戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 lity in order to identify optimal dosing for tuberculous meningitis.
2 mated to have been diagnosed and treated for tuberculous meningitis.
3 ilable and a negative result cannot rule out tuberculous meningitis.
4  or PCR (n = 2) and 11% (6/54) had confirmed tuberculous meningitis.
5 nes affect survival in dexamethasone-treated tuberculous meningitis.
6 ified childhood tuberculosis that was due to tuberculous meningitis.
7  disease severity and outcome in adults with tuberculous meningitis.
8 ring a hospital admission, and in those with tuberculous meningitis.
9 ical impact of dexamethasone, as observed in tuberculous meningitis.
10 he new Xpert MTB/RIF Ultra (Xpert Ultra) for tuberculous meningitis.
11 as the initial diagnostic test for suspected tuberculous meningitis.
12 rt MTB/RIF as initial diagnostic testing for tuberculous meningitis.
13 sible tuberculous meningitis, and two as not tuberculous meningitis.
14 ar stain improve the laboratory diagnosis of tuberculous meningitis.
15 HIV and 428 (85.8%) had definite or probable tuberculous meningitis.
16               Among the 24 000 children with tuberculous meningitis, 16 100 (14 900-17 300) were esti
17  in 2002 will have prevented 29,729 cases of tuberculous meningitis (5th-95th centiles, 24,063-36,192
18                          The pathogenesis of tuberculous meningitis, a devastating complication of tu
19 diagnosis; the rest had probable or possible tuberculous meningitis according to published criteria.
20               Among HIV-positive adults with tuberculous meningitis, adjunctive dexamethasone, as com
21 key parameters used as model inputs: risk of tuberculous meningitis after Mycobacterium tuberculosis
22 terized cerebral infarction in children with tuberculous meningitis and explored its relationship wit
23 equelae among children who had treatment for tuberculous meningitis and lived as for children who die
24                  We calculated the number of tuberculous meningitis and miliary tuberculosis cases th
25 consistently high efficacy against childhood tuberculous meningitis and miliary tuberculosis, but var
26  The polymorphisms were associated with both tuberculous meningitis and pulmonary tuberculosis and we
27 lware discusses the challenges of diagnosing tuberculous meningitis and the implications of the study
28 coccal meningitis, 48 with culture-confirmed tuberculous meningitis, and 2900 with culture-negative C
29 gitis, 46% (22 of 48) and 56% (27 of 48) for tuberculous meningitis, and 41% (1181 of 2900) and 49% (
30 ents with culture-confirmed pneumococcal and tuberculous meningitis, and all patients with culture-ne
31  developed tuberculous meningitis, died from tuberculous meningitis, and did not die from tuberculous
32 t approaches to prevent, diagnose, and treat tuberculous meningitis, and there are still too few answ
33 le tuberculous meningitis, three as possible tuberculous meningitis, and two as not tuberculous menin
34  after Mycobacterium tuberculosis infection, tuberculous meningitis as a proportion of tuberculosis n
35 d the prevalence of cryptococcal meningitis, tuberculous meningitis, bacterial meningitis, and cerebr
36 tuberculous meningitis, and did not die from tuberculous meningitis but had neurological sequelae in
37 gher rifampicin doses may improve outcome of tuberculous meningitis, but the desirable exposure and n
38 ants were classified as probable or definite tuberculous meningitis by uniform case definition, exclu
39 rden and attributable mortality of childhood tuberculous meningitis by WHO regions, age groups, treat
40 ns, and detected 64% of clinically diagnosed tuberculous meningitis cases, in a cohort of 603 clinica
41                 Consequently, differences in tuberculous meningitis characteristics across the lifesp
42 rospinal fluid of humans with HIV-associated tuberculous meningitis commonly express surface OX40 pro
43 87; 16 of 23 cases) for probable or definite tuberculous meningitis compared with 43% (23-66; 10/23)
44 uman immunodeficiency virus (HIV)-associated tuberculous meningitis despite limited data supporting t
45 6 in cerebrospinal fluid leukocytes improves tuberculous meningitis diagnosis.
