ライフサイエンスコーパス: tubal

1 Macaques developing gross tubal adhesions after the first chlamydial inoculation w

2 or relative resistance to rapid formation of tubal adhesions is correlated with expression of MHC cla

3 vely associated with good perinatal outcome; tubal (adjusted OR, 0.72 [95% CI, 0.60-0.86]) or uterine

4 rectomy (RRSO) lowers mortality from ovarian/tubal and breast cancers among BRCA1/2 mutation carriers

5 ng the initial surgery because of persistent tubal bleeding.

6 R = 1.52, 95% CI: 1.23, 1.87; P < 0.001) and tubal blockage (RR = 1.83, 95% CI: 1.20, 2.77; P = 0.005

7 y, particularly from ovulation disorders and tubal blockage, is associated with an increased GDM risk

8 Cediranib has activity in recurrent EOC, tubal cancer, and peritoneal cancer with predictable tox

9 ecisions among women at high risk of ovarian/tubal cancer.

10 3 had primary invasive epithelial ovarian or tubal cancers (iEOCs).

11 creening groups for both primary ovarian and tubal cancers as well as primary epithelial invasive ova

12 redictive values for all primary ovarian and tubal cancers were 89.4%, 99.8%, and 43.3% for MMS, and

13 42 (MMS) and 45 (USS) primary ovarian and tubal cancers were detected, including 28 borderline tum

14 For primary invasive epithelial ovarian and tubal cancers, the sensitivity, specificity, and positiv

15 l as primary epithelial invasive ovarian and tubal cancers.

16 y-naive patients with ovarian peritoneal and tubal carcinoma to establish a maximum-tolerated dose (M

17 Women sterilized by bipolar tubal coagulation before the age of 30 years had a proba

18 essing rates of symptomatic PID, subclinical tubal damage, and long-term reproductive sequelae after

19 s infection; better tools to measure PID and tubal damage; and studies on the natural history of repe

20 methotrexate for the treatment of women with tubal ectopic pregnancies is now common practice.

21 We studied 350 women with tubal ectopic pregnancies who were treated with methotre

22 Among women with tubal ectopic pregnancies, a high serum chorionic gonado

23 Tubal ectopic pregnancy (EP) is the most common cause of

24 matis and smoking are major risk factors for tubal ectopic pregnancy (EP), but the underlying mechani

25 Tubal ectopic pregnancy can be surgically treated by sal

26 ing - initiated by C. muridarum infection of tubal epithelial cells (serving as the first hit) - into

27 ubes (group 1) and 19 patients with complete tubal examination (group 2; sectioning and extensively e

28 Tubal factor infertility (TFI) represents 36% of female

29 study, we examined whether the proportion of tubal factor infertility (TFI) that is attributable to C

30 o the immunopathogenic process in women with tubal factor infertility (TFI).

31 5]; ectopic pregnancy, AHR 0.42 [0.39-0.44]; tubal factor infertility AHR 0.29 [0.25-0.33]).

32 ory disease (PID) is a leading cause of both tubal factor infertility and ectopic pregnancy.

33 nflammation or damage, ectopic pregnancy, or tubal factor infertility and no studies addressing the e

34 h anal anastomosis (IPAA) is associated with tubal factor infertility in female patients.

35 f pelvic inflammatory disease and subsequent tubal factor infertility in US women.

36 inflammatory disease, ectopic pregnancy, and tubal factor infertility) following chlamydia infection

37 tory disease, 0.6%; ectopic pregnancy, 0.2%; tubal factor infertility, 0.1%).

38 7]; ectopic pregnancy, AHR 1.31 [1.25-1.38]; tubal factor infertility, AHR 1.37 [1.24-1.52]) and 60%

39 mmatory disease, and possibly preterm birth, tubal factor infertility, and ectopic pregnancy in women

40 nfection include fallopian tube fibrosis and tubal factor infertility.

41 tory diseases, such as blinding trachoma and tubal factor infertility.

42 inflammatory disease, ectopic pregnancy, or tubal factor infertility.

43 itis, pelvic inflammatory disease (PID), and tubal factor infertility.

44 inflammatory disease, ectopic pregnancy, and tubal factor infertility.

45 pelvic inflammatory disease, cervicitis, and tubal factor infertility.

46 uct inflammation, are attenuated in inducing tubal fibrosis and are no longer able to colonize the ga

47 13, are essential for C. muridarum to induce tubal fibrosis; this may be induced by the gastrointesti

48 On T1-weighted images, hyperintense tubal fluid was significantly correlated with the presen

49 in situ hybridization evidence of persistent tubal infection was significantly more frequent among an

50 Hydrosalpinx is a pathological hallmark of tubal infertility associated with chlamydial infection.

51 in hydrosalpinx, a pathological hallmark for tubal infertility in women infected with C. trachomatis.

