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2 effect of prolactin on the basal activity of tuberoinfundibular DA neurons in females occurs via a ce
3 es the stimulatory effects of haloperidol on tuberoinfundibular DA neurons, and reveal that delayed i
4 ne the effects of acute hypoprolactinemia on tuberoinfundibular dopamine (DA) neurons using a rabbit
7 ion of Fos and its related antigens (FRA) in tuberoinfundibular dopamine (TIDA) neurons located in th
8 hormone prolactin is tonically inhibited by tuberoinfundibular dopamine (TIDA) neurons located in th
9 lly under tonic inhibition by neuroendocrine tuberoinfundibular dopamine (TIDA) neurons of the arcuat
11 ishes phasic discharge in rat neuroendocrine tuberoinfundibular dopamine (TIDA) neurons, the main inh
12 receptor-mediated activation of hypothalamic tuberoinfundibular dopaminergic (TIDA) neurons occurs vi
14 oids on regulation of the release of DA from tuberoinfundibular dopaminergic (TIDA), tuberohypophysea
15 d in the labeling of 40% of hypophysiotropic tuberoinfundibular dopaminergic (tyrosine hydroxylase-po
17 ophyseal dopaminergic neurons, while leaving tuberoinfundibular dopaminergic neurons and the vascular
20 ptide, orexins, prolactin-releasing peptide, tuberoinfundibular peptide 39 and neuropeptide B and W)
23 f the thalamus that contain the neuropeptide tuberoinfundibular peptide of 39 residues (TIP39) densel
24 recently identified natural peptide ligand, tuberoinfundibular peptide of 39 residues (TIP39) for th
30 second receptor (PTH2R) and its ligand, the tuberoinfundibular peptide of 39 residues (TIP39), in th
31 oral evidence suggests that the neuropeptide tuberoinfundibular peptide of 39 residues (TIP39), via i
32 crine dopaminergic neurons (NDNs) designated tuberoinfundibular (TIDA), tuberohypophyseal (THDA) and
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