-sensitive cardiac troponin T measurements demonstrated a majority of patie
218 cardiograms and concomitant high-sensitivity troponin-T measurements were performed in a cohort of 10
221 presenting different proteins (troponin I or troponin T), modifications (missense mutation, deletion,
223 in vivo, transgenic mice harboring the R92Q troponin-T mutation and wild-type littermates received a
224 f Drosophila cardiac tubes revealed that the troponin-T mutation prolongs systole and restricts diast
226 Measures: Concentrations of high-sensitivity troponin T, N-terminal pro-B-type natriuretic peptide, a
227 white blood cell count, fibrinogen, D-dimer, troponin T, N-terminal pro-brain natriuretic peptide, an
228 biomarkers of cardiac damage were measured (troponin T, N-terminal pro-brain natriuretic peptide, an
230 plicing patterns (for example, human cardiac troponin T) or affects other aspects of RNA biology.
231 erences in creatine kinase release (P=0.92), troponin T (P=0.85), or cardiac MRI-assessed infarct siz
233 In contrast, patients with NR had higher troponin T peak (P=0.006) but similar late gadolinium en
234 no effects on phosphorylation of troponin I, troponin T, phospholamban, or myosin light chain-1 or -2
235 TPase, increased levels of phospholamban and troponin T phosphorylation, and reduced phosphorylation
237 lective expression of EcSOD from the cardiac troponin-T promoter after systemic administration of AAV
239 superoxide dismutase (EcSOD) via the cardiac troponin-T promoter would protect the mouse heart agains
244 ylprednisolone significantly reduced cardiac troponin T release and the number of allograft infiltrat
245 myocardial damage evidenced by serum cardiac troponin T release in the rat and mouse cardiac allograf
246 m21G6 significantly reduced infarct size and troponin-T release, and led to marked preservation of ca
248 markers of cardiac (high-sensitivity cardiac troponin T), renal (creatinine and cystatin-C), and hepa
250 n natriuretic peptide (pro-BNP), and cardiac troponin T showed significant linear trends for increase
252 y more cardiomyocytes, determined by cardiac troponin-T staining, in the MI zone of the QHG213H hydro
254 tween or near residues 112 to 136 of cardiac troponin-T, the crucial TnT1 (N-terminal domain of tropo
255 oding cardiac troponin I (TNNI3) and cardiac troponin T (TNNT2) caused altered troponin protein stoic
256 cluding the Insulin Receptor (Insr), Cardiac Troponin T (Tnnt2), Lim Domain Binding 3 (Ldb3) RNAs in
257 roponin I (TNNI3p.98truncation ) and cardiac troponin T (TNNT2p.K217deletion ; also known as the p.K2
263 cardiomyocytes isolated from mice expressing troponin T (TnT) mutants (TnT-I79N, TnT-F110I, TnT-R278C
267 als also showed negligible interference from troponin T (TnT), bovine serum albumin (BSA) and urea un
270 ced expression of troponin I to 39% and 51%, troponin T to 64% and 53%, and troponin C to 73% and 97%
271 the binding of increasing concentrations of troponin-T to the immobilized antibodies on the ZnO surf
272 in-T, the crucial TnT1 (N-terminal domain of troponin-T)-tropomyosin-binding region, cause cardiomyop
274 -pro-B-type natriuretic peptide (NT-proBNP), troponin T (TropT) and C-reactive protein (CRP) predict
275 rrest of cardiomyocyte contraction either by troponin T type 2a (tnnt2a) MO or in weak atriumm58 (wea
277 diovascular disease in our study had cardiac troponin T values above the current myocardial infarctio
278 The effect of xenon on the change in the troponin-T values did not differ in patients with or wit
281 n A(1c), detectable high-sensitivity cardiac troponin T was associated with subsequent CVD (myocardia
294 iuretic peptide and high-sensitivity cardiac troponin T were measured 1, 2, and 7 days after enrollme
295 ntricular structure and function and cardiac troponin-T were among the top predictors for incident he
296 in the interactions between CTnC and cardiac troponin T, which are known to be necessary for ATPase a
298 ro-brain natriuretic peptide (NT-proBNP) and troponin T with liver involvement and the presence of ne
299 t patients would have had a negative cardiac troponin T with older assays); and Group 4, those with h
300 re- and postoperative high-sensitive cardiac troponin T with the occurrence of major adverse cardiac
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