52 Tubal infertility was not associated with the duration o

53 mice has been used as a surrogate marker for tubal infertility, the medical relevance of nontubal pat

54 nger in use, suggested that they might cause tubal infertility.

55 indicates no important effect of IUD use on tubal infertility.

56 lly leading to severe complications, such as tubal infertility.

57 better natural history data on the timing of tubal inflammation and damage after C. trachomatis infec

58 effect of chlamydia screening on subclinical tubal inflammation or damage, ectopic pregnancy, or tuba

59 id to chlamydial ascension and activation of tubal inflammation, we delivered plasmid-free C. muridar

60 sions rapidly, within 2 weeks after a single tubal inoculation with Chlamydia trachomatis, while in o

61 re not observed until 2 weeks after a second tubal inoculation.

62 Serous tubal intra-epithelial carcinoma (STIC) lesions are the

63 Serous tubal intraepithelial carcinoma (STIC; stage 0) has been

64 epithelial cells and also establishes serous tubal intraepithelial carcinoma as the precursor lesion

65 early fallopian tube lesions, called serous tubal intraepithelial carcinoma.

66 llopian tube lesions (p53 signatures, serous tubal intraepithelial carcinomas (STICs), and fallopian

67 g from small precursor lesions called serous tubal intraepithelial carcinomas (TICs, or more specific

68 lated to 1 case of UEC, and NGS of the other tubal lesion diagnosed as a STIC unexpectedly supported

69 In five of five cases, the peritoneal and tubal lesion shared an identical p53 mutation.

70 ques (Macaca fuscata) were ovariectomized or tubal-ligated (n=5/group) and returned to their natal tr

71 6% (p < 0.001) for noncarriers), history of tubal ligation (odds ratio = 0.68 (95% CI: 0.25, 1.90) f

72 endectomies (eight), liver biopsies (three), tubal ligation (one), and cholecystectomies (three).

73 Tubal ligation (RR = 0.66, 95% CI: 0.50, 0.87) was assoc

74 r relations across racial/ethnic groups, but tubal ligation and family history of breast or ovarian c

75 No interactions between ever having had a tubal ligation and other covariates were observed.

76 ere analyzed to examine the relation between tubal ligation and ovarian cancer mortality in a large p

77 s, transgastric liver biopsies, transgastric tubal ligation and transvaginal cholecystectomy without

78 Standard outpatient procedures such as tubal ligation are now being joined by ambulatory laparo

79 These data suggest that tubal ligation reduces the risk of fatal ovarian cancer.

80 femoral) and then omental fat biopsy during tubal ligation surgery.

81 Tubal ligation was significantly associated with a decre

82 ilarly, age at menopause, endometriosis, and tubal ligation were only associated with endometrioid an

83 actors of pregnancy, oral contraceptive use, tubal ligation, and body mass index.

84 sely associated with oral contraceptive use, tubal ligation, and childbearing.

85 ions associated with oral contraceptive use, tubal ligation, and parity.

86 btained from 37 women undergoing surgery for tubal ligation, ectopic pregnancy, or other gynecologic

87 cancer, duration of oral contraception use, tubal ligation, gravidity, education, and site.

88 Furthermore, tubal ligation, intrauterine device use, and infertility

89 Several gynecologic procedures including tubal ligation, oophorectomy, and partial hysterectomy h

90 Attributable fractions associated with tubal ligation, oral contraceptive use, and obesity were

91 f other, less studied contraceptive methods (tubal ligation, rhythm method, diaphragm, condoms, intra

92 e the most strongly related to pregnancy and tubal ligation, while clear cell tumors were the only ty

93 The existence of a post-tubal-ligation syndrome of menstrual abnormalities has b

94 ase patients) and from 0 of 44 women seeking tubal ligations (the control subjects) at Kenyatta Natio

95 ator for breast-feeding, and availability of tubal ligations); and (3) clinical outcomes, including s

96 f 17 to 55 years (mean 29 years) including 9 tubal ligations, 3 neurosurgeries, 3 cholecystectomies,

97 tubal PROKR1, leading to alterations in the tubal microenvironment that could predispose to EP.

98 eased tubal PROKR2, thereby predisposing the tubal microenvironment to ectopic implantation.

99 e contractility (SMC) and alterations in the tubal microenvironment.

100 ggesting serous malignancy originates in the tubal mucosa but grows preferentially at a remote perito

101 Twenty-four women with tubal obstruction after ligation reversal surgery underw

102 en with primary infertility who did not have tubal occlusion (infertile controls) and 584 primigravid

103 is not associated with an increased risk of tubal occlusion among nulligravid women whereas infectio

104 In analyses involving the women with tubal occlusion and the infertile controls, the odds rat

105 d the infertile controls, the odds ratio for tubal occlusion associated with the previous use of a co

106 d 358 women with primary infertility who had tubal occlusion documented by hysterosalpingography, as

107 between the previous use of a copper IUD and tubal occlusion.

