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1 ducts such as toyocamycin, sangivamycin, and tubercidin.
2 dNT1 transport and consequently resistant to tubercidin.
3  optimization efforts of the natural product tubercidin (7-deazaadenosine) involving modifications to
4 ection to study the mechanism of toxicity of tubercidin (7-deazaadenosine) to procyclic Trypanosoma b
5        Analogous treatment of the antibiotic tubercidin [7-deazaadenosine; 4-amino-7-(beta-D-ribofura
6 ggested that glycolysis could be a target of tubercidin action and that RNAi silencing of glycolytic
7 uclear speckles during stress induction with tubercidin, an adenosine analogue previously shown to af
8                                              Tubercidin, an inhibitor of SAHH, inhibits both chemotax
9  In this respect, hitherto 7-deazaadenosine (tubercidin) analogues attracted most attention.
10 ensitivity to the cytotoxic adenosine analog tubercidin and concurrently restores the ability of this
11 antitrypanosomal agents adenine arabinoside, tubercidin, and cordycepin, or they need to be combined
12 ceding article we reported that analogues of tubercidin are potent adenosine kinase (AK) inhibitors w
13                                      Several tubercidin based AKIs, including the ribo- and lyxo-fura
14 s, including adenine, 2'-deoxyadenosine, and tubercidin, but not for any oxopurines.
15                                              Tubercidin does not inhibit starvation-induced expressio
16                                              Tubercidin has no effect on either capping of Con A rece
17 nd that transport the toxic adenosine analog tubercidin in addition to the natural substrates.
18                    We conclude that 5 microm tubercidin kills trypanosomes by targeting glycolysis, e
19 cle, we demonstrate that substitution of the tubercidin molecule with aromatic rings at the N4- and t
20  structurally related to the natural product tubercidin, potently inhibited replication of poliovirus
21 1A mutation, Ser91Ala, and replacing A6 with tubercidin, purine, or 1-deazaadenine.
22 tion of glucose in the culture medium caused tubercidin resistance.
23 exokinase, a glycolytic enzyme, also yielded tubercidin-resistant parasites.
24 ic intermediates from parasites treated with tubercidin showed a dose-dependent increase in concentra
25 ression/selection using the toxic nucleoside tubercidin similarly yielded the TOR (toxic nucleoside r
26 RNAi-based genomic library, we used 5 microm tubercidin to select a drug-resistant cell line.
27                                 Furthermore, tubercidin triphosphate inhibited recombinant T. brucei
28 ed nucleosides, l-lyxofuranosyl analogues of tubercidin were tested as potential AKIs since the oppos