46 er of children aged 0-14 years who developed tuberculous meningitis, died from tuberculous meningitis
47                                              Tuberculous meningitis disproportionately affects young
48                                Children with tuberculous meningitis had baseline magnetic resonance i
49                                              Tuberculous meningitis has high mortality, linked to exc
50                 Despite treatment, childhood tuberculous meningitis has very poor outcomes.
51 00) children younger than 15 years developed tuberculous meningitis in 2019.
52         With composite standard, we detected tuberculous meningitis in 22 (17%) of 129 participants.
53                             Our estimates of tuberculous meningitis in children younger than 15 years
54 trolled clinical trial involving adults with tuberculous meningitis in Indonesia, South Africa, and U
55 HIV-positive adults (>=18 years of age) with tuberculous meningitis in Vietnam and Indonesia.
56                                              Tuberculous meningitis is a devastating brain infection
57                      Laboratory evidence for tuberculous meningitis is difficult to acquire due to th
58                                              Tuberculous meningitis is especially common in young chi
59                                              Tuberculous meningitis is fatal if untreated and can lea
60                                              Tuberculous meningitis is often lethal, and many survivo
61 ing the care of critically ill patients with tuberculous meningitis is poor and many patients do not
62                                              Tuberculous meningitis is the most severe form of tuberc
63 o received treatment to produce estimates of tuberculous-meningitis mortality by age group and HIV st
64 ountries report to WHO), and risk ratios for tuberculous-meningitis mortality by age group.
65                           Among persons with tuberculous meningitis, no evidence of beneficial effect
66 ed on CSF, offers diagnostic sensitivity for tuberculous meningitis of approximately 70%, although it
67 design in Vietnam with cases that had either tuberculous meningitis or pulmonary tuberculosis.
68 ents with health care-associated meningitis, tuberculous meningitis, or missing outcome were excluded
69                     Advances in the field of tuberculous meningitis predominantly focus on diagnosis,
70 logical differences adults and children with tuberculous meningitis receive similar treatment and are
71 omide or analogues in the treatment of other tuberculous meningitis-related complications requires fu
72  hyponatraemia, which frequently accompanies tuberculous meningitis, remain to be elucidated.
73                                              Tuberculous meningitis remains the most lethal form of t
74 search that were drafted at an international tuberculous meningitis research meeting organized by the
75 d new molecules and pathways associated with tuberculous meningitis severity and poor outcomes that c
76 ematic review and meta-analysis of childhood tuberculous meningitis studies published up to Oct 12, 2
77 and low threshold for empirical treatment of tuberculous meningitis suspects.
78                                              Tuberculous meningitis (TB meningitis) is the most sever
79 (LTA4H) polymorphisms in Zambian adults with tuberculous meningitis (TBM) and its association with mo
80 e cerebrospinal fluid (CSF) of patients with tuberculous meningitis (TBM) are associated with TBM-IRI
81                 Mortality and morbidity from tuberculous meningitis (TBM) are common, primarily due t
82                      Point-of-care tests for tuberculous meningitis (TBM) are needed.
83        Pretreatment predictors of death from tuberculous meningitis (TBM) are well established, but w
84                                    Pediatric tuberculous meningitis (TBM) commonly causes death or di
85  a fully automated PCR assay, as the initial tuberculous meningitis (TBM) diagnostic test.
86 e chain reaction (PCR) assay, as the initial tuberculous meningitis (TBM) diagnostic test.
87                                              Tuberculous meningitis (TBM) has a high fatality rate, w
88                             Murine models of tuberculous meningitis (TBM) have not reflected the seve
89      Rapid and reliable diagnostic tests for tuberculous meningitis (TBM) in children remain limited.
90 ome, and prognostic factors in children with tuberculous meningitis (TBM) in Europe are limited.