108 infected animals, suggesting the presence of tubal occlusion.

109 ho have family members with breast, ovarian, tubal, or peritoneal cancer with 1 of several screening

110 recurrent or persistent epithelial ovarian, tubal, or peritoneal carcinoma, measurable or detectable

111 ction) area between the OSE, mesothelium and tubal (oviductal) epithelium, as a previously unrecogniz

112 infected IL-10(-/-) mice were protected from tubal pathologies and infertility, whereas WT (IL-10(+/+

113 in rapid microbial clearance, which prevents tubal pathologies during infection.

114 chlamydial infection and the development of tubal pathologies.

115 cts mice, can induce hydrosalpinx in mice, a tubal pathology also seen in women infected with C. trac

116 llopian tubes and subsequent immune-mediated tubal pathology in females.

117 nding the etiology of immune system-mediated tubal pathology, we evaluated the regional recruitment o

118 earance of chlamydiae and the development of tubal pathology.

119 tory infiltrate that may also participate in tubal pathology.

120 epithelial ovarian cancer (EOC), one of the tubal/peritoneal cancers collectively referred to as pel

121 Invasive or intraepithelial ovarian/tubal/peritoneal neoplasms were detected in 25 (2.6%) of

122 ) early spontaneous abortions, and two (15%) tubal pregnancies.

123 hypertension (4.4%), proteinuria (4.4%), and tubal pregnancy (2.2%).

124 and older with a laparoscopically confirmed tubal pregnancy and a healthy contralateral tube were ra

125 In women with a tubal pregnancy and a healthy contralateral tube, salpin

126 sociated with infertility due to uterine and tubal problems, with relative risks of 7.7 (95% CI: 2.3,

127 f infertility is primarily due to uterine or tubal problems.

128 smoking predisposes women to EP by altering tubal PROKR1 expression.

129 rgets human FTs via nAChRalpha-7 to increase tubal PROKR1, leading to alterations in the tubal microe

130 FkappaB by C. trachomatis leads to increased tubal PROKR2, thereby predisposing the tubal microenviro

131 rgery underwent selective salpingography and tubal recanalization.

132 , as a second hit, to transmucosally convert tubal repairing - initiated by C. muridarum infection of

133 us, MMP-9 in particular could play a role in tubal scarring in response to gonococcal infection.

134 ade serous tumors can originate in fallopian tubal secretory epithelial cells and also establishes se

135 Contemporary IUDs rival tubal sterilisation in efficacy and are much safer than

136 ibroids were first visualized at the time of tubal sterilization (1978-1979 or 1985-1987) or who repo

137 A total of 9514 women who underwent tubal sterilization and 573 women whose partners underwe

138 a-analysis also found no association between tubal sterilization and breast cancer risk (odds ratio =

139 We investigated the association between tubal sterilization and breast cancer risk among 77,249

140 summary estimate for the association between tubal sterilization and breast cancer risk.

141 Women who have undergone tubal sterilization are no more likely than other women

142 lity of ectopic pregnancy for all methods of tubal sterilization combined was 7.3 per 1000 procedures

143 Tubal sterilization does not appear to be associated wit

144 A history of tubal sterilization does not rule out the possibility of

145 vided data on ovariectomy, hysterectomy, and tubal sterilization during in-person interviews.

146 Tubal sterilization is a common form of contraception in

147 Tubal sterilization is a common gynecological procedure

148 Tubal sterilization is an increasingly common method of

149 Specifically, tubal sterilization is unassociated with breast cancer r

150 urnal, Gaudet et al. argue successfully that tubal sterilization is unassociated with breast cancer r

151 Several studies suggest that tubal sterilization may decrease the risk of ovarian can

152 on with breast cancer risk was observed with tubal sterilization only or partial ovariectomy without

153 es was greater among women who had undergone tubal sterilization than among women who had not.

154 In the CPS-II Nutrition Cohort, tubal sterilization was not associated with breast cance

155 A total of 10,685 women undergoing tubal sterilization were followed in a multicenter, pros

156 Associations stratified by year of tubal sterilization, age, and time since surgery were al

157 for uterine fibroids among women undergoing tubal sterilization.

158 women who had undergone the common types of tubal sterilization.

159 Tubal swabs remained positive in 3 placebo-, 1 doxycycli

160 We propose that ligation of tubal TLR2 and activation of NFkappaB by C. trachomatis

161 nce of a dilated fallopian tube or thickened tubal wall and mucosal folds and the signal intensity of

162 and unifocal, with variable invasion of the tubal wall.