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1 (intracellular amastigotes and extracellular trypomastigotes).
2 hology so that the cells resemble mesocyclic trypomastigotes.
3 nd subsequently failed to differentiate into trypomastigotes.
4 uniformly expressed at the cell surfaces of trypomastigotes.
5 iate host cell invasion by Trypanosoma cruzi trypomastigotes.
6 induced Ca2+ transients and cell invasion by trypomastigotes.
7 DNA, procyclic trypanosomes, and bloodstream trypomastigotes.
8 bout 2.3 kilobases was observed in procyclic trypomastigotes.
9 oscopy of L6E9 myoblasts exposed to T. cruzi trypomastigotes.
10 of primary murine cardiomyocytes by T. cruzi trypomastigotes.
11 d before differentiation of amastigotes into trypomastigotes.
12 ved trypomastigotes but 55 kDa in metacyclic trypomastigotes.
13 ut distributed intracellularly in metacyclic trypomastigotes.
14 nd lacking in N-linked glycans in metacyclic trypomastigotes.
15 on both recombinant Tc-1 and Tc-1 of intact trypomastigotes.
16 protein that is expressed only in infective trypomastigotes.
17 at these genes are up-regulated in procyclic trypomastigotes.
18 roxide dismutase in COL and SYL (versus TCC) trypomastigotes.
19 invasion and the intracellular retention of trypomastigotes.
20 whereas TcHA2 is expressed predominantly in trypomastigotes.
21 e inhibitors of T.b. brucei bloodstream form trypomastigotes.
22 MP (cAMP), however, was unaltered in OPBnull trypomastigotes.
23 rk were enhanced in COL and SYL (versus TCC) trypomastigotes.
24 d cocultures of PBMCs with epimastigotes and trypomastigotes.
25 s detected in both procyclic and bloodstream trypomastigotes.
27 d by the cloned genes as surface proteins in trypomastigote and/or amastigote stages of T. cruzi.
29 pimastigotes readily converted to metacyclic trypomastigotes and efficiently infected mammalian cells
30 -3 binds to the surface of Trypanosoma cruzi trypomastigotes and human coronary artery smooth muscle
33 tudy, smooth muscle cells were infected with trypomastigotes, and immunoblot analysis revealed an inc
34 antigen-immunized groups, as well as higher trypomastigotes antibody-mediated lyses and cell invasio
38 opment of the parasite from amastigotes into trypomastigotes, are prevented by lactacystin, or by a p
41 lic trypomastigotes (MT), but not blood form trypomastigotes (BFT), are highly mucosally infective.
42 tes, amastigotes, and tissue culture-derived trypomastigotes but 55 kDa in metacyclic trypomastigotes
43 uced Ca2+ transients and myocyte invasion by trypomastigotes but are independent of Ca2+ entry into t
44 -kb transcript that is expressed in invasive trypomastigotes but not in noninvasive epimastigote form
45 y formed parasitophorous vacuoles containing trypomastigotes but not in the few parasite-containing v
46 ycoproteins are expressed by amastigotes and trypomastigotes but only amastigotes express the mannose
48 cells exposed to both wild-type and OPBnull trypomastigotes, but the signals triggered by the mutant
52 a developmentally regulated gene of T. cruzi trypomastigotes can be expressed in noninfectious epimas
53 n of TbFPPS double-stranded RNA in procyclic trypomastigotes caused specific degradation of mRNA.