91                                              Tuberculous meningitis (TBM) is a devastating form of tu
92                                              Tuberculous meningitis (TBM) is a devastating infection
93                                              Tuberculous meningitis (TBM) is a frequent cause of meni
94                                              Tuberculous meningitis (TBM) is a severe complication of
95                                    Stroke in tuberculous meningitis (TBM) is associated with signific
96                             The diagnosis of tuberculous meningitis (TBM) is difficult and poses a si
97                               Drug-resistant tuberculous meningitis (TBM) is difficult to diagnose an
98                                              Tuberculous meningitis (TBM) is difficult to diagnose.
99                                              Tuberculous meningitis (TBM) is the most devastating for
100                                              Tuberculous meningitis (TBM) is the most lethal form of
101                                              Tuberculous meningitis (TBM) is the most lethal form of
102                                              Tuberculous meningitis (TBM) is the most severe form of
103                                              Tuberculous meningitis (TBM) is the most severe form of
104                                              Tuberculous meningitis (TBM) is the most severe form of
105                                              Tuberculous meningitis (TBM) leads to death or disabilit
106                     The delayed diagnosis of tuberculous meningitis (TBM) leads to poor outcomes, yet
107 ulosis load in the brain of individuals with tuberculous meningitis (TBM) may reflect the host's abil
108                Neurological complications of tuberculous meningitis (TBM) often lead to raised intrac
109 noassay) in cerebral spinal fluid (CSF) from tuberculous meningitis (TBM) patients.
110                                              Tuberculous meningitis (TBM) remains a major cause of de
111                               Mortality from tuberculous meningitis (TBM) remains around 30%, with mo
112                                              Tuberculous meningitis (TBM) research is hampered by low
113             Early diagnosis and treatment of tuberculous meningitis (TBM) saves lives, but current la
114  a national molecular diagnostic service for tuberculous meningitis (TBM) using an in-house IS6110-ta
115 ibute to the high morbidity and mortality of tuberculous meningitis (TBM), but the link between infla
116                  We enrolled 135 adults with tuberculous meningitis (TBM), including 83% with HIV.
117  analyses in a large cohort of patients with tuberculous meningitis (TBM), the most severe manifestat
118 s reported as a common complication in adult tuberculous meningitis (TBM), yet few studies have syste
119                                              Tuberculous meningitis (TBM)-related deaths occur early,
120                                              Tuberculous meningitis (TBM)-the extrapulmonary form of
121 l fluid (CSF) inflammation, and outcome from tuberculous meningitis (TBM).
122 ng high-dose rifampicin (up to 35 mg/kg) for tuberculous meningitis (TBM).
123 l fluid (CSF) inflammation, and outcome from tuberculous meningitis (TBM).
124 udy of HIV-infected, ART-naive patients with tuberculous meningitis (TBM).
125 e available on their use in the diagnosis of tuberculous meningitis (TBM).
126 one is recommended as adjunctive therapy for tuberculous meningitis (TBM).
127 flammatory therapies may improve outcomes in tuberculous meningitis (TBM).
128 High-dose rifampicin may improve outcomes of tuberculous meningitis (TBM).
129 is evaluated in novel treatment regimens for tuberculous meningitis (TBM).
130 ION: Xpert Ultra detected significantly more tuberculous meningitis than did either Xpert or culture.
131  higher rate of survival among patients with tuberculous meningitis than standard treatment.
132 apeutic, and neurosurgical interventions for tuberculous meningitis that will improve morbidity and m
133 ause of death, but antibiotic treatments for tuberculous meningitis, the deadliest form of TB, are ba
134 ds are recommended as adjunctive therapy for tuberculous meningitis, the mechanism underlying their b
135                                           In tuberculous meningitis, the polymorphism is associated w
136 estions concerning the optimal management of tuberculous meningitis; these studies also form a platfo
137 se eight, three were categorised as probable tuberculous meningitis, three as possible tuberculous me
138                                              Tuberculous meningitis (TM) is difficult to diagnose and
139               Here, we use a rabbit model of tuberculous meningitis to evaluate the severity of disea
140 imated to be in children who did not receive tuberculous-meningitis treatment.
141 p and HIV status, and assumed that untreated tuberculous meningitis was always fatal.
142                                              Tuberculous meningitis was observed in animals given M.
143                      Using a rabbit model of tuberculous meningitis, we evaluated the protective effi
144 o hundred and eighty patients with suspected tuberculous meningitis were enrolled.
145 usly reported that rabbits with experimental tuberculous meningitis were protected from death by a co
146 fluid specimens from patients with suspected tuberculous meningitis were stained by conventional Zieh
147 infected adults with a clinical diagnosis of tuberculous meningitis who were admitted to one of two V

 
Page Top