55 , TcCLC and TcCHC at the posterior region of trypomastigote cells, coincident with the flagellar pock
56 th a T. cruzi expression vector carrying the trypomastigote CRP cDNA and produced fully functional re
57 e studies demonstrate the critical role that trypomastigote CRP plays in the protection of parasites
60 During T. cruzi infection, amastigotes and trypomastigotes disseminate in the mammalian host and in
62 t bloodstream-form Trypanosoma brucei brucei trypomastigotes engineered to overexpress reduced nicoti
65 results were obtained with bloodstream form trypomastigotes, except that the RNAi system in this cas
66 results were obtained with bloodstream form trypomastigotes, except that the RNAi system was leaky a
67 itative PCR (qPCR), and immunoglobulin (Ig)M trypomastigote excreted-secreted antigen (TESA)-blots at
72 AZ filament, and depletion of TbSAS-4 in the trypomastigote form disrupts the elongation of the new F
73 agellum in contact with the cell body of the trypomastigote form of Trypanosoma cruzi has been isolat
74 100% inhibition of growth of the bloodstream trypomastigote forms of T. brucei at a concentration of
75 exhibited trypanolytic activity against the trypomastigote forms of T. cruzi, a property known to co
77 of the insect (epimastigote) and infective (trypomastigote) forms by two-dimensional gel electrophor
78 tarts in the skin after an insect bite, when trypomastigotes get through the extracellular matrix to
80 is essential for the extreme potency of the trypomastigote GPI fraction, which is at least as active
82 The residual invasion capacity of OPBnull trypomastigotes in fibroblasts still involves lysosome r
84 und suppression in the number of circulating trypomastigotes in mice with an acute infection of T. cr
85 ine orange uptake in permeabilized procyclic trypomastigotes in the presence of different substrates
86 precipitation of labeled surface proteins of trypomastigotes indicated that the 62-kDa protein is a s
88 e first immediately after differentiation of trypomastigotes into amastigotes and the second before d
89 ion is induced during the differentiation of trypomastigotes into amastigotes, where TcPI-PLC associa
92 either chemotaxis or previously defined anti-trypomastigote mechanisms for the control of T. cruzi's
93 acted specifically with a 160-kDa protein in trypomastigote membrane protein preparations as well as
94 generally thought to be lethal (i.e., >10(6) trypomastigotes/ml), thus associating CD4+ T cell functi
95 of isolated myocytes with secreted/released trypomastigote molecules, is a common outcome of the car
96 ellular amastigote-like cells and metacyclic trypomastigotes more rapidly than wild-type parasites, s
98 nitric oxide-inducing activities of T.cruzi trypomastigote mucins were recovered quantitatively in a
101 endothelial cells (HUVEC) were infected with trypomastigotes of the Tulahuen strain of T. cruzi.
102 lular compartment in permeabilized procyclic trypomastigotes of Trypanosoma brucei, as measured by ac
103 that the transformation in axenic medium of trypomastigotes of Trypanosoma cruzi into amastigote-lik
105 hich is initiated by live infective T. cruzi trypomastigotes or stimulation of isolated myocytes with
106 MIP-1alpha, and MIP-1beta enhances T. cruzi trypomastigote phagocytosis in a dose peak response.
107 did not upon infection with the counterpart trypomastigote population expressing low trans-sialidase
108 Most interesting, HIMECs infected with a trypomastigote population expressing trans-sialidase eff
109 s Chagas disease, the elongated, flagellated trypomastigotes remodel into oval amastigotes with no ex
112 al 120-kDa alkaline peptidase contained in a trypomastigote soluble fraction (TSF) of Trypanosoma cru
113 identified as a member of a large family of trypomastigote-specific genes, and a complete cDNA was i
118 cted with a 55-kDa TcGP63 form in metacyclic trypomastigotes, suggesting stage-specific expression of
119 f an H-2Kb-restricted CTL epitope within two trypomastigote surface Ags encoded by members of the T.
120 ce antiparasite protective immunity, and the trypomastigote surface antigen 1 (TSA-1), a target of an
124 arly effects of defensin alpha-1 on invasive trypomastigotes that involve damage of the flagellar str
125 llular milieu of cultured cells by infective trypomastigotes, the enzyme is restricted to a small (20
130 rt and long chain polyPs was detected during trypomastigote to amastigote differentiation and during
132 e plasma membrane, in the differentiation of trypomastigotes to amastigotes, an essential step for th
133 he inability of oligopeptidase B null mutant trypomastigotes to mobilize Ca2+ from thapsigargin-sensi
134 unctival challenges with T. cruzi metacyclic trypomastigotes using a combination of immunohistochemic
135 m location was also found in amastigotes and trypomastigotes using a polyclonal antibody against a CO
136 f the OPB gene results in a marked defect in trypomastigote virulence, consistent with a greatly redu
138 ich are associated with effective killing of trypomastigotes, was not affected in CCR2(-/-) mice.
139 muM) against Trypanosoma brucei rhodesiense trypomastigotes were 5-31-fold more active against blood
141 of nonadherent TS- trypomastigotes, the TS+ trypomastigotes were isolated from the beads by specific
142 stigotes were relatively avirulent while TS+ trypomastigotes were more virulent than unfractionated p
143 the murine model of Chagas' disease, as TS- trypomastigotes were relatively avirulent while TS+ tryp
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